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1 rom uCT scans of adult OIM(-/-) and wild-type (WT) mice and used to calculate centroid sizes (CS) and interlandmark dista
2 , and ammonia (NH(3)) from stack emissions were modeled and used to calculate monthly high-resolution maps of global char
3 ) and concentrations of the asphaltene subfractions (C) are used to calculate adsorption dynamics and surface excess.
7 In this model, secondary structure prediction programs are used to calculate diversity indices, which are measures of un
8 ticle size-mass distribution function, which in turn can be used to calculate not only important 1D distributions, i.e. p
10 two decades, detrended fluctuation analysis (DFA) has been used to calculate scaling exponents of stride time (ST), stri
11 ned unaddressed; MS signal intensities are usually directly used to calculate fold changes for quantitative comparison.
14 al drug parameters (e.g. EC(50) and E(max)), which are then used to calculate chemical parameters such as affinity and ef
15 de Estadistica population projection for 2016 and 2018 was used to calculate the SAO provider density.
16 For each site, principal component analysis was used to calculate both a global fractional anisotropy compone
17 A self-controlled risk interval analysis was used to calculate the incidence rate ratio of bursitis in a r
18 the Trauma Information Exchange Program, and US Census was used to calculate an NBATS score for each trauma service area
19 e-water distribution ratios (D(lip/w)) of the chemicals was used to calculate D(FBS/w) and D(cell/w).
21 Conditional risk-set modeling for recurrent events was used to calculate hazard ratios with the use of the Prentice-
22 A decision analysis of a disease simulation model was used to calculate comparative cost-utility of intravitreal be
24 to mid-2011), and post-PCV13 (mid-2011 to 2016) periods was used to calculate incidence rate ratios (IRRs).
25 143-item semiquantitative food-frequency questionnaire was used to calculate 6- and 12-mo changes in CQI (categorized in
28 Cox proportional hazards regression was used to calculate hazard ratios (HRs) for any MACE, MALE, and
30 ucleotide polymorphisms associated with ischemic stroke was used to calculate a GRS in each patient and identify tertiles
34 sponding to the VF hemifield of more severe loss, which was used to calculate the paracentral total deviation (PaTD), or
35 den of Diseases, Injuries, and Risk Factors Study 2019 were used to calculate the prevalence and years of life lived with
36 Random models by confidence interval (CI: 95%) were used to calculate the pooled estimate of prevalence via Metap
37 ssociation study of children's aggressive behavior and were used to calculate individual-level genome-wide and gene-set P
39 th time-updated information on exposure and covariates were used to calculate the adjusted hazard ratios (HRs) of mortali
40 Center for Medicare and Medicaid Services data were used to calculate associated modeled costs in a hospital- or
41 nce, Epidemiology, and End Results (SEER) program data were used to calculate incidence rates for various cancer types in
42 Medicare claims and IRIS(R) Registry data were used to calculate Medicare Part B expenditures and patient co
44 verty level, overall and stratified by race/ethnicity, were used to calculate adjusted prevalence ratios (aPRs) and 95% c
45 nal hazards and conditional logistic regression models were used to calculate hazard ratios and odds ratios with adjustme
46 Unconditional logistic regression models were used to calculate lung cancer odds ratios and 95% confidence
47 xponential functions (f, D(p), D(f)) while Logan plots were used to calculate volume of distribution (V(T)).
48 Generalized modeling procedures were used to calculate multivariable-adjusted incidence rates for
49 ores and responsiveness to symptom induction protocols were used to calculate standardised mean differences (SMDs) betwee
50 sly using nuclear resonant inelastic X-ray scattering, were used to calculate the superconducting transition temperature