ライフサイエンスコーパス: uterine contraction

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1 ace electrodes has been attempted to monitor uterine contraction.

2 further prostaglandin production and further uterine contraction.

3 tocia), suggesting that it is detrimental to uterine contractions.

4 1) and admission to the hospital for preterm uterine contractions (50% vs 9%; P = .002).

5 ding uterus, WIN2 dose-dependently increased uterine contraction amplitude.

6 ffects only after HYPX, reducing both MT and uterine contraction amplitude.

7 noid receptor agonist WIN 55,212-2 (WIN2) on uterine contractions (amplitude and rate) and micturitio

8 Prostaglandins stimulate uterine contractions and are clinically used for cervica

9 lished roles of oxytocin (OT) is in inducing uterine contractions and labor.

10 PGs stimulate uterine contractions and prepare the cervix for parturit

11 eight, frequency of prenatal visits, preterm uterine contractions, antepartum hemorrhages, placenta p

12 The mechanisms used to coordinate uterine contractions are not known.

13 We assessed the frequency of uterine contractions as a predictor of the risk of spont

14 Each uterine contraction begins with a regional contraction,

15 In addition to uterine contractions during labor and milk ejection duri

16 Uterine contractions during labor are known to be associ

17 ometrial activation is required to establish uterine contractions during labor.

18 den infant death syndrome and for regulating uterine contractions during labor.

19 s on the occurrence of precisely coordinated uterine contractions during labour.

20 targeting uterine cells in-vitro, inhibiting uterine contractions ex-vivo, while doubling uterine dru

21 The measurement of the frequency of uterine contractions has not been useful for reducing th

22 Treatment intended to reduce uterine contractions include tocolytic agents, such as i

23 Uterine contraction is a central feature of PTB, so gain

24 Preterm labor caused by uterine contractions is a major contributor to neonatal

25 preterm labour, but their ability to repress uterine contractions lasts </= 48 h and their use does n

26 ery increases with an increased frequency of uterine contractions, measurement of this frequency is n

27 ng of the heart rate well after the onset of uterine contractions (odds ratio, 3.9; 95 percent confid

28 potheses formed to explain the regulation of uterine contraction/relaxation.

29 ceptor agonists that act via cAMP can reduce uterine contractions to delay preterm labour, but their

30 Uterine contractions, triggered by prostaglandins, excit

31 The regular uterine contractions were recorded via a balloon cathete

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