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1 between local radiation of tumor and active vaccine therapy.
2 mediated by T cells in the setting of cancer vaccine therapy.
3 ng tumor cells were implanted 14 days before vaccine therapy.
4 reast cancer cells is a potential target for vaccine therapy.
5 ls (DC) ex vivo as a model system for cancer vaccine therapy.
6 nant phenotype, would be an ideal target for vaccine therapy.
7 ccur within 3 months after completion of the vaccine therapy.
8 with tumors placed before the initiation of vaccine therapy.
9 develop vitiligo spontaneously or following vaccine therapy.
10 olecular adjuvants for specifically tailored vaccine therapies.
11 erial-derived small molecules as antigens in vaccine therapies.
12 oenvironment seems to impair the efficacy of vaccine therapies.
13 useful for the development of DC-based tumor vaccine therapies.
14 Provenge has brought new hope for anticancer vaccine therapies.
15 unosuppression toward potentiation of cancer vaccine therapies.
16 ciple and offers new practical solutions for vaccine therapy against cancer and other diseases in whi
22 cluding 3 who had minimal disease at time of vaccine therapy and remain free of tumor with 16-30 mont
26 targeted in more than 80 recent and ongoing vaccine therapy clinical trials involving QS-21 as a cri
27 cal tumor growth and an impaired response to vaccine therapy compared with CCR5 knockout (CCR5(-/-))
28 ases of the CNS may be adversely affected by vaccine therapies designed to boost autoreactive lymphoc
29 ll activation induced by a whole cancer cell vaccine therapy enhanced anticancer efficacy in a CD8(+)
30 Thus, HKL may be clinically effective in vaccine therapies for diseases such as allergy and asthm
34 ne response potentiation and dose-sparing in vaccine therapy given its exceedingly high level of pote
36 Nb-PM), working in a synergistic manner with vaccine therapy in an advanced mouse melanoma model.
39 Despite the efficacy of this approach for vaccine therapy, many questions remain regarding whether
41 In the setting of hepatectomy, multiagent vaccine therapy offers an advantage over single-agent th
42 uals enrolled in a trial of glycoprotein 160 vaccine therapy over the course of 3-5 years, lymphoprol
43 hypersensitivity (DTH) response to adjuvant vaccine therapy (P =.0001), and OS was significantly cor
44 ents revealed a significant OS advantage for vaccine therapy (P =.0009): 5-year OS was 39% for vaccin
45 during the past year with the approval of a vaccine therapy, second-line chemotherapy, and reported
46 se who are likely to benefit from a specific vaccine therapy; this approach may benefit future clinic
47 ey parameter relevant to the optimization of vaccine therapies through noninvasive MRI-based quantifi
48 adiation in combination with active specific vaccine therapy to elicit durable antitumor responses of
49 erical simulations of mixed chemo-immuno and vaccine therapy using both mouse and human parameters ar
50 However, neither radiation at this dose nor vaccine therapy was capable of inhibiting growth of 8-da
53 he 1,505 patients who were seen or who began vaccine therapy within 4 months after lymphadenectomy, a
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