ライフサイエンスコーパス: vagal reflex

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1 and pancreatic secretion evoked by the vago-vagal reflex.

2 re applied to investigate involvement of the vagal reflex.

3 estinal motility, which are mediated by vago-vagal reflexes.

4 of CCK to control gastric function via vago-vagal reflexes.

5 pacts the gain or even the direction of vago-vagal reflexes.

6 tion and modulation of gastrointestinal vago-vagal reflexes.

7 ed pyloric relaxation is mediated via a vago-vagal reflex and NO release.

8 We explored the ITR impact on vagal reflex apnea elicited by intravenuously injected 5

9 ry afferent nerves and the subsequent airway vagal reflex arcs.

10 vagal complex, where upper gastrointestinal vagal reflexes are integrated.

11 ecent reports which have suggested that vago-vagal reflexes are inverted by the metabolic status of t

12 ion are more sensitive in amplifying gastric vagal reflexes; (b) in the periphery, CCK8 is more poten

13 animals survived for several months and the vagal reflex behaviour, exemplified by citric acid-induc

14 ions as a consequence of its actions on vago-vagal reflex circuit elements.

15 e hindbrain is also the location of the vago-vagal reflex circuitry regulating gastric motility.

16 haps permanent changes in the sensitivity of vagal-reflex circuitry.

17 ests that TNF(alpha) action directly on vago-vagal reflex control circuits causes the autonomic misre

18 lucose on the central components of the vago-vagal reflex control of gastric function, we performed b

19 (CRF) on the central components of the vago-vagal reflex control of gastric function.

20 ion procedure targeted on the elimination of vagal reflex evoked by high frequency stimulation or an

21 central neural pathways involved in the vago-vagal reflex in pancreatic secretion; the demonstration

22 ts and to alter the function of gastric-vago-vagal reflexes in response to these stimuli.

23 bre terminals, which in turn initiate a vago-vagal reflex inhibition of gastric motor function.

24 ytin injections into the motor layer labeled vagal reflex interneurons that have radially directed de

25 We hypothesize that the vago-vagal reflex mediating pancreatic secretion in the rat i

26 t (ExStrT), an easily quantifiable marker of vagal reflexes, might identify high- and low-risk long Q

27 The vago-vagal reflex plays an important role in mediating pancre

28 It is suggested that the vago-vagal reflex plays an important role in mediating the ac

29 ensitivity, an established marker of reduced vagal reflexes, predicts low probability of symptoms amo

30 to control NST neurones responsible for vago-vagal reflex regulation of the stomach.

31 gestive process can powerfully regulate vago-vagal reflex sensitivity.

32 Intense exercise training, which potentiates vagal reflexes, should probably be avoided by LQT1 patie

33 obesity, suggesting that attenuation of vago-vagal reflex signalling may precede the development of o

34 ts from our parallel studies on gastric vago-vagal reflexes suggest that noradrenergic neurons in the

35 re, we report a role of delta9-THC in a vago-vagal reflex that can result in gastro-oesophageal reflu

36 , we report a role of Delta(9)-THC in a vago-vagal reflex that can result in gastro-oesophageal reflu

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