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1 ents of the human gastrointestinal tract and vagina.
2 herniation of the uterus into or through the vagina.
3 five swabs from the lateral wall of the mid-vagina.
4 sity as physician collected swabs of the mid-vagina.
5 ivity on bone, plasma lipids, hot flush, and vagina.
6 precursor lesions in both the cervix and the vagina.
7 red the titers of the challenge virus in the vagina.
8 nized monkeys was minimal and limited to the vagina.
9 monads is preparatory to colonization of the vagina.
10 ated in milk and C. pecorum dominated in the vagina.
11 eficiency virus (SHIV) infection through the vagina.
12 appeared to be nonspecific in the uterus and vagina.
13 primordia of the oviducts, uterus and upper vagina.
14 ell as epithelia of the oviduct, cervix, and vagina.
15 for sustained long-term colonization of the vagina.
16 primary role in creating the acidity of the vagina.
17 ut did not entirely prevent infection of the vagina.
18 els after controlled topical delivery to the vagina.
19 lective homing of T and B lymphocytes to the vagina.
20 approximately 1% of the concentration in the vagina.
21 in mucosal fluids of the eye as well as the vagina.
22 unction was not entirely eliminated from the vagina.
23 ns were not detected in either the uterus or vagina.
24 elia such as the oral cavity, esophagus, and vagina.
25 intermediate state between normal uterus and vagina.
26 did not increase plasma cell numbers in the vagina.
27 and specific IgG in both the cervix and the vagina.
28 4) known bacteria formerly isolated from the vagina.
29 elatively little to immune protection of the vagina.
30 ucts and sinovaginal bulbs, give rise to the vagina.
31 eutrophil transepithelial migration into the vagina.
32 eral human niches such as the mouth, gut and vagina.
33 e from semen, 69 from urine, and 21 from the vagina.
34 factors and secreted proteins in the macaque vagina.
35 ations: caries lesions, the stomach, and the vagina.
36 ations through natural orifices, such as the vagina.
37 phonuclear neutrophil (PMN) migration to the vagina.
38 clinically without further treatment of the vagina.
39 actors that influence drug movement into the vagina.
40 alic acid-containing mucus components in the vagina.
41 ay be more likely to have involvement of the vagina.
42 persistent interdigital webs and imperforate vaginas.
43 he endocervix and by patients from their own vaginas.
45 ce receiving recombinant IFN-alphaA (5-500 U/vagina) 24 h PI showed no significant survival in compar
49 , cervix plus urine, 86 and 49%; cervix plus vagina, 91 and 93%; and vagina plus urine, 94 and 79%.
50 ory mice showed high in vivo CTL activity in vagina, a strong recall response, and robust protection
56 of nectin-1 in epithelial cells of the mouse vagina and also in neuronal cells of the central nervous
57 he detection of GBS is culture of the distal vagina and anorectum in a selective broth medium followe
59 re part of the normal bacterial flora of the vagina and are typically considered contaminants when cu
60 to what was reported for studies of HSV-2 in vagina and brain and suggests that receptor requirements
61 EM is dispensable for HSV-2 infection of the vagina and brain, but is required for WT pathogenesis of
63 ) and clear cell adenocarcinoma (CCA) of the vagina and cervix is well known, yet there has been no s
66 ere preferentially found in the lower tract (vagina and cervix), whereas APCs and innate lymphoid cel
67 ased the number of viral target cells in the vagina and cervix, suggesting that the ratio of target c
69 -fire endorectal probe was inserted into the vagina and directed toward the posterior vaginal wall.
70 message was invariantly expressed by normal vagina and ectocervix and inflamed fallopian tube, but v
72 of the stratified squamous epithelium of the vagina and ectocervix, with the intensity of cellular st
74 e of Lactobacillus strains isolated from the vagina and enhances the color production of H(2)O(2)-pro
75 echanism that coordinates development of the vagina and feminization of the urethra, which may accoun
81 reased the number of IgG plasma cells in the vagina and increased vaginal secretion/serum titer ratio
82 ell types in the oviduct, uterus, cervix and vagina and is dependent upon specific mesenchymal-epithe
83 oundary structure between the uterus and the vagina and is key for the maintenance of pregnancy and t
84 de (H(2)O(2))-producing species found in the vagina and is under development as a probiotic for the t
87 tion defined as reduced bacterial numbers in vagina and prevention of pathological changes in the upp
88 Upon infection, pDCs are recruited to the vagina and produce large amounts of type I IFNs in a TLR
89 suggest that the mucosal environments of the vagina and rectum both impose a strong selection for the
91 ctions of challenge virus replication in the vagina and reduced latent viral loads in dorsal root gan
92 of innervation of epithelium of the proximal vagina and reduced proportions of VIP, CGRP, and SP cont
93 that IgG viral antibody was produced in the vagina and released into vaginal secretions at 6 weeks a
94 including stratified squamous epithelia from vagina and skin, as well as cuboidal epithelium from lun
95 s, and can colonize the periurethral area or vagina and subsequently ascend through the urethra to th
96 essing high levels of CCR5 reside within the vagina and that these cells are preferentially targeted
97 acterised by an abnormal opening between the vagina and the bladder or rectum, which results in conti
98 he squamous epithelium of the ectocervix and vagina and the columnar epithelium of the endocervical c
100 hat telomerase activity, particularly in rat vagina and uterus, appears to be associated with a cell
103 ecific concordance suggests that the cervix (vagina) and anus may serve as reservoirs for HPV infecti
104 cus, and groin) and their mothers (mouth and vagina) and were obtained from infants weekly until they
105 usly isolated from body sites other than the vagina, and (4) known bacteria formerly isolated from th
107 sentative UTI isolate present in the rectum, vagina, and bladder of a woman with UTI was chosen as th
109 tices (cleaning with the fingers, wiping the vagina, and inserting traditional substances) are associ
110 aea have been isolated from the human colon, vagina, and oral cavity, but have not been established a
111 that can harmlessly colonize the human gut, vagina, and rectum but can also cause pneumonia, sepsis,
112 ains from the three body sites (oral cavity, vagina, and rectum), regardless of host conditions.
114 cells of primitive neurons, the cervix, the vagina, and the endometrium in 5- to 400-fold higher num
115 cit functional and structural effects on the vagina, and therefore E4 appears promising as a therapeu
116 al tumor at all sites except paratesticular, vagina, and uterus, or with alveolar RMS were randomly a
119 ary lymphoid follicles in the ectocervix and vagina; and 3) concentrated on the path of virus entry b
122 outine Papanicolaou smears of cells from the vagina are commonly examined in women who have undergone
128 uding the oviducts, uterus, cervix and upper vagina, are derived from the Mullerian ducts, a pair of
129 innervate the distal urethra and the distal vagina, as well as the clitoris and perigenital skin and
130 f immunoglobulin G (IgG) plasma cells in the vagina at 6 weeks and 10 months after immunization, wher
131 hat vaginal lactobacilli could reacidify the vagina at the rate observed postcoitally following neutr
132 n serum and detectable levels of MAbs in the vagina at the time of infection, there was only modest p
137 clear p63 protein was localized to the skin, vagina, bladder, urethra, and basal columnar cells of th
138 ation (> or =10(5) cfu/mL) of E. coli in the vagina (both, P<.001) and urine (all <10(5) cfu/mL; P=.0
139 duced a strong IgG response in the serum and vagina but was inefficient in generating a mucosal IgA r
140 rkedly reduced T cells in blood, spleen, and vagina, but major histocompatibility complex class II an
141 g sustained local drug concentrations in the vagina can be challenging, due to the high permeability
144 llenge with herpes simplex virus in skin and vagina challenge models, and were clearly superior to th
145 resulted in an inflammatory response in the vagina characterized by neutrophils and infiltrating sub
147 an initial elevation in myeloid cells in the vagina (day 3) and uterine horns (day 7), followed by a
151 yed bimodal (e.g., gut) or multimodal (e.g., vagina) distributions of sample abundances, but not all
152 r titers in the corneas, trigeminal ganglia, vaginas, dorsal root ganglia, spinal cords, and brains o
153 th contributing to the overall health of the vagina due to its direct and indirect effects on pathoge
155 es in elastic fiber homeostasis in the mouse vagina during pregnancy and parturition were determined.
157 Epithelial cells derived from normal human vagina, ectocervix, and endocervix expressed mRNA for TL
158 the kidney and the epithelial layers of the vagina, ectocervix, endocervix, uterus, and fallopian tu
159 -related changes in innervation of the mouse vagina, effecting the distribution of neuropeptides with
160 lassical theory of Mullerian origin of upper vagina fails to explain complex urogenital malformations
161 ed body sites (gut, skin, oral, airways, and vagina), focusing on interpersonal and intrapersonal var
162 w outlines the anatomy and physiology of the vagina, focussing on areas relevant to drug delivery.
164 omized rats (very low proliferation) than in vagina from ovariectomized and estradiol-treated rats (h
166 re compared from the lymph nodes, blood, and vagina from uninfected and simian immunodeficiency virus
167 ward nanoparticle-based drug delivery in the vagina has been focused on HIV prevention, strategies fo
168 anti-candida host defence mechanisms in the vagina has developed slowly and, despite a growing list
169 Epithelial delivery of medication across the vagina has proven successful for administration of small
171 ccurs when semen temporarily neutralizes the vagina, HIV maintained its native surface charge and dif
173 We compared cervical and vulvar areas of the vagina in young nullipara and older multipara C57Bl/6 mi
174 le lower reproductive tracts (ie, cervix and vagina) in the human papillomavirus transgenic mouse mod
175 mmunolabeling of ganglia associated with the vagina indicated the likely origin of some peptidergic f
177 areas in intravaginal drug delivery, but the vagina is a challenging route of administration, due to
180 that a similar acute PMN migration into the vagina is mediated by chemotactic S100A8 and S100A9 alar
181 ermine the prevalence of each species in the vagina, its association with BV, and the utility of PCR
183 oliferation and indicators of cellularity in vagina, mammary gland, and uterus from virgin, pregnant,
185 sitive at the mouth, n = 26; positive at the vagina, n = 18; positive at both sites, n = 10) at the t
186 tivity was significantly higher (P=0.003) in vagina obtained from ovariectomized rats (very low proli
188 t to establish a productive infection in the vagina of 75% +/- 17% and in the spinal cord of 71% +/-
189 s) is a commensal of the human intestine and vagina of adult women but is the leading cause of invasi
191 f herpesvirus entry mediator nectin-1 in the vagina of human and mouse at different stages of their h
192 ological and functional impacts of E4 on the vagina of ovariectomized mice, and we determined the mol
193 Swab samples were obtained from the lower vagina of participants at admission for delivery and ino
195 oduced no pregnancies because the uterus and vagina of the MOER female mice were extremely atrophic.
197 quences were also found within the colon and vagina of the same animal, and within the peripheral blo
199 gens isolated from the rectum, urine, and/or vagina of women with UTIs and 54 E. coli isolates from t
200 he three MAbs and were used to inoculate the vaginas of C3H/HeJ mice which had been pretreated with p
201 the amount of chlamydiae recovered from the vaginas of mice that had received the two IgG2b MAbs, E4
203 , activated mononuclear blood cells into the vaginas of mice; four hours later, numerous double-stain
204 ols used a pressure device placed within the vagina or anal canal, or electromyographic (EMG) sensors
205 ty of virus transmission when applied to the vagina or rectum of a person at risk of sexually acquiri
206 he vaginal apex below the upper third of the vagina, or anterior or posterior vaginal wall prolapse b
207 s in at least one of three body sites (nose, vagina, or anus), with approximately 9% colonized vagina
210 sites of keratinized epithelium, such as the vagina, or for adherence of these bacteria to damaged ep
211 cally and/or mucosally into the nose, mouth, vagina, or rectum in a 4-dose schedule, followed by 2 do
213 oof that Langerhans cells (LCs) in the human vagina participate in dissemination of infectious human
214 letion of CD4(+) T cells was observed in the vagina, particularly among the CCR5(+)CD4(+) subset.
219 tious organisms (3 log(10)) from the cervico-vagina, produced a minimal inflammatory response in urog
220 ption factor for BMP4 signaling, was high in vagina-projecting sensory neurons after ovariectomy and
221 ry responses to infection in both cervix and vagina, recruits CD4(+) T cells to fuel this obligate ex
222 vical stroma, parametria and/or adnexae, and vagina, respectively, were 72%, 69%, 74%, and 85% for re
223 Specifically, HA depletion in the cervix and vagina resulted in inappropriate differentiation of epit
225 g single specimens, with the combined cervix-vagina results identifying the highest number of positiv
226 in sectioned murine lymph nodes draining the vagina revealed a profound cellular reorganization with
227 mmunocompartments matching blood, colon, and vagina samples from rhesus macaques were investigated.
228 or alpha, CXCL1, CCL2, CCL3, and CCL5 in the vagina, spinal cord, and/or brain stem than did wild-typ
229 lastin protein, and desmosine content in the vagina suggest that a burst of elastic fiber assembly an
230 e of the total number of lactobacilli in the vagina, suggest that vaginal lactobacilli could reacidif
231 quently identified in the cervix than in the vagina, suggesting that the expression levels of corecep
232 s and detectable levels of hemoglobin in the vagina, suggesting that the presence of hemoglobin in in
235 were more likely to be detected in the vulva/vagina than in the cervix (odds ratio, 4.38 [95% confide
237 bition of challenge virus replication in the vagina than when the virus was used to vaccinate mice in
238 her E. coli to the urethra, periurethra, and vagina, the authors reasoned that uropathogenic E. coli
240 highly expressed in the epithelium of human vagina throughout the menstrual cycle, whereas the mouse
242 thmic anastomosis enabling continuity of the vagina to be preserved following insertion of an isthmic
244 rom normal human endocervix, ectocervix, and vagina to characterize gonococcal epithelial interaction
247 osa explant model (endocervix and ectocervix/vagina) to mimic genital herpes infections caused by her
251 report here the construction of a functional vagina using autologous cells expanded from a small vagi
252 nically significant anaerobes from the human vagina using conventional approaches with systematic mol
253 ells expressing viral RNA and protein in the vagina, uterus, and pelvic and mesenteric lymph nodes in
257 strated tissue selectivity toward uterus and vagina versus breasts in a rodent model after oral admin
258 nd not seen in HPV-related precursors of the vagina, vulva, and penis further support the notion that
259 ral and nasal cavities, small intestine, and vagina was carried out in female rhesus macaques to dete
261 activity in the keratinized (differentiated) vagina was probably due to dilution of the number of tel
262 ound in human clitoral corpus cavernosum and vagina, we hypothesized that human arginase II is simila
263 cluding stool, oral gingiva, nares, skin and vagina were collected for each maternal-infant dyad.
264 s 14 days postinfection, both the uterus and vagina were found to be positive compared to those of sa
265 dermal LCs, the DCs in the epithelium of the vagina were found to be repopulated mainly by nonmonocyt
267 s, the numbers of T and B lymphocytes in the vagina were similar in vaginally and parenterally immuni
269 lamina propria of the cervical region of the vagina, where a higher number of fibers containing immun
270 ure downregulated RUNX1 in the fornix of the vagina, where DES-associated adenosis is frequently foun
272 were pure NOTES, through a SILS port in the vagina, whereas TV cholecystectomies were hybrid procedu
274 the Acien's hypothesis of Wolffian origin of vagina which explains the development of OHVIRA syndrome
275 s induced adenosis in the cranial portion of vagina, which mimicked the effect of developmental DES-e
276 bulin A chlamydia-specific antibodies in the vagina, while mice immunized with the detergent-extracte
277 igh levels of effector memory CD8 T cells in vagina, while the pool of memory T cells in spleen assum
280 he immune response was reoriented toward the vagina with strikingly higher CD8 T cell responses in th
281 communities in oral, nasal, stool, skin, and vagina, with Proteobacteria as the dominant phylum (60 %
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