ライフサイエンスコーパス: vaginal candidiasis

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1 ers, syphilis, bacterial vaginosis (BV), and vaginal candidiasis.

2 en was greater for oral candidiasis than for vaginal candidiasis.

3 role of gamma/delta T cells in experimental vaginal candidiasis.

4 in established murine models of systemic and vaginal candidiasis.

5 e virulence of C. albicans in a rat model of vaginal candidiasis.

6 transfer completely abrogated resistance to vaginal candidiasis.

7 ctive than rAls1p-N against oropharyngeal or vaginal candidiasis.

8 Gonococcal cervicitis (3.1, 1.1-9.8) and vaginal candidiasis (2.6, 1.2-5.4) were also correlated

9 significant protection against experimental vaginal candidiasis after a subcutaneous immunization wi

10 uggest a minimal role for antibodies against vaginal candidiasis, an experimental rat model has provi

11 Clinicians have traditionally diagnosed vaginal candidiasis, bacterial vaginosis, and vaginal tr

12 Although rodent models of experimental vaginal candidiasis have been useful, several difference

13 using an estrogen-dependent murine model of vaginal candidiasis have demonstrated little to no chang

14 es in currently used rat and mouse models of vaginal candidiasis have generated a large mass of data

15 Vaginal candidiasis, HIV-1 culture positivity, levels of

16 The incidences of vaginal candidiasis (HR, 0.84; 95% CI, 0.67-1.04) and tr

17 candidiasis (OPC) compared with systemic and vaginal candidiasis in female patients with AIDS has bee

18 Vaginal candidiasis is common during pregnancy.

19 Candida infections, the role of CMI against vaginal candidiasis is uncertain, both in humans and in

20 ed from clinical vaginal fluid specimens and vaginal candidiasis model systems.

21 analyzed in 137 human subjects with oral and vaginal candidiasis or carriage.

22 es and/or their limits in the acquisition of vaginal candidiasis remain unclear.

23 genetic relatedness of strains in recurrent vaginal candidiasis: strain maintenance without genetic

24 Results from an animal model of vaginal candidiasis suggest that Candida-specific cell-m

25 s with an estrogen-dependent murine model of vaginal candidiasis suggest that local cell-mediated imm

26 l specimens were studied using two models of vaginal candidiasis to determine how closely these model

27 ry end points were bacterial vaginosis (BV), vaginal candidiasis, trichomoniasis vaginalis (hereafter

28 immunity in protection against experimental vaginal candidiasis was evaluated through the quantifica

29 Protection from vaginal candidiasis was transferred to naive BALB/c mice

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