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1 dy fluids (e.g., semen and saliva, semen and vaginal secretions).
2 cipants had detectable ZIKV RNA in saliva or vaginal secretions.
3 ncluding potassium hydroxide preparation) of vaginal secretions.
4 retes putrescine that is readily detected in vaginal secretions.
5 cific antibody responses in the serum and in vaginal secretions.
6 ical Amsel criteria and direct Gram stain of vaginal secretions.
7 emonstrated by high levels of IgA in eye and vaginal secretions.
8 a significant effect on HIV-1 RNA levels in vaginal secretions.
9 ed Th2-MoPn produced higher levels of IgA in vaginal secretions.
10 gnant women for trichomoniasis by culture of vaginal secretions.
11 ell-free virus from matched blood plasma and vaginal secretions.
12 of Env-specific IgG antibodies in serum and vaginal secretions.
13 ificantly reduced the amount of HIV-1 RNA in vaginal secretions.
14 erline levels of mucosal Abs determined from vaginal secretions.
15 specific IgA antibody activity in serum and vaginal secretions.
16 al fluid (CSF) and semen, but transiently in vaginal secretions.
17 ld induce an antibody response detectable in vaginal secretions.
18 ti-PspA immunoglobulin G titers in serum and vaginal secretions.
19 culture of Trichomonas vaginalis from pooled vaginal secretions.
20 esting transepithelial transport of IgA into vaginal secretions.
21 c IgG and IgA responses in serum, feces, and vaginal secretions.
22 that cellular replication of HIV-1 occurs in vaginal secretions and can result in a virus population
25 immunization in inducing SIV-specific IgA in vaginal secretions and generated greater IgA responses i
26 late with Env-specific antibody responses in vaginal secretions and protection against infection.
28 r ratios and specific antibody activities in vaginal secretions and serum indicated that IgG viral an
29 s type 1 (HIV-1) RNA levels in blood plasma, vaginal secretions, and cervical mucus of 52 HIV-1-infec
30 ant biological fluids--blood, semen, saliva, vaginal secretions, and menstrual blood--in an attempt t
31 was produced in the vagina and released into vaginal secretions at 6 weeks and 10 months after vagina
33 d had yeast identified on wet preparation of vaginal secretions during the past 60 days (aOR, 4.06 [9
39 atinocyte-derived chemokine were elevated in vaginal secretions from infected BALB/c mice, but not in
43 pport the hypothesis that sexual exchange of vaginal secretions is a possible mechanism for acquisiti
44 ding of HIV-1 infected cells in cervical and vaginal secretions may be important determinants of sexu
45 The increase of proinflammatory cytokines in vaginal secretions may serve as a surrogate marker of un
46 correlates of HIV-1 shedding in cervical and vaginal secretions, most notably hormonal contraceptive
47 , significantly less IgG was detected in the vaginal secretions of B7KO mice than in those from wild-
48 vely, the data indicate that IgG antibody in vaginal secretions of immune mice provides early protect
49 t that antibodies are not readily present in vaginal secretions of infected mice and thus have a limi
50 ulin A (IgA) antibody activity in saliva and vaginal secretions of mice given AgI/II with LT-IIa or L
51 tigated the protective role of antibodies in vaginal secretions of mice that were immune to vaginal c
52 Large numbers of chlamydiae were found in vaginal secretions of progesterone-treated and combinati
53 hough the viral antibody titers in serum and vaginal secretions of recipient mice at the time of chal
54 3.12 vs. 3.00 log(10) copies/swab, P=1.0) in vaginal secretions of women receiving vitamin A, compare
56 he detection of proviral DNA in cervical and vaginal secretions (P<.001 and P = .030, respectively).
58 rual cycles in 25 women, HIV-1 RNA levels in vaginal secretions ranged from <1000 to 5.3x10(7) copies
60 IgG plasma cells in the vagina and increased vaginal secretion/serum titer ratios to 3.0- to 4.7-fold
61 in blood plasma was correlated with that in vaginal secretions (Spearman's rank correlation coeffici
63 , as was the median HIV-1 DNA copy number in vaginal secretions (undetectable versus 1.0 HIV-1 DNA co
64 mmunodeficiency virus (HIV) type 1 levels in vaginal secretions, vaginal lavage samples were collecte
65 products from cell-free viral RNA in paired vaginal secretion (VS) and blood plasma (BP) samples of
66 type 1 (HIV-1) RNA shedding in cervical and vaginal secretions was examined daily for 17 HIV-1-serop
67 e collected serum, urine, saliva, semen, and vaginal secretions weekly for the first month and then a
69 ll-free HIV-1 RNA levels in blood plasma and vaginal secretions were negatively correlated with CD4+
70 ore pronounced immune response in saliva and vaginal secretion, while rectal immunization was more po
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