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1 teral subdiaphragmatic vagotomy or were sham-vagotomized.
2 ed in the following groups of anaesthetized, vagotomized adult Sprague-Dawley rats (age 4 months), tr
7 e was also no significant difference between vagotomized and sham-vagotomized rats in the number of c
8 of the previously acquired CTA, because both vagotomized and sham-vagotomized rats rejected all of th
9 Experiments were conducted in anesthetized, vagotomized and spontaneously breathing rats while monit
10 prived) rats were anaesthetized, bilaterally vagotomized and underwent acute PVN inhibition by bilate
12 istered intrathecally (C4) to anaesthetized, vagotomized and ventilated male Sprague-Dawley rats befo
13 10 muM, 5 mul; 3 x 5 min), in anaesthetized, vagotomized and ventilated male Sprague-Dawley rats elic
15 c motor output was recorded in anesthetized, vagotomized, and mechanically ventilated rats with dual
17 fter intermittent hypoxia from anesthetized, vagotomized, and pump-ventilated control and C2 spinally
18 ditions stopped breathing in vagus-intact or vagotomized, anesthetized, spontaneously breathing adult
19 onses evoked by these stimuli were absent in vagotomized animals or in animals pretreated with the ga
20 njection of capsaicin in vagus-intact and in vagotomized animals was also abolished or attenuated aft
21 ose IL-1 beta, stimulation of plasma ACTH in vagotomized animals was also markedly diminished compare
22 s observed in sino-aortically denervated and vagotomized animals, we believe any contribution of the
25 riments were done in urethane-anaesthetized, vagotomized, aortic deafferented, ventilated rats except
27 +/- 0.2%) with intact vagus nerves and three vagotomized cats, AP-SND phase walk was characterized by
30 , additional experiments were performed in 5 vagotomized dogs to investigate vagally mediated mechani
31 ded phrenic activity in seven anaesthetized, vagotomized, glomectomized, paralysed and servo-ventilat
32 iled to increase preoptic PGE2 levels in the vagotomized guinea pigs (n = 10), whereas in their sham-
37 erve activity was measured in anaesthetized, vagotomized, neuromuscularly blocked and artificially ve
38 microA) stimulus intensity in anaesthetized, vagotomized, neuromuscularly blocked and artificially ve
39 (14-15 months old) which were anaesthetized, vagotomized, neuromuscularly blocked and ventilated: (1)
40 1 microM tetrodotoxin (TTX-R) was halved in vagotomized NGNs (21 +/- 8 vs. 56 +/- 8 % of total I(Na)
41 ecovery from inactivation was also slower in vagotomized NGNs (fast time constant, 2.8 +/- 0.4 vs. 1.
44 produced large, repetitive TTX-R I(Na) while vagotomized NGNs produced smaller TTX-S I(Na) that rapid
45 However, steady-state I(Na) inactivation in vagotomized NGNs was shifted -9 mV relative to control v
47 ial waveforms was increased by over 250 % in vagotomized NGNs with DAPs (19.0 +/- 2.1 pC) compared to
48 significantly different among control NGNs, vagotomized NGNs with DAPs and vagotomized NGNs without
51 control NGNs, vagotomized NGNs with DAPs and vagotomized NGNs without DAPs, averaging 54 +/- 7.9 (n =
56 dialysis) of conscious, subdiaphragmatically vagotomized or sham-operated guinea pigs following LPS a
58 not different from baseline levels either in vagotomized (P = 0.18) or intact (P > 0.05) animals.
59 eft C2 spinal cord hemisected, anesthetized, vagotomized, pancuronium paralyzed, and artificially ven
64 ing adult rats (n = 4) and in anaesthetized, vagotomized, paralysed and ventilated animals (n = 14).
65 isocapnic hypoxia in urethane-anaesthetized, vagotomized, paralysed and ventilated rats at different
66 vity was recorded in urethane-anaesthetized, vagotomized, paralysed and ventilated rats exposed to: (
67 ing aorta in 14 Dial-urethane anaesthetized, vagotomized, paralysed, artificially ventilated cats.
68 a in thirty-six Dial-urethane-anaesthetized, vagotomized, paralysed, artificially ventilated cats.
69 were from fifteen anaesthetized, bilaterally vagotomized, paralysed, artificially ventilated cats.
70 on (FI,O1), 0.1-0.12) was measured in twelve vagotomized, paralysed, artificially ventilated young ra
71 airway negative pressure in 15 decerebrated, vagotomized, paralyzed and artificially ventilated cats.
72 neurons in dl-pons in urethane-anesthetized, vagotomized, paralyzed, and servo-ventilated adult Sprag
73 Experiments were performed on anesthetized, vagotomized, paralyzed, ventilated, and spinally injured
74 irus (PRV) was injected into the pancreas of vagotomized rats and after 6 days survival, the pattern
76 cant difference between vagotomized and sham-vagotomized rats in the number of c-FLI-positive cells i
77 uired CTA, because both vagotomized and sham-vagotomized rats rejected all of the test intraoral infu
79 ne), artificially ventilated (FIO2=0.50) and vagotomized rats were presented with two or three, 5 min
80 In chloralose-anaesthetized, ventilated, vagotomized rats, acute hypoxia (10% O2, 60 s) evoked an
82 We used normal rats, subdiaphragmatically vagotomized rats, rats with denervated adrenal medullae
84 mited HPA activation in subdiaphragmatically vagotomized rats, the vagus nerve does not appear to be
94 e-anaesthetized, neuromuscularly blocked and vagotomized Sprague-Dawley rats, arterial blood pressure
97 ter exposure to CIH, urethane-anaesthetized, vagotomized, ventilated, paralysed rats had significantl
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