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1 teral subdiaphragmatic vagotomy or were sham-vagotomized.
2 ed in the following groups of anaesthetized, vagotomized adult Sprague-Dawley rats (age 4 months), tr
3                   In decerebrate, paralysed, vagotomized and artificially ventilated cats, we recorde
4 g inflation, were recorded in anaesthetized, vagotomized and artificially ventilated rats.
5 njected with a neuromuscular blocking agent, vagotomized and artificially ventilated.
6 ed intraperitoneally in subdiaphragmatically vagotomized and sham-operated (SHAM) rats.
7 e was also no significant difference between vagotomized and sham-vagotomized rats in the number of c
8 of the previously acquired CTA, because both vagotomized and sham-vagotomized rats rejected all of th
9  Experiments were conducted in anesthetized, vagotomized and spontaneously breathing rats while monit
10 prived) rats were anaesthetized, bilaterally vagotomized and underwent acute PVN inhibition by bilate
11               In the paralyzed, decerebrate, vagotomized and ventilated cat, baseline values for resp
12 istered intrathecally (C4) to anaesthetized, vagotomized and ventilated male Sprague-Dawley rats befo
13 10 muM, 5 mul; 3 x 5 min), in anaesthetized, vagotomized and ventilated male Sprague-Dawley rats elic
14 ated rats were then anesthetized, paralyzed, vagotomized, and artificially ventilated.
15 c motor output was recorded in anesthetized, vagotomized, and mechanically ventilated rats with dual
16                  The rats were anesthetized, vagotomized, and mechanically ventilated.
17 fter intermittent hypoxia from anesthetized, vagotomized, and pump-ventilated control and C2 spinally
18 ditions stopped breathing in vagus-intact or vagotomized, anesthetized, spontaneously breathing adult
19 onses evoked by these stimuli were absent in vagotomized animals or in animals pretreated with the ga
20 njection of capsaicin in vagus-intact and in vagotomized animals was also abolished or attenuated aft
21 ose IL-1 beta, stimulation of plasma ACTH in vagotomized animals was also markedly diminished compare
22 s observed in sino-aortically denervated and vagotomized animals, we believe any contribution of the
23 intradermal capsaicin in vagus-intact and in vagotomized animals.
24 a extravasation either in vagus-intact or in vagotomized animals.
25 riments were done in urethane-anaesthetized, vagotomized, aortic deafferented, ventilated rats except
26 neously in fifteen anaesthetized, paralysed, vagotomized, artificially ventilated adult cats.
27 +/- 0.2%) with intact vagus nerves and three vagotomized cats, AP-SND phase walk was characterized by
28               In baroreceptor denervated and vagotomized cats, the present study evaluated c-Fos acti
29                           In unanaesthetized vagotomized decerebrated rats, ligation of the internal
30 , additional experiments were performed in 5 vagotomized dogs to investigate vagally mediated mechani
31 ded phrenic activity in seven anaesthetized, vagotomized, glomectomized, paralysed and servo-ventilat
32 iled to increase preoptic PGE2 levels in the vagotomized guinea pigs (n = 10), whereas in their sham-
33                     The results show that in vagotomized lungs, there were less collagen staining, le
34          Administration of PCTR1 or ILC3s to vagotomized mice restored tissue resolution tone and hos
35 nd without vagal innervation in unilaterally vagotomized mice.
36 ctive role of LiCl on feeding was blunted in vagotomized mice.
37 erve activity was measured in anaesthetized, vagotomized, neuromuscularly blocked and artificially ve
38 microA) stimulus intensity in anaesthetized, vagotomized, neuromuscularly blocked and artificially ve
39 (14-15 months old) which were anaesthetized, vagotomized, neuromuscularly blocked and ventilated: (1)
40  1 microM tetrodotoxin (TTX-R) was halved in vagotomized NGNs (21 +/- 8 vs. 56 +/- 8 % of total I(Na)
41 ecovery from inactivation was also slower in vagotomized NGNs (fast time constant, 2.8 +/- 0.4 vs. 1.
42             A subset (approximately 25 %) of vagotomized NGNs displayed depolarizing after-potentials
43                   In conclusion, a subset of vagotomized NGNs have increased Ca(2+) currents and expr
44 produced large, repetitive TTX-R I(Na) while vagotomized NGNs produced smaller TTX-S I(Na) that rapid
45  However, steady-state I(Na) inactivation in vagotomized NGNs was shifted -9 mV relative to control v
46             Total I(Na) and I(Na) density in vagotomized NGNs were similar to control values.
47 ial waveforms was increased by over 250 % in vagotomized NGNs with DAPs (19.0 +/- 2.1 pC) compared to
48  significantly different among control NGNs, vagotomized NGNs with DAPs and vagotomized NGNs without
49 2+) influx were increased proportionately in vagotomized NGNs with DAPs.
50 compared to control NGNs (5.0 +/- 0.8 pC) or vagotomized NGNs without DAPs (7.0 +/- 0.8 pC).
51 control NGNs, vagotomized NGNs with DAPs and vagotomized NGNs without DAPs, averaging 54 +/- 7.9 (n =
52 tion underlies the decreased AP discharge of vagotomized NGNs.
53 a) activation, inactivation and repriming in vagotomized NGNs.
54 was abolished or attenuated in the brains of vagotomized or capsaicin-treated animals.
55          These measurements were repeated in vagotomized or haloperidol-treated mice, and in animals
56 dialysis) of conscious, subdiaphragmatically vagotomized or sham-operated guinea pigs following LPS a
57                            In groups of sham-vagotomized or vagotomized rats, we surgically removed o
58 not different from baseline levels either in vagotomized (P = 0.18) or intact (P > 0.05) animals.
59 eft C2 spinal cord hemisected, anesthetized, vagotomized, pancuronium paralyzed, and artificially ven
60        The rats were urethane anaesthetized, vagotomized, paralysed and artificially ventilated.
61        Animals were artificially ventilated, vagotomized, paralysed and given a pneumothorax.
62 awley rats were anaesthetized with urethane, vagotomized, paralysed and pump ventilated.
63                                 Decerebrate, vagotomized, paralysed and ventilated adult rats were st
64 ing adult rats (n = 4) and in anaesthetized, vagotomized, paralysed and ventilated animals (n = 14).
65 isocapnic hypoxia in urethane-anaesthetized, vagotomized, paralysed and ventilated rats at different
66 vity was recorded in urethane-anaesthetized, vagotomized, paralysed and ventilated rats exposed to: (
67 ing aorta in 14 Dial-urethane anaesthetized, vagotomized, paralysed, artificially ventilated cats.
68 a in thirty-six Dial-urethane-anaesthetized, vagotomized, paralysed, artificially ventilated cats.
69 were from fifteen anaesthetized, bilaterally vagotomized, paralysed, artificially ventilated cats.
70 on (FI,O1), 0.1-0.12) was measured in twelve vagotomized, paralysed, artificially ventilated young ra
71 airway negative pressure in 15 decerebrated, vagotomized, paralyzed and artificially ventilated cats.
72 neurons in dl-pons in urethane-anesthetized, vagotomized, paralyzed, and servo-ventilated adult Sprag
73  Experiments were performed on anesthetized, vagotomized, paralyzed, ventilated, and spinally injured
74 irus (PRV) was injected into the pancreas of vagotomized rats and after 6 days survival, the pattern
75                                          The vagotomized rats did not respond differently than their
76 cant difference between vagotomized and sham-vagotomized rats in the number of c-FLI-positive cells i
77 uired CTA, because both vagotomized and sham-vagotomized rats rejected all of the test intraoral infu
78                          Adult anesthetized, vagotomized rats were exposed to hypoxia (8% O2, mean=45
79 ne), artificially ventilated (FIO2=0.50) and vagotomized rats were presented with two or three, 5 min
80     In chloralose-anaesthetized, ventilated, vagotomized rats, acute hypoxia (10% O2, 60 s) evoked an
81                                           In vagotomized rats, GABA(A)ergic and glycinergic antagonis
82    We used normal rats, subdiaphragmatically vagotomized rats, rats with denervated adrenal medullae
83                                           In vagotomized rats, RTN neurons were inhibited to a variab
84 mited HPA activation in subdiaphragmatically vagotomized rats, the vagus nerve does not appear to be
85             In groups of sham-vagotomized or vagotomized rats, we surgically removed or denervated th
86 re recorded in anaesthetized, ventilated and vagotomized rats.
87 s and GES effects on all neurons examined in vagotomized rats.
88 d extracellularly in anaesthetized intact or vagotomized rats.
89 etized, ventilated, neuromuscularly blocked, vagotomized rats.
90 was studied using awake subdiaphragmatically vagotomized rats.
91  in anaesthetized sino-aortic denervated and vagotomized rats.
92  HF (n=5) to those observed in sham-operated vagotomized rats.
93  into the nodose ganglion ipsilateral to the vagotomized side.
94 e-anaesthetized, neuromuscularly blocked and vagotomized Sprague-Dawley rats, arterial blood pressure
95 heir excitability in spontaneously breathing vagotomized urethane-anesthetized adult rats.
96 on on respiration in spontaneously breathing vagotomized urethane-anesthetized rats.
97 ter exposure to CIH, urethane-anaesthetized, vagotomized, ventilated, paralysed rats had significantl

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