ライフサイエンスコーパス: vagus

1 ptin and electrical activity of the afferent vagus.

2 salis of Meynert and dorsal motor nucleus of vagus.

3 s solitaries and dorsal motor nucleus of the vagus.

4 s located in the dorsal motor nucleus of the vagus.

5 neural fulcrum, can be elicited from either vagus.

6 s solitarius and dorsal motor nucleus of the vagus.

7 solitary tract; and dorsal motor nucleus of vagus.

8 of parasympathetic function mediated by the vagus.

9 he penis (DNP) and abdominal branches of the vagus.

10 t (NTS), and the dorsal motor nucleus of the vagus.

11 marker of sensory nerve activation) of human vagus.

12 y tract, and the dorsal motor nucleus of the vagus.

13 ambiguus and the dorsal motor nucleus of the vagus.

14 the gut and ascend to the brainstem via the vagus.

15 itary tract, and dorsal motor nucleus of the vagus.

16 y volunteers were assessed and compared with vagus/airway nerve and cough responses in a cigarette sm

17 identified received indirect central inputs (vagus alone: 25%; stellate ganglion alone: 27%; both: 48

18 in response to electrical stimulation of the vagus and activation of cardiopulmonary, chemo- and baro

19 ct (NTS) and the dorsal motor nucleus of the vagus and affected within-meal satiation.

20 dies in rodents identified the hypothalamus, vagus and brainstem regions as potential sites of action

21 olarization of murine, guinea pig, and human vagus and firing of Adelta-fibers (not C-fibers), which

22 e of pharmacological studies on the isolated vagus and patch clamp and single-channel inside-out expe

23 ed to the reticuloendothelial system via the vagus and splenic nerves.

24 rated within the dorsal motor nucleus of the vagus and the hypoglossal nucleus.

25 he lateral NTS, dorsal medial nucleus of the vagus, and other areas of the dorsal hindbrain had no ef

26 voked depolarization of guinea pig and human vagus, and this was inhibited by a transient receptor po

27 t procedures such as deep brain stimulation, vagus, and trigeminal nerve stimulation are effective on

28 us coeruleus and dorsal motor nucleus of the vagus, but not in the nucleus basalis of Meynert, raphe

29 itarius, and the dorsal motor nucleus of the vagus, but not the area postrema.

30 ted no obvious disruption and lesions of the vagus caused some alterations to song behavior.

31 Dissection of the right vagus decreased peritoneal group 3 innate lymphoid cell

32 y nucleus [NST], dorsal motor nucleus of the vagus [DMN] and catecholaminergic neurons of the ventrol

33 tract (nTS) and dorsal motor nucleus of the vagus (DMNV).

34 mbiguus (NA) and dorsal motor nucleus of the vagus (DMNX).

35 solitarius (NTS) dorsal motor nucleus of the vagus (DMV) and area postrema) decrease gastric tone and

36 f neurons in the dorsal motor nucleus of the vagus (DMV) and nucleus ambiguus (nAmb) that express the

37 The dorsal motor nucleus of the vagus (DMV) contains preganglionic motor neurons that co

38 The dorsal motor nucleus of the vagus (DMV) in the brainstem consists primarily of vagal

39 alamus (PVN) and dorsal motor nucleus of the vagus (DMV) in the mouse brain as well as expression of

40 The Dorsal Motor Nucleus of Vagus (DMV) is degenerated in many patients with early s

41 re made from rat dorsal motor nucleus of the vagus (DMV) neurones in thin brainstem slices.

42 FD) dysregulated dorsal motor nucleus of the vagus (DMV) neurones, prior to the development of obesit

43 are modulated by dorsal motor nucleus of the vagus (DMV) neurones, whose activity is finely tuned by

44 eactivity in rat dorsal motor nucleus of the vagus (DMV) neurones.

45 orpus-projecting dorsal motor nucleus of the vagus (DMV) neurones.

46 neurones of the dorsal motor nucleus of the vagus (DMV) to GLP-1 and its analogues; (2) the effects

47 terneuron in the dorsal motor nucleus of the vagus (DMV), a nucleus that is intimately involved in re

48 d neurons in the dorsal motor nucleus of the vagus (DMV), a population of cholinergic neurons that sh

49 olitarius (NTS), dorsal motor nucleus of the vagus (DMV), and ventrolateral medulla (VLM).

50 ansmitter in the dorsal motor nucleus of the vagus (DMV).

51 lease in the rat dorsal motor nucleus of the vagus (DMV).

52 c neurons of the dorsal motor nucleus of the vagus (DMV).

53 m neurons in the dorsal motor nucleus of the vagus (DMV).

54 tarius (NTS) and dorsal motor nucleus of the vagus (DMV).

55 iginating in the dorsal motor nucleus of the vagus (DMV).

56 o neurons of the dorsal motor nucleus of the vagus (DMV).

57 on by injecting anterograde tracers into the vagus evoked potential (VEP) focus in Pf of macaque monk

58 al and dorsal trunks of the subdiaphragmatic vagus following electrical stimulation of the left or ri

59 ent fibres was connected to the intact right vagus in chronically instrumented dogs.

60 the afferent (sensory) and motor (efferent) vagus in regulation of appetite, mood, and the immune sy

61 The role of the vagus in the physiological, behavioral and neurochemical

62 o and depolarization of human and guinea pig vagus in vitro, and inhibited calcium influx in airway-s

63 em at the mid-pontine level blocks access of vagus-induced activity that ascends to the oculomotor nu

64 ing through the common hepatic branch of the vagus inhibits lard, but not chow, intake, directs fat d

65 nder control conditions stopped breathing in vagus-intact or vagotomized, anesthetized, spontaneously

66 The effect in vagus-intact rats was to slow the rhythm to a pace equiv

67 nerve fibres by stimulation of the cervical vagus is associated with NO production and release in th

68 omata, suggesting that direct input from the vagus may only account for a minority of the information

69 (including the parasympathetic dorsal motor vagus) mediated improved glucose homeostasis independent

70 The vagus motor neuron topographic map is therefore determin

71 that axon initiation is delayed in posterior vagus motor neurons independent of neuron birth time.

72 We focus on the vagus motor neurons, which are topographically arranged

73 re expressed in posterior, but not anterior, vagus motor neurons.

74 odels indicate various lesions affecting the vagus, muscle, enteric neurons, interstitial cells of Ca

75 the proximal end of the transected cervical vagus nerve (3 pulses, 100 Hz, 1 ms, 100-500 muA).

76 1)R in afferent and efferent branches of the vagus nerve (Cnr1(flox/flox); Phox2b-Cre mice).

77 Efferent activation of the cervical vagus nerve (cVN) dampens systemic inflammatory processe

78 ry tract (mNTS), dorsal motor nucleus of the vagus nerve (DMV) and the hypoglossal nucleus (HN).

79 ical stimulation of the central stump of the vagus nerve (ESV) transected at the spinal cervical leve

80 ients by closed-loop stimulation of the left vagus nerve (LVN).

81 their anatomical relation to branches of the vagus nerve (Xth cranial nerve).

82 iorates gut barrier dysfunction in sepsis by vagus nerve activation via central ghrelin receptors.

83 -inflammatory influences but also may reduce vagus nerve activity.

84 Here, we identify two populations of mouse vagus nerve afferents (P2ry1, Npy2r), each a few hundred

85 eus (Pf) in monkeys is strongly activated by vagus nerve afferents.

86 or in 12 dogs to stimulate the left cervical vagus nerve and a radiotransmitter for continuous record

87 ishes the anti-inflammatory potential of the vagus nerve and beta2-agonists to control inflammation i

88 This study indicates that vagus nerve and cholinergic agonists activate the sympat

89 Likewise, vagus nerve and cholinergic agonists fail to control inf

90 blish the anti-inflammatory potential of the vagus nerve and cholinergic agonists in immunocompromise

91 Thus, vagus nerve and cholinergic agonists inhibit systemic in

92 the anti-inflammatory potential of both the vagus nerve and cholinergic agonists, and abrogate their

93 e (ACh) is the major neurotransmitter of the vagus nerve and exerts its parasympathetic actions via a

94 n anti-inflammatory reflex arc involving the vagus nerve and memory T cells is necessary for resoluti

95 increased responses were observed in ex vivo vagus nerve and neuron cell bodies in the vagal ganglia.

96 c mechanism that involves recruitment of the vagus nerve and subsequent activation of endocannabinoid

97 was abolished by surgical transection of the vagus nerve and was not observed in other peripheral org

98 These results unveil CB(1)R in the vagus nerve as a key component underlying normal gastroi

99 omyography input and use it to stimulate the vagus nerve at specific time points of the respiratory c

100 ermine whether electrical stimulation of the vagus nerve attenuates kidney ischemia-reperfusion injur

101 blockade of the afferent transmission in the vagus nerve by topical application of local anaesthetic.

102 Furthermore neuromodulation of the vagus nerve can be used in the treatment of obesity.

103 A prototypical vagus nerve circuit, the inflammatory reflex, inhibits t

104 The vagus nerve contains afferent fibers that transmit senso

105 Thus, the vagus nerve contains intermingled sensory neurons consti

106 The vagus nerve contains primary visceral afferents that con

107 The vagus nerve controls inflammation in healthy, but not in

108 esponse to VCMS, providing evidence that the vagus nerve conveys VCMS directly from the genitalia to

109 tumor necrosis factor effect of the efferent vagus nerve could be a therapeutic target in IBD through

110 ugh it is known that peripheral axons of the vagus nerve degenerate and then regenerate to a limited

111 n subdiaphragmatically vagotomized rats, the vagus nerve does not appear to be the only route by whic

112 regulates leukocyte trafficking because the vagus nerve does not innervate endothelial cells.

113 s well as the pathophysiological outcomes of vagus nerve dysfunction resulting in obesity, mood disor

114 An additional mechanism regulating vagus nerve effects on the pancreas involves Group II an

115 ell as rats, posttraining stimulation of the vagus nerve enhances memory consolidation.

116 velop future pharmacotherapy targeted to the vagus nerve for the treatment of obesity are proposed.

117 e demonstrated the importance of splenic and vagus nerve functions in the inflammatory process throug

118 The vagus nerve has been implicated in the regulation of sev

119 These findings corroborate the role of the vagus nerve in regulating hippocampal activity and the i

120 review highlights evidence for a role of the vagus nerve in the development of obesity and how target

121 nce of the action of CCK at the level of the vagus nerve in the regulation of food intake, body weigh

122 Stimulation of the vagus nerve in the so-called cholinergic antiinflammator

123 Stimulation of the vagus nerve increases fibrogenic cytokines in humans, th

124 The efferent vagus nerve inhibits cytokine release through alpha7-nic

125 The vagus nerve innervates visceral organs and may contribut

126 The vagus nerve innervating the gut plays an important role

127 The vagus nerve is a key body-brain connection that monitors

128 The vagus nerve is a major conduit between lung and brain re

129 The common hepatic branch of the vagus nerve is a two-way highway of communication betwee

130 The vagus nerve is an important source of afferent informati

131 conclusion there is strong evidence that the vagus nerve is involved in the development of obesity an

132 The vagus nerve is thought to participate in signal transduc

133 It indicated that the vagus nerve may influence PF by enhancing fibrogenic fac

134 Pharmacologic stimulation of the vagus nerve may offer a novel approach of anti-sepsis th

135 The therapeutic potential of vagus nerve modulation to attenuate or reverse these pat

136 Aplysia californica, and in axons within the vagus nerve of a mammal, the musk shrew Suncus murinus.

137 the effects of electrical stimulation of the vagus nerve on learning and memory, mood, seizure suppre

138 st the relevance of CB(1)R in neurons of the vagus nerve on metabolic homeostasis and gastrointestina

139 s suggest an anti-fibrillatory action of the vagus nerve on the ventricle, although the exact mechani

140 Treatment with vagus nerve or sham stimulation was administered concurr

141 Stimulation of the vagus nerve prevents the damaging effects of cytokine re

142 teral electrical stimulation of the cervical vagus nerve produced bradypnoea by exclusively extending

143 The vagus nerve projects directly to the HSD2 neurons and th

144 In particular, the vagus nerve provides the parasympathetic innervation to

145 Transection of the vagus nerve proximal to the ganglion results in loss of

146 Thus, action potentials originating in the vagus nerve regulate T cells, which in turn produce the

147 These results indicate that the vagus nerve regulates both netrin-1 and pro-resolving li

148 n-initiated peritoneal inflammation that the vagus nerve regulates local expression of netrin-1, an a

149 Recent studies have implicated vagus nerve regulation of splenic cholinergic nicotinic

150 injections of both CTb and IB4 into the same vagus nerve resulted in labeling of two exclusive popula

151 Extracellular recordings in the isolated vagus nerve revealed that the conduction of action poten

152 rate variability (markers of low cholinergic/vagus nerve sensitivity).

153 We have previously shown that efferent vagus nerve signals regulate cytokine production through

154 the "inflammatory reflex," is dependent upon vagus nerve signals that inhibit cytokine production and

155 fluid percussion injury (FPI), FPI with sham Vagus Nerve Simulation (VNS), and FPI with chronic inter

156 ement anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selectively ac

157 We hypothesize that left-sided low-level vagus nerve stimulation (LL-VNS) can suppress sympatheti

158 Vagus nerve stimulation (up to four times daily) in RA p

159 Specifically, the authors demonstrate that vagus nerve stimulation (VNS) activates the cholinergic

160 The hypothesis that left vagus nerve stimulation (VNS) at the cervical level resu

161 ctive of this study was to determine whether vagus nerve stimulation (VNS) can enhance the consolidat

162 Vagus nerve stimulation (VNS) has been shown to exert ca

163 ABSTRACT: Vagus nerve stimulation (VNS) is an emerging therapy for

164 Noninvasive, transcutaneous vagus nerve stimulation (VNS) is currently used as a tre

165 e immunity, and modulation of this reflex by vagus nerve stimulation (VNS) is effective in various in

166 sought to investigate the effect of cervical vagus nerve stimulation (VNS) on cerebral blood flow (CB

167 he pilot study was to evaluate the effect of Vagus Nerve Stimulation (VNS) paired with sounds in chro

168 Recent research has shown that vagus nerve stimulation (VNS) paired with tones or with

169 We have previously shown that direct vagus nerve stimulation (VNS) reduces the slope of actio

170 evoked cardiac response to bipolar cervical vagus nerve stimulation (VNS) reflects a dynamic interac

171 Vagus nerve stimulation (VNS) therapy was shown to impro

172 ion Registry investigated whether adjunctive vagus nerve stimulation (VNS) with treatment as usual in

173 Only vagus nerve stimulation (VNS), which continues to develo

174 eloped a method of transcutaneous mechanical vagus nerve stimulation and then investigated whether th

175 erve stimulation (SNS) and right and/or left vagus nerve stimulation and was compared with DOR during

176 receive electrical, transcutaneous, or sham vagus nerve stimulation and were followed for survival o

177 epeatedly pairing tones with brief pulses of vagus nerve stimulation completely eliminated the physio

178 Left-sided low-level vagus nerve stimulation did not change vagal nerve activ

179 Transcutaneous vagus nerve stimulation dose-dependently reduced systemi

180 ntiinflammatory pathway." Here, we show that vagus nerve stimulation during endotoxemia specifically

181 Vagus nerve stimulation fails to control serum TNF level

182 Here we show that vagus nerve stimulation fails to inhibit tumor necrosis

183 US Food and Drug Administration approval of vagus nerve stimulation for recurrent treatment-resistan

184 hange in the direction of DOR, but bilateral vagus nerve stimulation increased and reversed DOR to ba

185 Axial stretch and vagus nerve stimulation induced cranial displacement of

186 Treatment with transcutaneous vagus nerve stimulation inhibited HMGB1 levels and impro

187 Electrical vagus nerve stimulation inhibits proinflammatory cytokin

188 Vagus nerve stimulation inhibits proinflammatory cytokin

189 Transcutaneous vagus nerve stimulation is an efficacious treatment for

190 vidence that the anti-fibrillatory effect of vagus nerve stimulation on the ventricle is mediated by

191 t may be exploited through either electrical vagus nerve stimulation or administration of alpha7 agon

192 Vagus nerve stimulation paired with exposure to conditio

193 It is unclear how vagus nerve stimulation regulates leukocyte trafficking

194 Vagus nerve stimulation significantly (P<0.01) decreased

195 Similarly, vagus nerve stimulation significantly attenuates neutrop

196 Left-sided low-level vagus nerve stimulation suppresses stellate ganglion ner

197 Together, these results establish that vagus nerve stimulation targeting the inflammatory refle

198 te that bilateral autonomic activity (SNS or vagus nerve stimulation) cause reversible shifts of apex

199 mulation, (3) surgical approaches, including vagus nerve stimulation, epidural electrical stimulation

200 n of nicotine, an alpha7 agonist that mimics vagus nerve stimulation, increases proinflammatory cytok

201 on celiac nerve abolishes TNF suppression by vagus nerve stimulation, suggesting that the cholinergic

202 During right or left vagus nerve stimulation, there was no change in the dire

203 timulation frequency-dependent manner during vagus nerve stimulation, with comparable increases seen

204 red for inhibition of cytokine production by vagus nerve stimulation.

205 r functional inhibition of TNF production by vagus nerve stimulation.

206 ugh neurostimulation techniques, such as the vagus nerve stimulator.

207 The vagus nerve supplies low-threshold chemo- and mechanosen

208 expansion via electrical stimulation of the vagus nerve to identify the brain circuits responsible f

209 ction or injury travels through the afferent vagus nerve to integrative regions in the brainstem, and

210 AchR) has been shown to be necessary for the vagus nerve to modulate the systemic inflammatory respon

211 Recent observations linking the vagus nerve to plasticity in the central nervous system

212 other to provide rhythmic motor drive to the vagus nerve to slow the heart.

213 by anorexigenic hormones, which act via the vagus nerve to stimulate feeding, are unknown.

214 s to exert axial stretch on the LES, and the vagus nerve was isolated in the neck for electrical stim

215 Human vagus nerve was used to confirm key observations in anim

216 perexcitability in large myelinated axons in vagus nerve which could contribute to autonomic dysfunct

217 ctively transected the hepatic branch of the vagus nerve while infusing resveratrol centrally.

218 development of obesity and how targeting the vagus nerve with neuromodulation or pharmacology can be

219 em regulates innate immune responses via the vagus nerve, a mechanism termed the cholinergic antiinfl

220 iginating in the dorsal motor nucleus of the vagus nerve, and the second postganglionic, originating

221 cants that affect the brain-gut axis via the vagus nerve, and then travel to higher centers, compromi

222 nflammatory potential of the parasympathetic vagus nerve, and they represent a potential pharmacologi

223 Afferent drive in cough is carried by the vagus nerve, and vagal afferent nerve terminals have bee

224 l pathways, with or without mediation by the vagus nerve, are known to participate in regulation of e

225 ependent of GLP-1 receptors (GLP-1Rs) in the vagus nerve, area postrema, and paraventricular nucleus.

226 us system, via an inflammatory reflex of the vagus nerve, can inhibit cytokine release and thereby pr

227 y, a severing of the peripheral axons of the vagus nerve, has been extensively utilized to determine

228 olish antiinflammatory signaling through the vagus nerve, indicating that peripheral muscarinic recep

229 e nucleus, medial lemniscus, pontine nuclei, vagus nerve, inferior olive, abducens nucleus, and motor

230 gical resection of the hepatic branch of the vagus nerve, negates the effects of central insulin and

231 Vagal blockade, which inhibits the vagus nerve, results in significant weight loss.

232 nvergence of the dorsal motor nucleus of the vagus nerve, sections of the medial retropharyngeal lymp

233 Effects may be mediated via the vagus nerve, spinal cord, or neuroendocrine systems.

234 a region which is densely innervated by the vagus nerve, suggesting that they could receive direct v

235 Defining the interactions among the vagus nerve, the enteric nervous system, and the intesti

236 at juxtaparanodes of myelinated axons in the vagus nerve, the primary conduit for parasympathetic inn

237 xpression in juxtaparanodes of the wild-type vagus nerve, the primary source of parasympathetic input

238 nervated by the gastroduodenal branch of the vagus nerve, the transection of which influences food in

239 vascular areas innervated by branches of the vagus nerve, whereas only cells in the carotid labyrinth

240 ervation from a nonspinal source through the vagus nerve, which innervates the distal colon as well.

241 uring sterile endotoxemia independent of the vagus nerve, without affecting innate immune cell activa

242 rallel, neural reflex circuits including the vagus nerve-based inflammatory reflex are physiological

243 ion of cytokine production, is essential for vagus nerve-mediated regulation of neutrophil activation

244 f fundoplication restored axial stretch- and vagus nerve-stimulated LES relaxation as well as LES cra

245 Here we show that an implantable vagus nerve-stimulating device in epilepsy patients inhi

246 eneral visceral information largely from the vagus nerve.

247 by parasympathetic signals delivered by the vagus nerve.

248 ry neurons traveling to the brainstem in the vagus nerve.

249 e ganglionic structure situated alone in the vagus nerve.

250 he gut and recruiting local afferents of the vagus nerve.

251 erent parasympathetic fibres of the cervical vagus nerve.

252 ount of direct viscerosensory input from the vagus nerve.

253 ytokines in sepsis through activation of the vagus nerve.

254 iple functions, including stimulation of the vagus nerve.

255 of ghrelin is mediated by activation of the vagus nerve.

256 diated signaling to the hypothalamus via the vagus nerve.

257 ting through hepatic branch afferents of the vagus nerve.

258 n is regulated by neural signals through the vagus nerve.

259 d wired to the spleen via this branch of the vagus nerve.

260 nt signaling at the central processes of the vagus nerve.

261 e brain from visceral regions is through the vagus nerve.

262 olarization of guinea pig and human isolated vagus nerve.

263 brain UGN augments fecal output through the vagus nerve.

264 nucleus of the solitary tract (NTS) via the vagus nerve.

265 (LXRalpha/beta) in the nodose ganglia of the vagus nerve.

266 and cells, which are closely related to the vagus nerve.

267 on, with these effects being mediated by the vagus nerve.

268 ubunit (CTb) and isolectin B4 (IB4) into the vagus nerve.

269 fibrillation ablation on the function of the vagus nerve/UGI system.

270 trally administered methoctramine stimulated vagus-nerve activity measured by changes in instantaneou

271 ulating the rostral cut ends of the cervical vagus nerves also evoked a sympathetic adrenergic nerve-

272 DAF-2 DA) during stimulation of the cervical vagus nerves and acetylcholine perfusion in the absence

273 rphology of the facial, glossopharyngeal and vagus nerves are abnormal in Spry1-/-;Spry2-/- embryos.

274 , phrenic (PN), hypoglossal (HN) and central vagus nerves from neonatal and juvenile rats in situ.

275 ed A- and Adelta-axons from excised cervical vagus nerves of young adult Kcna1-null mice and age-matc

276 The vagus nerves provide much of the capsaicin-sensitive noc

277 matory mechanism mediated by the sciatic and vagus nerves that modulates the production of catecholam

278 After the C-fibre conduction in both vagus nerves was blocked, right-atrial injection of caps

279 Electrical stimulation of the right and left vagus nerves was initiated 30min after the induction of

280 tension events from the glossopharyngeal and vagus nerves, respectively.

281 In rats with intact vagus nerves, RTN neurons were inhibited, also to a vari

282 acial nerve and not the glossopharyngeal and vagus nerves, suggesting that the facial nerve is most s

283 e autonomic nerves: the glossopharyngeal and vagus nerves.

284 rfusion or acidification are mediated by the vagus nerves.

285 e stimulation of the left and right cervical vagus nerves.

286 n neurons located in the dorsal motor of the vagus or indirectly by acting on pancreatic stellate cel

287 In addition, unilateral lesion of the vagus reduced Fos expression in the ipsilateral nucleus

288 rase in the CPEB2 KO dorsal motor nucleus of vagus resulted in hyperactivation of parasympathetic sig

289 n neurons in the dorsal motor nucleus of the vagus that project to the stomach.

290 roscopy, we observed a sparse input from the vagus to most HSD2 neurons.

291 nd efferent pathways of the subdiaphragmatic vagus to physiology and behavior.

292 n's disease, the dorsal motor nucleus of the vagus undergoes severe degeneration and pathological alp

293 efferent) components of the subdiaphragmatic vagus using unilateral rhizotomy combined with contralat

294 ostrema, and the dorsal motor nucleus of the vagus), ventrolateral periaqueductal gray, dorsal parabr

295 Lastly, the vagus was de-afferented with capsaicin, and 5HT3-recepto

296 ansmission in the KO dorsal motor nucleus of vagus was overactivated because KO mice lack CPEB2-suppr

297 nto those whose common hepatic branch of the vagus was treated with vehicle or capsaicin.

298 r neurons in the dorsal motor nucleus of the vagus were found only in mice.

299 stimulation of the abdominal branches of the vagus, which innervates portions of the female reproduct

300 Anterograde tracing from the vagus with CTb or IB4 was combined with immunohistochemi