ライフサイエンスコーパス: vagus nerve stimulation

1 red for inhibition of cytokine production by vagus nerve stimulation.

2 r functional inhibition of TNF production by vagus nerve stimulation.

3 y lung inflation (7.5 cm H2O) or mimicked by vagus nerve stimulation (50 Hz).

4 The place of vagus nerve stimulation among antiepileptic drugs and ot

5 , with the carrageenan air pouch model, both vagus nerve stimulation and cholinergic agonists signifi

6 eloped a method of transcutaneous mechanical vagus nerve stimulation and then investigated whether th

7 erve stimulation (SNS) and right and/or left vagus nerve stimulation and was compared with DOR during

8 receive electrical, transcutaneous, or sham vagus nerve stimulation and were followed for survival o

9 e association between temporal lobe surgery, vagus nerve stimulation, and other non-pharmacological t

10 te that bilateral autonomic activity (SNS or vagus nerve stimulation) cause reversible shifts of apex

11 epeatedly pairing tones with brief pulses of vagus nerve stimulation completely eliminated the physio

12 ement anti-inflammatory therapy via cervical vagus nerve stimulation (cVNS) one should selectively ac

13 Left-sided low-level vagus nerve stimulation did not change vagal nerve activ

14 Transcutaneous vagus nerve stimulation dose-dependently reduced systemi

15 ntiinflammatory pathway." Here, we show that vagus nerve stimulation during endotoxemia specifically

16 mulation, (3) surgical approaches, including vagus nerve stimulation, epidural electrical stimulation

17 Vagus nerve stimulation fails to control serum TNF level

18 Here we show that vagus nerve stimulation fails to inhibit tumor necrosis

19 US Food and Drug Administration approval of vagus nerve stimulation for recurrent treatment-resistan

20 Now nearly 5 years post-approval, vagus nerve stimulation has emerged as a major non-pharm

21 This review evaluates the role of vagus nerve stimulation in light of recently published r

22 hange in the direction of DOR, but bilateral vagus nerve stimulation increased and reversed DOR to ba

23 n of nicotine, an alpha7 agonist that mimics vagus nerve stimulation, increases proinflammatory cytok

24 Axial stretch and vagus nerve stimulation induced cranial displacement of

25 Treatment with transcutaneous vagus nerve stimulation inhibited HMGB1 levels and impro

26 Electrical vagus nerve stimulation inhibits proinflammatory cytokin

27 Vagus nerve stimulation inhibits proinflammatory cytokin

28 Transcutaneous vagus nerve stimulation is an efficacious treatment for

29 We hypothesize that left-sided low-level vagus nerve stimulation (LL-VNS) can suppress sympatheti

30 The effects of periodic left vagus nerve stimulation (LVNS) on chemically-induced sei

31 vidence that the anti-fibrillatory effect of vagus nerve stimulation on the ventricle is mediated by

32 Studies of the effect of vagus nerve stimulation on ventricular myocardial functi

33 t may be exploited through either electrical vagus nerve stimulation or administration of alpha7 agon

34 Vagus nerve stimulation paired with exposure to conditio

35 It is unclear how vagus nerve stimulation regulates leukocyte trafficking

36 Vagus nerve stimulation significantly (P<0.01) decreased

37 Similarly, vagus nerve stimulation significantly attenuates neutrop

38 In contrast to vagus nerve stimulation studies, no substantial cardiova

39 on celiac nerve abolishes TNF suppression by vagus nerve stimulation, suggesting that the cholinergic

40 Cervical vagus nerve stimulation suppressed power in the 8-12-Hz

41 Left-sided low-level vagus nerve stimulation suppresses stellate ganglion ner

42 Together, these results establish that vagus nerve stimulation targeting the inflammatory refle

43 During right or left vagus nerve stimulation, there was no change in the dire

44 a clinical study evaluating the capacity of vagus nerve stimulation to control epilepsy.

45 Vagus nerve stimulation (up to four times daily) in RA p

46 ical vagus nerve section, and during central vagus nerve stimulation (VAGSTIM).

47 Specifically, the authors demonstrate that vagus nerve stimulation (VNS) activates the cholinergic

48 The hypothesis that left vagus nerve stimulation (VNS) at the cervical level resu

49 ctive of this study was to determine whether vagus nerve stimulation (VNS) can enhance the consolidat

50 Vagus nerve stimulation (VNS) has been shown to exert ca

51 ABSTRACT: Vagus nerve stimulation (VNS) is an emerging therapy for

52 Noninvasive, transcutaneous vagus nerve stimulation (VNS) is currently used as a tre

53 e immunity, and modulation of this reflex by vagus nerve stimulation (VNS) is effective in various in

54 sought to investigate the effect of cervical vagus nerve stimulation (VNS) on cerebral blood flow (CB

55 m effects of episodic inspiratory-inhibitory vagus nerve stimulation (VNS) on phrenic nerve activity.

56 he pilot study was to evaluate the effect of Vagus Nerve Stimulation (VNS) paired with sounds in chro

57 Recent research has shown that vagus nerve stimulation (VNS) paired with tones or with

58 We have previously shown that direct vagus nerve stimulation (VNS) reduces the slope of actio

59 evoked cardiac response to bipolar cervical vagus nerve stimulation (VNS) reflects a dynamic interac

60 Vagus nerve stimulation (VNS) therapy was shown to impro

61 ion Registry investigated whether adjunctive vagus nerve stimulation (VNS) with treatment as usual in

62 We hypothesized that electrical vagus nerve stimulation (VNS) would suppress harmaline t

63 Only vagus nerve stimulation (VNS), which continues to develo

64 Vagus-nerve stimulation (VNS) is licensed in several cou

65 timulation frequency-dependent manner during vagus nerve stimulation, with comparable increases seen