ライフサイエンスコーパス: valve formation

1 se in integrin alpha9 expression at sites of valve formation.

2 tion protein Cx37 are required for lymphatic valve formation.

3 ed by the morphogenetic events that initiate valve formation.

4 Sema3A-Nrp1-PlexinA1 signaling in lymphatic valve formation.

5 formation, which is essential to subsequent valve formation.

6 aphorin3A (Sema3A) plays a role in lymphatic valve formation.

7 normal thoracic duct development, including valve formation.

8 canonical Wnt pathway at different stages of valve formation.

9 for the specification of the AV boundary and valve formation.

10 sed OFT mesenchyme with failure of semilunar valve formation.

11 icular cushion (AVC) is an important step in valve formation.

12 ndocardium during a critical window of heart valve formation.

13 eoclast RANKL function are not shared during valve formation.

14 ocardial expression of RANKL and Ctsk during valve formation.

15 ng a previously unappreciated step during AV valve formation.

16 of the developing heart, a critical step in valve formation.

17 c field to initiate and perpetuate embryonic valve formation.

18 l gene mutations are known to affect cardiac valve formation.

19 yocardium underlying the site of prospective valve formation.

20 ird chamber, diminished looping and impaired valve formation.

21 ment of the AVC and subsequent septation and valve formation.

22 ich are deficient in the initiation of heart valve formation.

23 xpression in lymphocytes and control cardiac valve formation.

24 her multiple TGFbeta isoforms participate in valve formation.

25 uction processes required for normal cardiac valve formation.

26 uce underlying endocardial cells to initiate valve formation.

27 to defective lymphatic vessel patterning and valve formation.

28 ort cardiac jelly deposition at the onset of valve formation.

29 embryos exhibited abnormal atrioventricular valve formation, a phenotype never seen in ECE-1 single

30 ad increased crypt presence, abnormal mitral valve formation and alterations in the compaction proces

31 These opposing defects are evident before valve formation and are the result of distinct gene misr

32 advances in the molecular genetics of aortic valve formation and calcification have recently been ide

33 mouse embryos led to defective lymphovenous valve formation and consequent subcutaneous edema.

34 utant enzymes were unable to restore cardiac valve formation and could only partially rescue cardiac

35 . further define the mechanisms of lymphatic valve formation and implicate oscillatory shear stress i

36 h signaling is required for proper lymphatic valve formation and regulates integrin alpha9 and fibron

37 llular processes and molecules that regulate valve formation and remodeling is required to develop ef

38 ition also suppressed the onset of lymphatic valve formation and subsequent valve maturation.

39 flow plays in fibrous ECM expression during valve formation and to test the underlying cellular mech

40 irth defect, yet few molecular regulators of valve formation are known.

41 Abnormalities of valve formation are the most common serious human congen

42 ults suggest that FOG-2 functions in cardiac valve formation as an attenuator of EMT by repressing GA

43 cardial Klf2 deficiency results in defective valve formation associated with loss of Wnt9b expression

44 endocardial cells and is required for normal valve formation, but its role in the fate of valve endoc

45 ing pathway is crucial for primitive cardiac valve formation by epithelial-mesenchymal transition, an

46 Using a zebrafish model of defective heart valve formation caused by morpholino oligonucleotide kno

47 During cardiac valve formation, Dll4-Notch1 signaling leads to epitheli

48 ion of several genes known to be involved in valve formation (Foxc2, Connexin37, EphrinB2, and Neurop

49 VEGF) signaling is one pathway implicated in valve formation, however its specific spatial and tempor

50 nt truncus arteriosus and impaired semilunar valve formation in humans.

51 alone or in combination result in defective valve formation in lymphatic collecting vessels, lymphed

52 omplex morphogenetic process of intraluminal valve formation in lymphatic vessels.

53 cesses and signaling pathways in controlling valve formation in veins and lymphatic vessels.

54 Cardiac valve formation is a complex process that involves cell

55 Heart valve formation is initiated by an epithelial-mesenchyma

56 How lymphatic valve formation is regulated remains largely unknown.

57 Yet, whether FGF signaling regulates OFT valve formation is unknown.

58 f collecting lymphatic vessels and lymphatic valve formation, uncovering an important role for fluid

59 terations in cardiomyocyte maturation, heart valve formation, vascular development, skeletal muscle d

60 Sema3A is selectively required for lymphatic valve formation, via interaction with Nrp1 and PlexinA1.

61 At the early stages of zebrafish heart valve formation, we show that endocardial cells are conv

62 ema3a(-/-) mice exhibit defects in lymphatic valve formation, which are not due to abnormal lymphatic

63 entrains sequential chamber contraction; and valve formation, which prevents retrograde fluid movemen

64 ial interactions coordinate the processes of valve formation with establishment of conduction delay.