ライフサイエンスコーパス: valve stenosis

1 ortant mitral valve regurgitation and mitral valve stenosis.

2 ng cardiovascular disease or calcific aortic valve stenosis.

3 gh-risk surgical patients with severe aortic valve stenosis.

4 r cardiovascular disease and calcific aortic valve stenosis.

5 functional characteristics of severe aortic valve stenosis.

6 nal properties of severe degenerative aortic valve stenosis.

7 iovascular disease (CVD) and calcific aortic valve stenosis.

8 e and coarctation of the aorta and pulmonary valve stenosis.

9 ostheses and those with predominant surgical valve stenosis.

10 atment of severe symptomatic calcific aortic valve stenosis.

11 approach to treat neonatal congenital aortic valve stenosis.

12 d regression of SMR following AVR for aortic valve stenosis.

13 ickening to severe calcification with aortic valve stenosis.

14 gnaling, and halts the progression of aortic valve stenosis.

15 therapeutic targets for prevention of aortic valve stenosis.

16 is an effective form of treatment for mitral valve stenosis.

17 Reasons for second MVR were prosthetic valve stenosis 24 (83%), thrombosis 4 (14%), and endocar

18 normalities, ocular hypertelorism, pulmonary valve stenosis, abnormal genitalia, retardation of growt

19 phic findings in patients with severe aortic valve stenosis after transcatheter aortic valve replacem

20 Thirty-five patients with aortic-valve stenosis and 10 healthy controls underwent a 27-mi

21 iming to prevent PPM in patients with aortic valve stenosis and concomitant SMR.

22 fectively reduce the risk of calcific aortic valve stenosis and CVD.

23 rominent cardiac defects in NS are pulmonary valve stenosis and hypertrophic cardiomyopathy.

24 Aortic valve stenosis and insufficiency develop over time and c

25 s does not always correlate with severity of valve stenosis and left ventricular (LV) function.

26 Patients >/= 70 years old with severe aortic valve stenosis and no significant coronary artery diseas

27 t highlight Lp(a) in CVD and calcific aortic valve stenosis and propose pathways to clinical registra

28 ms, thromboses and calcification; to truncal valve stenosis and regurgitation; to semilunar and atrio

29 is causally associated with calcific aortic valve stenosis and the need for aortic valve replacement

30 rdiomyopathy (HOCM), 10 patients with aortic valve stenosis, and 14 healthy individuals using [(11)C]

31 diagnostic tool in the assessment of aortic valve stenosis, and how the results compare with current

32 nical risk factors for development of aortic valve stenosis, and hypercholesterolemia is a putative t

33 haemodynamic cardiac consequences of aortic valve stenosis (AS) and aortic valve regurgitation (AR).

34 Aortic valve stenosis (AS) can cause angina despite unobstructe

35 lmonary hypertension (PHT) and severe aortic valve stenosis (AS) from 1987 to 1999.

36 Aortic valve stenosis (AS) induces compensatory alterations in

37 Calcific aortic valve stenosis (AS) is a life-threatening disease with n

38 presence of syncope in patients with aortic valve stenosis (AS) predicts a grave prognosis.

39 In aortic valve stenosis (AS), the occurrence of heart failure sym

40 ity impacted outcome in patients with aortic valve stenosis (AS).

41 ent can modify the natural history of aortic valve stenosis (AS).

42 Accurate quantification of aortic valve stenosis (AVS) is needed for relevant management d

43 Congenital aortic valve stenosis (AVS), coarctation of the aorta (COA) and

44 n was associated with the presence of aortic valve stenosis (AVS), no prospective study has suggested

45 vely associate with increased risk of aortic valve stenosis (AVS).

46 erity and hemodynamic consequences of aortic valve stenosis (AVS).

47 sed by FFR in 54 patients with severe aortic valve stenosis before and after transcatheter aortic val

48 r cardiovascular disease and calcific aortic valve stenosis, but no approved specific therapy exists

49 Calcific aortic valve stenosis (CAVS) is a major health problem facing a

50 iovascular disease (CVD) and calcific aortic valve stenosis (CAVS) is substantial.

51 trophy was also seen in patients with aortic valve stenosis: ERK(Thr188) phosphorylation was increase

52 with respect to echocardiographic indexes of valve stenosis, functional status, and quality of life.

53 is (AS) according to the new proposed aortic valve stenosis grading classification.

54 d risk factors described for critical aortic valve stenosis have been shown to be inapplicable to pat

55 a better prognosis when experiencing aortic valve stenosis, hypertrophic cardiomyopathy, or heart fa

56 alve sclerosis was present in 26% and aortic valve stenosis in 2% of the entire study cohort; in subj

57 f cardiovascular disease and calcific aortic valve stenosis in patients with elevated Lp(a) concentra

58 Although aortic-valve stenosis is clearly associated with adverse cardio

59 ng is the dominant mechanism by which mitral valve stenosis is relieved by this technique, and thus c

60 tension in patients with asymptomatic aortic valve stenosis is scarce.

61 Aortic valve stenosis may influence fractional flow reserve (FF

62 In contrast to patients with aortic valve stenosis, MEE was not improved in patients with HO

63 TAVR were moderate-severe prosthetic aortic valve stenosis (n=10, 21.7%), moderate-severe central pr

64 Patients with severe aortic valve stenosis (n=161) undergoing aortic valve replaceme

65 efects, atrial septal defects, and pulmonary valve stenosis) occurred in 2.0 per 1000 births, of whic

66 valve is frequently an antecedent to aortic valve stenosis or insufficiency and is often associated

67 pathophysiological conditions such as aortic valve stenosis or insufficiency, making it possible to p

68 tation was associated with lesser degrees of valve stenosis or regurgitation.

69 asurements were performed in HOCM and aortic valve stenosis patients 4 months after surgery.

70 valves (BAVs) are associated with premature valve stenosis, regurgitation, and ascending aortic aneu

71 values was found before and after the aortic valve stenosis removal (0.89+/-0.10 versus 0.89+/-0.13;

72 ronary hemodynamics are influenced by aortic valve stenosis removal.

73 diseases such as atherosclerosis and aortic valve stenosis, since it strongly suggests a genetic bas

74 ndomize all-comers with severe native aortic valve stenosis to either transcatheter aortic valve repl

75 ransplant recipients (KT) with severe aortic valve stenosis underwent transfemoral TAVI.

76 Tricuspid valve stenosis was not documented in any of the patients

77 e right sinus of Valsalva, congenital aortic valve stenosis (with bicuspid valve) and myocarditis.

78 Aortic valve stenosis (with or without aortic regurgitation and

79 the hypothesis that calcification and aortic valve stenosis would develop in genetically hypercholest