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1 he mainstay of surgical treatment for mitral valvular regurgitation.
2 aortic (AR), mitral (MR) and tricuspid (TR) valvular regurgitation.
3 ften failed to detect or accurately quantify valvular regurgitation.
4 mple and reliable measure of the severity of valvular regurgitation.
5 ight ventricular function, hemodynamics, and valvular regurgitation.
6 ociated with a low prevalence of significant valvular regurgitation.
7 been reported to be associated with cardiac valvular regurgitation.
8 n patients undergoing surgical correction of valvular regurgitation.
9 useful marker for evaluating the severity of valvular regurgitation.
10 on of hemodynamically significant shunts and valvular regurgitation.
11 with a powerful quantitative tool to assess valvular regurgitation.
12 been proposed as a new approach to quantify valvular regurgitation.
13 o 21 years) and tabulated visible shunts and valvular regurgitation.
14 luramine's and its isomer's association with valvular regurgitation.
15 his study was to determine the prevalence of valvular regurgitation and abnormal valve morphology in
16 of regurgitant flow, reflects the degree of valvular regurgitation and is measurable by color Dopple
18 assessed blindly to determine the degree of valvular regurgitation and valve leaflet thickness and m
20 Left ventricular dysfunction, significant valvular regurgitation, and inducible ischemia were iden
21 e side-by-side method of assessing change in valvular regurgitation appears to be the more reliable m
22 itral and tricuspid valve disease, primarily valvular regurgitation assessment, with an emphasis on t
23 assist devices, such as right heart failure, valvular regurgitation, cardiac arrhythmias, ventricular
25 n methods that have been used for evaluating valvular regurgitation, especially where they related to
26 limited information on long-term changes in valvular regurgitation following discontinuation of thes
28 al echocardiography, fenfluramine-associated valvular regurgitation improved or remained stable in mo
30 lowed by vegetations (in 43 and 34 percent), valvular regurgitation (in 25 and 28 percent), and steno
31 atherosclerosis in patients with nonischemic valvular regurgitation, in contrast to the marked decrea
32 al contribution to left ventricular filling, valvular regurgitation, increased ventricular rate or ir
35 e, 64 +/- 13 years) with isolated left-sided valvular regurgitation operated on from 1980 to 1991 was
36 disease in 3.8% (newly identified in 2.2%), valvular regurgitation or stenosis in 28.0% (newly ident
37 continued, development or progression of any valvular regurgitation over the following year is unlike
38 nd weight (P<0.001); a later onset of aortic valvular regurgitation (P=0.0039); increased preservatio
39 rature on prosthetic valve function and para-valvular regurgitation (PVR) after trans-catheter aortic
40 potential for stabilization or regression of valvular regurgitation should be taken into account when
44 ce among the groups, but when all degrees of valvular regurgitation were considered and when the two
46 r fractional shortening, and onset of aortic valvular regurgitation were serially assessed by echocar
47 died 601 patients with isolated, nonischemic valvular regurgitation who were operated on between 1980
48 e examined this possibility in patients with valvular regurgitation who, often in the absence of angi
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