ライフサイエンスコーパス: vanadate

1 tructure of an acyl transferase inhibited by vanadate.

2 y dissociation of the trapped nucleotide and vanadate.

3 in the absence of molybdate, with or without vanadate.

4 d in the presence or absence of molybdate or vanadate.

5 iazotrophically with or without molybdate or vanadate.

6 dition of BtuF and inhibited by sodium ortho-vanadate.

7 phosphate but was blocked by ADP-AlF3 or ADP-vanadate.

8 n, was reversed by the phosphatase inhibitor vanadate.

9 32-kDa fragment and abolished sensitivity to vanadate.

10 K(+) and a 4-5-fold decreased sensitivity to vanadate.

11 ) blocked the release of NO brought about by vanadate.

12 e I derived peptides as the apical ligand to vanadate.

13 sphorylated intermediate and is inhibited by vanadate.

14 ains is necessary to allow inhibition by ADP-vanadate.

15 he growth medium, and strongly stimulated by vanadate.

16 with the trapping of [14C]ADP in presence of vanadate.

17 ne H(+) gradient but completely abolished by vanadate.

18 ATP-hydrolysis transition state analogue ADP.vanadate.

19 e to the phosphoryl transition state analog, vanadate.

20 ated with the tyrosine phosphatase inhibitor vanadate.

21 bited by low concentrations of molybdate and vanadate.

22 by biotin and was only moderately reduced by vanadate.

23 binding cassette (ABC) transporter inhibitor vanadate.

24 nd in the absence of ATP and with or without vanadate.

25 is highly resistant to both thapsigargin and vanadate.

26 the Ca(2+)-pump inhibitors thapsigargin and vanadate.

27 state that is unable to bind thapsigargin or vanadate.

28 absence and in the presence of tungstate and vanadate.

29 ar levels by 1:1 complexes of catechols with vanadate.

30 1.52 A structure of the D10A mutant bound to vanadate.

31 igargin (0.8 micrometer) but is inhibited by vanadate (1 mM) or cyclopiazonic acid (1 microM).

32 Activity is abolished by vanadate (10 mM), but is less sensitive to phosphate (IC

33 by the tyrosine phosphatase inhibitor sodium vanadate (100 microm).

34 allize a quaternary complex containing Tdp1, vanadate, a DNA oligonucleotide, and a tyrosine-containi

35 ntibody but only minimally affected by 5 muM vanadate, a dynein inhibitor, or by anti-dynein antibody

36 vity, or the ATP-dependent transporters with vanadate, a general ATPase inhibitor.

37 ves enter the plant cell; and (3) blocked by vanadate, a known inhibitor/blocker of ATP-dependent tra

38 e level of pTyr-modified alphaII spectrin by vanadate, a phosphatase inhibitor, implies a dynamic bal

39 ific inhibitors of p38 MAP kinase and sodium vanadate, a potent protein-tyrosine phosphatase inhibito

40 corrosion mechanisms and apply it to bismuth vanadate, a state-of-the-art photoanode.

41 s H(2)O(2) or with the phosphatase inhibitor vanadate abrogates the inhibition of ERK1/2 phosphorylat

42 Treatment of cultured CGPs with vanadate activated ERKs and JNKs but not p38 MAPK and al

43 Vanadate activates the phosphoinositide3-kinase (PI3-K)/

44 Trapping of ADP (or 8-azido-ADP) and vanadate (ADP.Vi or 8-azido-ADP.Vi) at the catalytic sit

45 l transition state specifically, as shown by vanadate-ADP and beryllium fluoride-ADP trapping experim

46 Vanadate adsorbed to goethite, ferrihydrite, gibbsite, a

47 An extremely reduced vanadate affinity of this mutant indicates that the slow

48 Vanadate, an environmental toxicant, causes developmenta

49 metre-resolution structures of MsbA with ADP-vanadate and ADP reveal an unprecedented closed and an i

50 the absence of ligands and in complexes with vanadate and AMP.

51 omplete loss of sensitivity to inhibition by vanadate and an approximately 100-fold increase in the r

52 The complex of vanadate and benzohydroxamic acid is a competitive inhib

53 to MgATP and constants for the inhibition by vanadate and erythrosin B remain unchanged.

54 emoving Mg(2+) in the presence or absence of vanadate and F(-) to reduce general phosphatase activity

55 and reversibly inhibited by 1:1 complexes of vanadate and hydroxamic acids.

56 ed by low concentrations of 1:1 complexes of vanadate and hydroxamic acids.

57 lved in inhibition of the mutase activity by vanadate and identifies a water molecule, observed in th

58 Phosphorylation was inhibited by vanadate and sensitive to KOH and hydroxylamine, consist

59 the membrane transporter in the presence of vanadate and simultaneously loses its high affinity for

60 inhibition of alkaline phosphatase by sodium vanadate and sodium arsenate has been examined, and reve

61 acids form inhibitory ternary complexes with vanadate and the enzyme, although, in certain cases of t

62 nd to the phosphate transition state analogs vanadate and tungstate.

63 tence of occluded species in the presence of vanadate and/or ATP.

64 logy, and the effect of an ATPase inhibitor (vanadate) and a sulfyhdryl reagent (N-ethylmaleimide) wa

65 ontaining the yeast or human mutant protein, vanadate, and ATP.

66 lex between the catalytic cysteine of PTP1B, vanadate, and the peptide DADEYL, a fragment of a physio

67 tion characteristics of the first trinuclear vanadate are presented.

68 nd structure and charge transport of bismuth vanadate are systematically elucidated by ab initio calc

69 of the location and number of protons on the vanadate as a function of pH.

70 Taking advantage of the dual function of vanadate as a transition state analogue and as a photoac

71 idation, our study establishes ternary metal vanadates as a prolific class of photoanode materials fo

72 The vanadate assumes a trigonal bipyramidal geometry in both

73 a crystal structure of the VHZ complex with vanadate at 1.1 A resolution, with a detailed structural

74 al changes associated with calcium, ATP, and vanadate binding to purified PMCA.

75 Bismuth vanadate (BiVO(4)) has recently emerged as a promising m

76 Bismuth vanadate (BiVO4) has a band structure that is well-suite

77 Bismuth vanadate (BiVO4) is a promising photoelectrode material

78 in-tyrosine phosphatase inhibition by sodium vanadate both EGFR expressing Chinese hamster ovary (CHO

79 uptake is inhibited by cholorgenic acid and vanadate, both specific G6PT inhibitors.

80 With both ADP and vanadate bound, the stem and stalk emerge from the head

81 ATP could bind to the different vanadate-bound states of the enzyme either in the presen

82 [(C(4)H(9))(4)N](3)(V(3)O(9)) with the known vanadates [(C(4)H(9))(4)N](3)(HV(4)O(12)), [(C(4)H(9))(4

83 c leaflet of the bilayer and is sensitive to vanadate, Ca(2+), and modification by sulfhydryl reagent

84 Vanadate can often bind to phosphoryl transfer enzymes t

85 a photoactive chemical, we demonstrate that vanadate catalyzes the UV-dependent cleavage of the poly

86 modeling showed that a pentacoordinated 1:1 vanadate-catechol complex readily fits into the class C

87 mes) were competitively inhibited by the 1:1 vanadate-catechol complexes.

88 We conclude that vanadate causes NO release via a mechanism that involves

89 The vanadate cofactor changes its protonation from quadruply

90 The vanadate cofactor in the active site of VHPOs contains "

91 ries of active site models indicate that the vanadate cofactor is most likely anionic with one axial

92 formations were observed for some nucleotide/vanadate combinations.

93 n for specific taxa in columns that received vanadate compared to those that did not.

94 is spectra suggest that the structure of the vanadate complex bound to the enzyme may be very similar

95 his is significantly larger than that of the vanadate complex in free solution (3.62 ppm), suggesting

96 To test whether the enzyme-vanadate complex is a true transition state analogue bey

97 r modeling indicates that a pentacoordinated vanadate complex may well be able to snugly occupy the e

98 The vanadate complex of the hexose phosphate phosphatase BT4

99 ding a previously described hairpin ribozyme-vanadate complex, indicated the scissile bond can adopt

100 to nicked double-stranded DNA captured as a vanadate complex.

101 The 1:1 vanadate complexes are considerably better inhibitors of

102 The structures and properties of organic vanadate complexes are reviewed, and the potency of such

103 ken into account, "real" K(i) values for the vanadate complexes could be determined.

104 change of the nonbridging V--O bonds in the vanadate complexes does not correlate with the kinetic p

105 Detailed analysis of these vanadate compounds reveals the key role of VO4 structura

106 the formation of quaternary complexes around vanadate could be adapted to become a useful method for

107 The enzyme-vanadate dissociation constants in these enzymes are muc

108 ould be blocked either with H(2)O(2) or with vanadate, each of which inhibits PTPs.

109 re we show that annealing nanoporous bismuth vanadate electrodes at 350 degrees C under nitrogen flow

110 In contrast, vanadate-elicited Fas aggregation and Fas-FADD associati

111 Inhibition of MKP-1 by vanadate enhanced JNK phosphorylation and attenuated the

112 rther investigate the mechanism of action of vanadate, eNOS was immunoprecipitated and its associatio

113 Organic vanadate ester compounds are generally four coordinate o

114 damental chemistry and properties of organic vanadate esters impact their effects as inhibitors for p

115 vanadium compounds, specifically the organic vanadate esters, are reviewed with regard to their chemi

116 e unphosphorylated native enzyme, Ca(2+) and vanadate exert a strong influence over the global struct

117 ytic activity and affinity for tungstate and vanadate for a series of 20 AP variants.

118 The vanadate forms covalent linkages between the catalytic t

119 Furthermore, vanadate generated reactive oxygen species (ROS) in CGPs

120 The concentration of vanadate giving half-maximum V-nitrogenase activity when

121 f the 2.2 angstrom resolution structure of a vanadate-hairpin ribozyme complex with structures of pre

122 n-Type bismuth vanadate has been identified as one of the most promisin

123 Vanadate has previously been used as a transition state

124 (dPGM), complexed with the potent inhibitor vanadate, has been determined to a resolution of 1.30 A

125 magnesium pyrophosphate, MgAMPPNP, and MgADP.vanadate have been determined.

126 o the precipitation of either various silver vanadates having Ag/V ratios from 1/2 to 3/1 or the new

127 Resonances for these known vanadates, however, cannot be used to identify the peaks

128 sults are in accord with a model whereby the vanadate-hydroxamate-enzyme complex is a moderately good

129 ecause of the low formation constants of the vanadate-hydroxamic acid complex.

130 Vanadate in the Tdp1-vanadate structure has a trigonal b

131 transition state, the binding properties of vanadate in the Yersinia protein-tyrosine phosphatase (P

132 site is known to stabilize another oxyanion, vanadate, in trigonal bipyramidal geometry, but the exte

133 or the tyrosine phosphatase inhibitor sodium vanadate increased the level of phosphorylation of Tyr(3

134 bovine lung microvascular cells (BLMVEC) to vanadate increased the release of biologically active NO

135 ally, PLB decreased Ca-ATPase sensitivity to vanadate, increasing the IC50 value by 300 nM.

136 Here, we demonstrated that vanadate induced apoptosis in cultured cerebellar granul

137 triatal PTP by an intrastriatal injection of vanadate induced contralateral rotation in control rats

138 In addition, vanadate induced FasL production, Fas (CD95) aggregation

139 Furthermore, sodium vanadate induced primitive endoderm differentiation, eve

140 the molecular mechanisms of sodium vanadate (vanadate)-induced nitric oxide (NO) production.

141 Both hydrolysis of ATP and vanadate-induced [alpha-(32)P]-8-azido-ADP trapping (fol

142 Fas-FADD-caspase-8 pathway that accounts for vanadate-induced apoptosis of CGPs.

143 nd caspase-8 inhibitor completely eliminated vanadate-induced apoptosis.

144 may be an important signaling pathway in the vanadate-induced carcinogenesis, and ROS may play an imp

145 uced FasL expression and partially mitigated vanadate-induced cell death.

146 active oxygen species (ROS) were involved in vanadate-induced expression of HIF-1 and VEGF in DU145 c

147 drogen peroxide scavenger, catalase, blocked vanadate-induced FasL expression and partially mitigated

148 Vanadate-induced FasL expression was ROS-dependent but J

149 so studied the signaling pathway involved in vanadate-induced HIF-1 alpha and VEGF expression and fou

150 nduced NO production, they failed to inhibit vanadate-induced NO release.

151 tmannin, a PI3-K inhibitor, blocked both the vanadate-induced phosphorylation of eNOS and the increas

152 ) to stimulate ATP hydrolysis and facilitate vanadate-induced trapping of [alpha-(32)P]-8-azido-ADP i

153 , and Y401A/Y1044A mutants, TNP-ATP binding, vanadate-induced trapping of nucleotide, and ATP hydroly

154 In this study, we demonstrate that vanadate induces HIF-1 activity through the expression o

155 Sodium azide and vanadate inhibited sterol uptake, consistent with the pa

156 zation conditions fixed Na+,K+-ATPase in the vanadate-inhibited E2 conformation, and the crystals had

157 h low affinity and the unaffected IC(50) for vanadate inhibition observed in the C(27,30)A-substitute

158 enzyme kinetic, photoaffinity labeling, and vanadate inhibition studies.

159 There was no effect of vanadate (inhibitor of dynein) on motility, whereas 5'-a

160 olysis, and the extent to which sodium ortho-vanadate inhibits ATP hydrolysis all vary significantly.

161 t is concluded that H(+) gradient-dependent, vanadate-insensitive glucoside uptake is mediated by an

162 Vanadate is a phosphate mimic and causes protein misfold

163 Vanadate is a potent modulator of a number of biological

164 Therefore, it is concluded that vanadate is not a true transition state analogue for the

165 Vanadate is widely used in industry, and is a potent ind

166 Equilibration between the various vanadates is observed and characterized.

167 ion K(m) = 4.81 microm), and the presence of vanadate leads to enzyme inactivation (IC(50) = 24 micro

168 roup in the transition state highlights that vanadate may not always be an ideal transition state ana

169 coli dPGM structure which, once activated by vanadate, may dephosphorylate the active protein.

170 Thus, JNK signaling plays a major role in vanadate-mediated activation of the Fas-FADD-caspase-8 p

171 S) in CGPs; however, ROS was not involved in vanadate-mediated MAPK activation.

172 ng this phenomenon, we used the procedure of vanadate-mediated trapping of the Pgp transport cycle in

173 nesium, adenosine diphosphate, and inorganic vanadate (Mg.ADP.Vi) and the rough-chemotype lipopolysac

174 an affinity for SERCA2a approaching that of vanadate (micromolar), whereas PLB3 and PLB4 had much hi

175 Therefore, enzyme-bound vanadate moieties are often considered as transition sta

176 divanadate, but with evidence of additional vanadate moieties at either end, and representing a diff

177 rthermore, the ionization state of the bound vanadate moiety is not invariant across the PTPase varia

178 perchlorate, chlorate, monofluorophosphate, vanadate, molydate, and tungstate.

179 fructose-responsive gene, with tungstate or vanadate nonspecifically inhibited NaPi-2b mRNA expressi

180 ylsiloxy ligands, the transesterification of vanadate occurs via sigma-bond metathesis, and vanadium

181 Eight crystal structures of Tdp1 with vanadate, oligonucleotides, and peptides or peptide anal

182 The effects of protamine sulfate and vanadate on Nephrin phosphorylation and GSIR were studie

183 be modulated by changing CFTR activity with vanadate or adenylyl-imidodiphosphate, or by introducing

184 tant that traps nucleotide in the absence of vanadate or beryllium fluoride, the high to low affinity

185 itors of phosphotyrosine phosphatases (PTPs; vanadate or calpeptin) increases phosphorylation of p80

186 hich does not occur upon exposure to H2O2 or vanadate or if pervanadate is excluded during cell solub

187 In all cases, pretreatment with vanadate or N-ethylmaleimide inhibited the conversion of

188 yoprotectant solution became liganded to the vanadate or tungstate inhibitor molecules in a bidentate

189 hosphotyrosine phosphatase inhibitor, sodium vanadate, or expression of mutationally activated c-Src

190 s and geochemical simulations, no other lead vanadate, or vanadium minerals were detected.

191 n integrated workflow, we find eight ternary vanadate oxide photoanodes in the target band-gap range

192 The equatorial vanadate oxygen atoms bind to the P-loop, and the apical

193 ive antidiabetic compound such a the organic vanadate peptidic substrate was possible the toxicity pr

194 d it with two classical trigonal-bipyramidal vanadate-phosphatase complexes.

195 in the context of recent characterization of vanadate-phosphatase protein structures by data mining.

196 influx in Col-0 guard cells was impaired by vanadate pre-treatment or PM H(+) -ATPase mutation, sugg

197 ion remained unaffected by vanadate, whereas vanadate promoted recruitment of the 90-kDa heat shock p

198 Vanadium-dependent haloperoxidases contain a vanadate prosthetic group and utilize hydrogen peroxide

199 In contrast, in the mutant, the vanadate protonation is the same at pH 5.0 and 8.3, and

200 rrelated for tungstate (r(2) = 0.89) but not vanadate (r(2) = 0.23), indicating that the tungstate*AP

201 Vanadate reacts with catechols at submillimolar concentr

202 The phosphatase inhibitor vanadate reduced these effects.

203 rties (e.g. growth inhibition, flocculation, vanadate resistance, stress response) that are potential

204 Consistent with the vanadate-resistant ATPase activity, salt-stable closed c

205 ased and decreased by the addition of sodium vanadate, respectively, but these changes were eliminate

206 Treatment of BLMVEC with vanadate resulted in phosphorylation of both Akt and end

207 the W354F mutant obtained in the presence of vanadate reveals an unusual divanadate species with a cy

208 Inhibition of phosphatases with vanadate reveals site-specific regulatory mechanisms and

209 However, the phosphatase inhibitor vanadate reversed the inhibitory effect of RA on MAP kin

210 the limited information available on uranyl vanadates, room temperature Ca-U-V precipitation experim

211 reversible depending on the availability of vanadate salts and triclosan, a component of PIA.

212 f the tyrosine phosphatase inhibitor, sodium vanadate, selectively repressed Nanog transcription with

213 with varied classes of oxo compounds (i.e., vanadates, selenate, phosphate, sulfate, acetate, nitrat

214 as characterized as magnesium ion dependent, vanadate sensitive, and slightly stimulated by potassium

215 ueous two-phase partitioning and assayed for vanadate-sensitive ATPase and FCBP activity.

216 re very poor substrates for FAAH; however, a vanadate-sensitive enzymatic activity was detected in br

217 e is mediated by an H(+) antiporter, whereas vanadate-sensitive glutathione conjugate uptake is media

218 ng membrane vesicles in an ATP-dependent and vanadate-sensitive manner; net transfer of cholesterol w

219 s stereoselective; minimal ATP-dependent and vanadate-sensitive transfer of cholesteryl oleate, phosp

220 inding of ATP, does not progress to a normal vanadate-sensitive transition state, but hydrolyzes ATP

221 renamed here G6Pase-beta, is an acid-labile, vanadate-sensitive, endoplasmic reticulum-associated pho

222 None of these parameters was vanadate-sensitive.

223 lycerol but not serine, stereochemistry, and vanadate sensitivity) are similar to, but distinct from,

224 ruginosa cultures growing in the presence of vanadate showed differential expression of genes involve

225 The enzyme activity was inhibited by sodium vanadate, sodium fluoride, N-ethylmaleimide, and phenylg

226 After all of the vanadate species present in solution had been taken into

227 tavanadate in the active site from a dimeric vanadate species that previous crystallographic evidence

228 ydrolysis product, 8-azido-ADP, but not as a vanadate stabilized post-hydrolysis transition state com

229 We show that vanadate stably traps Pgp under two different conditions

230 imaged by cryo-electron microscopy or after vanadate staining by scanning transmission electron micr

231 Vanadate in the Tdp1-vanadate structure has a trigonal bipyramidal geometry t

232 olorimetric method based on formation of the vanadate-tartrate complex.

233 e (cyclosporin A) for or indirectly occlude (vanadate) the substrate-binding site prevent formation o

234 the expression of HIF-1 and VEGF induced by vanadate through PI3K/Akt may be an important signaling

235 Here, the ability of vanadate to accept a variety of different ligands is exp

236 Upon binding of vanadate to both enzymes, small increases in average met

237 ed in the presence of ATP, ADP, and ATP plus vanadate to identify the residues that are directly affe

238 periments, rigor cross-bridges and Ca2+ with vanadate to inhibit cross-bridge formation produce a sim

239 monas flagella in vivo, rendered immotile by vanadate, to be EI = 840 +/- 280 pNmum(2) and ks = 79.6

240 Addition of ouabain, but not vanadate, to the purified Na+/K+-ATPase/Src complex free

241 We have also determined the structure of a vanadate transition state mimic for the Cre-loxP reactio

242 hairpin ribozyme, including a complex with a vanadate transition state mimic, suggests an alternative

243 anic phosphate ground-state analogs and with vanadate transition-state analogs.

244 imicked the pro-R, nonbridging oxygen of the vanadate transition-state complex.

245 however, unlike the V-nitrogenase genes the vanadate transport genes were expressed in vegetative ce

246 uggesting that there was also a low-affinity vanadate transport system that functioned in the vupB mu

247 The genes for an ABC-type vanadate transport system, vupABC, were found in A. vari

248 High-affinity vanadate transport systems have not heretofore been iden

249 induce regeneration of [125I]IAAP binding to vanadate-trapped (or fluoroaluminate-trapped) Pgp withou

250 (beta,gamma-imino)triphosphate-bound and ADP/vanadate-trapped intermediates were conformationally dis

251 tent with this, limited trypsin digestion of vanadate-trapped Pgp shows protection by cis-(Z)-flupent

252 n of [alpha-(32)P]-8-azido-ADP (or ADP) from vanadate-trapped Pgp, which is essential for subsequent

253 toplasmic gate II remains closed also in the vanadate-trapped state, but it reopens in the ADP-bound

254 In a vanadate-trapped transition state intermediate, all free

255 Vanadate trapping further decreased the D/A ratio, indic

256 hereas substrate dissociation in response to vanadate trapping is considerably affected in its presen

257 Under conditions of vanadate trapping, most molecules displayed high FRET ef

258 well as on substrate dissociation coupled to vanadate trapping.

259 se of an 8-azido[alpha-(32)P]ATP-binding and vanadate-trapping assay allowed us to devise conditions

260 Like bisdioxopiperazines, vanadate traps the enzyme in a salt-stable closed confor

261 We measured the stiffness of 50 muM sodium vanadate treated bull sperm in the presence of 4 mM ADP,

262 of vinculin on tyrosine was reconstituted in vanadate treated COS-7 cells coexpressing c-Src.

263 nce of calcium/calmodulin, cell lysates from vanadate-treated cells exhibited greater NOS activity co

264 phosphorylation of alpha-actinin is seen in vanadate-treated COS-7 cells that are co-transfected wit

265 Vanadate treatment of cells revealed that the reduction

266 Vanadate treatment reversed the butein-induced down-regu

267 Vanadate treatment reversed the GS-induced down-regulati

268 P-2 is translocated to plasma membrane after vanadate treatment.

269 constants (km) for the dissolution of uranyl-vanadate (U-V) minerals predominant at Blue Gap/Tachee w

270 lexes is followed by the slower reduction of vanadate (V (V)) to vanadyl (V (IV)) and oxidation of th

271 process, we simultaneously added acetate and vanadate (V(5+)) to columns that were packed with aquife

272 template exchange between phosphate (P) and vanadate (V) anions enclosed in an {X(2)M(18)} cluster i

273 er oxidation catalyst based on redox-active (vanadate(V)-centered) polyoxometalate ligands, Na10[Co4(

274 estigated the molecular mechanisms of sodium vanadate (vanadate)-induced nitric oxide (NO) production

275 ug substrate-stimulable ATPase activity, and vanadate (Vi) blocks this activity effectively by trappi

276 Sodium vanadate (Vi) is a phosphate analog that inhibits force

277 ial Ca(2+) uptake rate was also increased by vanadate (Vi), acetate, ATP, or a non-hydrolyzable ATP a

278 P]-8-azidoADP was trapped in the presence of vanadate (Vi), and the release of trapped [alpha-(32)P]-

279 In this study we exploit the vanadate (Vi)-induced transition state conformation of P

280 e binding to intermediate levels with sodium vanadate (Vi).

281 rystallized with the phosphate (P(i)) analog vanadate (VO(3-)4 or V(i)).

282 Stoichiometry of [14C]ADP trapping by vanadate was 1 mol/mol P-glycoprotein at full inhibition

283 Inhibition by ADP-vanadate was slow and depended cooperatively on nucleoti

284 rnative V-dependent nitrogenase, transported vanadate well.

285 The effects of sodium vanadate were abrogated by Grb2 deficiency or by the add

286 by using V-nitrogenase unless high levels of vanadate were provided, suggesting that there was also a

287 -glycoprotein upon incubation with MgADP and vanadate were studied along with the trapping of [14C]AD

288 Vanadates were found to convert ethylating agents into e

289 red for HIF-1 and VEGF expression induced by vanadate, whereas mitogen-activated protein kinase pathw

290 yeast was inhibited to different extents by vanadate, whereas the latter yeast strain was more resis

291 -caveolin interaction remained unaffected by vanadate, whereas vanadate promoted recruitment of the 9

292 Antimony-alloyed bismuth vanadate, which is identified as a novel light absorber

293 We show that with bound ADP.vanadate, which mimics the transition state between ATP

294 Vanadate, which prevented Nephrin dephosphorylation afte

295 Addition of ADP and vanadate, which traps the posthydrolysis biochemical sta

296 s that were investigated, the 1:1 complex of vanadate with 4-nitrobenzohydroxamic acid, was 0.48 micr

297 er, the results show that the interaction of vanadate with biological systems is not solely reliant u

298 of formation of the 1:1 and 1:2 complexes of vanadate with catechol itself and with 2,3-dihydroxynaph

299 he presence of MgATP, Mg(2+), Mg-P(i), or Mg-vanadate with complete inhibition of activity.

300 with 4-nitrobenzo[(13)C]hydroxamic acid and vanadate yields a coordination-induced shift (CIS) of 7.