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1 oft-tissue infections, and infections due to vancomycin-resistant enterococci.
2 und in a subset of 875 recent US isolates of vancomycin-resistant enterococci.
3 - and vancomycin-resistant Staphylococci and vancomycin-resistant Enterococci.
4 There were no vancomycin-resistant enterococci.
5 tions such as relapses and infections due to vancomycin-resistant enterococci.
6 A and B two-component regulatory switches in vancomycin-resistant enterococci.
7 omologous D-Ala:D-lactate ligase produced by vancomycin-resistant enterococci.
8 aureus, vancomycin-resistant S. aureus, and vancomycin-resistant enterococci.
9 s used for molecular epidemiologic typing of vancomycin-resistant enterococci.
10 3%) environmental cultures were positive for vancomycin-resistant enterococci.
11 mug/mL), including enhanced potency against vancomycin-resistant enterococci.
12 ed off-label to treat patients infected with vancomycin-resistant enterococci.
13 stance to daptomycin during the treatment of vancomycin-resistant enterococci.
14 reus, vancomycin-intermediate S. aureus, and vancomycin-resistant Enterococci.
15 ureus, vancomycin-intermediate S. aureus and vancomycin-resistant enterococci.
16 with linezolid for bloodstream infections by vancomycin-resistant enterococci.
17 cocci, macrolide-resistant streptococci, and vancomycin-resistant enterococci.
18 etection of gastrointestinal colonization by vancomycin-resistant enterococci.
19 ese patients may help decrease the spread of vancomycin-resistant enterococci.
20 i and to assess molecular characteristics of vancomycin-resistant enterococci, 157 clinical blood iso
21 trol policies to prevent the transmission of vancomycin-resistant enterococci (22 of 25 [88 percent]
22 f the long-term care facilities screened for vancomycin-resistant enterococci (26 of 28 in 1998 [93 p
24 phalosporin-resistant enterobacteriaceae and vancomycin-resistant enterococci (acquired antibiotic re
26 ive against Gram-positive bacteria including vancomycin-resistant enterococci and methicillin-resista
27 th of antibiotic resistant superbugs such as vancomycin-resistant Enterococci and Staphylococci has b
28 h hydrophobic side chains are active against vancomycin-resistant enterococci and vancomycin-resistan
29 s aeruginosa, and vancomycin-susceptible and vancomycin-resistant enterococci and with 193 perianal s
30 -lactamase-producing Gram-negative bacteria, vancomycin-resistant enterococci, and Clostridium diffic
31 study of gastrointestinal colonization with vancomycin-resistant enterococci, and compared infection
32 including meticillin-resistant Staph aureus, vancomycin-resistant enterococci, and penicillin-resista
33 us aureus, coagulase-negative staphylococci, vancomycin-resistant enterococci, and resistance to thir
34 ococci; the number of days to acquisition of vancomycin-resistant enterococci; and other measurements
35 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococci), antifungal socks (whi
39 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococci as well as hemolytic ac
40 study of 51 patients who were colonized with vancomycin-resistant enterococci, as evidenced by the pr
42 control practices and screening policies for vancomycin-resistant enterococci at the acute care and l
44 meticillin-resistant Staphylococcus aureus, vancomycin-resistant enterococci, C difficile, and multi
45 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococci clinical isolates and i
46 ities that had had at least one patient with vancomycin-resistant enterococci declined from 15 in 199
47 e of D-serine for peptidoglycan synthesis in vancomycin-resistant enterococci expressing the VanC phe
48 tibacterial assays employing a unique set of vancomycin-resistant Enterococci faecalis and Enterococc
49 ncomycin-sensitive Staphylococcus aureus and vancomycin-resistant Enterococci faecalis as well as the
50 her derivative of vancomycin aglycon against vancomycin-resistant Enterococci faecalis previously rep
52 inst methicillin-resistant staphylococci and vancomycin-resistant enterococci has been purified, and
54 ceptional growth inhibitory activity against vancomycin-resistant Enterococci (IC50 40 nM), >270-fold
56 es only in preventing rectal colonization by vancomycin-resistant enterococci in a medical intensive
61 can reduce or eliminate the transmission of vancomycin-resistant enterococci in the health care faci
62 21 (23.9%) in the glove-only group acquired vancomycin-resistant enterococci in the medical intensiv
63 on was sought in assessing the prevalence of vancomycin-resistant enterococci in the region's facilit
64 oratories conducting active surveillance for vancomycin-resistant enterococci in three San Francisco
65 evention received a report of an outbreak of vancomycin-resistant enterococci in which 31 of 84 (36.9
68 s and Enterococcus gallinarum (intrinsically vancomycin-resistant enterococci [IVRE]) from Enterococc
70 p and 13 (14.8%) in the glove-only group had vancomycin-resistant enterococci on admission to the med
72 b-lacZ fusions report on expression from the vancomycin-resistant enterococci promoters of the type A
73 vanHAX are orthologous to genes found in vancomycin-resistant enterococci that encode enzymes pre
74 The number of patients becoming colonized by vancomycin-resistant enterococci; the number of days to
75 mice that have intestinal colonization with vancomycin-resistant enterococci, these agents promote h
76 ata from an interrupted time-series study of vancomycin-resistant enterococci transmission in a hemat
77 ising improvement of its bioactivity against vancomycin-resistant enterococci (Van A and Van B phenot
79 ent-specific risk factors for acquisition of vancomycin-resistant enterococci (VRE) among hospitalize
82 to selective enrichment broth for detecting vancomycin-resistant enterococci (VRE) and multidrug-res
83 gy laboratory to determine the prevalence of vancomycin-resistant enterococci (VRE) and multidrug-res
85 se of potential postpartum infections due to vancomycin-resistant enterococci (VRE) and the possible
86 ctively analyzed risk factors and outcome of vancomycin-resistant enterococci (VRE) and vancomycin-se
87 erococcus faecalis bacteremia caused by both vancomycin-resistant enterococci (VRE) and vancomycin-su
95 ta in the colon inhibit the establishment of vancomycin-resistant enterococci (VRE) colonization by d
97 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE) due to the scope
99 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE) for extended peri
100 r (BBL, Sparks, Md.) for direct detection of vancomycin-resistant enterococci (VRE) from 894 perianal
101 Xpert vanA assay for routine surveillance of vancomycin-resistant enterococci (VRE) from rectal swabs
102 ug/ml vancomycin (BEAV) for the isolation of vancomycin-resistant enterococci (VRE) from stool specim
104 incidence of colonization and infection with vancomycin-resistant enterococci (VRE) has increased dra
106 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant Enterococci (VRE) have now arisen a
107 rs were compared for their ability to detect vancomycin-resistant enterococci (VRE) in 750 stool spec
108 ve E. faecium, which implies that control of vancomycin-resistant enterococci (VRE) in hospitals also
109 -resistant Staphylococcus aureus (EMRSA) and vancomycin-resistant enterococci (VRE) in hospitals in E
112 he identification of patients colonized with vancomycin-resistant enterococci (VRE) is central to the
114 that a method of performing surveillance for vancomycin-resistant enterococci (VRE) is to screen spec
115 e gastrointestinal tracts are colonized with vancomycin-resistant enterococci (VRE) may serve as a re
116 esistant Staphylococcus aureus (MRSA) and/or vancomycin-resistant enterococci (VRE) on at least 1 occ
119 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE) that are known to
120 Vancomycin resistance is conferred upon vancomycin-resistant enterococci (VRE) through the repla
121 trains to the establishment of endemicity of vancomycin-resistant enterococci (VRE) were determined.
122 the Massachusetts General Hospital from whom vancomycin-resistant enterococci (VRE) were isolated fro
124 typic variation and stability of isolates of vancomycin-resistant enterococci (VRE) were studied to d
125 tant Staphylococcus aureus (MRSA) as well as vancomycin-resistant enterococci (VRE) with minimum inhi
126 tiresistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE), among patients i
127 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant enterococci (VRE), and ceftazidime-
128 , as well as glycopeptides effective against vancomycin-resistant enterococci (VRE), and fluoroquinol
129 vancomycin (BEAV) agar (84.8%) for detecting vancomycin-resistant enterococci (VRE), and the results
131 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE), has reached a cr
132 ibiotic-resistant bacterial species, such as vancomycin-resistant enterococci (VRE), necessitates the
133 anA/vanB DNA hybridization assay to identify vancomycin-resistant enterococci (VRE), was evaluated fo
148 rococcal bloodstream infection (BSI; 22 with vancomycin-resistant enterococci [VRE] and 61 with vanco
149 n-resistant Staphylococcus aureus [MRSA] and vancomycin-resistant enterococci [VRE]) or rapid screeni
150 (95 percent), high-density colonization with vancomycin-resistant enterococci was maintained (mean [+
155 ties, the risk factors for colonization with vancomycin-resistant enterococci were prior hospitalizat
156 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococci, without affecting the
157 mutant subsequently resulted in clearance of vancomycin-resistant enterococci, without plasmid transf
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