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1 oft-tissue infections, and infections due to vancomycin-resistant enterococci.
2 und in a subset of 875 recent US isolates of vancomycin-resistant enterococci.
3 - and vancomycin-resistant Staphylococci and vancomycin-resistant Enterococci.
4                                There were no vancomycin-resistant enterococci.
5 tions such as relapses and infections due to vancomycin-resistant enterococci.
6 A and B two-component regulatory switches in vancomycin-resistant enterococci.
7 omologous D-Ala:D-lactate ligase produced by vancomycin-resistant enterococci.
8  aureus, vancomycin-resistant S. aureus, and vancomycin-resistant enterococci.
9 s used for molecular epidemiologic typing of vancomycin-resistant enterococci.
10 3%) environmental cultures were positive for vancomycin-resistant enterococci.
11  mug/mL), including enhanced potency against vancomycin-resistant enterococci.
12 ed off-label to treat patients infected with vancomycin-resistant enterococci.
13 stance to daptomycin during the treatment of vancomycin-resistant enterococci.
14 reus, vancomycin-intermediate S. aureus, and vancomycin-resistant Enterococci.
15 ureus, vancomycin-intermediate S. aureus and vancomycin-resistant enterococci.
16 with linezolid for bloodstream infections by vancomycin-resistant enterococci.
17 cocci, macrolide-resistant streptococci, and vancomycin-resistant enterococci.
18 etection of gastrointestinal colonization by vancomycin-resistant enterococci.
19 ese patients may help decrease the spread of vancomycin-resistant enterococci.
20 i and to assess molecular characteristics of vancomycin-resistant enterococci, 157 clinical blood iso
21 trol policies to prevent the transmission of vancomycin-resistant enterococci (22 of 25 [88 percent]
22 f the long-term care facilities screened for vancomycin-resistant enterococci (26 of 28 in 1998 [93 p
23                           The VanA ligase of vancomycin-resistant enterococci, a D-Ala-D-lactate deps
24 phalosporin-resistant enterobacteriaceae and vancomycin-resistant enterococci (acquired antibiotic re
25                                              Vancomycin-resistant enterococci and Klebsiella pneumoni
26 ive against Gram-positive bacteria including vancomycin-resistant enterococci and methicillin-resista
27 th of antibiotic resistant superbugs such as vancomycin-resistant Enterococci and Staphylococci has b
28 h hydrophobic side chains are active against vancomycin-resistant enterococci and vancomycin-resistan
29 s aeruginosa, and vancomycin-susceptible and vancomycin-resistant enterococci and with 193 perianal s
30 -lactamase-producing Gram-negative bacteria, vancomycin-resistant enterococci, and Clostridium diffic
31  study of gastrointestinal colonization with vancomycin-resistant enterococci, and compared infection
32 including meticillin-resistant Staph aureus, vancomycin-resistant enterococci, and penicillin-resista
33 us aureus, coagulase-negative staphylococci, vancomycin-resistant enterococci, and resistance to thir
34 ococci; the number of days to acquisition of vancomycin-resistant enterococci; and other measurements
35 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococci), antifungal socks (whi
36                                              Vancomycin-resistant enterococci are a major cause of no
37 d, 900-bed, urban teaching hospital in which vancomycin-resistant enterococci are endemic.
38 l intensive care unit of a hospital in which vancomycin-resistant enterococci are endemic.
39 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococci as well as hemolytic ac
40 study of 51 patients who were colonized with vancomycin-resistant enterococci, as evidenced by the pr
41                    The overall prevalence of vancomycin-resistant enterococci at 30 facilities that p
42 control practices and screening policies for vancomycin-resistant enterococci at the acute care and l
43               A rapid protocol for subtyping vancomycin-resistant enterococci by pulsed-field gel ele
44  meticillin-resistant Staphylococcus aureus, vancomycin-resistant enterococci, C difficile, and multi
45 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococci clinical isolates and i
46 ities that had had at least one patient with vancomycin-resistant enterococci declined from 15 in 199
47 e of D-serine for peptidoglycan synthesis in vancomycin-resistant enterococci expressing the VanC phe
48 tibacterial assays employing a unique set of vancomycin-resistant Enterococci faecalis and Enterococc
49 ncomycin-sensitive Staphylococcus aureus and vancomycin-resistant Enterococci faecalis as well as the
50 her derivative of vancomycin aglycon against vancomycin-resistant Enterococci faecalis previously rep
51                           Two isolates among vancomycin-resistant enterococci from the greater Housto
52 inst methicillin-resistant staphylococci and vancomycin-resistant enterococci has been purified, and
53              Colonization and infection with vancomycin-resistant enterococci have been associated wi
54 ceptional growth inhibitory activity against vancomycin-resistant Enterococci (IC50 40 nM), >270-fold
55 screening and infection-control policies for vancomycin-resistant enterococci in 1998 and 1999.
56 es only in preventing rectal colonization by vancomycin-resistant enterococci in a medical intensive
57                   We examined the density of vancomycin-resistant enterococci in stool during and aft
58                          When the density of vancomycin-resistant enterococci in stool was at least 4
59                            For patients with vancomycin-resistant enterococci in stool, treatment wit
60                                The advent of vancomycin-resistant enterococci in the 1990s and the th
61  can reduce or eliminate the transmission of vancomycin-resistant enterococci in the health care faci
62  21 (23.9%) in the glove-only group acquired vancomycin-resistant enterococci in the medical intensiv
63 on was sought in assessing the prevalence of vancomycin-resistant enterococci in the region's facilit
64 oratories conducting active surveillance for vancomycin-resistant enterococci in three San Francisco
65 evention received a report of an outbreak of vancomycin-resistant enterococci in which 31 of 84 (36.9
66              Risk factors for acquisition of vancomycin-resistant enterococci induced length of stay
67 ains or hospitals in which the prevalence of vancomycin-resistant enterococci is low.
68 s and Enterococcus gallinarum (intrinsically vancomycin-resistant enterococci [IVRE]) from Enterococc
69                               All 465 (100%) vancomycin-resistant enterococci (MIC > or = 32 microgra
70 p and 13 (14.8%) in the glove-only group had vancomycin-resistant enterococci on admission to the med
71                       If the total number of vancomycin-resistant enterococci patients in the ICU wer
72 b-lacZ fusions report on expression from the vancomycin-resistant enterococci promoters of the type A
73     vanHAX are orthologous to genes found in vancomycin-resistant enterococci that encode enzymes pre
74 The number of patients becoming colonized by vancomycin-resistant enterococci; the number of days to
75  mice that have intestinal colonization with vancomycin-resistant enterococci, these agents promote h
76 ata from an interrupted time-series study of vancomycin-resistant enterococci transmission in a hemat
77 ising improvement of its bioactivity against vancomycin-resistant enterococci (Van A and Van B phenot
78                                In pathogenic vancomycin-resistant enterococci, vanX is part of a five
79 ent-specific risk factors for acquisition of vancomycin-resistant enterococci (VRE) among hospitalize
80                 The transmission dynamics of vancomycin-resistant enterococci (VRE) and factors contr
81        We analyzed surveillance cultures for vancomycin-resistant enterococci (VRE) and methicillin-r
82  to selective enrichment broth for detecting vancomycin-resistant enterococci (VRE) and multidrug-res
83 gy laboratory to determine the prevalence of vancomycin-resistant enterococci (VRE) and multidrug-res
84                    Surveillance cultures for vancomycin-resistant enterococci (VRE) and subsequent ch
85 se of potential postpartum infections due to vancomycin-resistant enterococci (VRE) and the possible
86 ctively analyzed risk factors and outcome of vancomycin-resistant enterococci (VRE) and vancomycin-se
87 erococcus faecalis bacteremia caused by both vancomycin-resistant enterococci (VRE) and vancomycin-su
88                                              Vancomycin-resistant enterococci (VRE) are an important
89                                              Vancomycin-resistant enterococci (VRE) are common hospit
90                                              Vancomycin-resistant enterococci (VRE) are increasingly
91                                              Vancomycin-resistant enterococci (VRE) are increasingly
92                                              Vancomycin-resistant enterococci (VRE) are nosocomial pa
93                    Surveillance cultures for vancomycin-resistant enterococci (VRE) are time-consumin
94                        Treatment options for vancomycin-resistant enterococci (VRE) bloodstream infec
95 ta in the colon inhibit the establishment of vancomycin-resistant enterococci (VRE) colonization by d
96  concentrations, did not promote significant vancomycin-resistant enterococci (VRE) colonization.
97 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE) due to the scope
98                                              Vancomycin-resistant enterococci (VRE) escape the bacter
99 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE) for extended peri
100 r (BBL, Sparks, Md.) for direct detection of vancomycin-resistant enterococci (VRE) from 894 perianal
101 Xpert vanA assay for routine surveillance of vancomycin-resistant enterococci (VRE) from rectal swabs
102 ug/ml vancomycin (BEAV) for the isolation of vancomycin-resistant enterococci (VRE) from stool specim
103        The treatment of infections caused by vancomycin-resistant enterococci (VRE) has become an imp
104 incidence of colonization and infection with vancomycin-resistant enterococci (VRE) has increased dra
105                                              Vancomycin-resistant enterococci (VRE) have emerged as n
106 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant Enterococci (VRE) have now arisen a
107 rs were compared for their ability to detect vancomycin-resistant enterococci (VRE) in 750 stool spec
108 ve E. faecium, which implies that control of vancomycin-resistant enterococci (VRE) in hospitals also
109 -resistant Staphylococcus aureus (EMRSA) and vancomycin-resistant enterococci (VRE) in hospitals in E
110                         Active screening for vancomycin-resistant enterococci (VRE) in rectal and sto
111                            As infection with vancomycin-resistant enterococci (VRE) increases in hosp
112 he identification of patients colonized with vancomycin-resistant enterococci (VRE) is central to the
113                     Vancomycin resistance in vancomycin-resistant enterococci (VRE) is due to an alte
114 that a method of performing surveillance for vancomycin-resistant enterococci (VRE) is to screen spec
115 e gastrointestinal tracts are colonized with vancomycin-resistant enterococci (VRE) may serve as a re
116 esistant Staphylococcus aureus (MRSA) and/or vancomycin-resistant enterococci (VRE) on at least 1 occ
117                                              Vancomycin-resistant enterococci (VRE) recently have eme
118 biquitous on plasmids isolated from clinical vancomycin-resistant enterococci (VRE) strains.
119 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE) that are known to
120      Vancomycin resistance is conferred upon vancomycin-resistant enterococci (VRE) through the repla
121 trains to the establishment of endemicity of vancomycin-resistant enterococci (VRE) were determined.
122 the Massachusetts General Hospital from whom vancomycin-resistant enterococci (VRE) were isolated fro
123           Among our patient population, more vancomycin-resistant enterococci (VRE) were recovered wi
124 typic variation and stability of isolates of vancomycin-resistant enterococci (VRE) were studied to d
125 tant Staphylococcus aureus (MRSA) as well as vancomycin-resistant enterococci (VRE) with minimum inhi
126 tiresistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE), among patients i
127 llin-resistant Staphylococcus aureus (MRSA), vancomycin-resistant enterococci (VRE), and ceftazidime-
128 , as well as glycopeptides effective against vancomycin-resistant enterococci (VRE), and fluoroquinol
129 vancomycin (BEAV) agar (84.8%) for detecting vancomycin-resistant enterococci (VRE), and the results
130                With increasing prevalence of vancomycin-resistant enterococci (VRE), appropriate anti
131 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant enterococci (VRE), has reached a cr
132 ibiotic-resistant bacterial species, such as vancomycin-resistant enterococci (VRE), necessitates the
133 anA/vanB DNA hybridization assay to identify vancomycin-resistant enterococci (VRE), was evaluated fo
134 ulty in detecting intermediate and low-level vancomycin-resistant enterococci (VRE).
135 agar plates as negative or "nonnegative" for vancomycin-resistant enterococci (VRE).
136 omycin per ml and evaluated for isolation of vancomycin-resistant enterococci (VRE).
137 epted method for detecting colonization with vancomycin-resistant enterococci (VRE).
138  to develop an antimicrobial peptide against vancomycin-resistant enterococci (VRE).
139 ater than 1000-fold was demonstrated against vancomycin-resistant Enterococci (VRE).
140  in antimicrobial potency (200-fold) against vancomycin-resistant Enterococci (VRE).
141 nsive care unit for rectal colonization with vancomycin-resistant enterococci (VRE).
142 han vancomycin and has activity against many vancomycin-resistant enterococci (VRE).
143 n-resistant Staphylococcus aureus (MRSA) and vancomycin-resistant Enterococci (VRE).
144 t contribute to the transmission dynamics of vancomycin-resistant enterococci (VRE).
145 in-resistant Staphylococcus aureus (MRSA) or vancomycin-resistant enterococci (VRE).
146 treatment of patients with stool carriage of vancomycin-resistant enterococci (VRE).
147        In 1996, the dominant (43%) strain of vancomycin-resistant enterococci (VRE; type A) at Massac
148 rococcal bloodstream infection (BSI; 22 with vancomycin-resistant enterococci [VRE] and 61 with vanco
149 n-resistant Staphylococcus aureus [MRSA] and vancomycin-resistant enterococci [VRE]) or rapid screeni
150 (95 percent), high-density colonization with vancomycin-resistant enterococci was maintained (mean [+
151                                       All 36 vancomycin-resistant enterococci were detected by the Ph
152                            Nineteen types of vancomycin-resistant enterococci were documented by puls
153                                In late 1996, vancomycin-resistant enterococci were first detected in
154 hicillin-resistant Staphylococcus aureus, or vancomycin-resistant enterococci were included.
155 ties, the risk factors for colonization with vancomycin-resistant enterococci were prior hospitalizat
156 hicillin-resistant Staphylococcus aureus and vancomycin-resistant Enterococci, without affecting the
157 mutant subsequently resulted in clearance of vancomycin-resistant enterococci, without plasmid transf

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