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1 ting abundance of replete ticks (Dermacentor variabilis).
2 intramembrane cleaving proteases in Anabaena variabilis.
3 ied through a genome-wide survey in Anabaena variabilis.
4 ization levels between I. fasciculata and I. variabilis.
5  from Rhodosporidium toruloides and Anabaena variabilis.
6 se inhibitor (KPI) from the tick Dermacentor variabilis.
7  in the ovaries of the hard tick Dermacentor variabilis.
8 GJB4) in the etiology of erythrokeratodermia variabilis.
9 rived cells from a natural host, Dermacentor variabilis.
10 ions and from salivary glands of Dermacentor variabilis.
11 vel with the vnfD gene product from Anabaena variabilis.
12 een for cold-induced transcripts in Anabaena variabilis.
13 e in the pathogenesis of erythrokeratodermia variabilis.
14 essential for the V-nitrogenase system in A. variabilis.
15 e transport system, vupABC, were found in A. variabilis about 5 kb from the major cluster of genes en
16 s and a sporadic case of erythrokeratodermia variabilis, all of which were not found in controls.
17      The similarity of the vnfEN genes of A. variabilis and A. vinelandii was not strong.
18 otypic differences, both erythrokeratodermia variabilis and erythrokeratodermia with ataxia map to a
19                                   Rhizomucor variabilis and Hormographiella aspergillata rarely cause
20 ndosymbionts (Chlorella heliozoae, Chlorella variabilis and Micractinium conductrix).
21 tous nitrogen-fixing cyanobacteria, Anabaena variabilis and Nostoc punctiforme.
22  (Ulva pertusa), and cyanobacteria (Anabaena variabilis and Synechococcus) have been investigated by
23 genetic heterogeneity in erythrokeratodermia variabilis, and emphasize that intercellular communicati
24 e biological species as the benthic Bolivina variabilis, and geochemical evidence that this ecologica
25 ed States, Amblyomma americanum, Dermacentor variabilis, and Ixodes scapularis are among the principa
26 presented with localized erythrokeratodermia variabilis, and one with erythrokeratodermia and ataxia.
27 topology, Absidia corymbifera and Rhizomucor variabilis appear to be misplaced taxonomically.
28 MITEs in the two bacterial genomes, Anabaena variabilis ATCC 29413 and Haloquadratum walsbyi DSM 1679
29  from cyanobacteria, in particular, Anabaena variabilis ATCC 29413 and Nostoc punctiforme ATCC 29133,
30                                     Anabaena variabilis ATCC 29413 fixes nitrogen in specialized cell
31                                     Anabaena variabilis ATCC 29413 is a filamentous heterocystous cya
32                                     Anabaena variabilis ATCC 29413 is unusual in that it has two Mo-d
33                  The cyanobacterium Anabaena variabilis ATCC 29413 uses nitrate and atmospheric N2 as
34 rate specificity from that known in Anabaena variabilis ATCC 29413.
35 utant strains of the cyanobacterium Anabaena variabilis ATCC 29413.
36 haplotype analyses place erythrokeratodermia variabilis between the marker D1S496 and D1S186 with a m
37 rved for the two active-site loops in the A. variabilis C503S/C565S double mutant, yielding a complet
38                                           A. variabilis C503S/C565S PAL is shown to be both more ther
39                        Examination of the A. variabilis C503S/C565S PAL structure, combined with anal
40 to antigens from four other algae: Chlorella variabilis, Coccomyxa subellipsoidea, Nannochloropsis oc
41  three antimicrobials, especially the two D. variabilis defensin isoforms, are markedly different, il
42 itor from the American dog tick (Dermacentor variabilis) (DvKPI) is suppressed by small interfering R
43 31 cause the skin disease erythrokeratoderma variabilis (EKV) and hearing loss with or without neurop
44          The skin disease erythrokeratoderma variabilis (EKV) has been shown to be associated with mu
45 a combination of SCA and erythrokeratodermia variabilis (EKV) in an autosomal dominant fashion was de
46 underlie the skin disease erythrokeratoderma variabilis (EKV) or sensorineural hearing loss with/with
47 ases, including familial erythrokeratodermia variabilis (EKV).
48                          Erythrokeratodermia variabilis (EKV, OMIM 133200) is an autosomal dominant g
49                          Erythrokeratodermia variabilis et progressiva (EKVP) is a rare, inherited sk
50  of GJB4 in 13 unrelated erythrokeratodermia variabilis families without detectable mutations in GJB3
51      The filamentous cyanobacterium Anabaena variabilis fixes nitrogen in the presence of vanadium (V
52                                     Anabaena variabilis fixes nitrogen under aerobic growth condition
53 ase from the established erythrokeratodermia variabilis gene region indicating genetic heterogeneity
54                                           A. variabilis grew better with increasing concentrations of
55                                     Anabaena variabilis grows heterotrophically using fructose, while
56                  The cyanobacterium Anabaena variabilis has two Mo-nitrogenases that function under d
57  calcium-dependent protease PrcA of Anabaena variabilis, HreP forms a new subfamily of bacterial subt
58             Expression of nifB2:lacZ from A. variabilis in anaerobic vegetative cells of Anabaena sp.
59 Connexin 31, which causes erythrokeratoderma variabilis, induces ER stress and p63-dependent epiderma
60                       We report a case of A. variabilis invasive wound infection in a 21-year-old mal
61                          Erythrokeratodermia variabilis is an autosomal dominant genodermatosis chara
62                                Apophysomyces variabilis is an emerging fungal pathogen that can cause
63 r miehei-Rhizomucor pusillus clade, while R. variabilis is nested within Mucor.
64   One of these disorders, erythrokeratoderma variabilis, is associated with germline mutations in the
65  isolates (three A. trapeziformis and two A. variabilis isolates).
66                                  In Anabaena variabilis it was shown that a NifE-N fusion protein enc
67 29 cells (mouse fibroblasts), implicating D. variabilis KPI as a bacteriostatic protein, a property t
68 kettsia montanensis, results in sustained D. variabilis KPI gene expression in the midgut.
69          Interestingly, we observe little D. variabilis KPI gene expression in the salivary gland and
70 rthermore, our in vitro studies show that D. variabilis KPI limits rickettsial colonization of L929 c
71 s anticoagulant properties indicates that D. variabilis KPI may be important for blood meal digestion
72 ole in the midgut during feeding and that D. variabilis KPI may be involved as part of the tick's def
73             However, our demonstration of D. variabilis KPI's anticoagulant properties indicates that
74 rotein, a property that may be related to D. variabilis KPI's trypsin inhibitory capability.
75  other Ixodid ticks, most proteins in the D. variabilis midgut cDNA library were intracellular.
76 and 8-heptadecene were detected in Chlorella variabilis NC64A (Trebouxiophyceae) and several Nannochl
77 re compared to PBCV-1 and its host Chlorella variabilis NC64A predicted proteomes.
78 n this case we reduced aggregation of the A. variabilis PAL by mutating two surface cysteine residues
79 l and biochemical characterization of the A. variabilis PAL C503S/C565S double mutant and carefully c
80 from the salivary glands of male Dermacentor variabilis persistently infected with A. marginale after
81 istics, and efficacy of recombinant Anabaena variabilis phenylalanine ammonia lyase (produced in Esch
82 re loaded with recombinant PAL from Anabaena variabilis (rAvPAL) and their ability to perform as bior
83 d to sustain a normal growth rate for the A. variabilis rca mutant.
84  family, maps within the erythrokeratodermia variabilis region and is an attractive candidate gene.
85 ermacentor occidentalis Marx and 30 adult D. variabilis Say ticks, collected chiefly in southern Cali
86                        The vnfEN genes of A. variabilis showed greater similarity to the vnfDK genes
87 sis demonstrated that these five putative A. variabilis site-2 proteases (S2Ps(Av)) have authentic pr
88 rine sponges Ircinia fasciculata and Ircinia variabilis that live in sympatry.
89 prise multiple operons; however, in Anabaena variabilis, the promoter for the first gene in the clust
90  and in Amblyomma americanum and Dermacentor variabilis ticks collected from a cattle herd in Missour
91 ysozyme, in the midguts and fat bodies of D. variabilis ticks that were challenged with R. montanensi
92 nactivation of rca reduced the ability of A. variabilis to elevate Rubisco activity under high light
93 salivary glands of the hard tick Dermacentor variabilis using a combination of gel filtration and hig
94            The patient first developed an R. variabilis var. regularior palate infection and later de
95   High-affinity transport of molybdate in A. variabilis was mediated by an ABC-type transport system
96 mal dominant skin disease erythrokeratoderma variabilis was segregating.
97 d in vivo and resulting mutant strains of A. variabilis were found to be incapable of synthesizing im
98 1 from Clostridium thermocellum and Anabaena variabilis, which are enzymatically indistinguishable fr
99                                     Anabaena variabilis, which can fix nitrogen by using an alternati
100 phore pathway in the cyanobacterium Anabaena variabilis, which was shown to be a bona fide DABA DC.
101  in this study, we show that challenge of D. variabilis with the spotted fever group rickettsia, Rick

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