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1 prophylaxis against Pneumocystis carinii and varicella zoster.
2  and pneumonia, and an increase observed for varicella zoster.
3 -mg dose of fingolimod: disseminated primary varicella zoster and herpes simplex encephalitis.
4 varicella-zoster virus vaccine in preventing varicella-zoster and herpes zoster is well documented, a
5 smosis, other infections (such as syphillis, varicella-zoster, and parvovirus B19), cytomegalovirus,
6  reaction (PCR) analysis for Herpes simplex, varicella zoster, cytomegalovirus, Epstein-Barr virus an
7 ependent promoter and a model activator, the varicella zoster IE62 protein, it was determined that HC
8                             Prophylaxis with varicella-zoster immunoglobulin can reduce the severity
9 number and critical immunization coverage of varicella-zoster infection in Belgium, Italy, Poland, an
10 y cells is presumably the initiating step of varicella-zoster infection.
11 virus (OPV), rubella, measles, yellow fever, varicella-zoster, multivalent measles/mumps/rubella, and
12 of herpes zoster disease, which is caused by Varicella zoster Nevertheless, the pathophysiological me
13                                   Only a few varicella-zoster nucleocapsids and cytoplasmic virions w
14 roidism, hypercholesterolemia, hypertension, varicella zoster, peripheral vascular disease, and autoi
15  clonal expansions in response to attenuated varicella-zoster vaccination in four pairs of adult iden
16 laria vaccine and the subunit glycoprotein E varicella zoster vaccine (both currently in phase III).
17                 Two doses of live-attenuated varicella-zoster vaccine are recommended for human immun
18 thy, such as prior herpes simplex keratitis, varicella-zoster viral keratitis, the linear form of Thy
19 e calculated an anterograde velocity for the varicella zoster virion of 5.55 mm/h or .0015 mm/s.
20  = 60), followed by tuberculosis (n = 8) and varicella zoster virus (n = 7).
21 s HSV1 and HSV2 (also termed HHV1 and HHV2), varicella zoster virus (VZV or HHV3), EBV (HHV4), cytome
22 tients showed a decreased ability to control varicella zoster virus (VZV) and Epstein-Barr virus (EBV
23                                              Varicella zoster virus (VZV) and the two herpes simplex
24                                              Varicella zoster virus (VZV) antibody titers (measured b
25                                              Varicella zoster virus (VZV) antigen was found in all of
26                     Vasculopathies caused by varicella zoster virus (VZV) are indicative of a product
27                                      CMV and varicella zoster virus (VZV) are significant causes of m
28  response biomarkers measuring antibodies to varicella zoster virus (VZV) by glycoprotein-based enzym
29 lex virus types 1 (HSV-1) and 2 (HSV-2), and varicella zoster virus (VZV) by weekly polymerase chain
30 se of herpes zoster caused by the attenuated varicella zoster virus (VZV) contained in Zostavax in a
31 , or no history of zoster (group 3) revealed varicella zoster virus (VZV) DNA in saliva samples from
32 rs who were immunized with Zostavax revealed varicella zoster virus (VZV) DNA in swabs of skin inocul
33                            Herpesvirions and varicella zoster virus (VZV) DNA were recently reported
34                                              Varicella zoster virus (VZV) establishes latency in dors
35                                              Varicella zoster virus (VZV) establishes lifelong persis
36  as an alternative to sampling of rashes for varicella zoster virus (VZV) genotyping and further char
37 s positive for herpes simplex virus (HSV) or varicella zoster virus (VZV) in 79% to 100% of cases of
38 Clinical reports observe the reactivation of varicella zoster virus (VZV) in people who have recovere
39                          Reactivation of the varicella zoster virus (VZV) increases during aging.
40                                              Varicella zoster virus (VZV) infections are a relevant c
41 virus (CMV), herpes simplex virus (HSV), and varicella zoster virus (VZV) infections were monitored i
42                                              Varicella zoster virus (VZV) is a neurotropic alphaherpe
43                                              Varicella Zoster Virus (VZV) is the causative agent of v
44                                              Varicella zoster virus (VZV) is the etiological agent of
45                                              Varicella zoster virus (VZV) reactivation results in zos
46                    An adjuvanted recombinant varicella zoster virus (VZV) subunit vaccine is being de
47  zoster (HZ) cases may play a larger role in varicella zoster virus (VZV) transmission.
48                                              Varicella zoster virus (VZV) typically causes chickenpox
49 ne responses to a high-titer live attenuated varicella zoster virus (VZV) vaccine (zoster vaccine), w
50    Since the introduction of live attenuated varicella zoster virus (VZV) vaccine in 1995 there has b
51 s, granulomatous aortitis, and intracerebral varicella zoster virus (VZV) vasculopathy.
52 portion of HZ cases caused by vaccine-strain varicella zoster virus (VZV), assessed the positive pred
53 c primers to detect DNA from JC virus (JCV), varicella zoster virus (VZV), cytomegalovirus (CMV), Eps
54 on childhood disease, chicken pox, caused by varicella zoster virus (VZV), over an 11-y period.
55 erpesviruses, herpes simplex virus (HSV) and varicella zoster virus (VZV), results in the rapid accum
56  immunogenicity of live-attenuated Oka/Merck varicella zoster virus (VZV)-containing vaccine (hereaft
57                                     Boost of varicella zoster virus (VZV)-specific cellular immunity
58     We investigated the relationship between varicella zoster virus (VZV)-specific memory CD4(+) T ce
59  virus type 1 (HSV-1) and type 2 (HSV-2) and varicella zoster virus (VZV)-was determined in autonomic
60 ses with age, which leads to reactivation of varicella zoster virus (VZV).
61  1 and 2 and the sequence-divergent pathogen varicella zoster virus (VZV).
62                    For herpes simplex virus, varicella zoster virus and cytomegalovirus, these advanc
63 RN, PCR-positive for herpes simplex virus or varicella zoster virus and evaluated between January 200
64 genes of both herpes simplex virus (HSV) and varicella zoster virus and functions, in part, by coupli
65  beyond CMV to other herpes viruses, such as varicella zoster virus and possibly Epstein-Barr virus.
66 ld decrease external boosting of immunity to varicella zoster virus and thereby increase incidence of
67  (as determined by testing lesions swabs for varicella zoster virus DNA by polymerase chain reaction)
68 ence of confirmed varicella (by detection of varicella zoster virus DNA or epidemiological link) from
69                                              Varicella zoster virus DNA was detected 2 months after t
70                                              Varicella zoster virus encodes an immediate-early (IE) p
71 were randomized 1:1 to receive either HZ/su (varicella zoster virus glycoprotein E; AS01B Adjuvant Sy
72 rveillance, combined with information from a Varicella Zoster Virus Identification Program, which use
73 cation) were associated with protection from varicella zoster virus infection (hazard ratio, 0.43; 95
74 contact dermatitis, infectious folliculitis, varicella zoster virus infection, fixed drug eruption, a
75 processes, including ubiquitin clearance and Varicella Zoster Virus infection.
76 nd increased susceptibility to bacterial and varicella zoster virus infections.
77                              Similarly, in a varicella zoster virus lytic infection, HCF-1, Set1, and
78                             One patient with varicella zoster virus meningitis and acute GVHD had iC9
79 e fills a notable gap in our knowledge about varicella zoster virus neuronal transportation.
80     Because there is no good animal model of varicella zoster virus reactivation from latency, this e
81 -Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex
82 ty of herpes simplex virus, cytomegalovirus, varicella zoster virus, and Epstein-Barr virus in our po
83                        Herpes simplex virus, varicella zoster virus, and pseudorabies virus are neuro
84 nstrate that childhood infections, including varicella zoster virus, are associated with an increased
85  for other infections (herpes simplex virus, varicella zoster virus, bacterial and fungal infections)
86 itis (AU), owing to either herpes simplex or varicella zoster virus, by using the Standardization of
87 se encephalitis virus, herpes simplex virus, varicella zoster virus, cytomegalovirus, dengue virus an
88 deficiency virus (HIV)-herpes simplex virus, varicella zoster virus, Epstein-Barr virus (EBV), and cy
89 gnificant members of the herpesvirus family: varicella zoster virus, human cytomegalovirus, and Epste
90 genes, Treponema pallidium, parvovirus, HIV, varicella zoster virus, Rubella, Cytomegalovirus, and He
91 highly dependent on the host cell, we tested varicella zoster virus-infected cell lysates and clinica
92 ese is also activated by the closely related varicella zoster virus.
93 ew mutations or recombination with wild-type Varicella zoster virus.
94  2, cytomegalovirus, Epstein-Barr virus, and varicella zoster virus.
95 1), P. jirovecii pneumonia (1.77; .42-7.47), varicella-zoster virus (1.51; .71-3.22), as well as over
96                                              Varicella-zoster virus (VZV) activates the phosphatidyli
97 SV functioned as a monopartite NLS, while in varicella-zoster virus (VZV) activity required an adjace
98 are the main architectural contrasts between varicella-zoster virus (VZV) and herpes simplex virus (H
99 d the Us9 homologs from two human pathogens, varicella-zoster virus (VZV) and herpes simplex virus ty
100                                  Intraocular varicella-zoster virus (VZV) and HSV type 1 (HSV-1) infe
101                   Although the homologs from varicella-zoster virus (VZV) and human cytomegalovirus (
102 simplex virus type 1 (HSV-1) is conserved in varicella-zoster virus (VZV) and pseudorabies virus (PRV
103 ype 1 (EHV-1), pseudorabies virus (PRV), and varicella-zoster virus (VZV) and their subsequent functi
104             Simian varicella virus (SVV) and varicella-zoster virus (VZV) are closely related alphahe
105 gument proteins encoded by ORF11 and ORF9 of varicella-zoster virus (VZV) are conserved among all alp
106 mmunity and protects against reactivation of varicella-zoster virus (VZV) as HZ.
107     Infection of human neurons in vitro with varicella-zoster virus (VZV) at a low multiplicity of in
108 y, IDE has been proposed as the receptor for varicella-zoster virus (VZV) attachment.
109                           The attenuation of varicella-zoster virus (VZV) by Takahashi in 1974 was a
110      Serum was tested for antibodies against varicella-zoster virus (VZV) by use of the previously va
111                                              Varicella-zoster virus (VZV) causes chicken pox and shin
112                                              Varicella-zoster virus (VZV) causes chickenpox and react
113 highly infectious, human-restricted pathogen varicella-zoster virus (VZV) causes chickenpox and shing
114 ating VZV from clinical specimens.IMPORTANCE Varicella-zoster virus (VZV) causes chickenpox and shing
115                                              Varicella-zoster virus (VZV) causes chickenpox upon prim
116                                              Varicella-zoster virus (VZV) causes varicella and establ
117                                              Varicella-zoster virus (VZV) causes varicella, establish
118                                              Varicella-zoster virus (VZV) characteristically forms mu
119 y throughout the study and were analyzed for varicella-zoster virus (VZV) DNA by use of both qualitat
120                                    Wild-type varicella-zoster virus (VZV) DNA was identified in all 3
121  15, pain was scored and saliva examined for varicella-zoster virus (VZV) DNA.
122       Herein we describe an episode of focal varicella-zoster virus (VZV) encephalitis in a healthy y
123                                              Varicella-zoster virus (VZV) establishes a lifelong late
124                  The neurotropic herpesvirus varicella-zoster virus (VZV) establishes a lifelong late
125                                              Varicella-zoster virus (VZV) establishes latency in huma
126 ts had similar magnitude memory responses to varicella-zoster virus (VZV) ex vivo restimulation measu
127                When grown in cultured cells, varicella-zoster virus (VZV) forms many aberrant light p
128                            Information about varicella-zoster virus (VZV) gB is limited, but homology
129                                              Varicella-zoster virus (VZV) glycoprotein E (gE) is esse
130                                              Varicella-zoster virus (VZV) glycoprotein E (gE) is requ
131 tive target for antiviral therapy.IMPORTANCE Varicella-zoster virus (VZV) has infected over 90% of pe
132          Mechanisms of neuronal infection by varicella-zoster virus (VZV) have been challenging to st
133 umoral and cell-mediated immune responses to varicella-zoster virus (VZV) have been evaluated after 1
134 (EBV) EB2, herpes simplex virus (HSV) ICP27, varicella-zoster virus (VZV) IE4/ORF4, and cytomegalovir
135                                          The varicella-zoster virus (VZV) IE62 protein is the major t
136                                              Varicella-zoster virus (VZV) immediate-early 63 protein
137 ction by enveloped, but not cell-associated, varicella-zoster virus (VZV) in a dose-dependent manner
138 ects immediate-early protein IE63 encoded by varicella-zoster virus (VZV) in the cytoplasm of product
139                                              Varicella-zoster virus (VZV) induces apoptosis in human
140      Previous studies have demonstrated that varicella-zoster virus (VZV) infection activates ERK1/2,
141                                              Varicella-zoster virus (VZV) infection causes varicella,
142                                      Primary varicella-zoster virus (VZV) infection in humans produce
143                                              Varicella-zoster virus (VZV) infection is usually mild i
144            Transcriptional changes following varicella-zoster virus (VZV) infection of cultured human
145 acaques (RMs) recapitulates the hallmarks of varicella-zoster virus (VZV) infection of humans, includ
146                                              Varicella-zoster virus (VZV) infection provides a valuab
147 he lytic, latent, and reactivating phases of varicella-zoster virus (VZV) infection were recapitulate
148 extensively studied the role of autophagy in varicella-zoster virus (VZV) infection, and have observe
149                                              Varicella-zoster virus (VZV) infections increasingly are
150                                              Varicella-zoster virus (VZV) is a common pathogen that c
151                                              Varicella-zoster virus (VZV) is a highly contagious agen
152                                              Varicella-zoster virus (VZV) is a highly neurotropic vir
153                                              Varicella-zoster virus (VZV) is a human alpha-herpesviru
154                                              Varicella-zoster virus (VZV) is a human alphaherpesvirus
155                                              Varicella-zoster virus (VZV) is a human alphaherpesvirus
156                                              Varicella-zoster virus (VZV) is a human neurotropic alph
157     The immediate early 62 protein (IE62) of varicella-zoster virus (VZV) is a major viral trans-acti
158                                              Varicella-zoster virus (VZV) is a member of the Herpesvi
159                                              Varicella-zoster virus (VZV) is a neurotropic alphaherpe
160                                              Varicella-zoster virus (VZV) is a ubiquitous pathogen th
161                                              Varicella-zoster virus (VZV) is a ubiquitous, highly cel
162                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
163                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
164                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
165                                              Varicella-zoster virus (VZV) is an extremely cell-associ
166                                              Varicella-zoster virus (VZV) is highly cell associated w
167 mary infection, latency, and reactivation by varicella-zoster virus (VZV) is incompletely understood.
168                                              Varicella-zoster virus (VZV) is renowned for its low tit
169                                              Varicella-zoster virus (VZV) is renowned for its very lo
170                                              Varicella-zoster virus (VZV) is the alphaherpesvirus tha
171                                              Varicella-zoster virus (VZV) is the causative agent of b
172                                              Varicella-zoster virus (VZV) is the causative agent of c
173                                              Varicella-zoster virus (VZV) is the etiological agent of
174                                          The varicella-zoster virus (VZV) major transactivator, IE62,
175       We report a case of AIDS presenting as varicella-zoster virus (VZV) meningomyeloradiculitis ass
176                      To efficiently generate varicella-zoster virus (VZV) mutants, we inserted a bact
177 f herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) on 695 consecutive cutaneou
178                                              Varicella-zoster virus (VZV) open reading frame (ORF) 63
179                                              Varicella-zoster virus (VZV) open reading frame (ORF) 63
180                                              Varicella-zoster virus (VZV) open reading frame 10 (ORF1
181        Based on comparative genome analyses, varicella-zoster virus (VZV) open reading frame 23 (ORF2
182                                          The varicella-zoster virus (VZV) open reading frame 54 (ORF5
183                                              Varicella-zoster virus (VZV) open reading frame 61 (ORF6
184                                Three loci in varicella-zoster virus (VZV) open reading frame 62 (ORF6
185                                              Varicella-zoster virus (VZV) open reading frame 63 (ORF6
186                                              Varicella-zoster virus (VZV) open reading frame 66 (ORF6
187                 The gene cluster composed of varicella-zoster virus (VZV) open reading frame 9 (ORF9)
188 f transcripts corresponding to all 68 unique varicella-zoster virus (VZV) open reading frames (ORFs)
189                                          The varicella-zoster virus (VZV) ORF61 protein is necessary
190                      The architecture of the varicella-zoster virus (VZV) origin of DNA replication (
191                                          The varicella-zoster virus (VZV) origin of DNA replication (
192     In this report, we show that ORF61p, the varicella-zoster virus (VZV) ortholog of ICP0, does not
193 ced syncytium formation, a characteristic of varicella-zoster virus (VZV) pathology in skin and senso
194 this minireview is to provide an overview of varicella-zoster virus (VZV) phylogenetics and phylogeog
195                                  We examined varicella-zoster virus (VZV) polymerase chain reaction (
196 er acyclovir prophylaxis should be given for varicella-zoster virus (VZV) prophylaxis after hematopoi
197                                  Analyses of varicella-zoster virus (VZV) protein expression during l
198                  ORF66p, a virion-associated varicella-zoster virus (VZV) protein, is a member of a c
199 nt is associated with increased incidence of varicella-zoster virus (VZV) reactivation in patients wi
200 IMPORTANCE The neurological damage caused by varicella-zoster virus (VZV) reactivation is commonly ma
201 a total of five major genotypes among the 22 varicella-zoster virus (VZV) strains or isolates for whi
202                                              Varicella-zoster virus (VZV) T-cell responses by interfe
203                                              Varicella-zoster virus (VZV) T-cell-mediated immunity (V
204                                          The varicella-zoster virus (VZV) terminase components (pORF2
205 r herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) to the BD Max system by usi
206                                   Studies of varicella-zoster virus (VZV) tropism for T cells support
207                                   Although a varicella-zoster virus (VZV) vaccine has been used for m
208 75 years) immunized with the live-attenuated varicella-zoster virus (VZV) vaccine.
209  of a high-potency live-attenuated Oka/Merck varicella-zoster virus (VZV) vaccine.
210                                              Varicella-zoster virus (VZV) vasculopathy produces strok
211 ish adverse events associated with wild-type varicella-zoster virus (VZV) versus those associated wit
212                                              Varicella-zoster virus (VZV), a double-stranded DNA alph
213 igated during the entire infectious cycle of varicella-zoster virus (VZV), a human herpesvirus.
214     The immediate early 62 protein (IE62) of varicella-zoster virus (VZV), a major viral trans-activa
215                       Primary infection with varicella-zoster virus (VZV), a neurotropic alphaherpesv
216          Regulation of gene transcription in varicella-zoster virus (VZV), a ubiquitous human neurotr
217 1) and pseudorabies virus (PRV) and ORF66 in varicella-zoster virus (VZV), affects several viral and
218                                              Varicella-zoster virus (VZV), an alphaherpesvirus restri
219                          Here we report that varicella-zoster virus (VZV), an alphaherpesvirus that i
220 pear healthy at 2 weeks after infection with varicella-zoster virus (VZV), and the cell culture mediu
221 ovirus, herpes simplex virus type 1 (HSV-1), varicella-zoster virus (VZV), and West Nile virus (WNV).
222 sviruses, herpes simplex virus 1 (HSV-1) and varicella-zoster virus (VZV), confirmed the expression o
223 ults for herpes simplex virus 1/2 (HSV-1/2), varicella-zoster virus (VZV), cytomegalovirus (CMV), or
224     In this study, quantitative PCR detected varicella-zoster virus (VZV), herpes simplex virus 1 (HS
225                      Levels of inhibition of varicella-zoster virus (VZV), human cytomegalovirus (HCM
226                                              Varicella-zoster virus (VZV), in both wild-type and live
227                                              Varicella-zoster virus (VZV), of the family Alphaherpesv
228 showed cytopathic changes, but HSV-1, unlike varicella-zoster virus (VZV), only rarely infected satel
229                                         Like varicella-zoster virus (VZV), simian varicella virus (SV
230 cilitate the generation of mutant viruses of varicella-zoster virus (VZV), the agent causing varicell
231 nation was more likely to identify wild-type varicella-zoster virus (VZV), whereas the presence of Ok
232                                           In varicella-zoster virus (VZV)-infected primary human brai
233  the risk of herpes zoster (HZ), we compared varicella-zoster virus (VZV)-specific and nonspecific T-
234                                              Varicella-zoster virus (VZV)-specific cell-mediated immu
235 n association with an age-related decline in varicella-zoster virus (VZV)-specific cell-mediated immu
236 udy were to evaluate the association between varicella-zoster virus (VZV)-specific humoral and cell-m
237                                   To measure varicella-zoster virus (VZV)-specific immune responses u
238 lication that can occur with reactivation of varicella-zoster virus (VZV).
239 itive GCA is associated with TA infection by varicella-zoster virus (VZV).
240 rus (CMV), herpes simplex viruses (HSV), and varicella-zoster virus (VZV).
241 rpes zoster caused by reactivation of latent Varicella-Zoster virus (VZV).
242 ly recognized component of the life cycle of varicella-zoster virus (VZV).
243  potency against hepatitis B virus (HBV) and varicella-zoster virus (VZV).
244               None have yet been reported in varicella-zoster virus (VZV; also known as human herpesv
245 onal and pathogen-specific stimulation (with varicella-zoster virus [VZV] and cytomegalovirus [CMV]).
246 cropsy of two monkeys inoculated with simian varicella-zoster virus and euthanized 117 days later.
247 ia in adults includes common agents, such as varicella-zoster virus and influenza virus, as well as r
248 ate changes in the molecular epidemiology of varicella-zoster virus and the effect of immunization wi
249                                              Varicella-zoster virus antigen was found in 45 of 70 GCA
250                                              Varicella-zoster virus antigen was frequently found in p
251 e whether herpes zoster antigen (also called varicella-zoster virus antigen) was detectable in tempor
252 gical boosting, through which reexposures to varicella-zoster virus are thought to reduce the individ
253 clonal antibodies against a major antigen of varicella-zoster virus called gE.
254 ES by the corresponding region from ORF61 of varicella-zoster virus did not rescue ND10 fusion.
255 th HSE (p.Leu297Val) and 1 in a patient with varicella-zoster virus encephalitis (p.Leu199Phe).
256 the phenotypic spectrum of TLR3 mutations to varicella-zoster virus encephalitis and support the role
257 rpes zoster is a common late complication of varicella-zoster virus exposure and can be further compl
258 hus, a 30-h delay after death did not affect varicella-zoster virus expression in latently infected g
259                                   Studies of varicella-zoster virus gene expression during latency re
260                                          The varicella-zoster virus geometric mean titer (GMT) and ge
261 ve (at months 0, 1, 3) three doses of 50 mug varicella-zoster virus glycoprotein E (gE) adjuvanted wi
262                                An adjuvanted varicella-zoster virus glycoprotein E (gE) subunit vacci
263 ubjects received 3 doses of HZ/su (50 microg varicella-zoster virus glycoprotein E [gE] combined with
264 g older adults, a subunit vaccine containing varicella-zoster virus glycoprotein E and the AS01B adju
265  zoster vaccine showed a greater increase in varicella-zoster virus gpELISA antibody compared with su
266 equences of wild-type and vaccine strains of varicella-zoster virus have been published and listed in
267 transfected cells, whereas expression of the varicella-zoster virus ICP22 homolog, ORF63, does not.
268 or who had resided in a country with endemic varicella-zoster virus infection for 30 years or more we
269 erties that may favor reactivation of latent varicella-zoster virus infection.
270                                        While varicella-zoster virus is also insensitive to interferon
271  regulate infection of host cells.IMPORTANCE Varicella-zoster virus is an important human pathogen, w
272                          Because features of varicella-zoster virus latency are similar in primate an
273                                          The varicella-zoster virus major transactivator, IE62, conta
274                                    Available varicella-zoster virus models can be classified in 3 mai
275 -coinfected children and were independent of varicella-zoster virus or herpes-simplex virus 1 coinfec
276 lives ranging from an estimated 50 years for varicella-zoster virus to more than 200 years for other
277 ng heat-inactivated or replication-defective varicella-zoster virus to prevent HZ in immunocompromise
278 The continued success of the live attenuated varicella-zoster virus vaccine in preventing varicella-z
279                The licensed live, attenuated varicella-zoster virus vaccine prevents herpes zoster in
280                                    HHV-1 and Varicella-Zoster virus were detected only twice and HHV-
281 ty for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were studied in 1079 6-year-old c
282 ncing to identify nosocomial transmission of varicella-zoster virus with fatal outcome.
283  2, human herpesvirus 6, human parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryp
284 antigens (vaccinia, measles, mumps, rubella, varicella-zoster virus, and Epstein-Barr virus) and nonr
285 Findings from skin biopsy, viral culture for varicella-zoster virus, and skin prick test to common fo
286  (HSV) and other alphaherpesviruses, such as varicella-zoster virus, depend upon the capacity to navi
287 es (parechovirus, dengue virus, Nipah virus, varicella-zoster virus, mumps virus, measles virus, lyss
288 h HLA-B27-associated (4460 [2465] pg/mL) and varicella-zoster virus-associated (5386 [1778] pg/mL) uv
289                 Among the 131 live births to varicella-zoster virus-seronegative women, there was no
290 in 50-59-year-old subjects were examined for varicella-zoster virus-specific antibody responses to va
291 1, CTLA-4, and TIM-3, whereas <2% of CMV- or varicella-zoster virus-specific CD4(+) T cells expressed
292  the change from baseline in IgG antibody to varicella-zoster virus-specific glycoproteins (gpELISA)
293 losely related to herpes simplex viruses and varicella-zoster virus.
294 G synthesis, and elevated antibody titers to varicella-zoster virus.
295 d, for example, in relation to pertussis and varicella-zoster virus.
296 ation were detected in OPV, mumps virus, and varicella-zoster virus.
297 uses herpes simplex virus type 1 (HSV-1) and varicella-zoster virus.
298 known as breakthrough disease, if exposed to varicella-zoster virus.
299 erpesviruses herpes simplex virus type 1 and varicella-zoster virus.
300 nuated varicella vaccine virus and wild-type varicella-zoster virus.

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