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1 processes, including ubiquitin clearance and Varicella Zoster Virus infection.
2 associated with decreased seroprevalence of varicella-zoster virus infection.
3 erties that may favor reactivation of latent varicella-zoster virus infection.
4 re critical host cell targets during primary varicella-zoster virus infection.
5 nd increased susceptibility to bacterial and varicella zoster virus infections.
6 s a promising new agent for the treatment of varicella-zoster virus infections.
7 oster-virus protects individuals with latent varicella-zoster virus infection against herpes zoster b
8 To evaluate a possible association between varicella-zoster virus infection and glioma, the authors
9 tient has had no infection except dermatomal varicella-zoster virus infection and no acute or chronic
10 contact dermatitis, infectious folliculitis, varicella zoster virus infection, fixed drug eruption, a
11 or who had resided in a country with endemic varicella-zoster virus infection for 30 years or more we
12 cation) were associated with protection from varicella zoster virus infection (hazard ratio, 0.43; 95
13 ir has been the mainstay for treating severe varicella-zoster virus infections; however, newer antivi
16 ikely than controls either to have had prior varicella-zoster virus infection or to have an immunoglo
17 dds ratio [OR], 0.28; 95% CI, 0.21-0.37) and varicella-zoster virus infections (OR, 0.29; 95% CI, 0.1
18 ulness of SVV infection as a model for human varicella-zoster virus infection, particularly virus rea
19 a32 is associated with the seroprevalence of varicella-zoster virus infection, we tested blood donors
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