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1 ese is also activated by the closely related varicella zoster virus.
2 ew mutations or recombination with wild-type Varicella zoster virus.
3  2, cytomegalovirus, Epstein-Barr virus, and varicella zoster virus.
4 losely related to herpes simplex viruses and varicella-zoster virus.
5 G synthesis, and elevated antibody titers to varicella-zoster virus.
6 d, for example, in relation to pertussis and varicella-zoster virus.
7 ation were detected in OPV, mumps virus, and varicella-zoster virus.
8 uses herpes simplex virus type 1 (HSV-1) and varicella-zoster virus.
9 known as breakthrough disease, if exposed to varicella-zoster virus.
10 erpesviruses herpes simplex virus type 1 and varicella-zoster virus.
11 nuated varicella vaccine virus and wild-type varicella-zoster virus.
12 gens herpes simplex virus type 1 (HSV-1) and varicella-zoster virus.
13 1), P. jirovecii pneumonia (1.77; .42-7.47), varicella-zoster virus (1.51; .71-3.22), as well as over
14 -Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex
15                    For herpes simplex virus, varicella zoster virus and cytomegalovirus, these advanc
16 RN, PCR-positive for herpes simplex virus or varicella zoster virus and evaluated between January 200
17 genes of both herpes simplex virus (HSV) and varicella zoster virus and functions, in part, by coupli
18  beyond CMV to other herpes viruses, such as varicella zoster virus and possibly Epstein-Barr virus.
19 ld decrease external boosting of immunity to varicella zoster virus and thereby increase incidence of
20 cropsy of two monkeys inoculated with simian varicella-zoster virus and euthanized 117 days later.
21 ia in adults includes common agents, such as varicella-zoster virus and influenza virus, as well as r
22 ate changes in the molecular epidemiology of varicella-zoster virus and the effect of immunization wi
23 ty of herpes simplex virus, cytomegalovirus, varicella zoster virus, and Epstein-Barr virus in our po
24                        Herpes simplex virus, varicella zoster virus, and pseudorabies virus are neuro
25  2, human herpesvirus 6, human parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryp
26 antigens (vaccinia, measles, mumps, rubella, varicella-zoster virus, and Epstein-Barr virus) and nonr
27 Findings from skin biopsy, viral culture for varicella-zoster virus, and skin prick test to common fo
28                                              Varicella-zoster virus antigen was found in 45 of 70 GCA
29                                              Varicella-zoster virus antigen was frequently found in p
30 e whether herpes zoster antigen (also called varicella-zoster virus antigen) was detectable in tempor
31 gical boosting, through which reexposures to varicella-zoster virus are thought to reduce the individ
32 nstrate that childhood infections, including varicella zoster virus, are associated with an increased
33 h HLA-B27-associated (4460 [2465] pg/mL) and varicella-zoster virus-associated (5386 [1778] pg/mL) uv
34  for other infections (herpes simplex virus, varicella zoster virus, bacterial and fungal infections)
35 itis (AU), owing to either herpes simplex or varicella zoster virus, by using the Standardization of
36 clonal antibodies against a major antigen of varicella-zoster virus called gE.
37 se encephalitis virus, herpes simplex virus, varicella zoster virus, cytomegalovirus, dengue virus an
38  (HSV) and other alphaherpesviruses, such as varicella-zoster virus, depend upon the capacity to navi
39 ES by the corresponding region from ORF61 of varicella-zoster virus did not rescue ND10 fusion.
40  (as determined by testing lesions swabs for varicella zoster virus DNA by polymerase chain reaction)
41 ence of confirmed varicella (by detection of varicella zoster virus DNA or epidemiological link) from
42                                              Varicella zoster virus DNA was detected 2 months after t
43 bstrates for gene suicide therapy or as anti-varicella zoster virus drugs.
44 th HSE (p.Leu297Val) and 1 in a patient with varicella-zoster virus encephalitis (p.Leu199Phe).
45 the phenotypic spectrum of TLR3 mutations to varicella-zoster virus encephalitis and support the role
46                                              Varicella zoster virus encodes a thymidine kinase respon
47                                              Varicella zoster virus encodes an immediate-early (IE) p
48                            The ORF29 gene of varicella-zoster virus encodes a single-stranded DNA bin
49 deficiency virus (HIV)-herpes simplex virus, varicella zoster virus, Epstein-Barr virus (EBV), and cy
50 rpes zoster is a common late complication of varicella-zoster virus exposure and can be further compl
51 hus, a 30-h delay after death did not affect varicella-zoster virus expression in latently infected g
52                                   Studies of varicella-zoster virus gene expression during latency re
53                                          The varicella-zoster virus geometric mean titer (GMT) and ge
54 were randomized 1:1 to receive either HZ/su (varicella zoster virus glycoprotein E; AS01B Adjuvant Sy
55 ve (at months 0, 1, 3) three doses of 50 mug varicella-zoster virus glycoprotein E (gE) adjuvanted wi
56                                An adjuvanted varicella-zoster virus glycoprotein E (gE) subunit vacci
57 ubjects received 3 doses of HZ/su (50 microg varicella-zoster virus glycoprotein E [gE] combined with
58 g older adults, a subunit vaccine containing varicella-zoster virus glycoprotein E and the AS01B adju
59  zoster vaccine showed a greater increase in varicella-zoster virus gpELISA antibody compared with su
60 equences of wild-type and vaccine strains of varicella-zoster virus have been published and listed in
61 gnificant members of the herpesvirus family: varicella zoster virus, human cytomegalovirus, and Epste
62 transfected cells, whereas expression of the varicella-zoster virus ICP22 homolog, ORF63, does not.
63 rveillance, combined with information from a Varicella Zoster Virus Identification Program, which use
64 highly dependent on the host cell, we tested varicella zoster virus-infected cell lysates and clinica
65 cation) were associated with protection from varicella zoster virus infection (hazard ratio, 0.43; 95
66 contact dermatitis, infectious folliculitis, varicella zoster virus infection, fixed drug eruption, a
67 processes, including ubiquitin clearance and Varicella Zoster Virus infection.
68 or who had resided in a country with endemic varicella-zoster virus infection for 30 years or more we
69 erties that may favor reactivation of latent varicella-zoster virus infection.
70 nd increased susceptibility to bacterial and varicella zoster virus infections.
71 s related to herpes simplex virus type 1 and varicella-zoster virus, infects a broad host range of ma
72                                        While varicella-zoster virus is also insensitive to interferon
73  regulate infection of host cells.IMPORTANCE Varicella-zoster virus is an important human pathogen, w
74                          Because features of varicella-zoster virus latency are similar in primate an
75                              Similarly, in a varicella zoster virus lytic infection, HCF-1, Set1, and
76                                          The varicella-zoster virus major transactivator, IE62, can a
77                                          The varicella-zoster virus major transactivator, IE62, conta
78                             One patient with varicella zoster virus meningitis and acute GVHD had iC9
79                                    Available varicella-zoster virus models can be classified in 3 mai
80 es (parechovirus, dengue virus, Nipah virus, varicella-zoster virus, mumps virus, measles virus, lyss
81  = 60), followed by tuberculosis (n = 8) and varicella zoster virus (n = 7).
82 e fills a notable gap in our knowledge about varicella zoster virus neuronal transportation.
83 -coinfected children and were independent of varicella-zoster virus or herpes-simplex virus 1 coinfec
84 stituents at C6 exhibit remarkable anti-VZV (varicella-zoster virus) potency and selectivity, and ana
85       Herpes Simplex Virus types 1 and 2 and Varicella-zoster virus produce neurotropic infections su
86     Because there is no good animal model of varicella zoster virus reactivation from latency, this e
87 genes, Treponema pallidium, parvovirus, HIV, varicella zoster virus, Rubella, Cytomegalovirus, and He
88                 Among the 131 live births to varicella-zoster virus-seronegative women, there was no
89 in 50-59-year-old subjects were examined for varicella-zoster virus-specific antibody responses to va
90 1, CTLA-4, and TIM-3, whereas <2% of CMV- or varicella-zoster virus-specific CD4(+) T cells expressed
91  the change from baseline in IgG antibody to varicella-zoster virus-specific glycoproteins (gpELISA)
92 e-based site-directed mutagenesis studies of varicella zoster virus thymidine kinase (VZVTK).
93 lives ranging from an estimated 50 years for varicella-zoster virus to more than 200 years for other
94 ng heat-inactivated or replication-defective varicella-zoster virus to prevent HZ in immunocompromise
95                                              Varicella-zoster virus vaccination to prevent herpes zos
96 The continued success of the live attenuated varicella-zoster virus vaccine in preventing varicella-z
97                The licensed live, attenuated varicella-zoster virus vaccine prevents herpes zoster in
98 s HSV1 and HSV2 (also termed HHV1 and HHV2), varicella zoster virus (VZV or HHV3), EBV (HHV4), cytome
99 tients showed a decreased ability to control varicella zoster virus (VZV) and Epstein-Barr virus (EBV
100                                              Varicella zoster virus (VZV) and the two herpes simplex
101                                              Varicella zoster virus (VZV) antibody titers (measured b
102                                              Varicella zoster virus (VZV) antigen was found in all of
103                     Vasculopathies caused by varicella zoster virus (VZV) are indicative of a product
104                                      CMV and varicella zoster virus (VZV) are significant causes of m
105  response biomarkers measuring antibodies to varicella zoster virus (VZV) by glycoprotein-based enzym
106 lex virus types 1 (HSV-1) and 2 (HSV-2), and varicella zoster virus (VZV) by weekly polymerase chain
107 se of herpes zoster caused by the attenuated varicella zoster virus (VZV) contained in Zostavax in a
108 , or no history of zoster (group 3) revealed varicella zoster virus (VZV) DNA in saliva samples from
109 rs who were immunized with Zostavax revealed varicella zoster virus (VZV) DNA in swabs of skin inocul
110                            Herpesvirions and varicella zoster virus (VZV) DNA were recently reported
111                                              Varicella zoster virus (VZV) establishes latency in dors
112                                              Varicella zoster virus (VZV) establishes lifelong persis
113  as an alternative to sampling of rashes for varicella zoster virus (VZV) genotyping and further char
114 s positive for herpes simplex virus (HSV) or varicella zoster virus (VZV) in 79% to 100% of cases of
115 Clinical reports observe the reactivation of varicella zoster virus (VZV) in people who have recovere
116                          Reactivation of the varicella zoster virus (VZV) increases during aging.
117                                              Varicella zoster virus (VZV) infections are a relevant c
118 virus (CMV), herpes simplex virus (HSV), and varicella zoster virus (VZV) infections were monitored i
119                                              Varicella zoster virus (VZV) is a neurotropic alphaherpe
120                                              Varicella Zoster Virus (VZV) is the causative agent of v
121                                              Varicella zoster virus (VZV) is the etiological agent of
122                                              Varicella zoster virus (VZV) reactivation results in zos
123                    An adjuvanted recombinant varicella zoster virus (VZV) subunit vaccine is being de
124  zoster (HZ) cases may play a larger role in varicella zoster virus (VZV) transmission.
125                                              Varicella zoster virus (VZV) typically causes chickenpox
126 ne responses to a high-titer live attenuated varicella zoster virus (VZV) vaccine (zoster vaccine), w
127    Since the introduction of live attenuated varicella zoster virus (VZV) vaccine in 1995 there has b
128 s, granulomatous aortitis, and intracerebral varicella zoster virus (VZV) vasculopathy.
129 portion of HZ cases caused by vaccine-strain varicella zoster virus (VZV), assessed the positive pred
130 c primers to detect DNA from JC virus (JCV), varicella zoster virus (VZV), cytomegalovirus (CMV), Eps
131 on childhood disease, chicken pox, caused by varicella zoster virus (VZV), over an 11-y period.
132 erpesviruses, herpes simplex virus (HSV) and varicella zoster virus (VZV), results in the rapid accum
133  immunogenicity of live-attenuated Oka/Merck varicella zoster virus (VZV)-containing vaccine (hereaft
134                                     Boost of varicella zoster virus (VZV)-specific cellular immunity
135     We investigated the relationship between varicella zoster virus (VZV)-specific memory CD4(+) T ce
136  virus type 1 (HSV-1) and type 2 (HSV-2) and varicella zoster virus (VZV)-was determined in autonomic
137 ses with age, which leads to reactivation of varicella zoster virus (VZV).
138  1 and 2 and the sequence-divergent pathogen varicella zoster virus (VZV).
139                                              Varicella-zoster virus (VZV) activates the phosphatidyli
140 SV functioned as a monopartite NLS, while in varicella-zoster virus (VZV) activity required an adjace
141 are the main architectural contrasts between varicella-zoster virus (VZV) and herpes simplex virus (H
142 d the Us9 homologs from two human pathogens, varicella-zoster virus (VZV) and herpes simplex virus ty
143                                  Intraocular varicella-zoster virus (VZV) and HSV type 1 (HSV-1) infe
144                   Although the homologs from varicella-zoster virus (VZV) and human cytomegalovirus (
145 simplex virus type 1 (HSV-1) is conserved in varicella-zoster virus (VZV) and pseudorabies virus (PRV
146 ype 1 (EHV-1), pseudorabies virus (PRV), and varicella-zoster virus (VZV) and their subsequent functi
147             Simian varicella virus (SVV) and varicella-zoster virus (VZV) are closely related alphahe
148 gument proteins encoded by ORF11 and ORF9 of varicella-zoster virus (VZV) are conserved among all alp
149 mmunity and protects against reactivation of varicella-zoster virus (VZV) as HZ.
150     Infection of human neurons in vitro with varicella-zoster virus (VZV) at a low multiplicity of in
151 y, IDE has been proposed as the receptor for varicella-zoster virus (VZV) attachment.
152                           The attenuation of varicella-zoster virus (VZV) by Takahashi in 1974 was a
153      Serum was tested for antibodies against varicella-zoster virus (VZV) by use of the previously va
154                                              Varicella-zoster virus (VZV) causes chicken pox and shin
155                                              Varicella-zoster virus (VZV) causes chickenpox and react
156 highly infectious, human-restricted pathogen varicella-zoster virus (VZV) causes chickenpox and shing
157 ating VZV from clinical specimens.IMPORTANCE Varicella-zoster virus (VZV) causes chickenpox and shing
158                                              Varicella-zoster virus (VZV) causes chickenpox upon prim
159                                              Varicella-zoster virus (VZV) causes varicella and establ
160                                              Varicella-zoster virus (VZV) causes varicella, establish
161                                              Varicella-zoster virus (VZV) characteristically forms mu
162 y throughout the study and were analyzed for varicella-zoster virus (VZV) DNA by use of both qualitat
163                                    Wild-type varicella-zoster virus (VZV) DNA was identified in all 3
164  15, pain was scored and saliva examined for varicella-zoster virus (VZV) DNA.
165 The attenuated Oka vaccine (V-Oka) strain of varicella-zoster virus (VZV) effectively reduces disease
166       Herein we describe an episode of focal varicella-zoster virus (VZV) encephalitis in a healthy y
167         The open reading frame 10 (ORF10) of varicella-zoster virus (VZV) encodes a tegument protein
168                                              Varicella-zoster virus (VZV) establishes a lifelong late
169                  The neurotropic herpesvirus varicella-zoster virus (VZV) establishes a lifelong late
170                                              Varicella-zoster virus (VZV) establishes latency in huma
171 ts had similar magnitude memory responses to varicella-zoster virus (VZV) ex vivo restimulation measu
172                When grown in cultured cells, varicella-zoster virus (VZV) forms many aberrant light p
173                            Information about varicella-zoster virus (VZV) gB is limited, but homology
174 d c-Jun are important for transactivation of varicella-zoster virus (VZV) genes.
175                                              Varicella-zoster virus (VZV) glycoprotein E (gE) is esse
176                                              Varicella-zoster virus (VZV) glycoprotein E (gE) is requ
177 tive target for antiviral therapy.IMPORTANCE Varicella-zoster virus (VZV) has infected over 90% of pe
178          Mechanisms of neuronal infection by varicella-zoster virus (VZV) have been challenging to st
179 umoral and cell-mediated immune responses to varicella-zoster virus (VZV) have been evaluated after 1
180 (EBV) EB2, herpes simplex virus (HSV) ICP27, varicella-zoster virus (VZV) IE4/ORF4, and cytomegalovir
181                                          The varicella-zoster virus (VZV) IE62 protein is the major t
182                                              Varicella-zoster virus (VZV) immediate-early 63 protein
183 ction by enveloped, but not cell-associated, varicella-zoster virus (VZV) in a dose-dependent manner
184 ects immediate-early protein IE63 encoded by varicella-zoster virus (VZV) in the cytoplasm of product
185                      Productive infection of varicella-zoster virus (VZV) in vitro is restricted almo
186                                              Varicella-zoster virus (VZV) induces apoptosis in human
187      Previous studies have demonstrated that varicella-zoster virus (VZV) infection activates ERK1/2,
188                                              Varicella-zoster virus (VZV) infection causes varicella,
189                                      Primary varicella-zoster virus (VZV) infection in humans produce
190                                              Varicella-zoster virus (VZV) infection is usually mild i
191            Transcriptional changes following varicella-zoster virus (VZV) infection of cultured human
192 acaques (RMs) recapitulates the hallmarks of varicella-zoster virus (VZV) infection of humans, includ
193                                              Varicella-zoster virus (VZV) infection provides a valuab
194 he lytic, latent, and reactivating phases of varicella-zoster virus (VZV) infection were recapitulate
195 extensively studied the role of autophagy in varicella-zoster virus (VZV) infection, and have observe
196                                              Varicella-zoster virus (VZV) infections increasingly are
197                                              Varicella-zoster virus (VZV) is a common pathogen that c
198                                              Varicella-zoster virus (VZV) is a highly contagious agen
199                                              Varicella-zoster virus (VZV) is a highly neurotropic vir
200                                              Varicella-zoster virus (VZV) is a human alpha-herpesviru
201                                              Varicella-zoster virus (VZV) is a human alphaherpesvirus
202                                              Varicella-zoster virus (VZV) is a human alphaherpesvirus
203                                              Varicella-zoster virus (VZV) is a human neurotropic alph
204     The immediate early 62 protein (IE62) of varicella-zoster virus (VZV) is a major viral trans-acti
205                                              Varicella-zoster virus (VZV) is a member of the Herpesvi
206                                              Varicella-zoster virus (VZV) is a neurotropic alphaherpe
207                                              Varicella-zoster virus (VZV) is a ubiquitous pathogen th
208                                              Varicella-zoster virus (VZV) is a ubiquitous, highly cel
209                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
210                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
211                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
212                                              Varicella-zoster virus (VZV) is an alphaherpesvirus that
213                                              Varicella-zoster virus (VZV) is an extremely cell-associ
214                                              Varicella-zoster virus (VZV) is highly cell associated w
215 mary infection, latency, and reactivation by varicella-zoster virus (VZV) is incompletely understood.
216                                              Varicella-zoster virus (VZV) is renowned for its low tit
217                                              Varicella-zoster virus (VZV) is renowned for its very lo
218                                              Varicella-zoster virus (VZV) is the alphaherpesvirus tha
219                                              Varicella-zoster virus (VZV) is the causative agent of b
220                                              Varicella-zoster virus (VZV) is the causative agent of c
221                                              Varicella-zoster virus (VZV) is the etiological agent of
222                                          The varicella-zoster virus (VZV) major transactivator, IE62,
223       We report a case of AIDS presenting as varicella-zoster virus (VZV) meningomyeloradiculitis ass
224                      To efficiently generate varicella-zoster virus (VZV) mutants, we inserted a bact
225 f herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) on 695 consecutive cutaneou
226                                              Varicella-zoster virus (VZV) open reading frame (ORF) 63
227                                              Varicella-zoster virus (VZV) open reading frame (ORF) 63
228                                              Varicella-zoster virus (VZV) open reading frame 10 (ORF1
229        Based on comparative genome analyses, varicella-zoster virus (VZV) open reading frame 23 (ORF2
230                                          The varicella-zoster virus (VZV) open reading frame 54 (ORF5
231                                              Varicella-zoster virus (VZV) open reading frame 61 (ORF6
232                                Three loci in varicella-zoster virus (VZV) open reading frame 62 (ORF6
233                                              Varicella-zoster virus (VZV) open reading frame 63 (ORF6
234                                              Varicella-zoster virus (VZV) open reading frame 63 (ORF6
235                                              Varicella-zoster virus (VZV) open reading frame 66 (ORF6
236                        We show here that the varicella-zoster virus (VZV) open reading frame 66 (ORF6
237                 The gene cluster composed of varicella-zoster virus (VZV) open reading frame 9 (ORF9)
238 f transcripts corresponding to all 68 unique varicella-zoster virus (VZV) open reading frames (ORFs)
239                                              Varicella-zoster virus (VZV) ORF29 encodes the viral sin
240                                          The varicella-zoster virus (VZV) ORF61 protein is necessary
241                                          The varicella-zoster virus (VZV) ORF62/63 intergenic region
242                      The architecture of the varicella-zoster virus (VZV) origin of DNA replication (
243                                          The varicella-zoster virus (VZV) origin of DNA replication (
244     In this report, we show that ORF61p, the varicella-zoster virus (VZV) ortholog of ICP0, does not
245 ced syncytium formation, a characteristic of varicella-zoster virus (VZV) pathology in skin and senso
246 this minireview is to provide an overview of varicella-zoster virus (VZV) phylogenetics and phylogeog
247                                  We examined varicella-zoster virus (VZV) polymerase chain reaction (
248 er acyclovir prophylaxis should be given for varicella-zoster virus (VZV) prophylaxis after hematopoi
249                                  Analyses of varicella-zoster virus (VZV) protein expression during l
250                  ORF66p, a virion-associated varicella-zoster virus (VZV) protein, is a member of a c
251 nt is associated with increased incidence of varicella-zoster virus (VZV) reactivation in patients wi
252 IMPORTANCE The neurological damage caused by varicella-zoster virus (VZV) reactivation is commonly ma
253 a total of five major genotypes among the 22 varicella-zoster virus (VZV) strains or isolates for whi
254                                              Varicella-zoster virus (VZV) T-cell responses by interfe
255                                              Varicella-zoster virus (VZV) T-cell-mediated immunity (V
256                                          The varicella-zoster virus (VZV) terminase components (pORF2
257 r herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) to the BD Max system by usi
258                                   Studies of varicella-zoster virus (VZV) tropism for T cells support
259                                   Although a varicella-zoster virus (VZV) vaccine has been used for m
260 75 years) immunized with the live-attenuated varicella-zoster virus (VZV) vaccine.
261  of a high-potency live-attenuated Oka/Merck varicella-zoster virus (VZV) vaccine.
262                                              Varicella-zoster virus (VZV) vasculopathy produces strok
263 ish adverse events associated with wild-type varicella-zoster virus (VZV) versus those associated wit
264                                              Varicella-zoster virus (VZV), a double-stranded DNA alph
265 igated during the entire infectious cycle of varicella-zoster virus (VZV), a human herpesvirus.
266     The immediate early 62 protein (IE62) of varicella-zoster virus (VZV), a major viral trans-activa
267                       Primary infection with varicella-zoster virus (VZV), a neurotropic alphaherpesv
268          Regulation of gene transcription in varicella-zoster virus (VZV), a ubiquitous human neurotr
269 1) and pseudorabies virus (PRV) and ORF66 in varicella-zoster virus (VZV), affects several viral and
270                                              Varicella-zoster virus (VZV), an alphaherpesvirus restri
271                          Here we report that varicella-zoster virus (VZV), an alphaherpesvirus that i
272 pear healthy at 2 weeks after infection with varicella-zoster virus (VZV), and the cell culture mediu
273 ovirus, herpes simplex virus type 1 (HSV-1), varicella-zoster virus (VZV), and West Nile virus (WNV).
274 sviruses, herpes simplex virus 1 (HSV-1) and varicella-zoster virus (VZV), confirmed the expression o
275 ults for herpes simplex virus 1/2 (HSV-1/2), varicella-zoster virus (VZV), cytomegalovirus (CMV), or
276     In this study, quantitative PCR detected varicella-zoster virus (VZV), herpes simplex virus 1 (HS
277                      Levels of inhibition of varicella-zoster virus (VZV), human cytomegalovirus (HCM
278                                              Varicella-zoster virus (VZV), in both wild-type and live
279                                              Varicella-zoster virus (VZV), of the family Alphaherpesv
280 showed cytopathic changes, but HSV-1, unlike varicella-zoster virus (VZV), only rarely infected satel
281                                         Like varicella-zoster virus (VZV), simian varicella virus (SV
282 cilitate the generation of mutant viruses of varicella-zoster virus (VZV), the agent causing varicell
283 nation was more likely to identify wild-type varicella-zoster virus (VZV), whereas the presence of Ok
284                                           In varicella-zoster virus (VZV)-infected primary human brai
285  the risk of herpes zoster (HZ), we compared varicella-zoster virus (VZV)-specific and nonspecific T-
286                                              Varicella-zoster virus (VZV)-specific cell-mediated immu
287 n association with an age-related decline in varicella-zoster virus (VZV)-specific cell-mediated immu
288 udy were to evaluate the association between varicella-zoster virus (VZV)-specific humoral and cell-m
289                                   To measure varicella-zoster virus (VZV)-specific immune responses u
290 lication that can occur with reactivation of varicella-zoster virus (VZV).
291 rus (CMV), herpes simplex viruses (HSV), and varicella-zoster virus (VZV).
292 rpes zoster caused by reactivation of latent Varicella-Zoster virus (VZV).
293 ly recognized component of the life cycle of varicella-zoster virus (VZV).
294  potency against hepatitis B virus (HBV) and varicella-zoster virus (VZV).
295 itive GCA is associated with TA infection by varicella-zoster virus (VZV).
296               None have yet been reported in varicella-zoster virus (VZV; also known as human herpesv
297 onal and pathogen-specific stimulation (with varicella-zoster virus [VZV] and cytomegalovirus [CMV]).
298                                    HHV-1 and Varicella-Zoster virus were detected only twice and HHV-
299 ty for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were studied in 1079 6-year-old c
300 ncing to identify nosocomial transmission of varicella-zoster virus with fatal outcome.

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