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1 ese is also activated by the closely related varicella zoster virus.
2 ew mutations or recombination with wild-type Varicella zoster virus.
3 2, cytomegalovirus, Epstein-Barr virus, and varicella zoster virus.
4 losely related to herpes simplex viruses and varicella-zoster virus.
5 G synthesis, and elevated antibody titers to varicella-zoster virus.
6 d, for example, in relation to pertussis and varicella-zoster virus.
7 ation were detected in OPV, mumps virus, and varicella-zoster virus.
8 uses herpes simplex virus type 1 (HSV-1) and varicella-zoster virus.
9 known as breakthrough disease, if exposed to varicella-zoster virus.
10 erpesviruses herpes simplex virus type 1 and varicella-zoster virus.
11 nuated varicella vaccine virus and wild-type varicella-zoster virus.
12 gens herpes simplex virus type 1 (HSV-1) and varicella-zoster virus.
13 1), P. jirovecii pneumonia (1.77; .42-7.47), varicella-zoster virus (1.51; .71-3.22), as well as over
14 -Barr virus, 3%; herpes simplex virus 1, 3%; varicella zoster virus, 3%; HHV7, 2%; and herpes simplex
16 RN, PCR-positive for herpes simplex virus or varicella zoster virus and evaluated between January 200
17 genes of both herpes simplex virus (HSV) and varicella zoster virus and functions, in part, by coupli
18 beyond CMV to other herpes viruses, such as varicella zoster virus and possibly Epstein-Barr virus.
19 ld decrease external boosting of immunity to varicella zoster virus and thereby increase incidence of
20 cropsy of two monkeys inoculated with simian varicella-zoster virus and euthanized 117 days later.
21 ia in adults includes common agents, such as varicella-zoster virus and influenza virus, as well as r
22 ate changes in the molecular epidemiology of varicella-zoster virus and the effect of immunization wi
23 ty of herpes simplex virus, cytomegalovirus, varicella zoster virus, and Epstein-Barr virus in our po
25 2, human herpesvirus 6, human parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryp
26 antigens (vaccinia, measles, mumps, rubella, varicella-zoster virus, and Epstein-Barr virus) and nonr
27 Findings from skin biopsy, viral culture for varicella-zoster virus, and skin prick test to common fo
30 e whether herpes zoster antigen (also called varicella-zoster virus antigen) was detectable in tempor
31 gical boosting, through which reexposures to varicella-zoster virus are thought to reduce the individ
32 nstrate that childhood infections, including varicella zoster virus, are associated with an increased
33 h HLA-B27-associated (4460 [2465] pg/mL) and varicella-zoster virus-associated (5386 [1778] pg/mL) uv
34 for other infections (herpes simplex virus, varicella zoster virus, bacterial and fungal infections)
35 itis (AU), owing to either herpes simplex or varicella zoster virus, by using the Standardization of
37 se encephalitis virus, herpes simplex virus, varicella zoster virus, cytomegalovirus, dengue virus an
38 (HSV) and other alphaherpesviruses, such as varicella-zoster virus, depend upon the capacity to navi
40 (as determined by testing lesions swabs for varicella zoster virus DNA by polymerase chain reaction)
41 ence of confirmed varicella (by detection of varicella zoster virus DNA or epidemiological link) from
45 the phenotypic spectrum of TLR3 mutations to varicella-zoster virus encephalitis and support the role
49 deficiency virus (HIV)-herpes simplex virus, varicella zoster virus, Epstein-Barr virus (EBV), and cy
50 rpes zoster is a common late complication of varicella-zoster virus exposure and can be further compl
51 hus, a 30-h delay after death did not affect varicella-zoster virus expression in latently infected g
54 were randomized 1:1 to receive either HZ/su (varicella zoster virus glycoprotein E; AS01B Adjuvant Sy
55 ve (at months 0, 1, 3) three doses of 50 mug varicella-zoster virus glycoprotein E (gE) adjuvanted wi
57 ubjects received 3 doses of HZ/su (50 microg varicella-zoster virus glycoprotein E [gE] combined with
58 g older adults, a subunit vaccine containing varicella-zoster virus glycoprotein E and the AS01B adju
59 zoster vaccine showed a greater increase in varicella-zoster virus gpELISA antibody compared with su
60 equences of wild-type and vaccine strains of varicella-zoster virus have been published and listed in
61 gnificant members of the herpesvirus family: varicella zoster virus, human cytomegalovirus, and Epste
62 transfected cells, whereas expression of the varicella-zoster virus ICP22 homolog, ORF63, does not.
63 rveillance, combined with information from a Varicella Zoster Virus Identification Program, which use
64 highly dependent on the host cell, we tested varicella zoster virus-infected cell lysates and clinica
65 cation) were associated with protection from varicella zoster virus infection (hazard ratio, 0.43; 95
66 contact dermatitis, infectious folliculitis, varicella zoster virus infection, fixed drug eruption, a
68 or who had resided in a country with endemic varicella-zoster virus infection for 30 years or more we
71 s related to herpes simplex virus type 1 and varicella-zoster virus, infects a broad host range of ma
73 regulate infection of host cells.IMPORTANCE Varicella-zoster virus is an important human pathogen, w
80 es (parechovirus, dengue virus, Nipah virus, varicella-zoster virus, mumps virus, measles virus, lyss
83 -coinfected children and were independent of varicella-zoster virus or herpes-simplex virus 1 coinfec
84 stituents at C6 exhibit remarkable anti-VZV (varicella-zoster virus) potency and selectivity, and ana
86 Because there is no good animal model of varicella zoster virus reactivation from latency, this e
87 genes, Treponema pallidium, parvovirus, HIV, varicella zoster virus, Rubella, Cytomegalovirus, and He
89 in 50-59-year-old subjects were examined for varicella-zoster virus-specific antibody responses to va
90 1, CTLA-4, and TIM-3, whereas <2% of CMV- or varicella-zoster virus-specific CD4(+) T cells expressed
91 the change from baseline in IgG antibody to varicella-zoster virus-specific glycoproteins (gpELISA)
93 lives ranging from an estimated 50 years for varicella-zoster virus to more than 200 years for other
94 ng heat-inactivated or replication-defective varicella-zoster virus to prevent HZ in immunocompromise
96 The continued success of the live attenuated varicella-zoster virus vaccine in preventing varicella-z
98 s HSV1 and HSV2 (also termed HHV1 and HHV2), varicella zoster virus (VZV or HHV3), EBV (HHV4), cytome
99 tients showed a decreased ability to control varicella zoster virus (VZV) and Epstein-Barr virus (EBV
105 response biomarkers measuring antibodies to varicella zoster virus (VZV) by glycoprotein-based enzym
106 lex virus types 1 (HSV-1) and 2 (HSV-2), and varicella zoster virus (VZV) by weekly polymerase chain
107 se of herpes zoster caused by the attenuated varicella zoster virus (VZV) contained in Zostavax in a
108 , or no history of zoster (group 3) revealed varicella zoster virus (VZV) DNA in saliva samples from
109 rs who were immunized with Zostavax revealed varicella zoster virus (VZV) DNA in swabs of skin inocul
113 as an alternative to sampling of rashes for varicella zoster virus (VZV) genotyping and further char
114 s positive for herpes simplex virus (HSV) or varicella zoster virus (VZV) in 79% to 100% of cases of
115 Clinical reports observe the reactivation of varicella zoster virus (VZV) in people who have recovere
118 virus (CMV), herpes simplex virus (HSV), and varicella zoster virus (VZV) infections were monitored i
126 ne responses to a high-titer live attenuated varicella zoster virus (VZV) vaccine (zoster vaccine), w
127 Since the introduction of live attenuated varicella zoster virus (VZV) vaccine in 1995 there has b
129 portion of HZ cases caused by vaccine-strain varicella zoster virus (VZV), assessed the positive pred
130 c primers to detect DNA from JC virus (JCV), varicella zoster virus (VZV), cytomegalovirus (CMV), Eps
132 erpesviruses, herpes simplex virus (HSV) and varicella zoster virus (VZV), results in the rapid accum
133 immunogenicity of live-attenuated Oka/Merck varicella zoster virus (VZV)-containing vaccine (hereaft
135 We investigated the relationship between varicella zoster virus (VZV)-specific memory CD4(+) T ce
136 virus type 1 (HSV-1) and type 2 (HSV-2) and varicella zoster virus (VZV)-was determined in autonomic
140 SV functioned as a monopartite NLS, while in varicella-zoster virus (VZV) activity required an adjace
141 are the main architectural contrasts between varicella-zoster virus (VZV) and herpes simplex virus (H
142 d the Us9 homologs from two human pathogens, varicella-zoster virus (VZV) and herpes simplex virus ty
145 simplex virus type 1 (HSV-1) is conserved in varicella-zoster virus (VZV) and pseudorabies virus (PRV
146 ype 1 (EHV-1), pseudorabies virus (PRV), and varicella-zoster virus (VZV) and their subsequent functi
148 gument proteins encoded by ORF11 and ORF9 of varicella-zoster virus (VZV) are conserved among all alp
150 Infection of human neurons in vitro with varicella-zoster virus (VZV) at a low multiplicity of in
153 Serum was tested for antibodies against varicella-zoster virus (VZV) by use of the previously va
156 highly infectious, human-restricted pathogen varicella-zoster virus (VZV) causes chickenpox and shing
157 ating VZV from clinical specimens.IMPORTANCE Varicella-zoster virus (VZV) causes chickenpox and shing
162 y throughout the study and were analyzed for varicella-zoster virus (VZV) DNA by use of both qualitat
165 The attenuated Oka vaccine (V-Oka) strain of varicella-zoster virus (VZV) effectively reduces disease
171 ts had similar magnitude memory responses to varicella-zoster virus (VZV) ex vivo restimulation measu
177 tive target for antiviral therapy.IMPORTANCE Varicella-zoster virus (VZV) has infected over 90% of pe
179 umoral and cell-mediated immune responses to varicella-zoster virus (VZV) have been evaluated after 1
180 (EBV) EB2, herpes simplex virus (HSV) ICP27, varicella-zoster virus (VZV) IE4/ORF4, and cytomegalovir
183 ction by enveloped, but not cell-associated, varicella-zoster virus (VZV) in a dose-dependent manner
184 ects immediate-early protein IE63 encoded by varicella-zoster virus (VZV) in the cytoplasm of product
187 Previous studies have demonstrated that varicella-zoster virus (VZV) infection activates ERK1/2,
192 acaques (RMs) recapitulates the hallmarks of varicella-zoster virus (VZV) infection of humans, includ
194 he lytic, latent, and reactivating phases of varicella-zoster virus (VZV) infection were recapitulate
195 extensively studied the role of autophagy in varicella-zoster virus (VZV) infection, and have observe
204 The immediate early 62 protein (IE62) of varicella-zoster virus (VZV) is a major viral trans-acti
215 mary infection, latency, and reactivation by varicella-zoster virus (VZV) is incompletely understood.
225 f herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) on 695 consecutive cutaneou
238 f transcripts corresponding to all 68 unique varicella-zoster virus (VZV) open reading frames (ORFs)
244 In this report, we show that ORF61p, the varicella-zoster virus (VZV) ortholog of ICP0, does not
245 ced syncytium formation, a characteristic of varicella-zoster virus (VZV) pathology in skin and senso
246 this minireview is to provide an overview of varicella-zoster virus (VZV) phylogenetics and phylogeog
248 er acyclovir prophylaxis should be given for varicella-zoster virus (VZV) prophylaxis after hematopoi
251 nt is associated with increased incidence of varicella-zoster virus (VZV) reactivation in patients wi
252 IMPORTANCE The neurological damage caused by varicella-zoster virus (VZV) reactivation is commonly ma
253 a total of five major genotypes among the 22 varicella-zoster virus (VZV) strains or isolates for whi
257 r herpes simplex virus 1 (HSV-1), HSV-2, and varicella-zoster virus (VZV) to the BD Max system by usi
263 ish adverse events associated with wild-type varicella-zoster virus (VZV) versus those associated wit
266 The immediate early 62 protein (IE62) of varicella-zoster virus (VZV), a major viral trans-activa
269 1) and pseudorabies virus (PRV) and ORF66 in varicella-zoster virus (VZV), affects several viral and
272 pear healthy at 2 weeks after infection with varicella-zoster virus (VZV), and the cell culture mediu
273 ovirus, herpes simplex virus type 1 (HSV-1), varicella-zoster virus (VZV), and West Nile virus (WNV).
274 sviruses, herpes simplex virus 1 (HSV-1) and varicella-zoster virus (VZV), confirmed the expression o
275 ults for herpes simplex virus 1/2 (HSV-1/2), varicella-zoster virus (VZV), cytomegalovirus (CMV), or
276 In this study, quantitative PCR detected varicella-zoster virus (VZV), herpes simplex virus 1 (HS
280 showed cytopathic changes, but HSV-1, unlike varicella-zoster virus (VZV), only rarely infected satel
282 cilitate the generation of mutant viruses of varicella-zoster virus (VZV), the agent causing varicell
283 nation was more likely to identify wild-type varicella-zoster virus (VZV), whereas the presence of Ok
285 the risk of herpes zoster (HZ), we compared varicella-zoster virus (VZV)-specific and nonspecific T-
287 n association with an age-related decline in varicella-zoster virus (VZV)-specific cell-mediated immu
288 udy were to evaluate the association between varicella-zoster virus (VZV)-specific humoral and cell-m
297 onal and pathogen-specific stimulation (with varicella-zoster virus [VZV] and cytomegalovirus [CMV]).
299 ty for CMV, EBV, herpes-simplex virus 1, and varicella-zoster virus were studied in 1079 6-year-old c
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