ライフサイエンスコーパス: varicosity

1 DCS (this ratio is 0.50 for axons, 0.55 for varicosities).

2 , largely because of reduced addition of new varicosities.

3 r and these depolarizations propagate to the varicosities.

4 were clustered opposite synapsin-containing varicosities.

5 vidence for functional NMDARs in basket cell varicosities.

6 ability and causing calcium influx at axonal varicosities.

7 tic specializations opposite to their axonal varicosities.

8 ong fibers characterized by regularly spaced varicosities.

9 n, vamp2, was present in a fraction of those varicosities.

10 GABA(B) receptors in a subpopulation of its varicosities.

11 ication of SIV adjacent to catecholaminergic varicosities.

12 he numerical density of the nLOT cholinergic varicosities.

13 ning-related formation of new sensory neuron varicosities.

14 s in retinal arbors with smaller than normal varicosities.

15 totagmin and SNAP-25 were localized to nerve varicosities.

16 of excitatory-like, presumably glutamatergic varicosities.

17 h or sparsely spiny, but never bear distinct varicosities.

18 ific clusters of dense terminal branches and varicosities.

19 d convergent synapses from TH-immunoreactive varicosities.

20 idine that measure cardiac sympathetic nerve varicosities.

21 rons and vasoactive intestinal peptide (VIP) varicosities.

22 retracting synaptic branches and presynaptic varicosities.

23 re accompanied by the emergence of dendritic varicosities.

24 2 weeks without changing the total number of varicosities.

25 and a slight decrease in the total number of varicosities.

26 within EYFP-positive or EYFP-negative axonal varicosities.

27 n the superficial layers of area TE and 8.74 varicosities/100 microm in the deep layers.

28 Labeled axons demonstrated 11.67 varicosities/100 microm on average in the superficial la

29 In area V1 we observed 8.24 varicosities/100 microm.

30 ENK colocalized with VGLUT2 in up to 77% of varicosities (17 +/- 21%, n = 21 neurons).

31 7% of somatostatin (SOM)-immunoreactive (IR) varicosities; 20 +/- 4.3% of substance P (SP)-IR varicos

32 holine transporter-immunoreactive (VAChT-IR) varicosities (80% +/- 1.7%, n = 4, P < 0.001) contained

33 sicular acetylcholine transporter (VAChT)-IR varicosities (88 +/- 3%, P < 0.001).

34 Among alpha-synuclein-IR varicosities, 96% +/- 0.9%, 99% +/- 0.6%, 83% +/- 1.9%,

35 tagmin-1-, synaptobrevin-2-, and CSPalpha-IR varicosities, 98% +/- 0.7%, 96% +/- 0.7%, 88% +/- 1.6%,

36 in (5HT) triggered growth of new presynaptic varicosities, a synaptic mechanism of long-term sensitiz

37 ter fibers giving rise to numerous spherical varicosities abutting type II hair cells and afferent pr

38 arget-dependent increases in axon growth and varicosities accompany the formation of functional synap

39 ated synapses at 24 hr, whereas newly formed varicosities account for 68%.

40 ould be differences in the density of axonal varicosities across regions, and that these differences,

41 packeted release of ATP from nerve terminal varicosities acting at postjunctional P2X receptors.

42 ggest that (i) ATP released from sympathetic varicosities activates the initial, transient, contracti

43 es in synaptic structure between cholinergic varicosities activating alpha7 and non-alpha7 classes of

44 ities along dendrites, decreases the size of varicosities after sublethal NMDA exposure, and protects

45 neurons with varicosities and the number of varicosities along dendrites, decreases the size of vari

46 More than half of presynaptic varicosities along its cholinergic axons make traditiona

47 he spinal cord, an increase in 5-HT-positive varicosities along motor-associated cholinergic neurons

48 imes upon entering the bladder and developed varicosities along their axon terminal endings.

49 toward the outer retina, exhibiting numerous varicosities along their course.

50 ns and the relative distribution of efferent varicosities among hair cells and afferents are also int

51 cosities; 20 +/- 4.3% of substance P (SP)-IR varicosities and 9 +/- 1.3% vasoactive intestinal polype

52 n of the newly synthesized sensorin from the varicosities and activation of the autocrine responses o

53 have a high number of densely packed, large varicosities and an extensive rostrocaudal (two or three

54 Multifocal axonal varicosities and axonal loss were found in deep cortex a

55 ng extensive process outgrowth with numerous varicosities and expression of neuronal and synaptic mar

56 aptic vesicle movement into previously empty varicosities and formation of new varicosities-may deter

57 and close appositions between PNMT-ir axonal varicosities and orexin-ir profiles were observed.

58 othalamic projections contain small synaptic varicosities and other features that resemble the modula

59 synapses between presynaptic climbing fiber varicosities and postsynaptic Purkinje cell spines and e

60 th of new neuritic processes and presynaptic varicosities and retracted growth at the highest LPA con

61 occur via a loose association between nerve varicosities and smooth muscle cells.

62 tion in the number of inhibitory perisomatic varicosities and synaptic GAD65/67 immunoreactivities in

63 ccompanied by the maintenance of presynaptic varicosities and target-dependent regulation of mRNA dis

64 c strength and enhanced the formation of new varicosities and target-dependent regulation of mRNA dis

65 We also found that the distribution of axon varicosities and terminal field locations show substanti

66 Axons from the medial CeA formed numerous varicosities and terminals enveloping the HSD2 neurons.

67 own increases the percentage of neurons with varicosities and the number of varicosities along dendri

68 laced in a region of the muscle containing a varicosity and held at a potential sufficient to oxidize

69 dendritic damage (characterized by swellings/varicosities), and dysregulated neuronal excitability (d

70 sis-dependent LTF, growth of new presynaptic varicosities, and activation of MAPK and its translocati

71 s were associated with enkephalin-containing varicosities, and enkephalin-induced clathrin- and dynam

72 e neuronal activities including MIP/SIFamide varicosities, and leads the contraction of myoepithelial

73 aintenance of synaptic efficacy, presynaptic varicosities, and mRNA distributions.

74 immunoreactivity was observed in processes, varicosities, and neuronal cell bodies of the olfactory

75 c filopodia, elaboration of undifferentiated varicosities, and potentiation of spontaneous release fr

76 the growth and formation of new presynaptic varicosities, and the target-dependent regulation of mRN

77 ly separated by at least 1.2 mum from NET-ir varicosities, and the two profile types were not seen to

78 ehavioral training), approximately 10% of PF varicosities appeared and disappeared over a period of 2

79 Importantly, EM2 and glutamate-containing varicosities appose spinal neurons that express MOR alon

80 d the nerve terminals change similarly to be varicosities apposed to these islands.

81 ansients evoked by release of ATP from nerve varicosities are elementary signals in the process of ne

82 We further show that axonal varicosities are induced by persistent Ca(2+) increase,

83 f long-term memory during which newly formed varicosities are labile and require sustained CPEB-depen

84 eflect changes in synaptic density, but axon varicosities are likely to be the most sensitive anatomi

85 These isolated varicosities are not found along particular topological

86 Some DLCST axon arbors with varicosities are seen near large neurons in the ventral

87 us but not in dendrites, indicating that the varicosities are sites of dopamine release.

88 These varicosities are thought to form largely nonjunctional-t

89 ermore, the number of synaptic-like terminal varicosities around PNs was decreased.

90 identical pathologic increases in dendritic varicosities as seen in Tat transgenic mice in vivo.

91 ar structures within axonal growth cones and varicosities as well as at axonal branch points in cultu

92 ine, were registered at noradrenergic axonal varicosities as well as at cell bodies.

93 onin appear to be selective for serotonergic varicosities, as dopaminergic and corazonergic varicosit

94 arkers accumulated abnormally in YFP-labeled varicosities associated with neuritic plaques.

95 rain, we find evidence for avoidance between varicosities at distances lower than 1.75 microm.

96 al neurons induced the formation of numerous varicosity-bearing tortuous processes, as well as the co

97 ellular injections of Neurobiotin to examine varicosities belonging to heart excitor (HE) neurons on

98 tic neuron, being present in spine-targeting varicosities but distinctly absent from those synapsing

99 ed by targeted retraction of the 5HT-induced varicosities but, rather, by an apparently arbitrary ret

100 presynaptic membrane were absent from DA-IPC varicosities, but the vesicular SNARE protein, vamp2, wa

101 rons from 6.0 +/- 0.9 to 2.3 +/- 0.4 CART-IR varicosities/cell.

102 itation in older kittens, corticospinal axon varicosities colocalize synaptophysin like adults, sugge

103 microscopic examination of a sample of these varicosities confirmed that labeled boutons formed synap

104 s that 5-HT activates ApCdc42 in a subset of varicosities contacting the postsynaptic motor neuron an

105 1 +/- 0.3% of biotinamide-labeled extrinsic varicosities contained alpha-synuclein-immunoreactivity

106 Within RTN, 51% of BDA-labelled axonal varicosities contained detectable levels of vesicular gl

107 d contorted or fragmented axons with swollen varicosities containing alphaSyn and Tau.

108 However, densities of terminal varicosities correlate with recovery of swimming behavio

109 icroscopy, we determined that mCherry-VGluT2 varicosities correspond to axon terminals, forming asymm

110 otential-evoked calcium transients in axonal varicosities, demonstrating the effectiveness of activat

111 Noticeably, the VGluT2+ and VIAAT+ varicosity density in Mo7 is 5-fold higher than in Mo5 a

112 excess exogenous serotonin decreases native varicosity density in older larvae, and these acute effe

113 aricosities increase substantially while the varicosity density remains relatively constant.

114 rotonin is an autoregulator for serotonergic varicosity density.

115 n terminal and preterminal branching, and in varicosity density.

116 mation is complete, motor neuron presynaptic varicosities develop into large bulbous protrusions that

117 rthermore, calcium indicators in basket cell varicosities did not report any change in intracellular

118 TRPCs) was similarly absent, although DA-IPC varicosities did show ryanodine receptor immunoreactivit

119 These anterogradely labeled varicosities displayed synaptophysin immunoreactivity, i

120 y of substance P (Sub P) to induce dendritic varicosities (DVs) or beads in neurons of the rostral ve

121 hese afferent axons provide numerous swollen varicosities, each presynaptic to many small profiles, a

122 k LTF and the rapid synthesis of sensorin at varicosities even in the absence of sensory neuron cell

123 the largest number of anterogradely labeled varicosities followed by vesicular GABA transporter (VGA

124 Focal swelling or varicosity formation in dendrites and loss of dendritic

125 synapse maturation and curtailed growth and varicosity formation of sensory neurons contacting L7, b

126 own that microtubule dynamics play a role in varicosity formation, very little is known about the pro

127 env, gag, and nef RNA, and catecholaminergic varicosities from the ANS were mapped by sucrose phospha

128 Unlike mature boutons, new varicosities have synaptic vesicles which are distribute

129 tween the absolute density of axons and axon varicosities; however, the ratio measures are strongly c

130 Among 1,136 DbH-positive varicosities identified within the parvocellular PVN in

131 ddressed was whether distally located DA-IPC varicosities, identified by tyrosine hydroxylase (TH) im

132 mate the number of motoneurons as well as of varicosities immunopositive for glutamate (VGluT1+, VGlu

133 remarkably high density of axons and axonal varicosities immunoreactive for serotonin (5-HT) and ore

134 erve endings from mouse cortex and in intact varicosities in a neuronal cell line using fluorescence

135 se of NET in endosomes of single boutons and varicosities in a Rab11-dependent manner.

136 ined that at least 55% of the tracer-labeled varicosities in areas TE and V1 colocalized synaptophysi

137 s of the hypothalamus (PVH), with fibers and varicosities in close apposition to a subset of melanoco

138 at the release face of bouton-like synaptic varicosities in contact with muscle cells, and lowest in

139 the number and size of glutamatergic axonal varicosities in cortical cultures.

140 d stimulation of glutamate receptors induces varicosities in dendrites but not in axons.

141 % vasoactive intestinal polypeptide (VIP)-IR varicosities in guinea pig rectal myenteric ganglia.

142 We found more 5-HT-positive varicosities in lamina X adjacent to central canal clust

143 71 +/- 0.8% of VAChT-IR varicosities in myenteric ganglia of human colon were al

144 tic bouton number and the ability to bud new varicosities in response to acute neuronal stimulation.

145 versican promotes enlargement of presynaptic varicosities in retinal axons.

146 We found that CHT-immunoreactive axon varicosities in the AVN displayed a smaller cross-sectio

147 that mechanical stress specifically induces varicosities in the axons but not the dendrites of centr

148 be the neuropil distribution of serotonergic varicosities in the brain and ventral nerve cord (VNC) o

149 studies of individual central vagal afferent varicosities in the caudal brainstem slice preparation s

150 nch contains large numbers of vesicles, with varicosities in the close vicinity of Purkinje dendrites

151 dritic arborization, and extensive dendritic varicosities in the cortical neurons of CM-infected brai

152 fixatives induce the formation of bead-like varicosities in the dendrites and axons of brain and spi

153 release from exocytosis events at individual varicosities in the Drosophila larval system by amperome

154 een in fiber staining which may reflect true varicosities in the fiber or simply varying densities of

155 CPA resulted in numerous labeled fibers with varicosities in the ipsilateral subnucleus reticularis d

156 m to examine the development of serotonergic varicosities in the larval CNS.

157 We found that TH immunoreactive varicosities in the outer retina possessed vesicular mon

158 vity is present in small diameter fibers and varicosities in the periphery of nerves located in the a

159 ced the appearance of NGF-receptor-IR axonal varicosities in the rat medial septum.

160 escence revealed a mixture of fine and thick varicosities in the SAP + IG but only fine fibers in con

161 the turnover of dendritic spines and axonal varicosities in the somatosensory cortex of mice lacking

162 findings, the density of 5-HT-immunoreactive varicosities in the superficial dorsal horn of the spina

163 OP) on substance P (SP)-immunoreactive (-ir) varicosities in the superficial dorsal horn of the spina

164 eurites/terminal swellings and vesicle-laden varicosities in the synaptic neuropil.

165 on between aggression and the number of axon varicosities in the telencephalic region proposed to be

166 the laminar distribution of labelled axonal varicosities in the terminal fields indicated the existe

167 Moreover, the number of new mossy fiber varicosities in these parts of the cerebellar nuclei is

168 ble, consistent with the formation of axonal varicosities in vivo induced by mechanical impact in a m

169 real density of cholinergic terminals (fiber varicosities) in the dentate gyrus.

170 Terminal varicosities increase in density rostral to the lesion s

171 e of the neuropil and number of serotonergic varicosities increase substantially while the varicosity

172 ound that the production of peptide-positive varicosities increased considerably during the last week

173 ppearance, a few fibers with en passant type varicosities (indicating synapses) were observed in the

174 fibers exhibited excitatory, vGLUT2-positive varicosities, indicating their synaptic integration into

175 ve retraction of newly formed sensory neuron varicosities induced by 5-HT.

176 the associated growth of new sensory neuron varicosities induced by repeated pulses of serotonin (5-

177 mitter from vesicles that accumulate in axon varicosities induces a local rise in cytoplasmic calcium

178 Ca(2+) transients in other varicosities initiated intracellular Ca(2+) waves in adj

179 Finally, axonal varicosity initiation can trigger action potentials to a

180 ere assessed at 6 weeks for need for further varicosity intervention, which was completed with either

181 ly via the conversion of some preexisting PF varicosities into multisynaptic terminals.

182 e that, in the retina, dopamine synthesis in varicosities is affected by the spiking activity of reti

183 rite extension, and formation of presynaptic varicosities is displayed by photoreceptors after retina

184 The density of DbetaH varicosities is greater in the prefrontal cortex than in

185 The ratio of varicosities is stable over a greater than seven-fold ra

186 onfocal imaging of individual sensory neuron varicosities labeled with three different fluorescent ma

187 ncluding cosmetic spider veins, asymptomatic varicosities, large painful varicose veins, edema, hyper

188 Presynaptic axonal varicosities, like postsynaptic spines, are dynamically

189 re characterized by contorted processes with varicosity-like structures, some containing both alpha-s

190 r in striatal dopaminergic presynaptic nerve varicosities, making them unusually susceptible to inhib

191 usly empty varicosities and formation of new varicosities-may determine intermediate- versus long-ter

192 loss of excitability in nerve terminals and varicosities, mediated by a Ca-activated K conductance.

193 apse, with larger varicosity size but normal varicosity number, indicating that these synaptic parame

194 agonist-evoked influx of Ca(2+) in dendritic varicosities of A17 amacrine cells from diabetic compare

195 ARs elicited small Ca(2+) transients in axon varicosities of cerebellar stellate cell interneurons.

196 estine (ICC-DMP) are closely associated with varicosities of enteric motor neurons and generate respo

197 lpha(+) ) cells, are closely associated with varicosities of enteric motor neurons and suggested to m

198 was localized in restricted fashion to axon varicosities of neurons recorded from laminae II-V, alth

199 Moreover, ATRP is present in peripheral varicosities of protraction motoneurons and enhances per

200 14 was used to stain thin axons and terminal varicosities of the crayfish neuromuscular junction.

201 light or GABA/glycine inhibitors but only in varicosities of the DA axon plexus, not in perikarya or

202 as shown by immunocytochemistry to reside in varicosities of the DA plexus but not in dendrites, indi

203 ularity is mirrored in the spacing of axonal varicosities of the stratified bipolar cells, which have

204 Outer retinal varicosities of this putative GABAergic IPC did colocali

205 ited by NT-3, without affecting the synaptic varicosity of the presynaptic terminals.

206 correlated with ongoing Ca(2+) transients in varicosities on the axons of presumably inhibitory motor

207 lesioned side, as well as the number of CART varicosities on the surface of TRH neurons from 6.0 +/-

208 15 +/- 1.4% of biotinamide-labeled extrinsic varicosities; only 1 +/- 0.3% of biotinamide-labeled ext

209 ributed upon stimulation toward the sites of varicosity outgrowth.

210 s capable of rapidly budding new presynaptic varicosities over the course of minutes in response to e

211 .9-fold in the vicinity of catecholaminergic varicosities (P < 0.0001).

212 es at 9 months further reduced the number of varicosities/PN.

213 to quantify the density of axons and axonal varicosities present in DCS (the latter represent presyn

214 to quantify the density of axons and axonal varicosities present in the FDLS.

215 E neurons still resulted in peptide-positive varicosity production.

216 ricosities, as dopaminergic and corazonergic varicosities remain qualitatively intact following serot

217 That these ChAT-positive varicosities represent presynaptic release sites were de

218 acquire the more stable properties of mature varicosities required for the persistence of LTF.

219 VGluT1+, VGluT2+, and VIAAT+ varicosities respectively represent: 28%, 41%, and 31% i

220 Interestingly, YFP-labeled varicosities revealed changes that corresponded with cha

221 are composed of a sequence of many beads or varicosities separated by intervaricose segments.

222 ndergo degeneration with formation of axonal varicosities sequestering transported proteins and progr

223 The fraction of dynamic PF varicosities significantly diminished during training on

224 ructural defects at the synapse, with larger varicosity size but normal varicosity number, indicating

225 ontrast, VGluT2 expression was restricted to varicosities, some of them coexpressing mCherry.

226 ry and higher-order dendrites exhibited rich varicosities, sometimes with dendritic spines.

227 l characteristics of CHT-immunoreactive axon varicosities specifically within the anteroventral thala

228 matched by activity in closely apposed nerve varicosities, suggesting EGCs were not only innervated b

229 Newly formed branches presented varicosities, suggesting that new axons were more than j

230 Also, somatic versus neuropil location of varicosities suggests that most of these afferents are i

231 enteric neurons, extensive OT-immunoreactive varicosities surrounded many others.

232 d in nerve trunks attached to the IMG and in varicosities surrounding IMG neurones.

233 mber of delta-opioid receptor-immunoreactive varicosities that appose the postsynaptic membrane of th

234 This pattern produces many varicosities that are clustered together but also includ

235 re clustered together but also includes some varicosities that are very isolated.

236 in contact with muscle cells, and lowest in varicosities that did not contact muscle cells.

237 Of SOM-IR, SP-IR, and VIP-IR varicosities that lacked VAChT-immunoreactivity, only 1

238 nt neurons give rise to numerous presynaptic varicosities that target hair cells and afferent process

239 itative changes in nerves including enlarged varicosities that were also seen following capsaicin and

240 ely localized to a limited population of its varicosities, the majority of likely synaptic-release si

241 examine the spatial pattern of serotonergic varicosities, the synaptic sites of serotonin release in

242 al layers and extends long processes without varicosities to cortical layers 3 and 4.

243 ion of PKM--caused the number of presynaptic varicosities to revert to the original, pretraining valu

244 We tested the sensitivity of serotonergic varicosities to serotonin by adding neurotransmitter at

245 expressed by genetic manipulation in Type II varicosities to study octopamine release in Drosophila.

246 haft atrophy, and nearby axons develop large varicosities, together leading to neurite breakage and l

247 Timing of varicosity treatment is controversial with delayed and s

248 o either simultaneous phlebectomy or delayed varicosity treatment.

249 hibiting rapid elongation and retraction and varicosity turnover.

250 s, characterized by an overextended synaptic varicosity, underdeveloped synaptic morphology and disru

251 e occurrence of immunolabeling in individual varicosities varied widely between cells (39 +/- 36%, n

252 The numerical density of cholinergic varicosities was also unchanged in aged rats.

253 Formation of varicosities was frequently preceded or accompanied by h

254 found that the density of cholinergic fiber varicosities was higher in epileptic rats versus control

255 The density of labeled varicosities was highest in the pons and lowest in MI.

256 mine beta-hydroxylase (DbetaH)-immunostained varicosities was performed on several cortical regions a

257 ed tonic, and production of peptide-positive varicosities was substantially reduced as well.

258 get region, a large percentage of the BDA-ir varicosities was VGLUT2-ir (41-83%).

259 These varicosities were absent in areas where there were nonfi

260 naptotagmin and SNAP-25-immunopositive nerve varicosities were concentrated in varicose regions of mo

261 Hcrt-1- and hcrt-2-positive fibers with varicosities were detected in almost all brainstem regio

262 ive synaptic contacts evident as bouton-like varicosities were detected in close apposition to latera

263 Although some BDA-labeled axons with varicosities were found bilaterally at all cervical leve

264 BDA-ir varicosities were found in the solitary tract nucleus (N

265 nto layers 2-5 of each area, labelled axonal varicosities were found ipsilaterally in the other corti

266 Serotonin fibers and varicosities were found throughout the spinal cord and w

267 cells, and most of the ChR2-positive axonal varicosities were immunoreactive for vesicular glutamate

268 V1-derived axonal varicosities were mostly (>80%) glycinergic and a third

269 Numerous VGAT and VGLUT2 labeled varicosities were observed apposed to dDpMe-labeled axon

270 The highest densities of C3 axon varicosities were observed in Lamina X and the intermedi

271 rve fibers until P14, when nerve fibers with varicosities were observed in the urethra and bladder ne

272 The less common Orx varicosities were occasionally apposed to TH- or GABA-la

273 d corticothalamic terminals and their beaded varicosities were plotted, and the digital reconstructio

274 opulation, nitrergic subpopulation and (VIP) varicosities were reduced.

275 opulation, nitrergic subpopulation and (VIP) varicosities were reduced.

276 part of the nucleus, orexin-A-immunopositive varicosities were relatively more abundant, located in c

277 Some double-labeled BDA/VGAT varicosities were seen apposed to small somata labeled f

278 Varicosities were seen in fiber staining which may refle

279 e DMV >95% mCherry-immunoreactive(ir) axonal varicosities were tyrosine hydroxylase (TH)-ir and the s

280 NA varicosities were visualized by immunoperoxidase labelin

281 Efferent fibers and varicosities were visualized in the semicircular canal o

282 and internalized mostly in growth cones and varicosities, where synaptic vesicle markers were also c

283 cells significantly increased the number of varicosities, whereas cone cells increased process growt

284 bindin, are colocalized with the presynaptic varicosities, whereas gephyrin, Na-K-2Cl cotransporter 1

285 nd (2) a slower generation of new functional varicosities which occurs between 12-18 hr and requires

286 lity failures limited to nerve terminals and varicosities, which account for the rapid decline in the

287 ated via the release of dopamine from nearby varicosities, which in turn leads to potentiation of the

288 ated via the release of dopamine from nearby varicosities, which in turn led to potentiation of the s

289 ncrease in sensorin synthesis, especially at varicosities, which precedes the secretion of sensorin.

290 hine treatment accelerated Tat-induced focal varicosities, which were accompanied by localized increa

291 butyric acid membrane transporter 1-positive varicosities, whose appearance resembles chandelier cart

292 GAC dendritic tree and endowed all dendritic varicosities with a small-center, strong-surround recept

293 in axon growth, the number of sensory neuron varicosities with release sites contacting the target, a

294 ic terminals by filling of preexisting empty varicosities with synaptic vesicles, which parallels int

295 Enrichment of empty varicosities with synaptophysin accounts for 32% of the

296 tion of delta-opioid receptor-immunoreactive varicosities with the labeling of the GABA-synthesizing

297 the ultrastructural relationship of NA axon varicosities with the somata and dendrites of identified

298 Sh) and the presence of orexin receptors and varicosities within the AccSh, we hypothesized that orex

299 can modulate the development of serotonergic varicosities within the fly central nervous system.

300 igantocellular preoptic area (gPOA) and axon varicosities within the ventral nucleus of the ventral t