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1 Saccharomycescerevisiae Ded1p and Drosophila Vasa.
2 egulating other multipotency factors such as vasa.
3 indle-class gene similar to the RNA helicase Vasa.
4 spermatogonia or spermatocytes and expressed VASA.
5 ated a human ortholog of the Drosophila gene vasa.
6 the germ cell markers nanos3, dnd, dazl and vasa.
7 n silencing, and perinuclear localization of Vasa.
8 ding modes and affinities for both Par-4 and VASA.
11 Genes encoding two zinc finger proteins, Vasa, a LIM domain protein, Sox and Jun-like transcripti
12 Although GLHs are homologous to Drosophila VASA, a polar granule component necessary for oogenesis
13 AMPPNP structure, and that of the Drosophila Vasa*AMPPNP*Mg2+*RNA complex, we targeted 20 positions i
14 er is nucleated by the DEAD box RNA helicase Vasa and contains the two Piwi proteins participating in
15 the regulatory sequence of medaka germ gene vasa and generated transgenic fish with visible GFP expr
17 ke two other known polar granule components, Vasa and Oskar, Aubergine remains cytoplasmic after pole
19 lts add to the growing body of evidence that vasa and piwi can play important roles in somatic develo
20 us PGCs and absence of instructive roles for vasa and piwi in PGC formation are reminiscent of mouse
21 amine germ line development and the roles of vasa and piwi orthologues in the common house spider Par
23 ess the germ-cell markers dazl, dnd, nanos3, vasa and piwil1 and the spermatogonial markers plzf and
24 script and protein expression patterns of Pt-vasa and Pt-piwi to show that primordial germ cells (PGC
25 e-related genes PRDM1, PRDM14, LIN28A, DAZL, VASA and SYCP3 induced direct conversion of somatic cell
28 RNA pathway in regulating the levels of OSK, VASA, and possibly other genes involved in germline dete
30 m cell-specific conditional knock-out (Cul4b(Vasa)),as well asCul4bglobal knock-out (Cul4b(Sox2)) mou
31 s developed normally and did not overexpress Vasa, as did embryos from a micromere deletion, implying
37 We attempted to use the germline-expressed Vasa-Cre transgene to engineer a mouse mutation, but obs
38 w tool for genetic analysis of the germline, Vasa-Cre(ERT2), showed that this pathway functions throu
42 MC proliferation in the central tail artery, vasa deferentia, seminal vesicles, prostate, and uterus,
47 stem cell population of the embryo, and that vasa expression in this embryo is restricted early by tr
48 on transplantation into fertile adults, thus vasa expression is correlated with the potential for gam
49 pleting maternal PIWI does not affect OSK or VASA expression or abdominal patterning but leads to fai
52 We show that ATP-dependent RNP remodeling by Vasa facilitates transfer of 5' sliced piRNA precursors
53 ms analyzed, Caenorhabditis elegans has four Vasa family members, the germline helicases GLH-1, GLH-2
56 These results suggest that, when present, Vasa functions are essential to contributing to developm
58 d adult tissues that expression of the human VASA gene is restricted to the ovary and testis and is u
61 a plasmid concentrations corresponded to the vasa gene, an important component of the nuage-piwi RNA
62 that Nile tilapia have a single copy of the vasa gene, we find evidence for at least three vasa gene
63 ed the zebrafish homologue of the Drosophila vasa gene, which, in the fly, encodes a germ-cell-specif
64 foundation for studying the role of multiple vasa genes in the development of tilapia gonads, and wil
65 stodes and trematodes have lost the piwi and vasa genes that are hallmark characters of the germline
69 ing and export, whereas the DEAD box protein Vasa has an established role in piRNA production and loc
75 Northern blotting revealed that zebrafish vasa homologue (vas) transcript is present in embryos ju
76 is necessary for the proper localization of Vasa, implying that Vasa is involved in the cyclin-depen
81 num homolog (Cf-vas) of the germ cell marker Vasa indicated that the B(4) blastomere in four cell-sta
83 ll micromeres, whereas overexpression of the Vasa-interacting domain of Gustavus (GusDeltaSOCS) resul
84 l retardation protein (FMRP), dFXR, and VIG (Vasa intronic gene), through their association with RISC
94 e proper localization of Vasa, implying that Vasa is involved in the cyclin-dependent cell cycle netw
95 the cyclin-dependent cell cycle network, and Vasa is required for the efficient translation of cyclin
97 uced Gurken accumulation and modification of Vasa - is very similar to the phenotype of the spindle-c
99 ing the maternal piwi dose increases OSK and VASA levels correspondingly and doubles and triples the
101 ariant Rhino, that nuage granules containing Vasa localize directly across the nuclear envelope from
102 to target a reporter construct to the DDX4 (vasa) locus in chicken primordial germ cells (PGCs).
103 cells determined in early development, that vasa may function in an early stem cell population of th
104 in a profound heterochrony, suggesting that vasa might play a homeostatic role in asexual developmen
112 c neuropathy results from destruction of the vasa nervorum and can be reversed by administration of a
114 ts from microvascular ischemia involving the vasa nervorum and suggest the feasibility of a novel tre
116 re was a profound reduction in the number of vasa nervorum associated with marked endothelial cell ap
117 ere uniquely localized in close proximity to vasa nervorum, and a smaller portion of these EPCs were
118 these drugs, resulting in destruction of the vasa nervorum, and accordingly that the neuropathy could
119 urbations affect neurons, Schwann cells, and vasa nervorum, which are held to be the primary cell typ
126 ility to test the function of these putative VASA positive germ cells is limited, these results demon
128 A knockdown of Gustavus protein reduces both Vasa protein abundance and its propensity for accumulati
130 ly throughout all cells of the early embryo, vasa protein accumulates selectively in the 16-cell stag
131 nes in isolated culture, including selective Vasa protein accumulation and transcriptional activation
132 has a conserved, positive regulatory role in Vasa protein accumulation during embryonic development.
133 egative cadherin each suggest that, although vasa protein accumulation in the small micromeres is fix
136 , paraffin-embedded tissue and characterized VASA protein expression in human germ cells at various s
137 rated polyclonal antibodies that bind to the VASA protein in formalin-fixed, paraffin-embedded tissue
141 he sea urchin Strongylocentrotus purpuratus, Vasa protein is enriched in the small micromeres despite
145 In this paper, I use immunodetection of Vasa protein to study germ cell development in the amphi
146 oved respond by significant up-regulation of vasa protein translation, followed by spatial restrictio
147 served germ plasm components, nanos mRNA and Vasa protein, revealed that germ plasm segregation is a
149 omolog), Xenopus laevis, and D. melanogaster Vasa proteins contain both symmetrical and asymmetrical
150 hese cells express germ cell markers such as vasa, pumilio and piwi, as well as sphingosine-1-phospha
151 movement through the walls of the ascending vasa recta (AVR) in the exposed papillae of 2-week-old S
152 ave suggested that the fenestrated ascending vasa recta (AVRs) drain the interstitial fluid in this l
153 ltage-operated Na+ conductance in descending vasa recta (DVR) pericytes isolated from the renal outer
154 soconstriction of outer medullary descending vasa recta (OMDVR) is modulated by adenosine, we examine
157 eases [Ca2+]i in pericytes of the descending vasa recta as part of its constrictor action and that th
161 in were both expressed on the endothelium of vasa recta in the renal medulla, the lymph node subcapsu
162 nd low urea reflection coefficient of UT3 in vasa recta may be important for the formation of a conce
163 disease who showed peritubular capillary and vasa recta thrombi and capillary basement membrane alter
164 ([NO]i) of pericytes and endothelium of the vasa recta were independently measured with the use of f
165 ase of [NO]i of 19+/-6 U in pericytes of the vasa recta when the vessels were adjacent to medullary t
168 elium of glomeruli, peritubular capillaries, vasa recta, and the principal cells (epithelial) of coll
169 s demonstrated syntaxin-4 mRNA in glomeruli, vasa recta, connecting tubules, and thin descending limb
170 urea rapidly as they traverse the ascending vasa recta, thereby preventing loss of urea from the med
171 nt exchange between ascending and descending vasa recta, to enhance the cortico-papillary osmolality
179 l tubule, thin descending limb of Henle, and vasa recta; AQP2, AQP3, and AQP4 in the collecting duct;
182 NPC)-like FG repeat domains are found in the VASA-related P-granule proteins GLH-1, GLH-2, and GLH-4
185 ponents, which include dead end, nanos1, and vasa RNAs, are initially present in a wide cortical band
186 These components assemble on the surface of Vasa's helicase domain, which functions as an RNA clamp
189 late of the mesenchyme blastula stage and Sp-vasa, Sp-nanos2, Sp-seawi, and Sp-SoxE transcripts are l
192 suggest an evolutionarily conserved role of Vasa that is independent of its function in germ line de
194 licase encoded by the "posterior" group gene vasa (vas) in control of localization of the mRNA encode
195 , we find that Bru interacts physically with Vasa (Vas), an RNA helicase that is a positive regulator
196 hetic axons embedded in a few arterioles and vasa vasora were recently shown to store tissue plasmino
197 espectively, P < 0.01) and in the density of vasa vasorum (1.84+/-0.05/mm2 vs. 4.73+/-0.24/mm2; respe
200 ed especially by an increase of second-order vasa vasorum and disorientation of normal vasa vasorum s
201 ansplanted islets received blood supply from vasa vasorum and had access to drainage through venous t
203 cytes and T lymphocytes, and the role of the vasa vasorum and surrounding perivascular adipose tissue
207 onstrate that rPAI-1(23) treatment decreased vasa vasorum area and length, which was supported by mic
209 reconstructed confocal microscopy images of vasa vasorum demonstrate that rPAI-1(23) treatment decre
211 prevents the increase in VEGF expression and vasa vasorum density of coronary arteries in experimenta
213 lloon-injured coronary arteries, adventitial vasa vasorum density was increased (3.16+/-0.17/mm2 vs.
218 culture adventitial fibroblasts (AdvFBs) and vasa vasorum endothelial cells (VVECs) from the adventit
219 ic cells and macrophages), progenitor cells, vasa vasorum endothelial cells and pericytes, and adrene
222 Recent attention has focused on the role of vasa vasorum in atherosclerotic and restenotic coronary
224 causes regression or collapse of adventitial vasa vasorum in hypercholesterolemic mice by stimulating
225 titate three-dimensional spatial patterns of vasa vasorum in normal and balloon injured porcine coron
226 ate the three-dimensional spatial pattern of vasa vasorum in normal and experimental hypercholesterol
227 ssessment of the therapeutic response of the vasa vasorum in patients with atherosclerotic plaque.
231 iew offers insight into the possible role of vasa vasorum in the development of intracranial vascular
232 These data demonstrate that formation of new vasa vasorum in vasculitis is regulated by inflammatory
233 arteries suggests that the formation of new vasa vasorum is determined by the nature of the immune r
234 ionally, neovascularization arising from the vasa vasorum may promote atherosclerotic plaque progress
235 es is accompanied by neovascularization from vasa vasorum microvessels extending through the tunica m
236 mplex layer of the vessel wall consisting of vasa vasorum microvessels, nerves, fibroblasts, immune c
237 mplex layer of the vessel wall consisting of vasa vasorum microvessels, nerves, fibroblasts, immune c
238 pport a role for the endogenous ET system in vasa vasorum neovascularization in early coronary athero
239 in (ET) receptor antagonism reduces coronary vasa vasorum neovascularization in experimental hypercho
240 , a promoter of adventitial inflammation and vasa vasorum neovascularization in experimental models o
241 hanistic role of the endogenous ET system in vasa vasorum neovascularization in hypercholesterolemia
242 Cs, in a process involving ET-1, to regulate vasa vasorum neovascularization occurring in the adventi
243 ed phases, the role of eccentric remodeling, vasa vasorum neovascularization, and mechanisms of plaqu
244 molecular mechanisms regulating adventitial vasa vasorum neovascularization, which occurs in the pul
245 ede "macrovascular endothelial dysfunction." Vasa vasorum neovascularization, with endothelial leakag
248 gests that adventitial neovascularization of vasa vasorum occurs in experimental hypercholesterolemic
251 es of vasa vasorum were defined: first-order vasa vasorum ran longitudinally parallel to the vessel a
258 ntiangiogenic effect of TSP-1, the number of vasa vasorum was reduced in aortas from diabetic rats.
262 on," which is composed of dysfunction of the vasa vasorum's endothelium as well as "microcellular end
263 dventitia, particularly within microvessels (vasa vasorum) but not in cells of the intima or media.
264 al nitric oxide synthase (due to ingrowth of vasa vasorum), neointima formation, and loss of smooth m
265 f helping detect and even grade intracranial vasa vasorum, and this may provide new insights into our
266 hat the coronary vessel wall, especially the vasa vasorum, as well as bone marrow-derived endothelial
267 In conclusion, rPAI-1(23) inhibits growth of vasa vasorum, as well as vessels within the adjacent pla
268 from the artery lumen and outer adventitial vasa vasorum, deposit proatherogenic plasma molecules, r
269 ix can be imaged, as can angiogenesis of the vasa vasorum, plaque inflammation, and fibrin deposits o
270 lammation; and to stimulate expansion of the vasa vasorum, which can act as a conduit for continued i
280 injury, the total vascular area comprised of vasa was significantly reduced in injured vessels compar
281 NA encoding the primordial germ-cell marker, vasa, was present for more than 30 days in embryo cells
282 sion of PGC markers nanos1 and TDRD7 but not vasa were down-regulated when dnd mutant proteins lackin
284 he diameters of first-order and second-order vasa were smaller in normal compared with balloon-treate
285 SN5 mutations also cause the modification of Vasa, which is known to be required for Gurken translati
286 Furthermore, we show that the RNA helicase Vasa, which is required for nanos RNA localization, also
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