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1 helial damage in peritubular capillaries and vasa recta.
2 porter in erythrocytes and kidney-descending vasa recta.
3 titium, and medulla including vessels in the vasa recta.
4 the outer medulla is expressed in descending vasa recta.
5 CAM-1 is prominent in the endothelium of the vasa recta.
6 the vascular bundles of the outer medullary vasa recta.
7 -sensitive NO production in pericytes of the vasa recta.
9 elium of glomeruli, peritubular capillaries, vasa recta, and the principal cells (epithelial) of coll
10 l tubule, thin descending limb of Henle, and vasa recta; AQP2, AQP3, and AQP4 in the collecting duct;
11 eases [Ca2+]i in pericytes of the descending vasa recta as part of its constrictor action and that th
12 movement through the walls of the ascending vasa recta (AVR) in the exposed papillae of 2-week-old S
13 ave suggested that the fenestrated ascending vasa recta (AVRs) drain the interstitial fluid in this l
16 s demonstrated syntaxin-4 mRNA in glomeruli, vasa recta, connecting tubules, and thin descending limb
17 ltage-operated Na+ conductance in descending vasa recta (DVR) pericytes isolated from the renal outer
19 in were both expressed on the endothelium of vasa recta in the renal medulla, the lymph node subcapsu
20 nd low urea reflection coefficient of UT3 in vasa recta may be important for the formation of a conce
21 soconstriction of outer medullary descending vasa recta (OMDVR) is modulated by adenosine, we examine
23 urea rapidly as they traverse the ascending vasa recta, thereby preventing loss of urea from the med
24 disease who showed peritubular capillary and vasa recta thrombi and capillary basement membrane alter
25 nt exchange between ascending and descending vasa recta, to enhance the cortico-papillary osmolality
26 ([NO]i) of pericytes and endothelium of the vasa recta were independently measured with the use of f
27 ase of [NO]i of 19+/-6 U in pericytes of the vasa recta when the vessels were adjacent to medullary t
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