ライフサイエンスコーパス: vascular anomaly

1 vena cava is a rare but important congenital vascular anomaly.

2 involves the focal development of a distinct vascular anomaly.

3 tissue from individuals with other types of vascular anomalies.

4 ent of the inferior vena cava (IVC) were the vascular anomalies.

5 ur understanding of vascular development and vascular anomalies.

6 effects of recent and previous therapies for vascular anomalies.

7 promote angiogenesis and the development of vascular anomalies.

8 icoagulation when confronted with underlying vascular anomalies.

9 that have minuscule function of irreparable vascular anomalies.

10 ns and susceptibility genes in patients with vascular anomalies, (3) recognition of new risk stratifi

11 the major thoracic and abdominal organs with vascular anomalies and multiple spleens.

12 e describe our approach in 5 recipients with vascular anomalies and significant comorbidities, includ

13 viewed include cholestatic diseases, tumors, vascular anomalies, and acquired diseases.

14 radial anomalies, and in DRS with deafness, vascular anomalies, and cognitive deficits, respectively

15 ory of multiple hospitalisations for foci of vascular anomalies appearing progressively in the upper

16 Vascular anomalies are an attractive model for the study

17 Thoracic vascular anomalies are common in TS, occurring in approx

18 We provide evidence that these vascular anomalies are explained by a light-response pat

19 National and international workshops on vascular anomalies are fostering clinical and basic scie

20 Vascular anomalies are usually diagnosed through their c

21 Vascular anomalies are variably associated with overgrow

22 antation because of the greater incidence of vascular anomalies, associated renal anomalies, and pred

23 rved for 2 years (May 2013 to June 2015) for vascular anomalies at a tertiary care referral center.

24 atic patients has suggested a range of other vascular anomalies, but the true prevalence of such lesi

25 ort here that this deletion also resulted in vascular anomalies characterized by extreme variability

26 emangioma," is a non-hereditary, congenital, vascular anomaly comprised of aberrant clusters of malfo

27 The vascular anomalies described in our cohort of AGS indivi

28 ovides information regarding hemodynamics of vascular anomalies, differentiating high- and low-flow v

29 We conclude that major vascular anomalies do not necessarily preclude transplan

30 RI) is an effective modality for classifying vascular anomalies due to its ability to delineate the e

31 Subjects who had intracranial vascular anomalies had the best outcomes of the group.

32 approach, focusing on Mendelian disorders of vascular anomalies, has been particularly fruitful for t

33 ntified in affected tissues of patients with vascular anomalies, implicating the Snrk-1-Dusp-5 signal

34 intracranial vessel abnormalities and other vascular anomalies in AGS.

35 s to characterize the nature and spectrum of vascular anomalies in AGS.

36 aging in initial assessment of cervicofacial vascular anomalies in children.

37 ished method for detection and monitoring of vascular anomalies in children.

38 Dominant mutations of either in humans cause vascular anomalies in the brain, known as cerebral caver

39 plication secondary to a rare and unexpected vascular anomaly in a pediatric renal transplant that re

40 was severely affected in the mutant embryos: vascular anomalies included decreased complexity of the

41 atomical mapping, the complete evaluation of vascular anomalies includes hemodynamic characterization

42 RIAL/METHODS: Twenty pediatric patients with vascular anomalies located in the cervicofacial region u

43 may have coexisting iatrogenic or congenital vascular anomalies making transplantation difficult.

44 Vascular anomalies may cause pulsatile tinnitus, but the

45 y based on the type, size, and extent of the vascular anomaly, necessitating accurate diagnosis and c

46 cavernous malformations (CCM) are congenital vascular anomalies of the brain that can cause significa

47 avernous malformations (CCMs) are congenital vascular anomalies of the central nervous system that ca

48 Cerebral cavernous malformations (CCMs) are vascular anomalies of the central nervous system, compri

49 not practical to take the left kidney due to vascular anomalies or asymmetric function.

50 Twenty-five patients (9%) had noncardiac vascular anomalies or events.

51 atile tinnitus suggests a vascular neoplasm, vascular anomaly, or vascular malformation.

52 Multidisciplinary vascular anomalies programs provide focused expertise in

53 -1 versus tie-1AS lncRNA is altered in human vascular anomaly samples.

54 webbing, increased chest diameter, and these vascular anomalies suggests that in utero, centrally loc

55 These data confirm a genetic cause of a vascular-anomaly syndrome that has phenotypic overlap wi

56 al cavernous malformations (CCMs) are common vascular anomalies that develop in the central nervous s

57 alformation (AVM) is a fast-flow, congenital vascular anomaly that may arise anywhere in the body.

58 Vascular anomalies were detected on CT scanning without

59 Eighty-seven vascular anomalies were evaluated by means of US.

60 Vascular anomalies were found using intracranial MR angi

61 Vascular anomalies were particularly striking in testis

62 Arteriovenous malformations (AVMs) are vascular anomalies where arteries and veins are directly

63 anol level was evaluated in 71 patients with vascular anomalies who underwent ethanol embolization, s

64 enotypic severity, we demonstrated that most vascular anomalies with overgrowth harbor postzygotic ga

65 netic variants associated with a spectrum of vascular anomalies with overgrowth, ranging from mild to

66 issue from a diverse cohort of subjects with vascular anomalies with overgrowth.