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1 ded by SAV, as measured by the expression of vascular cell adhesion molecule.
2 2.84-4.95] vs. 1.66 [1.22-2.63] p < 0.0001), vascular cell adhesion molecule 1 (539 [451-621] vs. 402
4 ated with the endothelial activation markers vascular cell adhesion molecule 1 (P < .001 for both) an
5 r adhesion molecule 1 (sICAM-1), and soluble vascular cell adhesion molecule 1 (sVCAM-1) were measure
6 shear stress to alpha4beta1 integrin ligands vascular cell adhesion molecule 1 (VCAM-1) and fibronect
8 elial dysfunctional markers, such as soluble vascular cell adhesion molecule 1 (VCAM-1) and intercell
11 tudies revealed that the interaction between vascular cell adhesion molecule 1 (VCAM-1) and very late
14 tercellular adhesion molecule 1 (ICAM-1) and vascular cell adhesion molecule 1 (VCAM-1) in the lungs
15 but interaction with the endothelial ligand vascular cell adhesion molecule 1 (VCAM-1) may also play
16 neutrophils adhering to surfaces expressing vascular cell adhesion molecule 1 (VCAM-1) motivated the
18 strate that the adhesion pathway mediated by vascular cell adhesion molecule 1 (VCAM-1) plays a role
19 s is monocyte recruitment through binding to vascular cell adhesion molecule 1 (VCAM-1) upregulated o
20 uman umbilical vein endothelial cell (HUVEC) vascular cell adhesion molecule 1 (VCAM-1) upregulation.
23 d serves as an adhesive ligand that captures vascular cell adhesion molecule 1 (VCAM-1)(+) metastatic
24 -activated antibody (pro-antibody) targeting vascular cell adhesion molecule 1 (VCAM-1), a marker of
25 e chemotactic protein 1 (MCP-1), IL-6, IL-8, vascular cell adhesion molecule 1 (VCAM-1), and E-select
26 intercellular adhesion molecule 1 (ICAM-1), vascular cell adhesion molecule 1 (VCAM-1), and E-select
27 essential for TNF-alpha-induced E-selectin, vascular cell adhesion molecule 1 (VCAM-1), and intercel
28 FimA, while RANTES, gamma interferon, IL-17, vascular cell adhesion molecule 1 (VCAM-1), and vascular
29 d factor, intracellular adhesion molecule 1, vascular cell adhesion molecule 1 (VCAM-1), E-selectin,
30 aluate expression of an inflammatory marker, vascular cell adhesion molecule 1 (VCAM-1), in atheroscl
31 ment and adherence to endothelium, including vascular cell adhesion molecule 1 (VCAM-1), intercellula
32 endothelial cells to synthesize E-selectin, vascular cell adhesion molecule 1 (VCAM-1), interleukin
33 ed significantly increased urinary levels of vascular cell adhesion molecule 1 (VCAM-1), P-selectin,
34 intercellular adhesion molecule 1 (ICAM-1), vascular cell adhesion molecule 1 (VCAM-1), platelet-end
35 ody fragment directed against mouse or human vascular cell adhesion molecule 1 (VCAM-1), recently has
36 acrophage chemoattractant protein 1 (MCP-1), vascular cell adhesion molecule 1 (VCAM-1), secretory ph
37 al variables, including the level of soluble vascular cell adhesion molecule 1 (VCAM-1), were assesse
38 cells to express adhesion molecules such as vascular cell adhesion molecule 1 (VCAM-1), which mediat
39 effect was specific for L-selectin, because vascular cell adhesion molecule 1 (VCAM-1)-mediated adhe
40 d intercellular adhesion molecule 1 (ICAM-1)/vascular cell adhesion molecule 1 (VCAM-1)-mediated adhe
41 uantitative behavioral outcomes, and a novel vascular cell adhesion molecule 1 (VCAM-1)-targeted magn
45 adhesion of eosinophils to various forms of vascular cell adhesion molecule 1 (VCAM-1, CD106), an in
47 of intercellular adhesion molecule 1 (ICAM1)/vascular cell adhesion molecule 1 (VCAM1), whose express
49 key cytokines, and cell adhesion molecules (vascular cell adhesion molecule 1 [VCAM-1] and intracell
50 issue inhibitor of metalloproteinases 1; and vascular cell adhesion molecule 1 [VCAM-1]) were measure
51 , vascular endothelial growth factor [VEGF], vascular cell adhesion molecule 1 [VCAM1], and GTP bindi
52 and alphaLbeta2 to their respective ligands, vascular cell adhesion molecule 1 and intercellular adhe
53 es with their endothelial counter-receptors, vascular cell adhesion molecule 1 and intercellular adhe
54 ws for adhesion to their respective ligands, vascular cell adhesion molecule 1 and intercellular adhe
56 ing in the posttranscriptional regulation of vascular cell adhesion molecule 1 expression and a reduc
57 othelial tumor necrosis factor-alpha-induced vascular cell adhesion molecule 1 expression and monocyt
58 s correlated with a marked reduction in lung vascular cell adhesion molecule 1 expression and product
61 ion of intracellular adhesion molecule 1 and vascular cell adhesion molecule 1 in cells and in kidney
62 ilonRII) cells; IL-4; IL-5; eosinophils, and vascular cell adhesion molecule 1 in the large airways a
63 on of monocyte chemoattractant protein-1 and vascular cell adhesion molecule 1 in wire-injured caroti
64 s/chemokines, leukotriene B(4) (LTB(4)), and vascular cell adhesion molecule 1 levels in the lungs, w
65 naling in the stromal cells and with reduced vascular cell adhesion molecule 1 production by them.
68 ing of intercellular adhesion molecule 1 and vascular cell adhesion molecule 1 under the migrating le
69 ntercellular adhesion molecule 1 and soluble vascular cell adhesion molecule 1 were reduced after whe
70 ased low-density lipoprotein uptake, VCAM-1 (vascular cell adhesion molecule 1) expression, apoptosis
71 esion (intracellular adhesion molecule 1 and vascular cell adhesion molecule 1) genes involved in mye
72 ctin, intercellular adhesion molecule 1, and vascular cell adhesion molecule 1) was slightly up- or d
73 idase [MPO], matrix metalloproteinase 9, and vascular cell adhesion molecule 1) would be increased an
74 se 2, intracellular adhesion molecule 1, and vascular cell adhesion molecule 1), increased expression
77 le p75 tumor necrosis factor alpha receptor, vascular cell adhesion molecule 1, and matrix metallopro
78 selectin, intercellular adhesion molecule 1, vascular cell adhesion molecule 1, and tissue factor mes
80 e intercellular adhesion molecule 1, soluble vascular cell adhesion molecule 1, C-peptide, insulin, a
81 sposed intercellular adhesion molecule 1 and vascular cell adhesion molecule 1, disruption or looseni
82 les (intercellular cell adhesion molecule 1, vascular cell adhesion molecule 1, E-selectin, and plate
83 scular endothelial growth factor receptor-2, vascular cell adhesion molecule 1, intercellular adhesio
84 in, von Willebrand factor (VWF), E-selectin, vascular cell adhesion molecule 1, intercellular adhesio
86 both diminished inflammation (expression of vascular cell adhesion molecule 1, plaque macrophage con
87 cells adhering to culture plates coated with vascular cell adhesion molecule 1, SDF-1 and stem cell f
88 lular adhesion molecule 1 (sICAM-1), soluble vascular cell adhesion molecule 1, soluble E-selectin, a
89 let), E-selectin (selectin endothelium), and vascular cell adhesion molecule 1, were also upregulated
96 d prevented monocyte adhesion by attenuating vascular cell adhesion molecule -1 and monocyte chemoatt
97 ctant protein-1 (-13% versus +0.2%), soluble vascular cell adhesion molecule-1 (-6% versus +1%), high
98 nce of early AMD was associated with soluble vascular cell adhesion molecule-1 (odds ratio per SD on
100 ellular adhesion molecule-1, E-selectin, and vascular cell adhesion molecule-1 (P < 0.05 above bacter
103 rix metalloproteinase 9 (MMP-9), and soluble vascular cell adhesion molecule-1 (sVCAM-1) were dramati
104 ercellular adhesion molecule-1 (sICAM-1) and vascular cell adhesion molecule-1 (sVCAM-1) were higher
105 intercellular adhesion molecule-1 (sICAM-1), vascular cell adhesion molecule-1 (sVCAM-1), E-selectin
107 lated up-regulation of the adhesion proteins vascular cell adhesion molecule-1 (VCAM) and intercellul
108 /mL, Group II: 0.14 (0.20) mug/mL, P = .02); vascular cell adhesion molecule-1 (VCAM-1) (Group I: 0.3
110 d by inhibition of TNF-induced expression of vascular cell adhesion molecule-1 (VCAM-1) and could be
111 ercellular adhesion molecule-1 (ICAM-1), and vascular cell adhesion molecule-1 (VCAM-1) and diabetes
112 levels than PDEs of the endothelial proteins vascular cell adhesion molecule-1 (VCAM-1) and endotheli
113 n inhibitory factor (rhMIF) in up-regulating vascular cell adhesion molecule-1 (VCAM-1) and intercell
115 177 as well as expression of redox-sensitive vascular cell adhesion molecule-1 (VCAM-1) and monocyte
116 ry signaling that controls expression of the vascular cell adhesion molecule-1 (VCAM-1) and monocyte
117 uction of the endothelial activation markers vascular cell adhesion molecule-1 (VCAM-1) and vascular
118 was abolished by CD29 blockade on hMSCs and vascular cell adhesion molecule-1 (VCAM-1) blockade on H
119 tercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule-1 (VCAM-1) expression by
120 tion, EGF treatment induced up-regulation of vascular cell adhesion molecule-1 (VCAM-1) expression in
122 cells was p38 MAPK-dependent while increased vascular cell adhesion molecule-1 (VCAM-1) expression wa
123 We hypothesized that molecular imaging of vascular cell adhesion molecule-1 (VCAM-1) expression wi
124 brain inflammation was used to induce global vascular cell adhesion molecule-1 (VCAM-1) expression.
125 iency also transiently augmented LPS-induced vascular cell adhesion molecule-1 (VCAM-1) expression.
126 e derivatives as novel, potent inhibitors of vascular cell adhesion molecule-1 (VCAM-1) expression.
127 y Wu et al presents additional evidence that vascular cell adhesion molecule-1 (VCAM-1) imaging in at
128 wnstream target genes tissue factor (TF) and vascular cell adhesion molecule-1 (VCAM-1) in diabetic a
129 ur angiogenesis, but increased expression of vascular cell adhesion molecule-1 (VCAM-1) in primary cu
132 croarray analysis of P0 and P3 revealed that vascular cell adhesion molecule-1 (VCAM-1) is up-regulat
133 vasive imaging of adhesion molecules such as vascular cell adhesion molecule-1 (VCAM-1) may identify
136 cess on CXC chemokine receptor 2 (CXCR2) and vascular cell adhesion molecule-1 (VCAM-1) using null st
137 effect of t-RA exposure on cell adhesion to vascular cell adhesion molecule-1 (VCAM-1), a well-estab
138 intercellular adhesion molecule-1 (ICAM-1), vascular cell adhesion molecule-1 (VCAM-1), and E-select
139 cellular adhesion molecule-1 (MAdCAM-1) and vascular cell adhesion molecule-1 (VCAM-1), endothelial
140 ctivator 2, decay accelerating factor (DAF), vascular cell adhesion molecule-1 (VCAM-1), manganese su
141 acrophage chemoattractant protein-1 (MCP-1), vascular cell adhesion molecule-1 (VCAM-1), nuclear fact
142 lium and subsequent induction of endothelial vascular cell adhesion molecule-1 (VCAM-1), which is req
143 with very late antigen-4 (VLA-4) binding to vascular cell adhesion molecule-1 (VCAM-1), which is upr
144 ssing the endothelial cell adhesion molecule vascular cell adhesion molecule-1 (VCAM-1), which normal
145 ly as a consequence of reduced expression of vascular cell adhesion molecule-1 (VCAM-1), which regula
154 ic kidney, which was reversed by blockade of vascular cell adhesion molecule-1 (VCAM1) and very late
159 oinflammatory proteins, cyclooxygenase-2 and vascular cell adhesion molecule-1 also in an Src kinase
160 decreased firm arrest of mouse monocytes on vascular cell adhesion molecule-1 and E-selectin in a sh
161 rabbits decreased collar-induced endothelial vascular cell adhesion molecule-1 and intercellular adhe
162 1 mRNA levels and inhibited cytokine-induced vascular cell adhesion molecule-1 and intercellular adhe
163 nockout mice showed significant decreases in vascular cell adhesion molecule-1 and intercellular adhe
165 le cells blocks Ang II-induced expression of vascular cell adhesion molecule-1 and intercellular adhe
166 tor T cells adhere to high concentrations of vascular cell adhesion molecule-1 and intercellular adhe
167 lished the blocking effect of kallistatin on vascular cell adhesion molecule-1 and monocyte chemoattr
168 pha-induced NF-kappaB activation, as well as vascular cell adhesion molecule-1 and monocyte chemoattr
169 repressor for inflammatory mediators such as vascular cell adhesion molecule-1 and monocyte chemotact
170 nhanced the expression of adhesion molecules vascular cell adhesion molecule-1 and monocyte chemotact
172 erns of vascular adhesion molecules, such as vascular cell adhesion molecule-1 and platelet endotheli
173 ctin, intercellular adhesion molecule-1, and vascular cell adhesion molecule-1 and secretion of the c
174 ntent), and endothelial dysfunction (soluble vascular cell adhesion molecule-1 and soluble intercellu
175 or inhibited adhesion of proerythroblasts to Vascular Cell Adhesion Molecule-1 and the fibronectin sp
176 nd demyelination by increasing expression of vascular cell adhesion molecule-1 and the transcytosis p
177 ement of tumor necrosis factor alpha-induced vascular cell adhesion molecule-1 and tissue factor expr
178 n, and its interactions with both macrophage Vascular Cell Adhesion Molecule-1 and with extracellular
179 82 augmented adhesion of proerythroblasts to Vascular Cell Adhesion Molecule-1 but not to the fibrone
180 ontin by 12% (95% CI, -14% to -10%), soluble vascular cell adhesion molecule-1 by 14% (95% CI, -18% t
181 ion of intercellular adhesion molecule-1 and vascular cell adhesion molecule-1 by, and adhesion of hu
182 atory biomarkers, and carotid P-selectin and vascular cell adhesion molecule-1 expression by contrast
184 osis factor alpha (TNF alpha) stimulation of vascular cell adhesion molecule-1 expression in endothel
185 romoted superoxide production, and increased vascular cell adhesion molecule-1 expression in human ao
187 ung injury, CPAP-30 and STEP-30/30 increased vascular cell adhesion molecule-1 expression, but the ty
188 ased endothelial ROS production, endothelial vascular cell adhesion molecule-1 expression, matrix met
189 eukocytes was associated with an increase in vascular cell adhesion molecule-1 expression, which itse
191 pendent increase in plasma levels of soluble vascular cell adhesion molecule-1 in sickle mice was not
192 elial cell adhesion molecules E-selectin and vascular cell adhesion molecule-1 in the microvasculatur
193 of endothelial nitric oxide, superoxide, and vascular cell adhesion molecule-1 in vitro improved sign
196 only volume-controlled ventilation increased vascular cell adhesion molecule-1 messenger RNA expressi
197 y be effectively replaced by transduction of vascular cell adhesion molecule-1 or intercellular adhes
198 with chronic PTSD had a greater increase in vascular cell adhesion molecule-1 over time (B = 0.003,
199 binant intercellular adhesion molecule 1 and vascular cell adhesion molecule-1 rather than endothelia
200 ion of intercellular adhesion molecule-1 and vascular cell adhesion molecule-1 surface expression in
201 alpha receptor 2, interleukin-6, and soluble vascular cell adhesion molecule-1 to the 20-year cumulat
203 mooth muscle cell actin, and upregulation of vascular cell adhesion molecule-1 was also suppressed in
205 nd their receptors and inhibition of VCAM-1 (vascular cell adhesion molecule-1) and ICAM-1 (intercell
206 and endothelial activation markers (soluble vascular cell adhesion molecule-1) decreased but remaine
207 ction (intercellular adhesion molecule-1 and vascular cell adhesion molecule-1) in 524 middle-aged wo
208 olecules (intercellular adhesion molecule-1, vascular cell adhesion molecule-1), inducible nitric oxi
209 olecules (intercellular adhesion molecule-1, vascular cell adhesion molecule-1), tumor necrosis facto
211 tissues, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, and E-selectin expres
213 protein, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, and leptin and decrea
214 or-alpha, intracellular adhesion molecule-1, vascular cell adhesion molecule-1, and monocyte chemotac
215 plasma intercellular adhesion molecule-1 and vascular cell adhesion molecule-1, and VEDS probands had
216 croM, Gal-1 increased eosinophil adhesion to vascular cell adhesion molecule-1, caused redistribution
217 sposed intercellular adhesion molecule-1 and vascular cell adhesion molecule-1, disruption or looseni
218 or-alpha, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, endothelial selectin,
219 le tumor necrosis factor receptor-1, soluble vascular cell adhesion molecule-1, granulocyte colony-st
220 pression (ie, ET-1, ET-A and ET-B receptors, vascular cell adhesion molecule-1, IL-6, and fractalkine
221 RvE1 or RvD1 inhibited the expressions of vascular cell adhesion molecule-1, IL-8, macrophage infl
222 leukin-6, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, inducible nitric oxid
223 ation; downregulated the local expression of vascular cell adhesion molecule-1, intercellular adhesio
224 tly inhibited thrombin-mediated induction of vascular cell adhesion molecule-1, intercellular adhesio
225 EC, increasing the expression of E-selectin, vascular cell adhesion molecule-1, intercellular adhesio
227 receptor 1, hypoxia-inducible factor-1alpha, vascular cell adhesion molecule-1, macrophage scavenger
228 red fluorescence signals with immunoreactive vascular cell adhesion molecule-1, macrophages, and cath
230 steopontin), and leukocyte adhesion (soluble vascular cell adhesion molecule-1, soluble intracellular
231 selectin, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, tumor necrosis factor
232 ecules intracellular adhesion molecule-1 and vascular cell adhesion molecule-1, which caused a fourfo
233 iency of monocytes sheared in whole blood on vascular cell adhesion molecule-1, while characterizing
234 ivo magnetic resonance imaging enhanced with vascular cell adhesion molecule-1-targeted nanoparticles
247 F-kappaB-responsive genes, but expression of vascular cell-adhesion molecule-1 (Vcam1) was particular
248 yr142-phosphorylated beta-catenin stimulates vascular cell-adhesion molecule-1 expression to increase
249 r to activate, its high-affinity state binds vascular cell adhesion molecule and fibronectin 100- to
251 crease in C-reactive protein, nitrotyrosine, vascular cell adhesion molecule and monocyte superoxide
252 CD11b-negative) vascular cells that express vascular cell adhesion molecule and variably alpha-smoot
253 necrotic cores, and proinflammatory VCAM-1 (vascular cell adhesion molecule) and MCP-1 (monocyte che
254 dothelial activation and circulating soluble vascular cell adhesion molecule), and a significant impr
255 eceptors, including nitric oxide synthase 2, vascular cell adhesion molecule, and intercellular cell
256 ble-intercellular adhesion molecule, soluble-vascular cell adhesion molecule, and soluble-E-selectin
257 levels of C-reactive protein, fibrinogen, or vascular cell adhesion molecule between the groups.
258 over, among control subjects, plasma soluble vascular cell adhesion molecule concentrations were sign
259 al pro-brain natriuretic peptide and soluble vascular cell adhesion molecule, has low predictive accu
260 monocyte chemoattractant protein-1, soluble vascular cell adhesion molecule, high-sensitivity C-reac
261 lls, TH2-related cytokines, eosinophils, and vascular cell adhesion molecule in the bronchopulmonary
262 with rigidity of the corresponding loops in vascular cell adhesion molecule, intercellular adhesion
263 which are regulated by NF-kappaB, including vascular cell adhesion molecule, intracellular adhesion
264 onitored by NF-kappaB activation and surface vascular cell adhesion molecule levels on aortic endothe
265 in (P = 0.001), E-selectin (P = 0.0005), and vascular cell adhesion molecule (P = 0.0468) with +4 por
266 erleukin-6, intercellular adhesion molecule, vascular cell adhesion molecule, P-selectin, and E-selec
267 opulation of melanoma cells that express the vascular cell adhesion molecule PECAM1, but not VEGFR-2,
268 tal kininogen; decreased total cathepsin-L1, vascular cell adhesion molecule, periostin, and neutroph
270 ellular adhesion molecule (sICAM)-1, soluble vascular cell adhesion molecule (sVCAM)-1, and sE-select
271 lycerides and lacked the increase in soluble vascular cell adhesion molecules (sVCAM)-1 evident in th
272 adhesion molecule type 1 (sICAM-1), soluble vascular cell adhesion molecule type 1 (sVCAM-1), and th
274 intercellular adhesion molecule (ICAM) 1 and vascular cell adhesion molecule (VCAM) 1 and for proper
275 ddressin cell adhesion molecule (MAdCAM) and vascular cell adhesion molecule (VCAM) appear to play ro
277 helial expression of the inflammatory marker vascular cell adhesion molecule (VCAM) in cells and anim
278 od pressure monitoring, serum interleukin-6, vascular cell adhesion molecule (VCAM), and asymmetric d
279 ndothelial cells increased the expression of vascular cell adhesion molecule (VCAM)-1 and intercellul
281 oxide (NO)-dependent signaling, and reduced vascular cell adhesion molecule (VCAM)-1 expression and
282 intercellular adhesion molecule (ICAM)-1 and vascular cell adhesion molecule (VCAM)-1 form nanocluste
283 acity as showed by the reduced expression of vascular cell adhesion molecule (VCAM)-1 in human cultur
285 X)-2, tumor necrosis factor (TNF)-alpha, and vascular cell adhesion molecule (VCAM)-1 was increased i
286 llular adhesion molecule (ICAM)-1, IL-8, and vascular cell adhesion molecule (VCAM)-1 were assessed b
287 of intercellular adhesion molecule (ICAM)-1, vascular cell adhesion molecule (VCAM)-1, and activated
288 leukin (IL)-6, white blood cell count (WBC), vascular cell adhesion molecule (VCAM)-1, and intercellu
289 actor (TNF)-alpha, endothelin (ET)-1, Ang-1, vascular cell adhesion molecule (VCAM)-1, and vascular e
290 , types I and IV collagen, and vitronectin), vascular cell adhesion molecule (VCAM)-1, or activating
291 of intercellular adhesion molecule (ICAM)-1, vascular cell adhesion molecule (VCAM)-1, platelet-endot
293 in; 3) tumor necrosis factor (TNF)-alpha; 4) vascular cell adhesion molecule (VCAM); 5) interleukin (
294 ms, we demonstrate that spatial variation in vascular cell adhesion molecule (VCAM-1) expression corr
295 ctivated neutrophils, alpha(5)-integrins, or vascular cell adhesion molecule (VCAM-1) were performed
296 p-regulated expression levels of E-selectin, vascular cell adhesion molecule (VCAM-1), and intercellu
297 1 represses the expression of E-selectin and vascular cell adhesion molecule (VCAM-1), two cytokine-i
298 cellular adhesion molecule (ICAM-1/CD54) and vascular cell adhesion molecule (VCAM/CD106); and the gr
299 (VEGF), soluble cell adhesion molecules (ie, vascular cell adhesion molecule [VCAM], intercellular ce
300 enhanced intercellular adhesion molecule and vascular cell adhesion molecule within the islets when s
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