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   1 ded by SAV, as measured by the expression of vascular cell adhesion molecule.                        
     2 2.84-4.95] vs. 1.66 [1.22-2.63] p < 0.0001), vascular cell adhesion molecule 1 (539 [451-621] vs. 402
  
     4 ated with the endothelial activation markers vascular cell adhesion molecule 1 (P < .001 for both) an
     5 r adhesion molecule 1 (sICAM-1), and soluble vascular cell adhesion molecule 1 (sVCAM-1) were measure
     6 shear stress to alpha4beta1 integrin ligands vascular cell adhesion molecule 1 (VCAM-1) and fibronect
  
     8 elial dysfunctional markers, such as soluble vascular cell adhesion molecule 1 (VCAM-1) and intercell
  
  
    11 tudies revealed that the interaction between vascular cell adhesion molecule 1 (VCAM-1) and very late
  
  
    14 tercellular adhesion molecule 1 (ICAM-1) and vascular cell adhesion molecule 1 (VCAM-1) in the lungs 
    15  but interaction with the endothelial ligand vascular cell adhesion molecule 1 (VCAM-1) may also play
    16  neutrophils adhering to surfaces expressing vascular cell adhesion molecule 1 (VCAM-1) motivated the
  
    18 strate that the adhesion pathway mediated by vascular cell adhesion molecule 1 (VCAM-1) plays a role 
    19 s is monocyte recruitment through binding to vascular cell adhesion molecule 1 (VCAM-1) upregulated o
    20 uman umbilical vein endothelial cell (HUVEC) vascular cell adhesion molecule 1 (VCAM-1) upregulation.
  
  
    23 d serves as an adhesive ligand that captures vascular cell adhesion molecule 1 (VCAM-1)(+) metastatic
    24 -activated antibody (pro-antibody) targeting vascular cell adhesion molecule 1 (VCAM-1), a marker of 
    25 e chemotactic protein 1 (MCP-1), IL-6, IL-8, vascular cell adhesion molecule 1 (VCAM-1), and E-select
    26  intercellular adhesion molecule 1 (ICAM-1), vascular cell adhesion molecule 1 (VCAM-1), and E-select
    27  essential for TNF-alpha-induced E-selectin, vascular cell adhesion molecule 1 (VCAM-1), and intercel
    28 FimA, while RANTES, gamma interferon, IL-17, vascular cell adhesion molecule 1 (VCAM-1), and vascular
    29 d factor, intracellular adhesion molecule 1, vascular cell adhesion molecule 1 (VCAM-1), E-selectin, 
    30 aluate expression of an inflammatory marker, vascular cell adhesion molecule 1 (VCAM-1), in atheroscl
    31 ment and adherence to endothelium, including vascular cell adhesion molecule 1 (VCAM-1), intercellula
    32  endothelial cells to synthesize E-selectin, vascular cell adhesion molecule 1 (VCAM-1), interleukin 
    33 ed significantly increased urinary levels of vascular cell adhesion molecule 1 (VCAM-1), P-selectin, 
    34  intercellular adhesion molecule 1 (ICAM-1), vascular cell adhesion molecule 1 (VCAM-1), platelet-end
    35 ody fragment directed against mouse or human vascular cell adhesion molecule 1 (VCAM-1), recently has
    36 acrophage chemoattractant protein 1 (MCP-1), vascular cell adhesion molecule 1 (VCAM-1), secretory ph
    37 al variables, including the level of soluble vascular cell adhesion molecule 1 (VCAM-1), were assesse
    38  cells to express adhesion molecules such as vascular cell adhesion molecule 1 (VCAM-1), which mediat
    39  effect was specific for L-selectin, because vascular cell adhesion molecule 1 (VCAM-1)-mediated adhe
    40 d intercellular adhesion molecule 1 (ICAM-1)/vascular cell adhesion molecule 1 (VCAM-1)-mediated adhe
    41 uantitative behavioral outcomes, and a novel vascular cell adhesion molecule 1 (VCAM-1)-targeted magn
  
  
  
    45  adhesion of eosinophils to various forms of vascular cell adhesion molecule 1 (VCAM-1, CD106), an in
  
    47 of intercellular adhesion molecule 1 (ICAM1)/vascular cell adhesion molecule 1 (VCAM1), whose express
  
    49  key cytokines, and cell adhesion molecules (vascular cell adhesion molecule 1 [VCAM-1] and intracell
    50 issue inhibitor of metalloproteinases 1; and vascular cell adhesion molecule 1 [VCAM-1]) were measure
    51 , vascular endothelial growth factor [VEGF], vascular cell adhesion molecule 1 [VCAM1], and GTP bindi
    52 and alphaLbeta2 to their respective ligands, vascular cell adhesion molecule 1 and intercellular adhe
    53 es with their endothelial counter-receptors, vascular cell adhesion molecule 1 and intercellular adhe
    54 ws for adhesion to their respective ligands, vascular cell adhesion molecule 1 and intercellular adhe
  
    56 ing in the posttranscriptional regulation of vascular cell adhesion molecule 1 expression and a reduc
    57 othelial tumor necrosis factor-alpha-induced vascular cell adhesion molecule 1 expression and monocyt
    58 s correlated with a marked reduction in lung vascular cell adhesion molecule 1 expression and product
  
  
    61 ion of intracellular adhesion molecule 1 and vascular cell adhesion molecule 1 in cells and in kidney
    62 ilonRII) cells; IL-4; IL-5; eosinophils, and vascular cell adhesion molecule 1 in the large airways a
    63 on of monocyte chemoattractant protein-1 and vascular cell adhesion molecule 1 in wire-injured caroti
    64 s/chemokines, leukotriene B(4) (LTB(4)), and vascular cell adhesion molecule 1 levels in the lungs, w
    65 naling in the stromal cells and with reduced vascular cell adhesion molecule 1 production by them.   
  
  
    68 ing of intercellular adhesion molecule 1 and vascular cell adhesion molecule 1 under the migrating le
    69 ntercellular adhesion molecule 1 and soluble vascular cell adhesion molecule 1 were reduced after whe
    70 ased low-density lipoprotein uptake, VCAM-1 (vascular cell adhesion molecule 1) expression, apoptosis
    71 esion (intracellular adhesion molecule 1 and vascular cell adhesion molecule 1) genes involved in mye
    72 ctin, intercellular adhesion molecule 1, and vascular cell adhesion molecule 1) was slightly up- or d
    73 idase [MPO], matrix metalloproteinase 9, and vascular cell adhesion molecule 1) would be increased an
    74 se 2, intracellular adhesion molecule 1, and vascular cell adhesion molecule 1), increased expression
  
  
    77 le p75 tumor necrosis factor alpha receptor, vascular cell adhesion molecule 1, and matrix metallopro
    78 selectin, intercellular adhesion molecule 1, vascular cell adhesion molecule 1, and tissue factor mes
  
    80 e intercellular adhesion molecule 1, soluble vascular cell adhesion molecule 1, C-peptide, insulin, a
    81 sposed intercellular adhesion molecule 1 and vascular cell adhesion molecule 1, disruption or looseni
    82 les (intercellular cell adhesion molecule 1, vascular cell adhesion molecule 1, E-selectin, and plate
    83 scular endothelial growth factor receptor-2, vascular cell adhesion molecule 1, intercellular adhesio
    84 in, von Willebrand factor (VWF), E-selectin, vascular cell adhesion molecule 1, intercellular adhesio
  
    86  both diminished inflammation (expression of vascular cell adhesion molecule 1, plaque macrophage con
    87 cells adhering to culture plates coated with vascular cell adhesion molecule 1, SDF-1 and stem cell f
    88 lular adhesion molecule 1 (sICAM-1), soluble vascular cell adhesion molecule 1, soluble E-selectin, a
    89 let), E-selectin (selectin endothelium), and vascular cell adhesion molecule 1, were also upregulated
  
  
  
  
  
  
    96 d prevented monocyte adhesion by attenuating vascular cell adhesion molecule -1 and monocyte chemoatt
    97 ctant protein-1 (-13% versus +0.2%), soluble vascular cell adhesion molecule-1 (-6% versus +1%), high
    98 nce of early AMD was associated with soluble vascular cell adhesion molecule-1 (odds ratio per SD on 
  
   100 ellular adhesion molecule-1, E-selectin, and vascular cell adhesion molecule-1 (P < 0.05 above bacter
  
  
   103 rix metalloproteinase 9 (MMP-9), and soluble vascular cell adhesion molecule-1 (sVCAM-1) were dramati
   104 ercellular adhesion molecule-1 (sICAM-1) and vascular cell adhesion molecule-1 (sVCAM-1) were higher 
   105 intercellular adhesion molecule-1 (sICAM-1), vascular cell adhesion molecule-1 (sVCAM-1), E-selectin 
  
   107 lated up-regulation of the adhesion proteins vascular cell adhesion molecule-1 (VCAM) and intercellul
   108 /mL, Group II: 0.14 (0.20) mug/mL, P = .02); vascular cell adhesion molecule-1 (VCAM-1) (Group I: 0.3
  
   110 d by inhibition of TNF-induced expression of vascular cell adhesion molecule-1 (VCAM-1) and could be 
   111 ercellular adhesion molecule-1 (ICAM-1), and vascular cell adhesion molecule-1 (VCAM-1) and diabetes 
   112 levels than PDEs of the endothelial proteins vascular cell adhesion molecule-1 (VCAM-1) and endotheli
   113 n inhibitory factor (rhMIF) in up-regulating vascular cell adhesion molecule-1 (VCAM-1) and intercell
  
   115 177 as well as expression of redox-sensitive vascular cell adhesion molecule-1 (VCAM-1) and monocyte 
   116 ry signaling that controls expression of the vascular cell adhesion molecule-1 (VCAM-1) and monocyte 
   117 uction of the endothelial activation markers vascular cell adhesion molecule-1 (VCAM-1) and vascular 
   118  was abolished by CD29 blockade on hMSCs and vascular cell adhesion molecule-1 (VCAM-1) blockade on H
   119 tercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule-1 (VCAM-1) expression by
   120 tion, EGF treatment induced up-regulation of vascular cell adhesion molecule-1 (VCAM-1) expression in
  
   122 cells was p38 MAPK-dependent while increased vascular cell adhesion molecule-1 (VCAM-1) expression wa
   123    We hypothesized that molecular imaging of vascular cell adhesion molecule-1 (VCAM-1) expression wi
   124 brain inflammation was used to induce global vascular cell adhesion molecule-1 (VCAM-1) expression.  
   125 iency also transiently augmented LPS-induced vascular cell adhesion molecule-1 (VCAM-1) expression.  
   126 e derivatives as novel, potent inhibitors of vascular cell adhesion molecule-1 (VCAM-1) expression.  
   127 y Wu et al presents additional evidence that vascular cell adhesion molecule-1 (VCAM-1) imaging in at
   128 wnstream target genes tissue factor (TF) and vascular cell adhesion molecule-1 (VCAM-1) in diabetic a
   129 ur angiogenesis, but increased expression of vascular cell adhesion molecule-1 (VCAM-1) in primary cu
  
  
   132 croarray analysis of P0 and P3 revealed that vascular cell adhesion molecule-1 (VCAM-1) is up-regulat
   133 vasive imaging of adhesion molecules such as vascular cell adhesion molecule-1 (VCAM-1) may identify 
  
  
   136 cess on CXC chemokine receptor 2 (CXCR2) and vascular cell adhesion molecule-1 (VCAM-1) using null st
   137  effect of t-RA exposure on cell adhesion to vascular cell adhesion molecule-1 (VCAM-1), a well-estab
   138  intercellular adhesion molecule-1 (ICAM-1), vascular cell adhesion molecule-1 (VCAM-1), and E-select
   139  cellular adhesion molecule-1 (MAdCAM-1) and vascular cell adhesion molecule-1 (VCAM-1), endothelial 
   140 ctivator 2, decay accelerating factor (DAF), vascular cell adhesion molecule-1 (VCAM-1), manganese su
   141 acrophage chemoattractant protein-1 (MCP-1), vascular cell adhesion molecule-1 (VCAM-1), nuclear fact
   142 lium and subsequent induction of endothelial vascular cell adhesion molecule-1 (VCAM-1), which is req
   143  with very late antigen-4 (VLA-4) binding to vascular cell adhesion molecule-1 (VCAM-1), which is upr
   144 ssing the endothelial cell adhesion molecule vascular cell adhesion molecule-1 (VCAM-1), which normal
   145 ly as a consequence of reduced expression of vascular cell adhesion molecule-1 (VCAM-1), which regula
  
  
  
  
  
  
  
  
   154 ic kidney, which was reversed by blockade of vascular cell adhesion molecule-1 (VCAM1) and very late 
  
  
  
  
   159 oinflammatory proteins, cyclooxygenase-2 and vascular cell adhesion molecule-1 also in an Src kinase 
   160  decreased firm arrest of mouse monocytes on vascular cell adhesion molecule-1 and E-selectin in a sh
   161 rabbits decreased collar-induced endothelial vascular cell adhesion molecule-1 and intercellular adhe
   162 1 mRNA levels and inhibited cytokine-induced vascular cell adhesion molecule-1 and intercellular adhe
   163 nockout mice showed significant decreases in vascular cell adhesion molecule-1 and intercellular adhe
  
   165 le cells blocks Ang II-induced expression of vascular cell adhesion molecule-1 and intercellular adhe
   166 tor T cells adhere to high concentrations of vascular cell adhesion molecule-1 and intercellular adhe
   167 lished the blocking effect of kallistatin on vascular cell adhesion molecule-1 and monocyte chemoattr
   168 pha-induced NF-kappaB activation, as well as vascular cell adhesion molecule-1 and monocyte chemoattr
   169 repressor for inflammatory mediators such as vascular cell adhesion molecule-1 and monocyte chemotact
   170 nhanced the expression of adhesion molecules vascular cell adhesion molecule-1 and monocyte chemotact
  
   172 erns of vascular adhesion molecules, such as vascular cell adhesion molecule-1 and platelet endotheli
   173 ctin, intercellular adhesion molecule-1, and vascular cell adhesion molecule-1 and secretion of the c
   174 ntent), and endothelial dysfunction (soluble vascular cell adhesion molecule-1 and soluble intercellu
   175 or inhibited adhesion of proerythroblasts to Vascular Cell Adhesion Molecule-1 and the fibronectin sp
   176 nd demyelination by increasing expression of vascular cell adhesion molecule-1 and the transcytosis p
   177 ement of tumor necrosis factor alpha-induced vascular cell adhesion molecule-1 and tissue factor expr
   178 n, and its interactions with both macrophage Vascular Cell Adhesion Molecule-1 and with extracellular
   179 82 augmented adhesion of proerythroblasts to Vascular Cell Adhesion Molecule-1 but not to the fibrone
   180 ontin by 12% (95% CI, -14% to -10%), soluble vascular cell adhesion molecule-1 by 14% (95% CI, -18% t
   181 ion of intercellular adhesion molecule-1 and vascular cell adhesion molecule-1 by, and adhesion of hu
   182 atory biomarkers, and carotid P-selectin and vascular cell adhesion molecule-1 expression by contrast
  
   184 osis factor alpha (TNF alpha) stimulation of vascular cell adhesion molecule-1 expression in endothel
   185 romoted superoxide production, and increased vascular cell adhesion molecule-1 expression in human ao
  
   187 ung injury, CPAP-30 and STEP-30/30 increased vascular cell adhesion molecule-1 expression, but the ty
   188 ased endothelial ROS production, endothelial vascular cell adhesion molecule-1 expression, matrix met
   189 eukocytes was associated with an increase in vascular cell adhesion molecule-1 expression, which itse
  
   191 pendent increase in plasma levels of soluble vascular cell adhesion molecule-1 in sickle mice was not
   192 elial cell adhesion molecules E-selectin and vascular cell adhesion molecule-1 in the microvasculatur
   193 of endothelial nitric oxide, superoxide, and vascular cell adhesion molecule-1 in vitro improved sign
  
  
   196 only volume-controlled ventilation increased vascular cell adhesion molecule-1 messenger RNA expressi
   197 y be effectively replaced by transduction of vascular cell adhesion molecule-1 or intercellular adhes
   198  with chronic PTSD had a greater increase in vascular cell adhesion molecule-1 over time (B = 0.003, 
   199 binant intercellular adhesion molecule 1 and vascular cell adhesion molecule-1 rather than endothelia
   200 ion of intercellular adhesion molecule-1 and vascular cell adhesion molecule-1 surface expression in 
   201 alpha receptor 2, interleukin-6, and soluble vascular cell adhesion molecule-1 to the 20-year cumulat
  
   203 mooth muscle cell actin, and upregulation of vascular cell adhesion molecule-1 was also suppressed in
  
   205 nd their receptors and inhibition of VCAM-1 (vascular cell adhesion molecule-1) and ICAM-1 (intercell
   206  and endothelial activation markers (soluble vascular cell adhesion molecule-1) decreased but remaine
   207 ction (intercellular adhesion molecule-1 and vascular cell adhesion molecule-1) in 524 middle-aged wo
   208 olecules (intercellular adhesion molecule-1, vascular cell adhesion molecule-1), inducible nitric oxi
   209 olecules (intercellular adhesion molecule-1, vascular cell adhesion molecule-1), tumor necrosis facto
  
   211  tissues, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, and E-selectin expres
  
   213  protein, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, and leptin and decrea
   214 or-alpha, intracellular adhesion molecule-1, vascular cell adhesion molecule-1, and monocyte chemotac
   215 plasma intercellular adhesion molecule-1 and vascular cell adhesion molecule-1, and VEDS probands had
   216 croM, Gal-1 increased eosinophil adhesion to vascular cell adhesion molecule-1, caused redistribution
   217 sposed intercellular adhesion molecule-1 and vascular cell adhesion molecule-1, disruption or looseni
   218 or-alpha, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, endothelial selectin,
   219 le tumor necrosis factor receptor-1, soluble vascular cell adhesion molecule-1, granulocyte colony-st
   220 pression (ie, ET-1, ET-A and ET-B receptors, vascular cell adhesion molecule-1, IL-6, and fractalkine
   221    RvE1 or RvD1 inhibited the expressions of vascular cell adhesion molecule-1, IL-8, macrophage infl
   222 leukin-6, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, inducible nitric oxid
   223 ation; downregulated the local expression of vascular cell adhesion molecule-1, intercellular adhesio
   224 tly inhibited thrombin-mediated induction of vascular cell adhesion molecule-1, intercellular adhesio
   225 EC, increasing the expression of E-selectin, vascular cell adhesion molecule-1, intercellular adhesio
  
   227 receptor 1, hypoxia-inducible factor-1alpha, vascular cell adhesion molecule-1, macrophage scavenger 
   228 red fluorescence signals with immunoreactive vascular cell adhesion molecule-1, macrophages, and cath
  
   230 steopontin), and leukocyte adhesion (soluble vascular cell adhesion molecule-1, soluble intracellular
   231 selectin, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, tumor necrosis factor
   232 ecules intracellular adhesion molecule-1 and vascular cell adhesion molecule-1, which caused a fourfo
   233 iency of monocytes sheared in whole blood on vascular cell adhesion molecule-1, while characterizing 
   234 ivo magnetic resonance imaging enhanced with vascular cell adhesion molecule-1-targeted nanoparticles
  
  
  
  
  
  
  
  
  
  
  
  
   247 F-kappaB-responsive genes, but expression of vascular cell-adhesion molecule-1 (Vcam1) was particular
   248 yr142-phosphorylated beta-catenin stimulates vascular cell-adhesion molecule-1 expression to increase
   249 r to activate, its high-affinity state binds vascular cell adhesion molecule and fibronectin 100- to 
  
   251 crease in C-reactive protein, nitrotyrosine, vascular cell adhesion molecule and monocyte superoxide 
   252  CD11b-negative) vascular cells that express vascular cell adhesion molecule and variably alpha-smoot
   253  necrotic cores, and proinflammatory VCAM-1 (vascular cell adhesion molecule) and MCP-1 (monocyte che
   254 dothelial activation and circulating soluble vascular cell adhesion molecule), and a significant impr
   255 eceptors, including nitric oxide synthase 2, vascular cell adhesion molecule, and intercellular cell 
   256 ble-intercellular adhesion molecule, soluble-vascular cell adhesion molecule, and soluble-E-selectin 
   257 levels of C-reactive protein, fibrinogen, or vascular cell adhesion molecule between the groups.     
   258 over, among control subjects, plasma soluble vascular cell adhesion molecule concentrations were sign
   259 al pro-brain natriuretic peptide and soluble vascular cell adhesion molecule, has low predictive accu
   260  monocyte chemoattractant protein-1, soluble vascular cell adhesion molecule, high-sensitivity C-reac
   261 lls, TH2-related cytokines, eosinophils, and vascular cell adhesion molecule in the bronchopulmonary 
   262  with rigidity of the corresponding loops in vascular cell adhesion molecule, intercellular adhesion 
   263  which are regulated by NF-kappaB, including vascular cell adhesion molecule, intracellular adhesion 
   264 onitored by NF-kappaB activation and surface vascular cell adhesion molecule levels on aortic endothe
   265 in (P = 0.001), E-selectin (P = 0.0005), and vascular cell adhesion molecule (P = 0.0468) with +4 por
   266 erleukin-6, intercellular adhesion molecule, vascular cell adhesion molecule, P-selectin, and E-selec
   267 opulation of melanoma cells that express the vascular cell adhesion molecule PECAM1, but not VEGFR-2,
   268 tal kininogen; decreased total cathepsin-L1, vascular cell adhesion molecule, periostin, and neutroph
  
   270 ellular adhesion molecule (sICAM)-1, soluble vascular cell adhesion molecule (sVCAM)-1, and sE-select
   271 lycerides and lacked the increase in soluble vascular cell adhesion molecules (sVCAM)-1 evident in th
   272  adhesion molecule type 1 (sICAM-1), soluble vascular cell adhesion molecule type 1 (sVCAM-1), and th
  
   274 intercellular adhesion molecule (ICAM) 1 and vascular cell adhesion molecule (VCAM) 1 and for proper 
   275 ddressin cell adhesion molecule (MAdCAM) and vascular cell adhesion molecule (VCAM) appear to play ro
  
   277 helial expression of the inflammatory marker vascular cell adhesion molecule (VCAM) in cells and anim
   278 od pressure monitoring, serum interleukin-6, vascular cell adhesion molecule (VCAM), and asymmetric d
   279 ndothelial cells increased the expression of vascular cell adhesion molecule (VCAM)-1 and intercellul
  
   281  oxide (NO)-dependent signaling, and reduced vascular cell adhesion molecule (VCAM)-1 expression and 
   282 intercellular adhesion molecule (ICAM)-1 and vascular cell adhesion molecule (VCAM)-1 form nanocluste
   283 acity as showed by the reduced expression of vascular cell adhesion molecule (VCAM)-1 in human cultur
  
   285 X)-2, tumor necrosis factor (TNF)-alpha, and vascular cell adhesion molecule (VCAM)-1 was increased i
   286 llular adhesion molecule (ICAM)-1, IL-8, and vascular cell adhesion molecule (VCAM)-1 were assessed b
   287 of intercellular adhesion molecule (ICAM)-1, vascular cell adhesion molecule (VCAM)-1, and activated 
   288 leukin (IL)-6, white blood cell count (WBC), vascular cell adhesion molecule (VCAM)-1, and intercellu
   289 actor (TNF)-alpha, endothelin (ET)-1, Ang-1, vascular cell adhesion molecule (VCAM)-1, and vascular e
   290 , types I and IV collagen, and vitronectin), vascular cell adhesion molecule (VCAM)-1, or activating 
   291 of intercellular adhesion molecule (ICAM)-1, vascular cell adhesion molecule (VCAM)-1, platelet-endot
  
   293 in; 3) tumor necrosis factor (TNF)-alpha; 4) vascular cell adhesion molecule (VCAM); 5) interleukin (
   294 ms, we demonstrate that spatial variation in vascular cell adhesion molecule (VCAM-1) expression corr
   295 ctivated neutrophils, alpha(5)-integrins, or vascular cell adhesion molecule (VCAM-1) were performed 
   296 p-regulated expression levels of E-selectin, vascular cell adhesion molecule (VCAM-1), and intercellu
   297 1 represses the expression of E-selectin and vascular cell adhesion molecule (VCAM-1), two cytokine-i
   298 cellular adhesion molecule (ICAM-1/CD54) and vascular cell adhesion molecule (VCAM/CD106); and the gr
   299 (VEGF), soluble cell adhesion molecules (ie, vascular cell adhesion molecule [VCAM], intercellular ce
   300 enhanced intercellular adhesion molecule and vascular cell adhesion molecule within the islets when s
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