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1 ffect binding of VLA-4 to its ligand VCAM-1 (vascular cell adhesion molecule 1).
2 ctin, intercellular adhesion molecule 1, and vascular cell adhesion molecule 1.
3 f endothelial activation, angiopoietin 2 and vascular cell adhesion molecule 1.
4 nd intercellular adhesion molecule 1 but not vascular cell adhesion molecule 1.
5 llular cell adhesion molecule 1, and soluble vascular cell adhesion molecule 1.
6 in very late antigen 4 binding to its ligand vascular cell adhesion molecule 1.
7 1, interleukin 1beta, interferon gamma, and vascular cell adhesion molecule 1.
8 , IL-16, monocyte chemotactic protein-1, and vascular cell adhesion molecule-1.
9 tumor necrosis factor-mediated expression of vascular cell adhesion molecule-1.
10 mooth muscle cell actin and up-regulation of vascular cell adhesion molecule-1.
11 levels of IL-6, serum amyloid P, and soluble vascular cell adhesion molecule-1.
12 h a peptide mimicking monocyte engagement by vascular cell adhesion molecule-1.
13 natriuretic peptide (NT-proBNP) and soluble vascular cell adhesion molecule-1.
14 late antigen-4 (VLA-4), which interacts with vascular cell adhesion molecule-1.
15 f TRPC3 channels and also B cell adhesion to vascular cell adhesion molecule-1.
16 ha(4)beta(1) and blocks its interaction with vascular cell adhesion molecule-1.
17 1beta-induced expression of IL-6, MCP-1, and vascular cell adhesion molecule-1.
18 on and directed migration on fibronectin and vascular cell adhesion molecule-1.
19 lpha4beta1 and loss of binding to its ligand vascular cell adhesion molecule-1.
20 coclustering within focal adhesive sites on vascular cell adhesion molecule-1.
21 2.84-4.95] vs. 1.66 [1.22-2.63] p < 0.0001), vascular cell adhesion molecule 1 (539 [451-621] vs. 402
22 ctant protein-1 (-13% versus +0.2%), soluble vascular cell adhesion molecule-1 (-6% versus +1%), high
25 I domain-mediated cell spreading and soluble vascular cell adhesion molecule-1 (alpha4beta1 ligand)-i
26 oinflammatory proteins, cyclooxygenase-2 and vascular cell adhesion molecule-1 also in an Src kinase
28 d prevented monocyte adhesion by attenuating vascular cell adhesion molecule -1 and monocyte chemoatt
29 marrow depended on constitutively expressed vascular cell adhesion molecule 1 and endothelial select
30 and alphaLbeta2 to their respective ligands, vascular cell adhesion molecule 1 and intercellular adhe
31 es with their endothelial counter-receptors, vascular cell adhesion molecule 1 and intercellular adhe
32 ws for adhesion to their respective ligands, vascular cell adhesion molecule 1 and intercellular adhe
34 decreased firm arrest of mouse monocytes on vascular cell adhesion molecule-1 and E-selectin in a sh
35 pression potently inhibited the induction of vascular cell adhesion molecule-1 and endothelial adhesi
36 1 mRNA levels and inhibited cytokine-induced vascular cell adhesion molecule-1 and intercellular adhe
37 nockout mice showed significant decreases in vascular cell adhesion molecule-1 and intercellular adhe
39 le cells blocks Ang II-induced expression of vascular cell adhesion molecule-1 and intercellular adhe
41 tor T cells adhere to high concentrations of vascular cell adhesion molecule-1 and intercellular adhe
42 rabbits decreased collar-induced endothelial vascular cell adhesion molecule-1 and intercellular adhe
43 lished the blocking effect of kallistatin on vascular cell adhesion molecule-1 and monocyte chemoattr
44 pha-induced NF-kappaB activation, as well as vascular cell adhesion molecule-1 and monocyte chemoattr
45 repressor for inflammatory mediators such as vascular cell adhesion molecule-1 and monocyte chemotact
46 nhanced the expression of adhesion molecules vascular cell adhesion molecule-1 and monocyte chemotact
48 erns of vascular adhesion molecules, such as vascular cell adhesion molecule-1 and platelet endotheli
49 ture, S18886 also prevented the induction of vascular cell adhesion molecule-1 and prevented the decr
50 ctin, intercellular adhesion molecule-1, and vascular cell adhesion molecule-1 and secretion of the c
51 ntent), and endothelial dysfunction (soluble vascular cell adhesion molecule-1 and soluble intercellu
52 or inhibited adhesion of proerythroblasts to Vascular Cell Adhesion Molecule-1 and the fibronectin sp
53 nd demyelination by increasing expression of vascular cell adhesion molecule-1 and the transcytosis p
54 ement of tumor necrosis factor alpha-induced vascular cell adhesion molecule-1 and tissue factor expr
55 n, and its interactions with both macrophage Vascular Cell Adhesion Molecule-1 and with extracellular
56 nd their receptors and inhibition of VCAM-1 (vascular cell adhesion molecule-1) and ICAM-1 (intercell
57 le p75 tumor necrosis factor alpha receptor, vascular cell adhesion molecule 1, and matrix metallopro
58 selectin, intercellular adhesion molecule 1, vascular cell adhesion molecule 1, and tissue factor mes
60 increased C3 deposition and increased AT1-R, vascular cell adhesion molecule-1, and collagen expressi
61 tissues, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, and E-selectin expres
62 in, thromboxane B2, 6-ketoprostaglandin F1a, vascular cell adhesion molecule-1, and E-selectin were m
66 of mRNA for adhesion molecules (P-selectin, vascular cell adhesion molecule-1, and intercellular adh
67 protein, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, and leptin and decrea
68 or-alpha, intracellular adhesion molecule-1, vascular cell adhesion molecule-1, and monocyte chemotac
69 plasma intercellular adhesion molecule-1 and vascular cell adhesion molecule-1, and VEDS probands had
71 ion of intercellular adhesion molecule 1 and vascular cell adhesion molecule 1 as well as production
72 82 augmented adhesion of proerythroblasts to Vascular Cell Adhesion Molecule-1 but not to the fibrone
73 ontin by 12% (95% CI, -14% to -10%), soluble vascular cell adhesion molecule-1 by 14% (95% CI, -18% t
74 ion of intercellular adhesion molecule-1 and vascular cell adhesion molecule-1 by, and adhesion of hu
75 e intercellular adhesion molecule 1, soluble vascular cell adhesion molecule 1, C-peptide, insulin, a
76 croM, Gal-1 increased eosinophil adhesion to vascular cell adhesion molecule-1, caused redistribution
78 and endothelial activation markers (soluble vascular cell adhesion molecule-1) decreased but remaine
79 sposed intercellular adhesion molecule 1 and vascular cell adhesion molecule 1, disruption or looseni
80 sposed intercellular adhesion molecule-1 and vascular cell adhesion molecule-1, disruption or looseni
81 les (intercellular cell adhesion molecule 1, vascular cell adhesion molecule 1, E-selectin, and plate
82 or-alpha, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, endothelial selectin,
83 ighly intercellular adhesion molecule-1- and vascular cell adhesion molecule-1-enriched vertical micr
84 ing in the posttranscriptional regulation of vascular cell adhesion molecule 1 expression and a reduc
85 othelial tumor necrosis factor-alpha-induced vascular cell adhesion molecule 1 expression and monocyt
86 s correlated with a marked reduction in lung vascular cell adhesion molecule 1 expression and product
89 atory biomarkers, and carotid P-selectin and vascular cell adhesion molecule-1 expression by contrast
91 osis factor alpha (TNF alpha) stimulation of vascular cell adhesion molecule-1 expression in endothel
92 romoted superoxide production, and increased vascular cell adhesion molecule-1 expression in human ao
94 ung injury, CPAP-30 and STEP-30/30 increased vascular cell adhesion molecule-1 expression, but the ty
95 ased endothelial ROS production, endothelial vascular cell adhesion molecule-1 expression, matrix met
96 eukocytes was associated with an increase in vascular cell adhesion molecule-1 expression, which itse
99 yr142-phosphorylated beta-catenin stimulates vascular cell-adhesion molecule-1 expression to increase
100 ased low-density lipoprotein uptake, VCAM-1 (vascular cell adhesion molecule 1) expression, apoptosis
101 esion (intracellular adhesion molecule 1 and vascular cell adhesion molecule 1) genes involved in mye
102 le tumor necrosis factor receptor-1, soluble vascular cell adhesion molecule-1, granulocyte colony-st
103 pression (ie, ET-1, ET-A and ET-B receptors, vascular cell adhesion molecule-1, IL-6, and fractalkine
104 RvE1 or RvD1 inhibited the expressions of vascular cell adhesion molecule-1, IL-8, macrophage infl
105 increased apoptosis and reduced inflammatory vascular cell adhesion molecule-1 immunostaining but no
106 ion of intracellular adhesion molecule 1 and vascular cell adhesion molecule 1 in cells and in kidney
107 ilonRII) cells; IL-4; IL-5; eosinophils, and vascular cell adhesion molecule 1 in the large airways a
108 on of monocyte chemoattractant protein-1 and vascular cell adhesion molecule 1 in wire-injured caroti
109 pendent increase in plasma levels of soluble vascular cell adhesion molecule-1 in sickle mice was not
110 elial cell adhesion molecules E-selectin and vascular cell adhesion molecule-1 in the microvasculatur
111 of endothelial nitric oxide, superoxide, and vascular cell adhesion molecule-1 in vitro improved sign
113 ction (intercellular adhesion molecule-1 and vascular cell adhesion molecule-1) in 524 middle-aged wo
114 se 2, intracellular adhesion molecule 1, and vascular cell adhesion molecule 1), increased expression
116 olecules (intercellular adhesion molecule-1, vascular cell adhesion molecule-1), inducible nitric oxi
117 leukin-6, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, inducible nitric oxid
118 scular endothelial growth factor receptor-2, vascular cell adhesion molecule 1, intercellular adhesio
119 IL-8, IL-15, and RANTES), adhesion molecule (vascular cell adhesion molecule 1, intercellular adhesio
120 in, von Willebrand factor (VWF), E-selectin, vascular cell adhesion molecule 1, intercellular adhesio
121 ation; downregulated the local expression of vascular cell adhesion molecule-1, intercellular adhesio
122 tly inhibited thrombin-mediated induction of vascular cell adhesion molecule-1, intercellular adhesio
123 EC, increasing the expression of E-selectin, vascular cell adhesion molecule-1, intercellular adhesio
126 s/chemokines, leukotriene B(4) (LTB(4)), and vascular cell adhesion molecule 1 levels in the lungs, w
127 receptor 1, hypoxia-inducible factor-1alpha, vascular cell adhesion molecule-1, macrophage scavenger
128 red fluorescence signals with immunoreactive vascular cell adhesion molecule-1, macrophages, and cath
129 only volume-controlled ventilation increased vascular cell adhesion molecule-1 messenger RNA expressi
131 nce of early AMD was associated with soluble vascular cell adhesion molecule-1 (odds ratio per SD on
133 y be effectively replaced by transduction of vascular cell adhesion molecule-1 or intercellular adhes
134 elial cells on immobilized thrombospondin-1, vascular cell adhesion molecule-1, or recombinant N-term
135 with chronic PTSD had a greater increase in vascular cell adhesion molecule-1 over time (B = 0.003,
136 ated with the endothelial activation markers vascular cell adhesion molecule 1 (P < .001 for both) an
138 ellular adhesion molecule-1, E-selectin, and vascular cell adhesion molecule-1 (P < 0.05 above bacter
140 ation was assessed by measurement of soluble vascular cell adhesion molecule 1, P-selectin, L-selecti
141 both diminished inflammation (expression of vascular cell adhesion molecule 1, plaque macrophage con
142 naling in the stromal cells and with reduced vascular cell adhesion molecule 1 production by them.
143 LDL, Db-HAGE-LDL induced significant soluble vascular cell adhesion molecule-1 production (2.3-fold),
145 binant intercellular adhesion molecule 1 and vascular cell adhesion molecule-1 rather than endothelia
146 cells adhering to culture plates coated with vascular cell adhesion molecule 1, SDF-1 and stem cell f
147 lular adhesion molecule 1 (sICAM-1), soluble vascular cell adhesion molecule 1, soluble E-selectin, a
148 steopontin), and leukocyte adhesion (soluble vascular cell adhesion molecule-1, soluble intracellular
149 ion of intercellular adhesion molecule-1 and vascular cell adhesion molecule-1 surface expression in
150 r adhesion molecule 1 (sICAM-1), and soluble vascular cell adhesion molecule 1 (sVCAM-1) were measure
152 othelial growth factor (VEGF) and/or soluble vascular cell adhesion molecule-1 (sVCAM-1) are associat
154 rix metalloproteinase 9 (MMP-9), and soluble vascular cell adhesion molecule-1 (sVCAM-1) were dramati
155 ercellular adhesion molecule-1 (sICAM-1) and vascular cell adhesion molecule-1 (sVCAM-1) were higher
156 intercellular adhesion molecule-1 (sICAM-1), vascular cell adhesion molecule-1 (sVCAM-1), E-selectin
157 ivo magnetic resonance imaging enhanced with vascular cell adhesion molecule-1-targeted nanoparticles
158 alpha receptor 2, interleukin-6, and soluble vascular cell adhesion molecule-1 to the 20-year cumulat
161 olecules (intercellular adhesion molecule-1, vascular cell adhesion molecule-1), tumor necrosis facto
162 selectin, intercellular adhesion molecule-1, vascular cell adhesion molecule-1, tumor necrosis factor
163 ing of intercellular adhesion molecule 1 and vascular cell adhesion molecule 1 under the migrating le
164 shear stress to alpha4beta1 integrin ligands vascular cell adhesion molecule 1 (VCAM-1) and fibronect
166 elial dysfunctional markers, such as soluble vascular cell adhesion molecule 1 (VCAM-1) and intercell
169 tudies revealed that the interaction between vascular cell adhesion molecule 1 (VCAM-1) and very late
172 endothelial CD40, which allowed induction of vascular cell adhesion molecule 1 (VCAM-1) in a CD40-dep
174 tercellular adhesion molecule 1 (ICAM-1) and vascular cell adhesion molecule 1 (VCAM-1) in the lungs
175 (17-beta E(2)) attenuates the expression of vascular cell adhesion molecule 1 (VCAM-1) in vivo at ph
176 but interaction with the endothelial ligand vascular cell adhesion molecule 1 (VCAM-1) may also play
177 neutrophils adhering to surfaces expressing vascular cell adhesion molecule 1 (VCAM-1) motivated the
179 strate that the adhesion pathway mediated by vascular cell adhesion molecule 1 (VCAM-1) plays a role
180 s is monocyte recruitment through binding to vascular cell adhesion molecule 1 (VCAM-1) upregulated o
181 uman umbilical vein endothelial cell (HUVEC) vascular cell adhesion molecule 1 (VCAM-1) upregulation.
184 d serves as an adhesive ligand that captures vascular cell adhesion molecule 1 (VCAM-1)(+) metastatic
185 -activated antibody (pro-antibody) targeting vascular cell adhesion molecule 1 (VCAM-1), a marker of
186 e chemotactic protein 1 (MCP-1), IL-6, IL-8, vascular cell adhesion molecule 1 (VCAM-1), and E-select
187 intercellular adhesion molecule 1 (ICAM-1), vascular cell adhesion molecule 1 (VCAM-1), and E-select
188 essential for TNF-alpha-induced E-selectin, vascular cell adhesion molecule 1 (VCAM-1), and intercel
189 e (ADMA), intercellular adhesion molecule 1, vascular cell adhesion molecule 1 (VCAM-1), and monocyte
190 FimA, while RANTES, gamma interferon, IL-17, vascular cell adhesion molecule 1 (VCAM-1), and vascular
191 d factor, intracellular adhesion molecule 1, vascular cell adhesion molecule 1 (VCAM-1), E-selectin,
192 aluate expression of an inflammatory marker, vascular cell adhesion molecule 1 (VCAM-1), in atheroscl
193 ment and adherence to endothelium, including vascular cell adhesion molecule 1 (VCAM-1), intercellula
194 endothelial cells to synthesize E-selectin, vascular cell adhesion molecule 1 (VCAM-1), interleukin
195 ed significantly increased urinary levels of vascular cell adhesion molecule 1 (VCAM-1), P-selectin,
196 intercellular adhesion molecule 1 (ICAM-1), vascular cell adhesion molecule 1 (VCAM-1), platelet-end
197 ody fragment directed against mouse or human vascular cell adhesion molecule 1 (VCAM-1), recently has
198 acrophage chemoattractant protein 1 (MCP-1), vascular cell adhesion molecule 1 (VCAM-1), secretory ph
199 al variables, including the level of soluble vascular cell adhesion molecule 1 (VCAM-1), were assesse
200 cells to express adhesion molecules such as vascular cell adhesion molecule 1 (VCAM-1), which mediat
201 effect was specific for L-selectin, because vascular cell adhesion molecule 1 (VCAM-1)-mediated adhe
202 d intercellular adhesion molecule 1 (ICAM-1)/vascular cell adhesion molecule 1 (VCAM-1)-mediated adhe
203 uantitative behavioral outcomes, and a novel vascular cell adhesion molecule 1 (VCAM-1)-targeted magn
210 adhesion of eosinophils to various forms of vascular cell adhesion molecule 1 (VCAM-1, CD106), an in
213 lated up-regulation of the adhesion proteins vascular cell adhesion molecule-1 (VCAM) and intercellul
214 /mL, Group II: 0.14 (0.20) mug/mL, P = .02); vascular cell adhesion molecule-1 (VCAM-1) (Group I: 0.3
216 d by inhibition of TNF-induced expression of vascular cell adhesion molecule-1 (VCAM-1) and could be
217 ercellular adhesion molecule-1 (ICAM-1), and vascular cell adhesion molecule-1 (VCAM-1) and diabetes
218 levels than PDEs of the endothelial proteins vascular cell adhesion molecule-1 (VCAM-1) and endotheli
219 mposed of B78H1 cells transfected with human vascular cell adhesion molecule-1 (VCAM-1) and human mon
220 n inhibitory factor (rhMIF) in up-regulating vascular cell adhesion molecule-1 (VCAM-1) and intercell
222 ry signaling that controls expression of the vascular cell adhesion molecule-1 (VCAM-1) and monocyte
223 177 as well as expression of redox-sensitive vascular cell adhesion molecule-1 (VCAM-1) and monocyte
224 uction of the endothelial activation markers vascular cell adhesion molecule-1 (VCAM-1) and vascular
225 was abolished by CD29 blockade on hMSCs and vascular cell adhesion molecule-1 (VCAM-1) blockade on H
226 tercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule-1 (VCAM-1) expression by
227 tion, EGF treatment induced up-regulation of vascular cell adhesion molecule-1 (VCAM-1) expression in
229 cells was p38 MAPK-dependent while increased vascular cell adhesion molecule-1 (VCAM-1) expression wa
230 We hypothesized that molecular imaging of vascular cell adhesion molecule-1 (VCAM-1) expression wi
231 brain inflammation was used to induce global vascular cell adhesion molecule-1 (VCAM-1) expression.
232 iency also transiently augmented LPS-induced vascular cell adhesion molecule-1 (VCAM-1) expression.
233 e derivatives as novel, potent inhibitors of vascular cell adhesion molecule-1 (VCAM-1) expression.
234 y Wu et al presents additional evidence that vascular cell adhesion molecule-1 (VCAM-1) imaging in at
235 pression of inducible NO synthase (iNOS) and vascular cell adhesion molecule-1 (VCAM-1) in cultured r
236 wnstream target genes tissue factor (TF) and vascular cell adhesion molecule-1 (VCAM-1) in diabetic a
237 timulates the expression of pro-inflammatory vascular cell adhesion molecule-1 (VCAM-1) in endothelia
238 (1-100 microm) stimulated the expression of vascular cell adhesion molecule-1 (VCAM-1) in human coro
239 ur angiogenesis, but increased expression of vascular cell adhesion molecule-1 (VCAM-1) in primary cu
242 very late antigen-4 (VLA-4) with its ligand vascular cell adhesion molecule-1 (VCAM-1) is required f
243 tensile forces in their normal function, and vascular cell adhesion molecule-1 (VCAM-1) is typical in
244 croarray analysis of P0 and P3 revealed that vascular cell adhesion molecule-1 (VCAM-1) is up-regulat
245 vasive imaging of adhesion molecules such as vascular cell adhesion molecule-1 (VCAM-1) may identify
247 ctions mediated via alpha4beta1 integrin and vascular cell adhesion molecule-1 (VCAM-1) on myeloma ce
248 free of endotoxin were incapable of inducing vascular cell adhesion molecule-1 (VCAM-1) or tumor necr
249 (very late activation antigen-4 [VLA-4]) and vascular cell adhesion molecule-1 (VCAM-1) play a major
250 dhesion mediated by integrin alpha4beta1 and vascular cell adhesion molecule-1 (VCAM-1) plays a cruci
253 cess on CXC chemokine receptor 2 (CXCR2) and vascular cell adhesion molecule-1 (VCAM-1) using null st
254 y associated with survival were defined, and vascular cell adhesion molecule-1 (VCAM-1) was the gene
255 bit neurite outgrowth on recombinant soluble vascular cell adhesion molecule-1 (VCAM-1), a known liga
256 effect of t-RA exposure on cell adhesion to vascular cell adhesion molecule-1 (VCAM-1), a well-estab
257 intercellular adhesion molecule-1 (ICAM-1), vascular cell adhesion molecule-1 (VCAM-1), and E-select
258 e synthase (iNOS), cyclooxygenase-2 (COX-2), vascular cell adhesion molecule-1 (VCAM-1), and manganes
259 rcellular adhesion molecule-1 (ICAM-1), anti-vascular cell adhesion molecule-1 (VCAM-1), anti-fibrin,
260 cellular adhesion molecule-1 (MAdCAM-1) and vascular cell adhesion molecule-1 (VCAM-1), endothelial
261 ion strengthening on the alpha4beta1 ligand, vascular cell adhesion molecule-1 (VCAM-1), in shear flo
262 ctivator 2, decay accelerating factor (DAF), vascular cell adhesion molecule-1 (VCAM-1), manganese su
263 acrophage chemoattractant protein-1 (MCP-1), vascular cell adhesion molecule-1 (VCAM-1), nuclear fact
264 lium and subsequent induction of endothelial vascular cell adhesion molecule-1 (VCAM-1), which is req
265 with very late antigen-4 (VLA-4) binding to vascular cell adhesion molecule-1 (VCAM-1), which is upr
266 ssing the endothelial cell adhesion molecule vascular cell adhesion molecule-1 (VCAM-1), which normal
267 ly as a consequence of reduced expression of vascular cell adhesion molecule-1 (VCAM-1), which regula
278 key cytokines, and cell adhesion molecules (vascular cell adhesion molecule 1 [VCAM-1] and intracell
279 issue inhibitor of metalloproteinases 1; and vascular cell adhesion molecule 1 [VCAM-1]) were measure
280 ts of SLPs (fibrinogen, fibronectin, soluble vascular cell adhesion molecule-1 [VCAM-1], intercellula
281 ty lipoprotein (VLDL)-stimulated endothelial vascular cell adhesion molecule 1 (VCAM1) induction and
282 of intercellular adhesion molecule 1 (ICAM1)/vascular cell adhesion molecule 1 (VCAM1), whose express
284 ic kidney, which was reversed by blockade of vascular cell adhesion molecule-1 (VCAM1) and very late
287 F-kappaB-responsive genes, but expression of vascular cell-adhesion molecule-1 (Vcam1) was particular
288 , vascular endothelial growth factor [VEGF], vascular cell adhesion molecule 1 [VCAM1], and GTP bindi
290 ellular adhesion molecule 1, E-selectin, and vascular cell adhesion molecule 1 was up-regulated on th
291 mooth muscle cell actin, and upregulation of vascular cell adhesion molecule-1 was also suppressed in
292 ctin, intercellular adhesion molecule 1, and vascular cell adhesion molecule 1) was slightly up- or d
293 ntercellular adhesion molecule 1 and soluble vascular cell adhesion molecule 1 were reduced after whe
295 let), E-selectin (selectin endothelium), and vascular cell adhesion molecule 1, were also upregulated
297 ecules intracellular adhesion molecule-1 and vascular cell adhesion molecule-1, which caused a fourfo
298 se-9, intercellular adhesion molecule-1, and vascular cell adhesion molecule-1, which might act in co
299 iency of monocytes sheared in whole blood on vascular cell adhesion molecule-1, while characterizing
300 idase [MPO], matrix metalloproteinase 9, and vascular cell adhesion molecule 1) would be increased an
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