ライフサイエンスコーパス: vascular change

1 use axonal injury and other forms of diffuse vascular change.

2 -714 PET, especially in diseases that induce vascular change.

3 flow and sensory afferent to the ACC-induced vascular change.

4 was graded 1 to 4 for increasing severity of vascular change.

5 findings in AMN, with attention to choroidal vascular changes.

6 ated maculopathy (ARM) is related to adverse vascular changes.

7 d renal function, with minimal effect on the vascular changes.

8 P inhibitors for 1 hour affects the diabetic vascular changes.

9 studied to document progressive intracranial vascular changes.

10 important mediator for these early and late vascular changes.

11 esult of neuronal dysfunction with secondary vascular changes.

12 ung disease associated with severe pulmonary vascular changes.

13 sies were graded 0 to 4 for interstitial and vascular changes.

14 culature, and astrocytic mechanosensation of vascular changes.

15 europathogenesis were analyzed for pulmonary vascular changes.

16 amination instead of by evaluation of actual vascular changes.

17 administration of STI571 reversed pulmonary vascular changes.

18 eration (72%), optic nerve pallor (68%), and vascular changes (64%) were present.

19 In conclusion, the vascular changes after anti-VEGF therapy can have a cont

20 that, in anti-VEGF-refractory murine tumors, vascular changes after VEGF ablation result in reduced d

21 Hot flashes may mark adverse underlying vascular changes among midlife women.

22 Although evidence suggests adverse vascular changes among women with hot flashes, it is unk

23 the effects of AG on aging-related renal and vascular changes and AGE accumulation were studied in no

24 therapeutics and imaging agents to sites of vascular changes and astrogliosis in diseases associated

25 performed after 2008, and demonstrated early vascular changes and decreased platelet counts.

26 shed sterol 27-hydroxylase activity leads to vascular changes and identify Cyp27a1-/- mice as a model

27 tion of VEGF and MIP-1 alpha, leading to the vascular changes and microglial activation necessary to

28 igate the effect of actual delivered dose on vascular changes and to test the appropriateness of the

29 Although vascular changes and transplant vasculopathy have been d

30 We monitored cell infiltration, vascular changes and tumor progression during inflammati

31 risk factor for both large and small vessel vascular changes, and conversely other vascular risk fac

32 athogenesis of early lesions of scleroderma, vascular changes, and fibrosis and perhaps lead us towar

33 be expanded to include torpedo maculopathy, vascular changes, and hemorrhagic retinopathy.

34 ood, but it is suspected that the associated vascular changes are a result of anti-HLA Ab-mediated in

35 These vascular changes are driven by the immune response to th

36 These vascular changes are due to tracing, as opposed to probl

37 -retinal barrier (BRB) breakdown and related vascular changes are implicated in several ocular diseas

38 These data support the concept that early vascular changes are involved in the pathogenesis of OAG

39 lecular components underlying these dramatic vascular changes are not well characterized.

40 However, the mechanisms producing these vascular changes are not well defined.

41 ic hernia (CDH), where the typical pulmonary vascular changes are present before birth and are accomp

42 In the neonatal stage, inflammation and vascular changes are reversible and may define an import

43 ologic studies have identified apoptosis and vascular changes as significant novel findings in human

44 thrombospondin-1 KO background reversed the vascular changes as well as the delayed catagen exhibite

45 that this pathway underlies both matrix and vascular changes associated with chronic liver disease.

46 Cs and LECs and thereby regulates matrix and vascular changes associated with chronic liver injury.

47 ithfully replicates many of the hallmarks of vascular changes associated with human retinopathy, thus

48 in activation of the immune system and cause vascular changes associated with PAH.

49 cal or pathological processes, including the vascular changes associated with sepsis.

50 types, pathways, and molecules that control vascular changes before and after ischemia may result in

51 Cardiac oxidation, independently of vascular changes, can lead to uncoupled cardiac NOS and

52 de the established clinical and morphometric vascular changes caused by DM and represent a paradigm s

53 utic targets for intervention in progressive vascular changes common to cerebrovascular disease and A

54 fferential epigenetic markers and neuro-glio-vascular changes compared with nondiabetic mice.

55 iopsies revealed that tertiary follicles and vascular changes developed in the absence of acute skin

56 urpose of this study was to map and quantify vascular changes during progression of DR by VESsel GENe

57 s of psoriasis, including the characteristic vascular changes, epidermal alterations, and inflammator

58 Choroidal vascular changes have also been noted in AMD.

59 be helpful in understanding dynamic retinal vascular changes in a number of common ocular diseases,

60 e noninvasive semiquantitative evaluation of vascular changes in a streptozotocin (STZ)-induced mouse

61 In this study, we evaluated metabolic and vascular changes in AGER1 transgenic mice (AGER1-tg) sub

62 Histopathological vascular changes in all forms of PAH are qualitatively s

63 However, there were vascular changes in allografts from GN-treated mice, inc

64 Such vascular changes in astronauts could compromise the main

65 n prevent and reverse these diabetic retinal vascular changes in both new and established diabetes.

66 5LO pathway is important for mediating early vascular changes in diabetes.

67 ms allow for objective assessment of retinal vascular changes in eyes with DR that are correlated to

68 ional magnetic resonance imaging to evaluate vascular changes in HCC after sunitinib treatment.

69 fic drug-induced side effects in relation to vascular changes in healthy tissues.

70 In contrast, vascular changes in isografts of hypertensive hosts were

71 he patients in this study, important retinal vascular changes in MacTel 2 occur in the deep capillary

72 RATIONALE: Early vascular changes in metabolic disease that precipitate t

73 Although the mediators responsible for these vascular changes in mice have not been identified, VEGF-

74 s study, therefore, was to analyse lymphatic vascular changes in obese mice and to determine whether

75 quantitative PCR were used to assess retinal vascular changes in relation to apoptosis, and expressio

76 ic magnetic resonance (MR) imaging to depict vascular changes in response to cyclooxygenase-2 (COX-2)

77 ture could underlie attenuation of pulmonary vascular changes in response to elevated pressure, as we

78 matory mediators, and local inflammatory and vascular changes in response to subcutaneous injection o

79 to maternal high fat diets produces systemic vascular changes in the offspring.

80 stational hypertensive disorders may lead to vascular changes in the offspring.

81 in rat granulation tissue and corresponding vascular changes in the retina, sciatic nerve, and aorta

82 the major causes of blindness caused by neo-vascular changes in the retina.

83 function and prevents neurodegeneration and vascular changes in the retinas of streptozotocin (STZ)-

84 e current study, we investigated the retinal vascular changes in these animals.

85 The character of coronary vascular changes in transplanted hearts was distinctly a

86 g novel therapeutics expected to cause acute vascular changes in tumors.

87 Vascular changes included progressive subendothelial hya

88 ina3n but not SOCS3 gene expression, whereas vascular changes including angiogenesis and blood-brain

89 the best technique for measuring CNV and the vascular changes induced by anti-VEGF-A monoclonal antib

90 this combined blockade reversed normal organ vascular changes induced by Dll4 blockade alone.

91 gelatinases in particular reverse the renal vascular changes induced by pregnancy or relaxin adminis

92 re the role of nitric oxide in mediating the vascular changes induced by VEGF.

93 duced choroidal neovascularization (CNV) and vascular changes induced on CNV by an anti-VEGF-A monocl

94 Here, we consider whether vascular changes instead play a central role in disease

95 ut the pathogenesis of these As(III)-induced vascular changes is unknown.

96 ution of portal myofibroblasts (PMFs) to the vascular changes leading to cirrhosis.

97 The pathological vascular changes led to major disruption of inner retina

98 Pathological vascular changes may also include less well characterize

99 Alzheimer pathology and stroke through which vascular changes may influence dementia risk.

100 oning and cognition, with an emphasis on how vascular changes may mediate this disorder.

101 direct allograft infection, suggesting that vascular changes may not be limited to the allograft.

102 These vascular changes may represent a previously unrecognized

103 Early vascular changes occur in the absence of vEVTs, suggesti

104 c dissection of the hormonal, metabolic, and vascular changes occurring in such women during pregnanc

105 ade may attenuate the cerebral and pulmonary vascular changes of IH while augmenting cerebral angioge

106 le of angiopoietins and the Tie2 receptor in vascular changes of psoriasis.

107 nucleotidohydrolase induced hemodynamic and vascular changes of pulmonary hypertension in rats (n=8)

108 niques that can more precisely visualize the vascular changes of the whole joint can further enhance

109 ffort to provide the STandards for ReportIng Vascular changes on nEuroimaging (STRIVE).

110 s of SVD (as per the Standards for Reporting Vascular Changes on Neuroimaging [STRIVE] guidelines) an

111 form of pulmonary hypertension in which the vascular changes originate in the small pulmonary veins

112 Time-course experiments revealed that vascular changes preceded node transformation.

113 Because these metabolic and vascular changes predispose to atherosclerosis, monitori

114 to moderate myocardial fibrosis, and various vascular changes ranging from focal intimal thickening t

115 basis of the migraine attack with secondary vascular changes, reconciling previous theories into a n

116 , but the time course and development of any vascular changes remain largely unexplored.

117 The molecular mechanisms of these vascular changes remain to be determined.

118 in these dogs, but the pathogenesis of this vascular change remains unknown.

119 tive effect of this compound, MEM can reduce vascular changes seen in diabetic retinas.

120 chidonic acid and that it contributes to the vascular changes seen in this model.

121 These vascular changes should alert healthcare practitioners t

122 containing 30% galactose experience retinal vascular changes similar to those in human diabetic reti

123 cate that hyperhexosemia can trigger retinal vascular changes similar to those seen in human DR inclu

124 shaped macula was likely caused by choroidal vascular changes, similar to central serous chorioretino

125 medium-size collaterals representing a major vascular change that contributed to the restoration of v

126 sufficient to protect against the structural vascular changes that accompany hypertension in the sett

127 targets that regulate liver fibrosis and the vascular changes that accompany it and may have broad im

128 al step in liver fibrosis and the sinusoidal vascular changes that accompany this process.

129 iated with adverse structural and functional vascular changes that are most pronounced in children ex

130 e of CsA-treated rats may be involved in the vascular changes that are seen in this model.

131 ct of PAI-1 on intimal hyperplasia and other vascular changes that develop after arterial injury in a

132 n and renin in the kidney may underlie renal vascular changes that develop in diabetes.

133 ading depression is associated with profound vascular changes that may be a significant factor in the

134 mine release and granulocyte activity in the vascular changes that precede the onset of necrotic lesi

135 dothelial cells (ECs) and nonlytically cause vascular changes that result in hemorrhagic fever with r

136 plays a role in the chronic hypoxia-induced vascular changes that underlie pulmonary hypertension.

137 phoblast-specific transgene caused placental vascular changes that were consistent with excess VEGF a

138 mage might partly originate from HIV-induced vascular changes, the timing suggests that myelin and/or

139 ffects of aminoguanidine on diabetes-induced vascular changes versus parameters of nonenzymatic glyca

140 Associated with these vascular changes was increased elastin content in Adipo-

141 These vascular changes were independent of islet size and were

142 ryos with a vasoconstrictor, indicating that vascular changes were not a factor.

143 The vascular changes were not congophilic.

144 Vascular changes were not noted in gray matter 3 mm dist

145 No significant vascular changes were observed in isografts (mean medial

146 owed for 6 months, killed, and rejection and vascular changes were scored blinded to the group.

147 A consistent set of retinal vascular changes were seen in the eyes with MacTel 2.

148 These vascular changes were suppressed by the administration o

149 ressed--as well as Muller glia and secondary vascular changes, which were associated at later times w

150 The vascular changes, with the exception of PNAd, appear to