1  patients (4 with meniscus pathology, 2 with vascular insufficiency).

2 bition of healing, most likely from regional vascular insufficiency.

3 irreversible complication of lower extremity vascular insufficiency.

4 ular responses in a mouse hind limb model of vascular insufficiency.

5 g aetiology of ischaemic damage arising from vascular insufficiency.

6 uding symptoms and signs of inflammation and vascular insufficiency, acute-phase reactants (APRs), an

7 , and the reminder perished due to placental vascular insufficiency and compromised vessel integrity.

8                                              Vascular insufficiency and retinal ischemia precede many

9 Localized GCA symptoms are the end-result of vascular insufficiency and tissue ischemia.

10 mains in question, the presence of lymphatic vascular insufficiency can be ascertained through imagin

11                              Upper extremity vascular insufficiency dominated the clinical presentati

12                                              Vascular insufficiency has been reported to be a cause o

13                                              Vascular insufficiency in PAD occurs despite higher circ

14               In a rabbit hind limb model of vascular insufficiency, intramuscular activation of Akt1

15 G and non-glaucoma controls, suggesting that vascular insufficiency or dysregulation by themselves ma

16 from the management strategies for lymphatic vascular insufficiency states.

17       Adverse events consistent with ongoing vascular insufficiency such as limb necrosis or gangrene

18 ature in order to circumvent or mitigate the vascular insufficiency that leads to edema and tissue de

19 w experimental strategy for the treatment of vascular insufficiency, uses the administration of media