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1 lear phagocytes and exclude cells within the vascular lumen.
2 tly regulate the exit of leukocytes from the vascular lumen.
3 tin sulfate, and hyaluronic acid) lining the vascular lumen.
4 n the perivascular space, but not within the vascular lumen.
5 space with occasional processes probing the vascular lumen.
6 eliver antithrombotic drugs to the pulmonary vascular lumen.
7 om the circulation and from the walls of the vascular lumen.
8 helium and no localization to the irradiated vascular lumen.
9 mor thrombi that had completely occluded the vascular lumen.
10 ulates angiogenic genes and the formation of vascular lumens.
11 esion, which together drive the emergence of vascular lumens.
12 ll viability and establishment of perfusable vascular lumens.
14 P-selectin is rapidly translocated to the vascular lumen after tissue injury to initiate the adhes
15 helial cell shape, and formation of a patent vascular lumen all require defined endothelial cell pola
16 etween the endothelial cells, which line the vascular lumen, and associated mural cells, namely vascu
17 sed by 47 fold in the tissue surrounding the vascular lumen, as compared with non-targeted liposomes.
19 nging from focal intimal thickening to total vascular lumen blockade due to smooth muscle cell prolif
20 ve eicosanoids that are generated within the vascular lumen by leukocytes and transcellular biosynthe
21 his purpose if they could be anchored to the vascular lumen by targeting stably expressed, noninterna
24 ion defects, regression of valves, and focal vascular lumen collapse, which triggered generalized lym
25 supposed to exert therapeutic action in the vascular lumen (e.g., antithrombotic proteins), to the s
26 ons in humans; the lesions display disrupted vascular lumens, enlarged capillary cavities, loss of pr
28 during embryogenesis, including insufficient vascular lumen formation as well as defective arteriogen
30 herin in regulating endothelial polarity and vascular lumen formation is mediated through its interac
31 work has shown that FMNL3 is also needed for vascular lumen formation, a critical element of the form
37 s (WPBs) that release their content into the vascular lumen in response to specific agonists that rai
38 ation-induced P-selectin localization to the vascular lumen increased in time-dependent manner, until
39 oduction in the endothelial cells lining the vascular lumen is an important component of many vascula
40 on-induced localization of P-selectin to the vascular lumen is specific to the microvasculature of ma
41 lycosaminoglycan (GAG)-rich layer lining the vascular lumen, is associated with the onset of kidney i
43 radiation-induced P-selectin staining in the vascular lumen of neoplasms is associated with aggregati
44 ergo an early swelling with narrowing of the vascular lumen, resulting in prolonged renal hypoperfusi
45 step in the migration of leukocytes from the vascular lumen to the extravascular tissue, but fundamen
47 and its primary branches with regard to the vascular lumen, vessel wall anatomy, and vessel wall ede
48 the opportunity to assess the progression of vascular lumen volume in vivo after balloon angioplasty.
50 liomas showed P-selectin localization to the vascular lumen within 1 h, whereas blood vessels in norm
52 was accompanied by PMN degranulation within vascular lumen without PMN transmigration, likely becaus
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