ライフサイエンスコーパス: vascular occlusion

1 odialysis who subsequently developed retinal vascular occlusion.

2 Rs also enhance thrombotic and nonthrombotic vascular occlusion.

3 ncy procedure in 4 patients due to prolonged vascular occlusion.

4 tion, incompetent blood-retinal barrier, and vascular occlusion.

5 are ineligible for IV t-PA but have a large vascular occlusion.

6 toring blood flow to ischemic tissue after a vascular occlusion.

7 -threatening diseases that result from acute vascular occlusion.

8 were imaged before, during, and after renal vascular occlusion.

9 id adaptation of the lymphatic pump to local vascular occlusion.

10 silateral parietal cortex to ensure adequate vascular occlusion.

11 and the most common cause of acute pulmonary vascular occlusion.

12 heparin may be useful in preventing painful vascular occlusion.

13 ndothelium is important to the generation of vascular occlusion.

14 rease collateral blood flow after mesenteric vascular occlusion.

15 crosis and possibly plaque rupture and acute vascular occlusion.

16 estinal viability after segmental mesenteric vascular occlusion.

17 osure and secure hemostasis while preventing vascular occlusion.

18 state, resulting in neointima formation and vascular occlusion.

19 the key to hemostasis and also to pathologic vascular occlusion.

20 M)beta(2) microdomains leads to acute lethal vascular occlusions.

21 ed toxicities were chorioretinal atrophy and vascular occlusions.

22 Mice deficient in VN (n = 12) developed vascular occlusion 77 +/- 11 minutes after injury, inter

23 lial cells has been thought to contribute to vascular occlusion, a major cause of morbidity in sickle

24 on incidence also was increased with retinal vascular occlusions (aHR, 10, 95% CI, 3.0-33), retinal v

25 ge-related macular degeneration, and retinal vascular occlusions, among others.

26 Vascular occlusion and complex plexiform lesions are hal

27 This patient highlights the vascular occlusion and endothelial cell injury in the me

28 es inflammatory lung pathologic findings and vascular occlusion and hemorrhage.

29 urs during surgery involving renal or aortic vascular occlusion and is one of the leading causes of p

30 et activation during disease states leads to vascular occlusion and ischemic damage.

31 stologic examination of the same rete showed vascular occlusion and moderate intraluminal foreign bod

32 ive pulmonary vascular remodeling, including vascular occlusion and plexiform-like lesions, resemblin

33 echanism of reversing established neointimal vascular occlusion and pulmonary hypertension.

34 pholipid antibodies in patients with retinal vascular occlusion and the spectrum of ophthalmologic fi

35 systemic conditions associated with retinal vascular occlusions and offer guidelines for appropriate

36 ical examination shows no objective signs of vascular occlusion, and this can lead the clinician to s

37 and transiently immediately after pulmonary vascular occlusion, and this rise was blunted significan

38 n microscopic analysis of tumor sections for vascular occlusion; and analysis of tumor cryosections f

39 myocardial infarction, stroke and peripheral vascular occlusion are the leading causes of morbidity a

40 Vascular occlusions are common structural modifications

41 is a possible co-factor in atherogenesis and vascular occlusion, but its ability to actively infect m

42 soluble TM, prevents platelet activation and vascular occlusion by clots.

43 thus be attributed to impaired perfusion and vascular occlusion caused by deposition of Abeta and fib

44 Sickle cell disease (SCD) results in vascular occlusions, chronic hemolytic anemia, and cumul

45 frequency (24+/-2%) and severity (9+/-4%) of vascular occlusion compared with wild-type controls (n=7

46 lls into Rag2-deficient mice greatly reduced vascular occlusion compared with WT cells, clearly demon

47 athophysiology depicted with imaging include vascular occlusion, compensatory collateral flow, result

48 In patients with persistent vascular occlusions despite intravenous tissue plasminog

49 ls in 28, umbrella devices in 6 and a Grifka vascular occlusion device in 1.

50 tched on age and smoking who did not develop vascular occlusion during the prospective follow-up peri

51 he patient had no classical risk factors for vascular occlusions, except for the presence of Flammer

52 in vascular injury in sickle cell anemia, in vascular occlusion following the rupture of atherosclero

53 otein precursor, A beta PP) in the organized vascular occlusions harvested from patients with this di

54 emodialysis and subsequently develop retinal vascular occlusion have not been examined.

55 Eyes with recent retinal vascular occlusion illustrating paracentral acute middle

56 and parenchymal destruction with more severe vascular occlusion in grafts from IL-10 -/- recipients.

57 helial proteins is believed to contribute to vascular occlusion in sickle cell disease.

58 of irreversible PEm to ask whether pulmonary vascular occlusion in the absence of reperfusion is itse

59 ere significantly diminished, and thrombotic vascular occlusion in vivo was significantly blunted in

60 D) is characterized by recurring episodes of vascular occlusion in which neutrophil activation plays

61 suring hemostasis and thrombosis by inducing vascular occlusion in zebrafish larvae.

62 The presence of patent neovessels within vascular occlusions in chronic thromboembolic pulmonary

63 ce R rickettsii infection induces thrombotic vascular occlusions in patients with Rocky Mountain Spot

64 we concluded that the extensive formation of vascular occlusions in PD-susceptible grapevines does no

65 ear-resistant adhesions ex vivo and unstable vascular occlusions in vivo.

66 Retinal vascular occlusion is considered a risk factor for cardi

67 liferative processes that ultimately lead to vascular occlusion is enhanced by the abnormal metabolic

68 Vascular occlusion is the major cause of morbidity and m

69 d strategies targeting gpIIbIIIa may prevent vascular occlusion, maintain tissue viability, and provi

70 strategies that target gpIIbIIIa may prevent vascular occlusion, maintain tissue viability, and provi

71 get platelet adherence molecules may prevent vascular occlusion, maintain tissue viability, and reduc

72 bed with the following categories: bleeding, vascular occlusion (myocardial infarction, stroke and pu

73 were acute stroke (n = 3), aneurysm (n = 3), vascular occlusion (n = 3), and disk herniation (n = 2).

74 ) receptors, whereas, in contrast, (3) brief vascular occlusion not associated with appreciable adeno

75 vessel wall is believed to contribute to the vascular occlusion observed in patients with sickle call

76 The intermittent vascular occlusion occurring in sickle cell disease (SCD

77 Thrombotic vascular occlusion occurs in disorders of diverse etiolo

78 Vascular occlusions of the retina and choroid can cause

79 iuretic peptide, and percentage of pulmonary vascular occlusion on chest computed tomography.

80 ase, such as diabetic retinopathy or retinal vascular occlusion, one would refer to the lesion as PAM

81 nce of substantial progression (P=0.008), or vascular occlusion (P=0.005) when compared with the mode

82 ical dynamics for oxygen, growth factors and vascular occlusions, replicate key clinical observations

83 Thrombotic vascular occlusion resulting in infarction occurs during

84 lead to pathological thrombus formation and vascular occlusion, resulting in stroke, myocardial infa

85 l toxoplasmosis, retinal vasculitis, retinal vascular occlusions, rhegmatogenous and serous retinal d

86 terial neointimal formation (quantified by a vascular occlusion score) was more severe in vehicle-tre

87 score, 1.98 +/- 0.02) than in Group PMS5-35 (vascular occlusion score, 0.59 +/- 0.46; p < 0.001).

88 lls was more severe in vehicle-treated rats (vascular occlusion score, 1.98 +/- 0.02) than in Group P

89 parenchymal infiltration and less prominent vascular occlusion than day 2 murine CTLA4Ig-treated or

90 vascular endothelium could contribute to the vascular occlusion that characterizes the acute crisis o

91 After 7 days, all embolic agents caused vascular occlusion that led to necrosis and fibrosis.

92 Patients develop vascular occlusions that cause cardiovascular, cerebrova

93 This study focuses on vascular occlusions that form in stem secondary xylem of

94 Patients develop painful neuropathy and vascular occlusions that progressively lead to cardiovas

95 diurnal variation in the time to thrombotic vascular occlusion (TTVO) subsequent to a photochemical

96 Patients who develop thrombotic vascular occlusions usually have underlying vascular dis

97 ques that precipitate acute atherothrombotic vascular occlusion ("vulnerable plaques") are abundant i

98 Retinal vascular occlusion was introduced by rose Bengal and las

99 Vascular occlusion was not permanent, and even when embo

100 essed vasodilatory response to SNP caused by vascular occlusion was reversed with training.

101 Vascular occlusion was substantially increased when BM-d

102 omocysteine concentration, a risk factor for vascular occlusion, was examined in a representative sam

103 By adjusting the severity of the vascular occlusion, we were able to generate hypertensiv

104 nically relevant model of stroke utilizing a vascular occlusion with a freshly formed clot, and evalu

105 may be possible to inhibit platelet-mediated vascular occlusion with a minimal effect on primary hemo

106 large areas of splenic necrosis secondary to vascular occlusion with hepatocytes, whereas direct sple

107 na spacing were recorded and correlated with vascular occlusion with logistic regression analysis.