ライフサイエンスコーパス: vascular tumor

1 re closed using particles filling the entire vascular tumor.

2 should be aware of this low-grade cutaneous vascular tumor.

3 antile hemangioma (IH) is a common childhood vascular tumor.

4 ead to novel therapeutic treatments for this vascular tumor.

5 y to show direct evidence of EPCs in a human vascular tumor.

6 he pathogenesis of both benign and malignant vascular tumors.

7 diagnosis of congenital hemangioma for these vascular tumors.

8 a mutant FOS protein we identified in these vascular tumors.

9 g treatment strategy because GBMs are highly vascular tumors.

10 ential therapeutic agent in this category of vascular tumors.

11 erefore was not due to the selection of more vascular tumors.

12 rocess may be useful in treating cancers and vascular tumors.

13 gioendotheliomas and perhaps other cutaneous vascular tumors.

14 essing cells were present at the site of the vascular tumors.

15 a higher incidence of metastasis than poorly vascular tumors.

16 ype, including those of benign and malignant vascular tumors (0 of 3 hemangiomas, 0 of 1 hemangioendo

17 ment for Kaposi's sarcoma (KS), an enigmatic vascular tumor and a model for pathologic angiogenesis.

18 ely, animals lacking both genes develop many vascular tumors and also present with medulloblastomas n

19 in as a novel treatment for nonmetastasizing vascular tumors and for Kasabach-Merritt syndrome.

20 y of beta adrenergic inhibition on malignant vascular tumors and have laid the groundwork for a promi

21 rt that the loss of Notch1 caused widespread vascular tumors and organism lethality secondary to mass

22 Hif-1alpha did not affect the development of vascular tumors and polycythemia, nor did it suppress th

23 nt HIF in the pathogenesis of VHL-associated vascular tumors and that pharmacologic targeting of HIF-

24 Kaposi's sarcoma is a highly vascular tumor, and recently both hypoxia-inducible fact

25 genic human HCC cells into highly aggressive vascular tumors, and inhibition of AEG1 abrogated tumori

26 11 can cause certain types of rare childhood vascular tumors, and we have now identified causal recur

27 Vascular tumors are among the most common neoplasms in i

28 Vascular tumors are endothelial cell neoplasms whose mec

29 Vascular tumors are not a known complication of antiangi

30 -twin transfusion syndrome, lung masses, and vascular tumors, are highlighted.

31 Vascular tumors arising after intradermal injection of i

32 Kaposi's sarcoma (KS), a vascular tumor associated with human immunodeficiency vi

33 n the therapeutic algorithm for IH and other vascular tumors awaits controlled study.

34 The angiograms showed an intense vascular tumor blush in recurrent mass lesions supplied

35 Purpose To quantify initial changes in the vascular tumor burden (VTB), a measure of the area of va

36 changes ranging from erythematous maculae to vascular tumors, by the presence of spindle and inflamma

37 Beta blockade is selective for vascular tumor cells over normal endothelial cells and s

38 Conversely, S6K signaling was increased in vascular tumor cells where Akt3 was silenced, and the gr

39 Kaposi's sarcoma is a vascular tumor commonly associated with human immunodefi

40 blocking autocrine VEGF signaling abolished vascular tumor development and growth.

41 that local application of imiquimod inhibits vascular tumor enlargement in the mouse vascular tumor m

42 r soft part sarcoma (ASPS) is a rare, highly vascular tumor, for which no effective standard systemic

43 etiologic agent of Kaposi's sarcoma (KS), a vascular tumor frequently found in immunodeficient indiv

44 del describes the spatiotemporal dynamics of vascular tumor growth and treats cells as distinct entit

45 ilding upon previous work, here we develop a vascular tumor growth model by coupling a continuous gro

46 We apply our recently developed vascular tumor growth model which couples a continuous g

47 potential for PDT-BPD to inhibit selectively vascular tumor growth was tested in a mouse angiosarcoma

48 a, (2) mechanistic modeling of avascular and vascular tumor growth, and (3) modeling of cancer initia

49 ation, where Akt1 promotes and Akt3 inhibits vascular tumor growth.

50 transgenic animals expressing vGPCR manifest vascular tumors histologically identical to human KS, wi

51 with the adjacent myocardium, malignant and vascular tumors hyper-enhanced, whereas stromal tumors a

52 wledge, angiomyolipomas are the first benign vascular tumor in which the vascular cells, rather than

53 ative lesions from vascular malformations to vascular tumors in adult mice.

54 n tumors, male hepatocellular tumors, and in vascular tumors in both sexes.

55 ation of endothelial cell-derived intramural vascular tumors in the implantation site.

56 ence that chronic Notch1 inhibition leads to vascular tumors in the liver and decreased survival, whi

57 cally that the development of VHL-associated vascular tumors in the liver depends on functional ARNT.

58 t typically regress by 5 years of age; other vascular tumors include congenital tufted angiomas (TAs)

59 It is characterized clinically by vascular tumors including benign hemangioblastomas of th

60 e characterized by the development of highly vascular tumors including hemangioblastomas of the retin

61 es in the global gene expression patterns of vascular tumors, including alterations in the expression

62 Lindau disease (VHL) patients develop highly vascular tumors, including central nervous system hemang

63 red in a series of central cartilaginous and vascular tumors, including samples from syndromic and no

64 choroidal hemangioma is an uncommon, benign vascular tumor manifesting as an orange-red mass in the

65 were indeed present and therefore aggressive vascular tumors may similarly show increased susceptibil

66 bits vascular tumor enlargement in the mouse vascular tumor model suggests a novel, less toxic means

67 Vascular tumors occur in approximately 10% of all infant

68 represent a heterogeneous group of malignant vascular tumors occurring not only in different anatomic

69 angiofibromas (JNAs) are locally aggressive vascular tumors occurring predominantly in adolescent ma

70 ogically associated with Kaposi's sarcoma, a vascular tumor of endothelial cells.

71 Kaposi's sarcoma (KS) is a vascular tumor of proliferative endothelial cells caused

72 ndle cell hemangioma (SCH) is a rare, benign vascular tumor of the dermis and subcutis.

73 he pathogenesis, diagnosis and management of vascular tumors of infancy in the past year.

74 ds to VHL disease, which is characterized by vascular tumors of the central nervous system, renal cle

75 sia syndromes and is characterized by highly vascular tumors of the eyes, brain, and spine, as well a

76 tify GNA14 mutations as a cause of childhood vascular tumors, offer insight into mechanisms of oncoge

77 rus 8 (KSHV/ HHV8), has not been reported in vascular tumors other than KS.

78 to the nanodrug to inhibit the expression of vascular tumor protein laminin-411 in order to block tum

79 Vascular tumors ranged in severity from angiomas to hema

80 ent 129/J mice with life-threatening primary vascular tumors reduced tumor volume 7.5-fold and almost

81 Histology disclosed intramural vascular tumors resembling hemangiomas in the VEGF-myobl

82 ession of only KDR observed in the malignant vascular tumors studied.

83 ession of these receptors on more aggressive vascular tumors such as hemangioendotheliomas and angios

84 markable efficacy in the treatment of benign vascular tumors such as infantile hemangiomas.

85 cept for the therapeutic utility of treating vascular tumors, such as angiosarcomas, with S6K inhibit

86 Available therapies for vascular tumors, such as systemic corticosteroids, vincr

87 ies have not fully realized their promise in vascular tumors, suggesting that these tumors do not dep

88 Congenital hemangioma is a rare vascular tumor that forms in utero.

89 In addition, GCT is a vascular tumor that may occasionally rupture and result

90 liomas are categorized as intermediate-grade vascular tumors that are commonly localized in the lungs

91 Congenital hemangiomas (CHs) are rare benign vascular tumors that differ from common infantile hemang

92 gnant gliomas are locally aggressive, highly vascular tumors that have a dismal prognosis, and presen

93 Renal angiomyolipomas are highly vascular tumors that occur sporadically, in women with p

94 Congenital hemangiomas are uncommon benign vascular tumors that present fully formed at birth.

95 Kaposi's sarcoma (KS) is an enigmatic vascular tumor thought to be a consequence of dysregulat

96 ry efficiently formed highly invasive and/or vascular tumors upon intracerebral implantation into imm

97 information were gathered, and sections from vascular tumors were stained with vascular endothelial g

98 Hepatocellular cancer is a highly vascular tumor, where the neovasculature is unique in th

99 elioid hemangioendothelioma (HEHE) is a rare vascular tumor which has an intermediate aggressive beha

100 These are highly vascular tumors which overproduce angiogenic peptides su

101 Infantile hemangioma is a common vascular tumor with a unique lifecycle: rapid growth in

102 Hepatocellular carcinoma (HCC) is a vascular tumor with poor prognosis.