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1 y to form tube-like structures on collagen ("vasculogenic mimicry").
2 ell dissemination by increasing invasion and vasculogenic mimicry.
3  cell invasion, migration, and inhibition of vasculogenic mimicry.
4 ddition to tumor cell plasticity as shown by vasculogenic mimicry.
5  adapted to a pseudoendothelial phenotype in vasculogenic mimicry.
6 sis that VE-cadherin is critical in melanoma vasculogenic mimicry.
7 in tyrosine kinases are involved in melanoma vasculogenic mimicry.
8 may not be applicable to tumors that express vasculogenic mimicry.
9 noma mitogen, laminin, a phenomenon known as vasculogenic mimicry.
10              In addition, Notch4 function in vasculogenic mimicry and anchorage-independent growth in
11  an essential role during the acquisition of vasculogenic mimicry and angiogenic properties associate
12 events that regulate the process of melanoma vasculogenic mimicry and identify new signal transductio
13 he ability of these tumor cells to engage in vasculogenic mimicry and neovascularization.
14 osome nucleation, motility, neoangiogenesis, vasculogenic mimicry, and osteoclastogenesis in the abse
15      MEF2C was found to mediate VEGF-induced vasculogenic mimicry, angiogenesis and migration/invasio
16 lial-cadherin, which plays a pivotal role in vasculogenic mimicry, as well as interleukin-8, fibronec
17 /or its cleavage fragments, are required for vasculogenic mimicry by aggressive melanoma cells.
18 y human melanoma cells in vivo and in vitro: vasculogenic mimicry," by Maniotis and colleagues, which
19                                              Vasculogenic mimicry describes a process where aggressiv
20 erized to invade aggressively and to undergo vasculogenic mimicry, expressed Mig-7.
21                          Recently, the term "vasculogenic mimicry" has been used by our laboratory an
22                                              Vasculogenic mimicry is posited to contribute to melanom
23 lular and molecular determinants of melanoma vasculogenic mimicry is presented.
24                     These data indicate that vasculogenic mimicry-like laminin networks, in addition
25    Mig-7 protein primarily co-localized with vasculogenic mimicry markers factor VIII-associated anti
26 EF2C on cell proliferation, and VEGF-induced vasculogenic mimicry, migration/invasion as well as angi
27  aggressive melanoma cells to participate in vasculogenic mimicry, particularly their expression of e
28 highly invasive uveal melanoma cells forming vasculogenic mimicry patterns after exposure to a lamini
29  tissue samples, uveal melanoma cells within vasculogenic mimicry patterns assumed the spindle A morp
30 s of primary uveal melanomas lacking looping vasculogenic mimicry patterns either did not stain for o
31   The histological detection of laminin-rich vasculogenic mimicry patterns in human primary uveal mel
32 on of human rather than mouse laminin in the vasculogenic mimicry patterns in this model demonstrates
33                      Thus, the generation of vasculogenic mimicry patterns is accompanied by dampenin
34                                              Vasculogenic mimicry patterns were reconstructed from se
35 highly invasive uveal melanoma cells forming vasculogenic mimicry patterns, and genes associated with
36                                              Vasculogenic mimicry patterns, composed of human laminin
37      In primary melanomas containing looping vasculogenic mimicry patterns, strong osteopontin staini
38                                After forming vasculogenic mimicry patterns, uveal melanoma cells inva
39 ith their increased invasion, migration, and vasculogenic mimicry plasticity.
40 echanical and molecular events that regulate vasculogenic mimicry provide opportunities for the devel
41      The unique patterning characteristic of vasculogenic mimicry provides an opportunity to design n
42 an carcinoma cells could engage in molecular vasculogenic mimicry reflected by their plasticity, comp
43         We recently have introduced the term vasculogenic mimicry to describe the unique ability of a
44 egulated, aggressive tumor cells was termed "vasculogenic mimicry" to emphasize their de novo generat
45                                              Vasculogenic mimicry (VM) describes the unique ability o
46 s found in patient tumor slices displaying a vasculogenic mimicry (VM) phenotype.
47                                   Tumor cell vasculogenic mimicry (VM) refers to the plasticity of ag
48 E-cadherin) and cytokeratins consistent with vasculogenic mimicry (VM), a process whereby tumour cell
49  display the ability to drive blood-perfused vasculogenic mimicry (VM), an alternative microvascular
50 in) and TIE1 and are associated with CD31(-) vasculogenic mimicry (VM), an established biomarker asso
51 alpha expression and an increase in CD144(+) vasculogenic mimicry (VM), leading to formation of chann
52                                              Vasculogenic mimicry (VM), the formation of matrix-rich
53 -2 expression with known prognostic factors, vasculogenic mimicry (VM), the mature vasculature (von W
54 ng vessel-like channels, a phenomenon termed vasculogenic mimicry (VM).
55 ternative microvascular circulation known as vasculogenic mimicry (VM).
56 nd participate in a PECAM1-dependent form of vasculogenic mimicry (VM).
57 rs have recently described a process termed "vasculogenic mimicry," which consists of the formation o

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