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1 P) as a second messenger (e.g., secretin and vasoactive intestinal polypeptide).
2 toxin-insensitive G(s)-coupled receptors for vasoactive intestinal polypeptide.
3 tion in response to sodium nitroprusside and vasoactive intestinal polypeptide.
4 Rs that do not distinguish between PACAP and vasoactive intestinal polypeptide.
5 a response not elicited by stimulation with vasoactive intestinal polypeptide.
6 sequence homology with both PACAP-27/38 and vasoactive intestinal polypeptide.
7 injured unmyelinated afferents labeled with vasoactive intestinal polypeptide.
8 rtical interneurons express either Reelin or vasoactive intestinal polypeptide.
9 AC receptor homo-oligomers were modulated by vasoactive intestinal polypeptide.
10 dal cells and GABA interneurons coexpressing vasoactive intestinal polypeptide.
12 ehavior, combined with genetic disruption of vasoactive intestinal polypeptide, a key SCN signaling m
13 al afferents, and contains neurons producing vasoactive intestinal polypeptide and gastrin-releasing
14 er, GTP gamma S binding induced by CCK-8 and vasoactive intestinal polypeptide and the binding capaci
16 ceptor or of the neuropeptides somatostatin, vasoactive intestinal polypeptide, and choleocystokinin.
18 neurons contained the inhibitory transmitter vasoactive intestinal polypeptide, and some were immunor
19 nolol, methysergide, substance P antagonist, vasoactive intestinal polypeptide antagonist, apamin, an
20 found in SCN cells, arginine vasopressin and vasoactive intestinal polypeptide appeared to be in cont
21 and the receptors for prostaglandin E(2) and vasoactive intestinal polypeptide, are not expressed or
22 AC1-VPAC2 hetero-oligomers were modulated by vasoactive intestinal polypeptide binding, whereas the s
23 co-localize with either cholecystokinin- or vasoactive intestinal polypeptide, but does with vasopre
24 n at least acetylcholine, adrenergic agents, vasoactive intestinal polypeptide, calcitonin gene-relat
25 pressing cyclooxygenase-2 (22%, p < 0.05) or vasoactive intestinal polypeptide-containing interneuron
26 unctions such as motility and secretion (eg, vasoactive intestinal polypeptide, cystic fibrosis trans
29 re, we used a transgenic mouse line in which vasoactive intestinal polypeptide-expressing (VIP+) GABA
30 at in contrast to somatostatin-expressing or vasoactive intestinal polypeptide-expressing interneuron
31 tory neurons reduced their activity, whereas vasoactive intestinal polypeptide-expressing interneuron
32 ubstance P, calcitonin gene-related peptide, vasoactive intestinal polypeptide, galanin, somatostatin
33 bodies with immunoreactive (IR) vasopressin, vasoactive intestinal polypeptide, gastrin-releasing pep
36 signaling pathways induced by kisspeptin and vasoactive intestinal polypeptide in GnRH neuronal cell
37 for the specification of neuropeptide Y and vasoactive intestinal polypeptide, indicating that a sub
39 ndin, calretinin, parvalbumin, somatostatin, vasoactive intestinal polypeptide, neuropeptide Y, or ch
42 ons, but not nonpyramidal neurons containing vasoactive intestinal polypeptide or neuropeptide Y.
43 light acting weakly upon a strongly rhythmic vasoactive intestinal polypeptide oscillation can explai
45 nitric oxide synthase (eNOS)-expressing and vasoactive intestinal polypeptide-positive enteric neuro
46 inergic, adrenergic, and nitrergic axons and vasoactive intestinal polypeptide-positive terminals, so
47 ealed an abnormal number and distribution of vasoactive intestinal polypeptide-producing neurons, sug
48 d by agonists such as prostaglandin E(2) and vasoactive intestinal polypeptide, promotes proliferatio
49 function, including cholinergic, adrenergic, vasoactive intestinal polypeptide, purinergic, androgen,
52 in, or with thyrotropin releasing hormone or vasoactive intestinal polypeptide resulted in abundant e
54 phorbol ester, epidermal growth factor, and vasoactive intestinal polypeptide stimulated p38 MAP kin
55 e exchange of action potentials that release vasoactive intestinal polypeptide, striking a compromise
56 gene-related peptide, tyrosine hydroxylase, vasoactive intestinal polypeptide, substance P, corticot
57 y preabsorption of PACAP-27/38 antisera with vasoactive intestinal polypeptide, suggesting that a sub
59 oxylase, neuronal nitric oxide synthase, and vasoactive intestinal polypeptide to visualize neural el
60 de for two other members of this family, the vasoactive intestinal polypeptide type 1 and calcitonin
62 (MT), corticotropin-releasing hormone (CRH), vasoactive intestinal polypeptide, tyrosine hydroxylase,
63 We demonstrate that interneurons expressing vasoactive intestinal polypeptide (VIP(+)) play a causal
64 Here, we show that interneurons expressing vasoactive intestinal polypeptide (VIP(+)) regulate the
65 of two neuropeptides synthesized in the SCN, vasoactive intestinal polypeptide (VIP) and arginine vas
66 hat type 3 IS (IS3) cells that coexpress the vasoactive intestinal polypeptide (VIP) and calretinin c
67 th light microscopic immunocytochemistry for vasoactive intestinal polypeptide (VIP) and cytoarchitec
68 -dependent coupling process mediated by both vasoactive intestinal polypeptide (VIP) and GABAA signal
70 also simulated clock phase shifts induced by vasoactive intestinal polypeptide (VIP) and matched expe
71 opalatine ganglion, which appears to utilize vasoactive intestinal polypeptide (VIP) and nitric oxide
72 an serous cells secrete fluid in response to vasoactive intestinal polypeptide (VIP) and other agents
73 adenomas, we investigated the expression of vasoactive intestinal polypeptide (VIP) and PACAP bindin
74 s this cyclic information to GnRH neurons is vasoactive intestinal polypeptide (VIP) and that it may
75 een the pelvic visceral afferent transmitter vasoactive intestinal polypeptide (VIP) and the delta-op
77 e used to ascertain the relationship between vasoactive intestinal polypeptide (VIP) and tyrosine hyd
78 L-arginine methyl ester (L-NAME), but not by vasoactive intestinal polypeptide (VIP) antiserum, guane
79 r AVD is a redundant system in which ACh and vasoactive intestinal polypeptide (VIP) are co-released
80 recent data implicating the neurotransmitter vasoactive intestinal polypeptide (VIP) as the key synch
81 racellular cAMP, was sufficient to stimulate vasoactive intestinal polypeptide (VIP) biosynthesis at
83 ) of an antibody raised by immunization with vasoactive intestinal polypeptide (VIP) cleaved this pep
84 , we investigated whether the projections of vasoactive intestinal polypeptide (VIP) from the SCN to
85 olar infusion of the VPAC1/2 receptor ligand vasoactive intestinal polypeptide (VIP) had no effect on
88 e cyclase-activating polypeptide (PACAP) and vasoactive intestinal polypeptide (VIP) have been found
89 e cyclase activating polypeptide (PACAP) and vasoactive intestinal polypeptide (VIP) immunoreactive (
90 ked sexual dimorphism in the distribution of vasoactive intestinal polypeptide (VIP) immunoreactive f
91 K), and receive input from galanin (GAL) and vasoactive intestinal polypeptide (VIP) immunoreactive f
93 d determine the role of mast cells (MCs) and vasoactive intestinal polypeptide (VIP) in barrier regul
94 were immunoreactive for vasopressin (AVP) or vasoactive intestinal polypeptide (VIP) in wild type and
96 and alpha5-knockout mice, lower activity of vasoactive intestinal polypeptide (VIP) interneurons res
102 ow that a class of interneurons that express vasoactive intestinal polypeptide (VIP) mediates disinhi
104 upled via gamma-aminobutyric acid (GABA) and vasoactive intestinal polypeptide (VIP) neurotransmitter
106 ssion and odor detection performance require vasoactive intestinal polypeptide (VIP) or its receptor
107 that contained arginine vasopressin (AVP) or vasoactive intestinal polypeptide (VIP) or neither.
109 nd these constituted a sub-population of the vasoactive intestinal polypeptide (VIP) positive cells.
110 ampal neurons in culture to demonstrate that vasoactive intestinal polypeptide (VIP) promotes neurona
111 All splice variants of PAC1 were found, but vasoactive intestinal polypeptide (VIP) receptor (VPAC)
116 ne hydroxylase, nitric oxide synthetase, and vasoactive intestinal polypeptide (VIP) to detect neural
120 ansmission was simulated via the addition of vasoactive intestinal polypeptide (VIP), a pelvic viscer
121 OR myenteric neurons were immunoreactive for vasoactive intestinal polypeptide (VIP), and about 31% w
122 ce labeling for nitric oxide synthase (NOS), vasoactive intestinal polypeptide (VIP), and choline ace
123 rneurons expressing neuropeptide Y (NPY) and vasoactive intestinal polypeptide (VIP), and the numeric
124 ne-related peptide (CGRP), substance P (SP), vasoactive intestinal polypeptide (VIP), and tyrosine hy
125 sine hydroxylase (TH), neuropeptide Y (NPY), vasoactive intestinal polypeptide (VIP), calcitonin gene
126 e (nNOS), choline acetyl transferase (ChAT), vasoactive intestinal polypeptide (VIP), calcitonin gene
127 presence and colocalization of the peptides vasoactive intestinal polypeptide (VIP), calcitonin-gene
128 e cyclase-activating polypeptide (PACAP) and vasoactive intestinal polypeptide (VIP), on ECL cell pro
130 r GnIH inhibits the action of kisspeptin and vasoactive intestinal polypeptide (VIP), positive regula
132 the nucleus characterized by a population of vasoactive intestinal polypeptide (VIP)-containing neuro
133 circuit in frontal cortex that originates in vasoactive intestinal polypeptide (VIP)-expressing inter
134 , but not that of somatostatin-expressing or vasoactive intestinal polypeptide (VIP)-expressing inter
135 OS)-, choline acetyltransferase (ChAT)-, and vasoactive intestinal polypeptide (VIP)-immunoreactiviti
136 stance P (SP)-IR varicosities and 9 +/- 1.3% vasoactive intestinal polypeptide (VIP)-IR varicosities
137 ion-induced release of substance P (SP)- and vasoactive intestinal polypeptide (VIP)-like immunoreact
145 N, physiological evidence suggests that only vasoactive intestinal polypeptide (VIP)/gastrin-releasin
149 s of somatostatin(+) (SST) (MGE-derived) and vasoactive intestinal polypeptide(+) (VIP) (CGE-derived)
150 ed on immunocytochemistry, that synthesis of vasoactive intestinal polypeptide was increased upon lid
151 acetyltransferase, and substance P, whereas vasoactive intestinal polypeptide was more abundant in v
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