ライフサイエンスコーパス: vasoconstricting

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1 ut requires nitric oxide release to suppress vasoconstricting 20-HETE synthesis.

2 toxin (PTX) (400 ng/ml) for 2 h blocked the vasoconstricting action of adenosine or N(6)-cyclohexyla

3 helin-1 (ET-1) are not limited to its potent vasoconstricting activity.

4 lar endothelium acts as a buffer against the vasoconstricting agent Ang II via release of vasodilator

5 and an increase in sensitivity to adrenergic vasoconstricting agents.

6 Because of the potent vasoconstricting and mitogenic effects of ET-1 and its i

7 The vasoconstricting and profibrotic cytokine endothelin-1 w

8 cular resistance is regulated in part by the vasoconstricting and relaxing effects of compounds such

9 nal glial cells are a possible source of the vasoconstricting ATP.

10 blood flow may involve both vasodilating and vasoconstricting components.

11 Vasoconstricting concentration-response relationships we

12 between endothelium-derived vasodilating and vasoconstricting factors and coronary vasoconstriction i

13 nhanced contractile responses to endothelial vasoconstricting factors, which increase the risk of cor

14 allowing it to be regulated by a variety of vasoconstricting hormones and neurotransmitters.

15 tive K(+) channels are inhibited by multiple vasoconstricting hormones via the protein kinase C (PKC)

16 of ET-1, an endothelium-derived peptide with vasoconstricting, mitogenic and angiogenic properties, i

17 Norepinephrine (NE) is a vasoconstricting neurotransmitter released from sympathe

18 al hyperplasia for endothelins (ETs), potent vasoconstricting peptides that also exert growth-promoti

19 These observations indicate that vasoconstricting post-junctional alpha(2)-adrenoceptors

20 Although the vasoconstricting properties of cocaine underlie its cere

21 mediated, in part, by the generation of the vasoconstricting prostanoid thromboxane A2 (TXA2).

22 The vasoconstricting protein endothelin-1 (ET-1) has been im

23 monary vascular resistance and the pulmonary vasoconstricting response to alveolar hypoxia, suggestin

24 tentiates an exaggerated afferent arteriolar vasoconstricting response to angiotensin II and opens po

25 lled by the balance between vasodilating and vasoconstricting systems that could finely tune local bl

26 levels cause hyperthermia during exercise by vasoconstricting the skin.

27 scle is trans-inhibited by activation of the vasoconstricting thromboxane A(2) prostanoid receptor (T

28 ce in vasodilating (prostacyclin [PGI2]) and vasoconstricting (thromboxane A2 [TxA2]) eicosanoids may

29 lance of several counteracting forces, i.e., vasoconstricting versus vasodilating, growth-promoting v

30 o of Tx-M to PGI-M, used to express relative vasoconstricting vs vasodilating effects, was 24% higher

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