ライフサイエンスコーパス: vasoconstriction

1 te 20-hydroxyeicosatetraenoic acid-dependent vasoconstriction.

2 terial carbon dioxide (P aC O2) and cerebral vasoconstriction.

3 e; 5 microM) prevented acute hypoxia-induced vasoconstriction.

4 m and chloride delivery, leading to afferent vasoconstriction.

5 ed spontaneous neuronal activity rather than vasoconstriction.

6 mechanism capable of attenuating sympathetic vasoconstriction.

7 el blockers, such as 4-aminopyridine, induce vasoconstriction.

8 ation, which uncouples NO/Hb interaction and vasoconstriction.

9 level of sympathetic activity there is more vasoconstriction.

10 ligatory role of AT1 Rb for pressure-induced vasoconstriction.

11 cular coupling response from vasodilation to vasoconstriction.

12 kinase activation, which conjointly trigger vasoconstriction.

13 ed contractions and increased URP-associated vasoconstriction.

14 s to the ability to blunt alpha1 -adrenergic vasoconstriction.

15 promotes smooth muscle cell contraction and vasoconstriction.

16 d to a marked impairment in reflex cutaneous vasoconstriction.

17 ole of astrocytes in PA flow/pressure-evoked vasoconstriction.

18 induction of inflammation and independent of vasoconstriction.

19 e ability of ATP to blunt alpha1 -adrenergic vasoconstriction.

20 tiazem or nifedipine attenuated S1P-mediated vasoconstriction.

21 protein kinase A inhibitor peptide-mediated vasoconstriction.

22 the ability to attenuate alpha1 -adrenergic vasoconstriction.

23 ainst these alterations, including augmented vasoconstriction.

24 erial innervation and sympathetic control of vasoconstriction.

25 W2871 each evoked modest afferent arteriolar vasoconstriction.

26 e it is accompanied by reduced ET-1-mediated vasoconstriction.

27 tric oxide leading to systemic and pulmonary vasoconstriction.

28 lter depolarization (60 mmol/L K(+))-induced vasoconstriction.

29 attenuated S1P-mediated afferent arteriolar vasoconstriction.

30 ible with a tacrolimus-induced preglomerular vasoconstriction.

31 ed MaxiK channel trans-inhibition as well as vasoconstriction.

32 tional assays as inhibitors of ACE-dependent vasoconstriction.

33 sodilatation, and enhanced hypoxic pulmonary vasoconstriction.

34 lter depolarization-induced (60 mmol l K(+)) vasoconstriction.

35 erebral blood flow (CBF), caused by cortical vasoconstriction.

36 ty, inhibition of transient BK currents, and vasoconstriction.

37 teinase K LFH <3 kDa inhibited ACE-dependent vasoconstriction.

38 a(2+)](i) that cause acute hypoxic pulmonary vasoconstriction.

39 on ECE activity and inhibited ECE-dependent vasoconstriction.

40 relaxation as well as phenylephrine-induced vasoconstriction.

41 bral blood flow from hypoxia were related to vasoconstriction.

42 cy and efficacy without changing URP induced vasoconstriction.

43 at perivascular adipose tissue (PVAT) causes vasoconstriction.

44 f Ang II, and led to complete suppression of vasoconstriction.

45 linking GPR75 activation to 20-HETE-mediated vasoconstriction.

46 A1-3 agonist VPC31143 induced dose-dependent vasoconstriction.

47 t depolarize smooth muscle cells, leading to vasoconstriction.

48 hannels to cause membrane depolarization and vasoconstriction.

49 skeletal muscle to blunt alpha1 -adrenergic vasoconstriction.

50 o mesenteric vessel muscle cells, leading to vasoconstriction.

51 zation that is required for pressure-induced vasoconstriction.

52 jacent vascular smooth muscle cells, causing vasoconstriction.

53 , which comprises bradycardia and peripheral vasoconstriction.

54 atment with an antioxidant regimen increased vasoconstriction.

55 in metabolic activation and blockade of skin vasoconstriction.

56 the key signal mediating activity-dependent vasoconstrictions.

57 mesenteric and hindquarters, but not renal, vasoconstrictions.

58 At the site of implantation, vasoconstriction (-10% mean reduction) was observed duri

59 mild exercise did not attenuate PE-mediated vasoconstriction (-32 +/- 5 and -46 +/- 3%).

60 Rho-kinase-dependent vasoconstriction accounted for approximately 60% of the

61 induced an ECM-like syndrome, causing brain vasoconstriction, adherence of activated leukocytes in t

62 e TEBV exhibited flow-mediated vasodilation, vasoconstriction after exposure to 1 muM phenylephrine a

63 hibited pressure- and depolarization-induced vasoconstriction, although CCt was a far more effective

64 We conclude that hypoxic pulmonary vasoconstriction and a profound catecholamine surge occu

65 al mechanisms by which uric acid could cause vasoconstriction and a progressive ateriolopathy were es

66 lls (PASMC) is a major trigger for pulmonary vasoconstriction and an important stimulus for PASMC pro

67 al inputs contribute to the daily control of vasoconstriction and blood pressure and suggest that clo

68 ving cells and the role of IKK2 in mediating vasoconstriction and blood pressure regulation.

69 Although acute hypoxia causes pulmonary vasoconstriction and chronic hypoxia can cause vascular

70 ressurized cerebral arteries rapidly induced vasoconstriction and depolarized cVSMCs.

71 in a development of significantly increased vasoconstriction and endothelial dysfunction.

72 may have opposing roles in the regulation of vasoconstriction and endothelium-dependent vasorelaxatio

73 on on cardiac function are likely because of vasoconstriction and ensuing ischemia.

74 itric oxide (NO) in the vasculature, causing vasoconstriction and eventually cardiovascular complicat

75 and changes in blood markers associated with vasoconstriction and fibrinolysis, suggesting that OO su

76 s into the bursa of wild-type mice prevented vasoconstriction and follicle rupture.

77 -body cooling would elicit greater cutaneous vasoconstriction and greater increases in skin sympathet

78 in myocardial injury as a result of coronary vasoconstriction and heightened oxidative stress.

79 rs were reactive to hyperoxygenation-induced vasoconstriction and hypercapnia-induced vasodilatation.

80 a form of fetal stress that stimulates renal vasoconstriction and ischaemia as a consequence of the p

81 larial is limited by its potent induction of vasoconstriction and its rapid degradation within minute

82 In conclusion, blunted peripheral vasoconstriction and lower stroke volume contribute to c

83 We tested the hypothesis that muscle vasoconstriction and muscle sympathetic nerve activity (

84 bolism by hypoxia to acute hypoxic pulmonary vasoconstriction and progression of pulmonary hypertensi

85 However, it enhanced ET-1 vasoconstriction and prolonged the increase in blood pre

86 esized that serelaxin could ameliorate renal vasoconstriction and renal dysfunction in patients with

87 Hypoxic pulmonary vasoconstriction and responses to exogenous sphingomyeli

88 ding follicle rupture in wild-type mice, but vasoconstriction and rupture were absent in Amhr2(cre/+)

89 ntral thermoregulatory control, and prevents vasoconstriction and shivering in blocked areas.

90 n, synchronously reducing the thresholds for vasoconstriction and shivering.

91 , spaceflight-induced reductions in myogenic vasoconstriction and stiffness and increases in maximal

92 ebral perfusion are associated with enhanced vasoconstriction and structural remodeling of cerebral a

93 y circulation to a low oxygen environment is vasoconstriction and structural remodelling of pulmonary

94 hese pathological responses promote regional vasoconstriction and subsequent blood vessel remodeling.

95 -dependent contributions to reflex cutaneous vasoconstriction and vascular adrenergic sensitivity wer

96 ), and that this mechanism explains cortical vasoconstriction and vascular dysfunction after CSD.

97 erial hypertension (PAH) is characterized by vasoconstriction and vascular remodeling.

98 n VVS, as in hemorrhage, impaired adrenergic vasoconstriction and venoconstriction result in hypotens

99 endent sensitivity to phenylephrine-mediated vasoconstriction and with decreased acetylcholine-mediat

100 amage, epicardial and microvascular coronary vasoconstriction and/or spasm, and increased cardiac wor

101 ectors studied (thermopreferendum, tail-skin vasoconstriction, and brown fat thermogenesis), thus sug

102 inhibition promoted increased proliferation, vasoconstriction, and inflammation.

103 ing skeletal muscle to attenuate sympathetic vasoconstriction, and is critical to ensure proper blood

104 d hyperthermia by moderately inhibiting skin vasoconstriction, and labetalol was ineffective.

105 in humans are sweating, arteriovenous shunt vasoconstriction, and shivering.

106 n on acute pulmonary hypertension induced by vasoconstriction, and to demonstrate denervation of the

107 The magnitude of vasoconstriction appeared graded with the number of cons

108 tricted diffusion, contrast enhancement, and vasoconstriction are all compatible with a diagnosis.

109 Importantly, we identified peripheral vasoconstriction as a critical mechanism underlying the

110 function and may relate as much to increased vasoconstriction as with decreased vasodilation.

111 We hypothesized that vasoconstriction at the apex is essential for rupture.

112 These results demonstrate that vasoconstriction at the apex of the follicle is essentia

113 by hypoxia, regulates neoplastic growth and vasoconstriction but its role in the regulation of pulmo

114 y adults is not due to augmented sympathetic vasoconstriction, but rather due to impairments in local

115 or of the renin-angiotensin system, promotes vasoconstriction by activating angiotensin AT1 receptors

116 Endothelin-induced vasoconstriction by hepatic stellate cells, and not plat

117 hile blunting the attenuation of sympathetic vasoconstriction by sensory nerves.

118 TXA2 induces vasoconstriction by way of activation of the thromboxane

119 Initial PO2-dependent vasoconstriction causes functional DA closure within min

120 means of preventing the damaging effects of vasoconstriction, central to ARF, but design of drugs wi

121 Extreme renal vasoconstriction characterizes hepatorenal syndrome, a f

122 y modulation of SNA preferentially increases vasoconstriction compared to a frequency-matched tonic p

123 Our results indicate that sympathetic vasoconstriction competes with endothelium-dependent dil

124 ther augmented SNS-mediated alpha-adrenergic vasoconstriction contributes to the age-associated impai

125 Electrically-induced vasoconstriction could be induced in seconds while blood

126 th mental activity, physical stimulation and vasoconstriction/dilation along with accurate determinat

127 on focus on the relationship between SNA and vasoconstriction during a pressor stimulus, which increa

128 ains the ability to blunt alpha1 -adrenergic vasoconstriction during combined KIR channel and Na(+) /

129 KCa channel remodeling, which contributes to vasoconstriction during diabetes mellitus.

130 Protocol 2) did not enhance alpha1 -mediated vasoconstriction during exercise (Protocol 1: -27 +/- 3%

131 ents with HF and SDB have more severe muscle vasoconstriction during hypoxia and hypercapnia than HF

132 one reduced feelings of warmth and increased vasoconstriction during social inclusion, especially for

133 bitors dose- and time-dependently attenuated vasoconstriction elicited by diverse agonists, suggestin

134 optosis (TWEAK), promoted pericyte-dependent vasoconstriction followed by pericyte detachment from ca

135 old-induced vascular response, consisting of vasoconstriction followed by vasodilatation, is critical

136 The results indicate that the impaired vasoconstriction following spaceflight occurs through th

137 nists able to induce systemic and mesenteric vasoconstriction have shown their usefulness in reducing

138 Hypoxic pulmonary vasoconstriction (HPV) maintains blood oxygenation durin

139 Hypoxic pulmonary vasoconstriction (HPV) optimizes pulmonary ventilation-p

140 (AEA) is a key mediator of hypoxic pulmonary vasoconstriction (HPV) via fatty acid amide hydrolase (F

141 -threatening hypoxemia via hypoxic pulmonary vasoconstriction (HPV) which matches perfusion to ventil

142 This phenomenon, hypoxic pulmonary vasoconstriction (HPV), preserves the overall efficiency

143 s been proposed to mediate hypoxic pulmonary vasoconstriction (HPV).

144 is proposed to result from hypoxic pulmonary vasoconstriction (HPV).

145 racting skeletal muscle to blunt sympathetic vasoconstriction (i.e. 'functional sympatholysis'), whic

146 ular effects, such as arterial and pulmonary vasoconstriction, impaired left ventricular (LV) relaxat

147 in healthy subjects is associated with acute vasoconstriction in a conductance artery and found sugge

148 cell TNF augments resistance artery myogenic vasoconstriction in a diabetes model that induces a smal

149 n astrocytes with intracellular BAPTA causes vasoconstriction in adjacent arterioles.

150 d subsequent impairments in reflex cutaneous vasoconstriction in ageing.

151 dulatory effects on hUII- and URP-associated vasoconstriction in an ex vivo rat aortic ring bioassay.

152 t, spironolactone inhibited alpha-adrenergic vasoconstriction in arterioles from mice and hypertensiv

153 et and inversion of NVC from vasodilation to vasoconstriction in brain slices obtained from subarachn

154 t necessary to properly modulate sympathetic vasoconstriction in contracting muscle, and that age-ass

155 er showed that therapy attenuated adrenergic vasoconstriction in contracting muscle.

156 Sympathetic vasoconstriction in contracting skeletal muscle is blunt

157 vasodilatation can blunt alpha1 -adrenergic vasoconstriction in contracting skeletal muscle of human

158 othelium-dependent regulation of sympathetic vasoconstriction in contracting skeletal muscle, and spe

159 4; 55 +/- 2 years) and (ii) augmented reflex vasoconstriction in HTN would be mediated by an increase

160 for sympathetic reflex control of cutaneous vasoconstriction in HTN.

161 ing skeletal muscle to attenuate sympathetic vasoconstriction in humans.

162 While Sphk1 is important in mediating vasoconstriction in hypertension, Sphk1(-/-) mice were c

163 ociated with decreased aortic and mesenteric vasoconstriction in hypertensive Sphk1(-/-) mice.

164 XA2 antagonists can inhibit the PVAT-induced vasoconstriction in male and female PCAs, respectively.

165 SRF controls vasoconstriction in mesenteric arteries via vascular SM

166 e the role of cardiac output and sympathetic vasoconstriction in neurally mediated (pre)syncope.

167 ced from vascular endothelial cells, induces vasoconstriction in physiological conditions.

168 ve activity but modulates blood pressure and vasoconstriction in POTS women during orthostatic stress

169 al activity but modulates blood pressure and vasoconstriction in POTS women during tilting.

170 0.001-10 muM) evoked concentration-dependent vasoconstriction in preglomerular microvessels, predomin

171 of sympathetic innervation and to defective vasoconstriction in resistance arteries.

172 ', however, ADRA2A and ADRA2C, implicated in vasoconstriction in response to cold and pain stimuli, s

173 process, and in the physiological process of vasoconstriction in response to hypoxia, remains unclear

174 ables the endothelium to moderate adrenergic vasoconstriction in response to sympathetic nerve activi

175 Stimulation of alpha-adrenoceptors elicits vasoconstriction in resting skeletal muscle that is blun

176 e ability of ATP to blunt alpha1 -adrenergic vasoconstriction in resting skeletal muscle would be ind

177 izing resistance vessels to pressure-induced vasoconstriction in systolic HF.

178 egulating vascular tone are shifted to favor vasoconstriction in the absence of GRK2 expression and t

179 contractility with an exaggerated peripheral vasoconstriction in the CHF state.

180 y to test the hypothesis that cocaine causes vasoconstriction in the human coronary microcirculation.

181 ha2(-/-) resistance arteries, insulin caused vasoconstriction in the presence of PVAT, and AMPKalpha2

182 Our findings indicate that LPA causes vasoconstriction in VSMCs, mediated by LPA1-, Gi-, and C

183 Impaired postsynaptic alpha1-adrenergic vasoconstriction in young adults with VVS can be correct

184 icantly higher pressor responses and greater vasoconstrictions in the offspring.

185 Throughout MA networks, vasoconstriction increased with PNS frequency (1-16 Hz)

186 ventilation, polycythemia, hypoxic pulmonary vasoconstriction-increased intracellular oxidative enzym

187 (STS)-135 shuttle mission] enhances myogenic vasoconstriction, increases medial wall thickness, and e

188 obin scavenges nitric oxide, which can cause vasoconstriction, induce inflammation, and activate plat

189 However, CCF was able to inhibit vasoconstriction induced by the thromboxane A2 receptor

190 he aorta and small retinal arterioles to the vasoconstriction-inducing drug U46619 was reduced.

191 s effects of angiotensin (Ang) II, including vasoconstriction, inflammation, water and salt retention

192 Therefore, we named the peptide vasoconstriction-inhibiting factor (VIF).

193 Excessively increased peripheral vasoconstriction is a hallmark of heart failure (HF).

194 Intravascular pressure-induced vasoconstriction is a smooth muscle cell-specific mechan

195 ested the hypothesis that alpha1 -adrenergic vasoconstriction is augmented during exercise following

196 in-1 (ET-1) in levels sufficient to initiate vasoconstriction is considered to be a hallmark feature

197 Hypoxic pulmonary vasoconstriction is correlated with pulmonary vascular r

198 We tested the hypothesis that SNS-mediated vasoconstriction is greater in older than young adults a

199 Reflex cutaneous vasoconstriction is impaired in older adults; however, t

200 TGF-mediated vasoconstriction is limited by the simultaneous release

201 The initial cold-induced vasoconstriction is mediated via TRPA1-dependent superox

202 Vasoconstriction is not simply a response to reduced thi

203 nal pathologies, the underlying mechanism of vasoconstriction is understood incompletely.

204 quent exercise training on low-flow mediated vasoconstriction (L-FMC) has not previously been studied

205 from perivascular sympathetic nerves, causes vasoconstriction largely via P2X1 receptors.

206 Oxygen-induced vasoconstriction may serve as a protective mechanism to

207 an important component of hypoxic pulmonary vasoconstriction, memory, and circadian rhythms, and dis

208 ay variations in response to agonist-induced vasoconstriction, myosin phosphorylation, and ROCK2 acti

209 nded periods of waking, and terminated, with vasoconstriction, near the nadir of daily ambient temper

210 rocesses, including catecholamine synthesis, vasoconstriction, neuronal function, and inflammation.

211 ockers on HPV and also on normoxic pulmonary vasoconstriction (NPV) stimulated by prostaglandin F2alp

212 on, dilatation and inhibition of sympathetic vasoconstriction observed in Young MAs were lost in Old

213 Hypoxic pulmonary vasoconstriction occurred following WLST, and was associ

214 r angiotensin 2) into the bursa restored the vasoconstriction of apical vessels and ovulation.

215 Vasoconstriction of apical vessels occurred within hours

216 PM4 currents in cerebral artery myocytes and vasoconstriction of cerebral arteries.

217 ological inhibition of S1P synthesis reduced vasoconstriction of mesenteric arteries.

218 50 4 (Cyp4)-derived eicosanoid that enhances vasoconstriction of renal vessels and induces hypertensi

219 lial cell apoptosis, hypercoagulability, and vasoconstriction of specific arteries and arterioles sup

220 P<0.0001), in keeping with exercise-induced vasoconstriction of stenosed epicardial segments and dil

221 TGF-induced vasoconstriction of the afferent arteriole results from

222 thelial-dependent vasomotor responses showed vasoconstriction of the arteries of the ApoE(-/-) (-22.2

223 cause some of its effects may be mediated by vasoconstriction of the coronary vasculature rather than

224 we studied the consequences of ET-1-mediated vasoconstriction of the middle cerebral artery in a rat

225 beta-methylene ATP (300 nm) evoked sustained vasoconstrictions of 18.7 +/- 3.2% (P < 0.05).

226 ROS in contributing to the fetal peripheral vasoconstriction, part of the fetal defence to hypoxia.

227 maintained the ability to blunt PE-mediated vasoconstriction (PE-mediated DeltaFVC: KIR blockade alo

228 e to reduced thickness of the follicle wall; vasoconstriction persisted in wild-type mice when thinni

229 physiologic responses such as cell adhesion, vasoconstriction, platelet aggregation, angiogenesis, in

230 cterized by reduced alpha-adrenergic-induced vasoconstriction, reduced endothelium-dependent vasodila

231 Sympathetic vasoconstriction regulates peripheral circulation and co

232 ATP attenuated PE-mediated vasoconstriction relative to adenosine (ADO) and sodium

233 Vasoconstriction required metabotropic glutamate recepto

234 ed vessel density, and displayed a decreased vasoconstriction response compared to Day 0 networks.

235 mice and showed no weakness in the tail skin vasoconstriction response or thermogenic response to col

236 Arterioles displayed a vasoconstriction response to KCl and endothelin for each

237 tinib treatment attenuated hypoxic pulmonary vasoconstriction responses and increased susceptibility

238 ex III is required for the ROS signaling and vasoconstriction responses to acute hypoxia in pulmonary

239 ng mild exercise significantly attenuated PE vasoconstriction similar to levels observed during moder

240 onary arterial hypertension (PAH), promoting vasoconstriction, smooth muscle proliferation, and infla

241 Angiotensin II also reduced vasoconstriction stimulated by Psora-4 or 4-aminopyridin

242 olipstatin also augmented the Ang II-induced vasoconstriction, suggesting that endocannabinoid releas

243 TS: Intravascular ATP attenuates sympathetic vasoconstriction (sympatholysis) similar to what is obse

244 r ATP attenuate sympathetic alpha-adrenergic vasoconstriction (sympatholysis).

245 Reversible cerebral vasoconstriction syndrome (RCVS) is a clinical-angiograp

246 Reversible cerebral vasoconstriction syndrome (RCVS) is characterized by rec

247 romuscular dysplasia and reversible cerebral vasoconstriction syndrome was described.

248 ous sinus thrombosis and reversible cerebral vasoconstriction syndrome.

249 Simultaneous [Ca(2+)]i fluorescence and vasoconstriction testing showed reduced Ca(2+), leading

250 expression may contribute to the heightened vasoconstriction that characterizes pulmonary hypertensi

251 nSMase had synergistic effects on pulmonary vasoconstriction that involved TRPC6, phospholipase C, a

252 of AZ10419369 exerted an extracranial tissue vasoconstriction that was comparable to the less blood-b

253 genous nSMase caused TRPC6 translocation and vasoconstriction that were blocked by CFTR inhibition.

254 ivery of blood to active brain regions cause vasoconstriction that would limit cerebral blood flow.

255 h as croup, may be related to airway mucosal vasoconstriction through a nongenomic mechanism.

256 itric oxide (NO) attenuates alpha-adrenergic vasoconstriction, thus optimizing perfusion.

257 iods would induce adverse effects (pulmonary vasoconstriction, tissue injury, inflammation, and plate

258 Hg) of intravascular pressures, and reduced vasoconstriction to iberiotoxin and vasodilation to NS16

259 scle cells responsible for hypoxic pulmonary vasoconstriction to limit perfusion of poorly ventilated

260 Vasoconstriction to NE was also determined in mesenteric

261 Robust vasoconstriction to PE was observed during vasodilator i

262 CK inhibitor H-1152 abolished basal tone and vasoconstrictions to ET-1 and PDBu.

263 to caffeine increased pressor responses and vasoconstrictions to phenylephrine, accompanied by enhan

264 tually masks oxygen consumption and balances vasoconstriction toward adulthood.

265 y and increased endothelin 1 (ET-1)-mediated vasoconstriction, two abnormalities contributing to vasc

266 levations in brain temperature and sustained vasoconstriction, two critical factors associated with M

267 gh Tie2-CYP4F2-Tr aortas displayed increased vasoconstriction, vasorelaxation and blood pressure were

268 sient BK currents in myocytes and stimulated vasoconstriction via a PKC-dependent mechanism that requ

269 1P evokes segmentally distinct preglomerular vasoconstriction via activation of S1P1 and/or S1P2 rece

270 travascular ATP modulates alpha1 -adrenergic vasoconstriction via pathways independent of KIR channel

271 Surprisingly, vasoconstriction was also diminished in vessels lacking

272 Vasoconstriction was attenuated in O.

273 Here, PE-mediated vasoconstriction was blunted at rest (blockade: -20 +/-

274 In vitro, flow/pressure-evoked vasoconstriction was blunted when the astrocytic syncyti

275 PE-mediated vasoconstriction was calculated (%DeltaFVC) in each cond

276 BAPTA-induced vasoconstriction was eliminated by a general COX blocker

277 muscle contraction to attenuate NE-mediated vasoconstriction was impaired, resulting in functional i

278 f local application of ZM323881, significant vasoconstriction was observed in the venules of diabetic

279 Diesel exhaust-related vasoconstriction was primarily observed in the variant a

280 Significant paradoxical vasoconstriction was seen in groups B and C.

281 Similarly, PGF2alpha-mediated vasoconstriction was symmetrically regulated by co-treat

282 channels in buffering phenylephrine-induced vasoconstrictions was decreased, whole cell BKCa current

283 tream molecular events specific to transient vasoconstriction, we studied the consequences of ET-1-me

284 is, where respiratory modulated increases in vasoconstriction were mediated by a noradrenergic mechan

285 Forearm and calf alpha1-adrenergic vasoconstriction were unimpaired in VVS and unaffected b

286 Vasoconstrictions were also observed in response to sing

287 R microangiography depicted retinal arterial vasoconstriction when the animals were breathing oxygen

288 agonist suramin blunted flow/pressure-evoked vasoconstriction, whereas K(+) and 20-HETE signaling blo

289 vasodilations were followed by a VP-mediated vasoconstriction, which acted to limit the magnitude of

290 drugs also induced dose-dependent peripheral vasoconstriction, which appears to be a primary mechanis

291 ABSTRACT: Myogenic vasoconstriction, which reflects the intrinsic ability o

292 This anomalous vasoconstriction, which would potentiate hypoxia, raises

293 y reduced the sensitivity of MAs to alpha1AR vasoconstriction while blunting the attenuation of sympa

294 in their environment: increases in PO2 cause vasoconstriction while decreases in PO2 result in vasodi

295 ntioxidants augmented diesel exhaust-related vasoconstriction with a mean change in BAd of -0.18 mm (

296 Therapeutic targeting of renal vasoconstriction with serelaxin in the rat models increa

297 urrent thunderclap headaches and evidence of vasoconstriction with subsequent resolution.

298 Regional reductions in CBF, and associated vasoconstriction, within the default mode network in hyp

299 nSMase- and hypoxia-induced vasoconstriction, yet not TRPC6 translocation, were bloc

300 ng skeletal muscle to blunt the stimulus for vasoconstriction, yet the underlying signalling of this