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1 c acid to both epoxyeicosatrienoic acids and vasoconstrictive 19,20-hydroxyeicosatetraenoic acid, the
4 I (UII) is a cyclic neuropeptide with strong vasoconstrictive activity in the peripheral vasculature.
5 , fibroblast growth factor-2, and the potent vasoconstrictive agent endothelin-1 as compared with con
7 ngiotensin II receptor AGTR1, which mediates vasoconstrictive and inflammatory signaling in vascular
10 oxyeicosatetraenoic acid (20-HETE), a potent vasoconstrictive and proinflammatory arachidonic acid me
14 s a propensity to hemolyze in vivo, releases vasoconstrictive cell-free hemoglobin over days, worsens
15 ge, 14.8 years) during passive exposure to a vasoconstrictive cold chamber (8 degrees C to 10 degrees
19 wise, cross-linking of free Hb may limit its vasoconstrictive effect by preventing abluminal movement
21 nd its analogues, such as octreotide, have a vasoconstrictive effect in cirrhotic patients and experi
26 eprandial conditions octreotide enhances the vasoconstrictive effect of dependent vasoconstrictors.
27 d vein dilation (dynamic analysis) and had a vasoconstrictive effect on both arteries and veins (stat
28 protein kinase C, octreotide exerts a local vasoconstrictive effect on vascular smooth muscle of SMA
33 result of iNOS activity, may antagonize the vasoconstrictive effects of a number of mediators (i.e.,
34 's and Bergman's(1) discovery in 1898 of the vasoconstrictive effects of an extract of rabbit kidney
35 a vasoregulatory peptide that modulates the vasoconstrictive effects of angiotensin II by acting on
37 e hypothesize that the cellular toxicity and vasoconstrictive effects of depolarizations act in syner
38 opose that OA-NO2 and Nrf2 may alleviate the vasoconstrictive effects of ET-1 by removing it from the
40 lly, instead of 100% oxygen, to minimize the vasoconstrictive effects of pure O(2) on retinal blood f
46 ithrombotic effects used in the treatment of vasoconstrictive/ischemic diseases including pulmonary a
47 distinct vascular responses and identify the vasoconstrictive mechanism as Neuropeptide Y (NPY) actin
48 ses the possibility that both myocardial and vasoconstrictive mechanisms may contribute to the increa
49 opathy by causing renal ischemia mediated by vasoconstrictive metabolites of the prostanoid pathway.
50 rotein acts as an inhibitor of physiological vasoconstrictive pathways, and a low RGS2 level is assoc
52 plays a central role in counterbalancing the vasoconstrictive, proliferative, and hypertensive peptid
53 bolic acidosis, possibly attributable to the vasoconstrictive properties of smokable "crystal" metham
54 ptic conditions, S-EITU was expected to have vasoconstrictive properties only in the septic phase.
61 lity (both P < 0.05), and a 33% reduction in vasoconstrictive reactivity to 5-HT trending toward sign
63 % confidence interval (CI): 1.2, 4.3) and 2) vasoconstrictive recreational drugs (defined as cocaine,
64 st, the % max SkBF-[BQ788] curve indicated a vasoconstrictive response (Hill slope -4.69 +/- 3.85, -4
65 of this sensitivity may be due to an altered vasoconstrictive response induced by lipopolysaccharide
68 Hepatic microcirculatory dysfunction and the vasoconstrictive response to endothelin-1 were also obse
69 te hepatic fibrogenesis, enhance the hepatic vasoconstrictive response to endothelin-1, and aggravate
70 n, microcirculatory dysfunction, an enhanced vasoconstrictive response to endothelin-1, and an increa
72 sent study demonstrates an enhanced coronary vasoconstrictive response to pathophysiological doses of
73 endothelial dysfunction is characterized by vasoconstrictive response to the endothelium-dependent v
76 fferent molecular dimensions correlates with vasoconstrictive responses in the microcirculation is un
77 pulmonary rat artery ring segments increased vasoconstrictive responses to Ang II and ET-1, and impli
82 ACE inhibitors alter the balance between the vasoconstrictive, salt-retentive, and hypertrophic prope
84 d volume that leads to maximal activation of vasoconstrictive systems, renal vasoconstriction, and re
85 d volume that leads to maximal activation of vasoconstrictive systems, renal vasoconstriction, and re
86 es renal function and reduces free radicals, vasoconstrictive thromboxanes, and tubulointerstitial fi
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