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1 erized 1 or 5 days before treatment with the vasoconstrictor agents.
2 nhibited by intracellular ATP ([ATP](i)) and vasoconstrictor agents and can be activated by vasodilat
3 cterized by nonresponsiveness to sympathetic vasoconstrictor agents and the subsequent irreversible f
4 or, or withdrawal of, conventional inotropic vasoconstrictor agents (i.e., dopamine and norepinephrin
6 d by various physiological stimuli including vasoconstrictor agents such as noradrenaline, depletion
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