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1 g in significantly greater production of the vasodilating 11,12-EET and 14,15-EET.
2 helial dysfunction and (2) resistance to the vasodilating action of insulin.
3 These peptides mediate antiproliferative and vasodilating actions.
4                                              Vasodilating agents induce relaxation of mesangial cells
5 per group), the perfusion responses to these vasodilating agents were similar in all three AIA groups
6  in glial cells, triggering glial release of vasodilating agents.
7  (NO) and carbon monoxide (CO) are important vasodilating agents.
8 ome P-450 epoxygenase that possess important vasodilating and anti-inflammatory properties.
9 with nebivolol, a beta1-selective agent with vasodilating and antioxidant properties.
10  underlying this effect may involve a direct vasodilating and antiproliferative action of endogenous
11 at regulation of blood flow may involve both vasodilating and vasoconstricting components.
12  by an imbalance between endothelium-derived vasodilating and vasoconstricting factors and coronary v
13 laries are controlled by the balance between vasodilating and vasoconstricting systems that could fin
14 ), which have natriuretic, renin-inhibiting, vasodilating, and lusitropic properties.
15 have effects on the RV, in addition to their vasodilating/antiproliferative effects in pulmonary arte
16  and safety of a fixed-dose combination of a vasodilating beta blocker (nebivolol) and an angiotensin
17                            Carvedilol, a new vasodilating beta-adrenoceptor antagonist and a potent a
18                          Carvedilol, a novel vasodilating beta-blocker with antioxidant activity, sig
19            We hypothesized that nebivolol, a vasodilating beta-blocker, could improve endothelial dys
20 ity and cardiovascular event data on the new vasodilating beta-blockers (eg, carvedilol and nebivolol
21                                              Vasodilating beta-blockers have beneficial effects on ce
22 eview is to summarize the latest findings on vasodilating beta-blockers in terms of central effects o
23                                              Vasodilating beta-blockers preferentially improve centra
24                                 Furthermore, vasodilating beta-blockers, particularly nebivolol, have
25                                    The newer vasodilating beta-blocking agents and calcium antagonist
26  coronary arteries would lead to a decreased vasodilating capacity and increased risk of coronary spa
27 nhibitors were first-line antiarrhythmic and vasodilating drugs.
28               We conclude that the pulmonary vasodilating effect of inhaled nitrite results from its
29 nd carotid arteries and in portal veins, the vasodilating effect of isoproterenol was reduced in mice
30 ontrol of the heart; 5) the discovery of the vasodilating effect of sympathetic stimulation on blood
31 oflurane is a volatile anesthetic that has a vasodilating effect on cerebral vessels producing a cere
32 elaxing factor (nitric oxide) exerts a tonic vasodilating effect on coronary collateral channels deve
33                However, in these vessels the vasodilating effect was only abolished in double KO mice
34          These findings demonstrate that the vasodilating effects of enalapril in the skeletal muscle
35 eptor blockade with ifetroban on the chronic vasodilating effects of enalapril in the skeletal muscle
36 ular smooth muscle cells, where they mediate vasodilating effects of endogenous catecholamines.
37 asodilator that can potentiate the pulmonary vasodilating effects of inhaled NO.
38                                   If so, the vasodilating effects of nitrite would be attenuated by f
39 tance blood vessels (mediating dose-limiting vasodilating effects) and even at supratherapeutic conce
40 used to express relative vasoconstricting vs vasodilating effects, was 24% higher (95% CI, 6%-45%) in
41 lcium increases B-cells/NPCs release ATP and vasodilating factors that activate purinergic receptors
42 acting forces, i.e., vasoconstricting versus vasodilating, growth-promoting versus growth-inhibiting,
43  peptide hormone relaxin-2, is a pleiotropic vasodilating hormone that has been studied in patients w
44 tive K(+) channels are activated by multiple vasodilating hormones and neurotransmitters via PKA.
45 dioprotective potency relative to the potent vasodilating K(ATP) openers (e.g., cromakalim).
46 etic peptide (BNP) is a cardiac peptide with vasodilating, lusitropic, and natriuretic properties tha
47                                      BNP has vasodilating, natriuretic, and renin-inhibiting properti
48 atriuretic peptide is a cardiac hormone with vasodilating, natriuretic, renin-angiotensin inhibiting,
49 ationale of this study was based on BNP as a vasodilating, natriuretic, renin-inhibiting and lusitrop
50 erioles via hypoxia-facilitated reduction to vasodilating nitric oxide.
51 redundant signal compensates for the loss of vasodilating PGs, and (3). NO and PG signals appear to c
52 erapy may improve hepatic blood flow via the vasodilating properties of PGE1.
53 ckers, have additional endothelium-dependent vasodilating properties specifically related to microcir
54 c, glomerular filtration rate-enhancing, and vasodilating properties that go beyond myocardial action
55 oves oxygenation and has selective pulmonary vasodilating properties without causing significant syst
56                              An imbalance in vasodilating (prostacyclin [PGI2]) and vasoconstricting
57 ed the hypothesis that nitric oxide (NO) and vasodilating prostaglandins (PGs) contribute independent
58 at endothelium-derived nitric oxide (NO) and vasodilating prostaglandins (PGs) contribute to the augm
59  of synthesis of either nitric oxide (NO) or vasodilating prostaglandins (PGs) would not alter exerci
60 vergent effects on the vascular synthesis of vasodilating prostaglandins.
61                                     However, vasodilating prostanoids do not appear to be important.
62 erfusion pressure and totally normalized the vasodilating response of the liver vasculature to acetyl
63  free-radical gas with potent antimicrobial, vasodilating, smooth muscle relaxant, and growth factor
64 tion occurs in the human forearm, and if the vasodilating substance nitric oxide contributes to this
65 ion intensity, prior to alterations in local vasodilating substances that influence vascular tone.
66 urs of treatment, wild-type controls stopped vasodilating to nitroglycerin, and the vascular sensitiv
67 ile at the same time prolonging diastole and vasodilating with acetylcholine (ACh) to maximize corona

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