ライフサイエンスコーパス: vasodilation

1 tibody significantly attenuated NaHS-induced vasodilation.

2 istance were acquired at baseline and during vasodilation.

3 r quality of life despite exerting pulmonary vasodilation.

4 NO activates myocyte BK channels and induces vasodilation.

5 s released, which induces local and systemic vasodilation.

6 ) sensitivity in arterial myocytes to induce vasodilation.

7 increased vasoconstriction as with decreased vasodilation.

8 ronal activation, enhancing the accompanying vasodilation.

9 steroid-sensing site in BK beta1, rendering vasodilation.

10 the ability of stored RBCs to effect hypoxic vasodilation.

11 te cyclase (sGC), resulting in cGMP-mediated vasodilation.

12 ia ratio, and maintained endothelium-derived vasodilation.

13 ng mechanisms, and how obesity disturbs this vasodilation.

14 ide, leading to smooth muscle relaxation and vasodilation.

15 annels to reduce myogenic tone, facilitating vasodilation.

16 s consistent with decreased angiogenesis and vasodilation.

17 ) sensitivity of IK and SK channels to cause vasodilation.

18 sease severity, endothelial dysfunction, and vasodilation.

19 trite is a physiological effector of hypoxic vasodilation.

20 with age, which was caused by reduced local vasodilation.

21 stimulation of IK and SK channels to promote vasodilation.

22 this bioactivation process is essential for vasodilation.

23 itical for NO-stimulated cGMP production and vasodilation.

24 to the adjacent smooth muscle cells causing vasodilation.

25 nophosphate (cGMP) pathway and is coupled to vasodilation.

26 olarizing factor, or prostaglandins to cause vasodilation.

27 Ca(V)1.2 channel proximal N terminus induces vasodilation.

28 d nitrite aerosol both resulted in pulmonary vasodilation.

29 aintenance of resting and agonist-stimulated vasodilation.

30 cing total Ca(V)1.2 and inducing the largest vasodilation.

31 (38 mm Hg), suggesting significant pulmonary vasodilation.

32 lar coupling, suppressing activity-dependent vasodilation.

33 s can produce selective and potent pulmonary vasodilation.

34 sociated with impaired endothelium-dependent vasodilation.

35 and NF-kappaB subunit knockdown, leading to vasodilation.

36 stimulating vascular DP1 receptors to cause vasodilation.

37 ic calcium (Ca(2+)) signaling to cause local vasodilation.

38 rtly restores NOS activity and NOS-dependent vasodilation.

39 nflux via TRPA1 causes endothelium-dependent vasodilation.

40 gnaling, integrity and endothelium-dependent vasodilation.

41 synthesis, and impairs endothelium-dependent vasodilation.

42 due to an inability to increase NO dependent vasodilation.

43 he classic physiological response of hypoxic vasodilation.

44 le guanylate cyclase (sGC) and cGMP-mediated vasodilation.

45 P from the red cell which has been linked to vasodilation.

46 n important physiologic regulator of hypoxic vasodilation.

47 at is independent of phasic, neuronal-evoked vasodilation.

48 echanisms underlying GLP-1R agonist-mediated vasodilation.

49 t vascular tone following activity-dependent vasodilation.

50 g the desensitization of GPCRs important for vasodilation.

51 as the dominant mechanism underlying hypoxic vasodilation.

52 emia/reperfusion injury, cytoprotection, and vasodilation.

53 ylation of KV1 channels in cVSMCs to promote vasodilation.

54 xpression and diminished BK channel-mediated vasodilation.

55 duced the following: (1) a massive cutaneous vasodilation; (2) drastic drops in deep brain temperatur

56 assessed the contribution of MPs to arterial vasodilation, a mechanism that contributes to portal hyp

57 o-photon imaging revealed prompt peritubular vasodilation after fluvoxamine treatment, which was bloc

58 ains has been implicated in red cell-induced vasodilation, although the mechanism for this process ha

59 ated a significant increase in flow-mediated vasodilation and a decrease in systolic blood pressure w

60 s revealed a hypoxia response and changes in vasodilation and angiogenesis genes that strongly suppor

61 fy the inflammatory response, but also cause vasodilation and angiogenesis.

62 of such association may compromise cerebral vasodilation and blood flow.

63 2Y2 or Gq/G11 deficiency lacked flow-induced vasodilation and developed hypertension that was accompa

64 SAH, signaling events that normally lead to vasodilation and enhanced delivery of blood to active br

65 Here we show in rats that NMDA-induced vasodilation and hemodynamic responses evoked by whisker

66 s is controversial because of concerns about vasodilation and hypotension.

67 and its supplying vasculature, resulting in vasodilation and increased blood flow.

68 O2/H(+) where an increase in CO2/H(+) causes vasodilation and increased blood flow.

69 effect in normoxia, but produced significant vasodilation and increased nitrosylation during hypoxaem

70 Administration of nimodipine induced vasodilation and increased pial blood flow.

71 anylyl cyclase generates cGMP, which induces vasodilation and inhibits platelet activation.

72 (RHI), which measures endothelium-dependent vasodilation and is a surrogate marker of endothelial fu

73 brachial artery macrovascular flow-mediated vasodilation and microvascular reactive hyperemia (p < 0

74 impact of IFN-alpha on mediators that induce vasodilation and modulate inflammation, including endoth

75 is associated with impaired insulin-induced vasodilation and mTORC1 signaling.

76 e diverse physiological functions, including vasodilation and neurotransmission.

77 e-induced improvement in nutrient-stimulated vasodilation and nutrient delivery to muscle rather than

78 may be a novel mechanism underlying impaired vasodilation and oxygen delivery during hypoxemia with a

79 CB(1) cannabinoid receptors (CB(1)R) induces vasodilation and reduces blood pressure, we have tested

80 n, which acted to limit the magnitude of the vasodilation and served to reset vascular tone following

81 ure supports not only the role of splanchnic vasodilation and systemic vasoconstriction but also hear

82 vascular smooth muscle is implicated in the vasodilation and vascular hyporeactivity underlying sept

83 ibution of purinergic signaling to disturbed vasodilation and vascular remodeling during atherosclero

84 Its opening and closure lead to vasodilation and vasoconstriction, respectively.

85 helium-dependent and -independent arteriolar vasodilation and venular leukocyte and platelet adhesion

86 rease that results primarily from peripheral vasodilation, and a subsequent increase driven by metabo

87 the modern era that is related to excessive vasodilation, and most frequently caused by obesity, art

88 ocytic cells activates NADPH oxidase, limits vasodilation, and promotes renal IRI.

89 r glucose uptake and utilization, autophagy, vasodilation, and proton removal, as demonstrated by qua

90 quantify plaque area, vascular permeability, vasodilation, and stiffness and post-triggering to ident

91 ignificant mediator of nitroglycerin-induced vasodilation, and tolerance to nitroglycerin is associat

92 ose produced by angiotensin-(1-7), including vasodilation, antifibrosis, antihypertensive, and centra

93 g other biological functions of NO including vasodilation, antimicrobial, anticancer, and neurotransm

94 The initial vasodilation appears caused by respiratory depression-in

95 for this stress-induced shift in mediator of vasodilation are proposed.

96 physiological ramifications of RBC-mediated vasodilation are unknown, and the apparently essential n

97 d endothelial markers, endothelium-dependent vasodilation, arteriolar glycocalyx size, and glomerular

98 solution of retinal detachment and choroidal vasodilation as well as improved visual acuity.

99 y, magnetic resonance imaging, flow-mediated vasodilation (assessed as the FMD%), the ankle-brachial

100 c S1PR2 agonists may serve to counteract the vasodilation associated with anaphylactic shock.

101 The concurrent vasodilation associated with increases in Ca(2+) event f

102 blood pressure, contributing to the arterial vasodilation associated with portal hypertension.

103 liotransmitters is widely assumed to trigger vasodilation associated with rapid increases in neuronal

104 was also used to elucidate mechanisms of SCS vasodilation at these higher frequencies.

105 othelial function, measured by flow-mediated vasodilation before and 6 hours after severe acute pancr

106 independently associated with flow-mediated vasodilation (beta = 0.1, p = 0.03), reactive hyperemia

107 and bacteria, being involved in signalling, vasodilation, blood clotting and immunity and as an inte

108 nsduces signals important for flow-dependent vasodilation, blood vessel remodeling, and atheroscleros

109 helium disruption inhibits carvacrol-induced vasodilation, but block of nitric-oxide synthase and cyc

110 ctive in selectively enhancing the pulmonary vasodilation by imatinib and sildenafil.

111 nitrate, GTN) in blood vessels, resulting in vasodilation by nitric oxide (NO) or a related species.

112 a role in S-nitrosothiol (SNO)-based hypoxic vasodilation by RBCs.

113 that astrocytes provide tonic, steady-state vasodilation by releasing prostaglandin messengers.

114 na, 100% O(2) reduced light-evoked arteriole vasodilations by 3.9-fold and increased vasoconstriction

115 ohemoglobin], which has been shown to induce vasodilation, by a rapid radical-radical reaction of any

116 significantly improved endothelium-dependent vasodilation, by both forearm venous occlusion plethysmo

117 mpaired cardiac function or primary arterial vasodilation can delay or prevent recovery from ischemic

118 We found that DAR induced hepatic sinusoidal vasodilation, caused more transplanted cells to be depos

119 tor and better understanding of its roles in vasodilation, cell permeability, platelet function, infl

120 it causes a strong side effect of cutaneous vasodilation, commonly called flushing.

121 ercholesterolemia, K(+)(Ca) channel-mediated vasodilation compensates for the reduced nitric oxide ac

122 Local increases in blood flow--'hypoxic vasodilation'--confer cellular protection in the face of

123 (BK) channels prevented both neurally evoked vasodilation (control) and vasoconstriction (SAH).

124 leads to a predominant nitric oxide-mediated vasodilation (Delta19 +/- 2%).

125 nel and transient BK current activation, and vasodilation did not involve Rab11A S177.

126 us CAD, resulting in an altered capacity for vasodilation during disease.

127 edly less sensitive to nitroglycerin-induced vasodilation (EC(50)=39.2 +/- 10.7 mumol/L) than wild-ty

128 art rate, contractility, lusitropy, arterial vasodilation, endothelial function, and venous return.

129 xtensive evidence now indicates that hypoxic vasodilation entails S-nitrosothiol-based (SNO-based) va

130 ounds (922+/-38mg GAE/kg), it did not induce vasodilation, even at the highest dose tested (0.206g/L)

131 Propionate, a SCFA shown to induce vasodilation ex vivo, produces an acute hypotensive resp

132 atus was assessed by measuring flow-mediated vasodilation (FMD), brachial pulse wave velocity (bPWV),

133 ere changes in brachial artery flow-mediated vasodilation (FMD), carotid-femoral pulse wave velocity

134 the change in brachial artery flow-mediated vasodilation (FMD).

135 Recent work has proposed iFR as a vasodilation-free alternative to FFR for making mechanic

136 5% CI, 0.90-1.31; P=0.39), and flow-mediated vasodilation (HR, 0.85; 95% CI, 0.69-1.04; P=0.12) were

137 itric oxide levels and endothelium-dependent vasodilation in a murine model and ex vivo human tissues

138 peroxide and inhibited nitric oxide-mediated vasodilation in a SIRP-alpha-dependent manner.

139 (JNK) in db/db PVAT restored insulin-induced vasodilation in an adiponectin-dependent manner.

140 re-establishing a physiological mechanism of vasodilation in arterioles from subjects with CAD.

141 , elevation of external K(+) to 10 mM caused vasodilation in brain slices from control animals but ca

142 Despite the importance of vasodilation in cardiovascular homeostasis and therapy,

143 expression and BK-channel-mediated coronary vasodilation in diabetic mice.

144 on and restored BK channel-mediated coronary vasodilation in diabetic mice.

145 into liposomal vesicles results in prolonged vasodilation in distal pulmonary arterioles.

146 ng, and, hence, reduced NO-mediated coronary vasodilation in DM patients.

147 Systemic arterial vasodilation in early pregnancy is accompanied by a comp

148 -NMMA and TEA attenuated bradykinin-mediated vasodilation in healthy and hypercholesterolemic subject

149 holine, stimulates K(+)(Ca) channel-mediated vasodilation in healthy subjects, whereas in hypercholes

150 ed pressure gradient/concentration-dependent vasodilation in isolated endothelium-intact and -denuded

151 Coronary PVAT also diminished H2O2-mediated vasodilation in lean and, to a lesser extent, in obese a

152 ulatory mechanism for processes ranging from vasodilation in mammals to communal behavior in bacteria

153 formulations produced pulmonary preferential vasodilation in MCT induced PAH rats.

154 gates whether PVAT regulates insulin-induced vasodilation in muscle, the underlying mechanisms, and h

155 emonstrate that GrC activity causes a robust vasodilation in nearby capillaries via the NMDA receptor

156 ODS AND H2O2 induced endothelium-independent vasodilation in non-CAD adipose arterioles, which was re

157 H2O2 induced endothelium-independent vasodilation in non-CAD adipose arterioles, which was re

158 MPs also compromised vasodilation in perfused microvessels.

159 on, we hypothesized that curcumin may induce vasodilation in peripheral arterioles, to improve perfus

160 er niacin regarding the FFA reduction versus vasodilation in rats and dogs.

161 -mediated dilation and endothelium-dependent vasodilation in response to acetylcholine were improved

162 ction by measuring the endothelial-dependent vasodilation in response to acetylcholine.

163 Vasodilation in response to carvacrol is inhibited when

164 eters of athletes would be larger, have less vasodilation in response to cuff occlusion, but more con

165 lost the ability to induce NO formation and vasodilation in response to flow and consequently develo

166 eled by measuring parenchymal arteriole (PA) vasodilation in response to neuronal stimulation in amyg

167 giotensin II-induced hypertension, decreased vasodilation in response to NO correlates with GC1 thiol

168 sting arteriole diameter but fails to affect vasodilation in response to vibrissae stimulation.

169 icited more pulmonary specific and sustained vasodilation in SUGEN-5416/hypoxia-induced PAH rats than

170 as signalling in the respiratory system and vasodilation in the cardiovascular system.

171 ra tissue in Franz diffusion cells suggested vasodilation in the conjunctival vasculature in the pres

172 igh sodium diets, and ADRB2-mediated forearm vasodilation in the high sodium condition.

173 indicate that obestatin causes NO-dependent vasodilation in the human circulation.

174 cellular calcium was not required for normal vasodilation in the IP3R2-KO animals.

175 lower endothelium-dependent and -independent vasodilation in the macrocirculation.

176 signed to right heart catheterization in the Vasodilation in the Management of Acute Congestive Heart

177 al. describe alterations in pressure-induced vasodilation in the populations most prone to developmen

178 on and with decreased acetylcholine-mediated vasodilation in the renal microvasculature.

179 s significantly to nitrite-dependent hypoxic vasodilation in vivo and ex vivo.

180 se oxidation underlies nitroglycerin-induced vasodilation in vivo, we used a Cys42Ser PKG1alpha knock

181 ls leading to impaired endothelium-dependent vasodilation, increased vascular tone, and hypertension.

182 ) increases tissue glucose uptake and causes vasodilation independent of insulin.

183 ly partially restores eNOS-mediated coronary vasodilation, indicating that other critical factors tri

184 H/I blunted vasodilation induced by the Katp agonists cromakalim, ca

185 xide release, improved endothelial-dependent vasodilation, inhibited vascular inflammation, and suppr

186 Thus, the effect of LPS on vasodilation involves up-regulation of K(ATP) channel ex

187 We sought to confirm that early hypoxic vasodilation is a nitric oxide (NO)-mediated phenomenon

188 Hypoxic vasodilation is a physiological response to low oxygen t

189 In conclusion, acute hypoxic vasodilation is an adaptive NO-mediated response conferr

190 In the postischemic heart, coronary vasodilation is impaired due to loss of endothelial nitr

191 We hypothesized that nitroglycerin-induced vasodilation is mediated by disulfide activation of PKG1

192 Vasodilation is observed to be a major determinant of ba

193 paradigm in showing that the major player in vasodilation is the keratinocyte, which produces PGE2, s

194 anoid PGE(2), which induces angiogenesis and vasodilation, is diminished in diabetic skin, suggesting

195 For the terminal arteriole: vasodilation logEC(50) -10.3+/-0.2, peak dilation +39+/-

196 hysiological role in processes as diverse as vasodilation, maintenance of vascular tone, neurotransmi

197 Niacin causes vasodilation, manifest as rubor (redness) of the head an

198 e diverse physiological processes, including vasodilation, neurotransmission, and myocardial function

199 s in diverse physiological processes such as vasodilation, neurotransmission, and the innate immune r

200 of the hemodynamic (mostly mediated through vasodilation of capacitance and conductance arteries) an

201 nflux via endothelial TRPA1 channels elicits vasodilation of cerebral arteries by a mechanism involvi

202 Vasodilation of detector vessels was diminished when exp

203 Carvacrol elicits vasodilation of intact cerebral arteries (EC(50) = 4.1 m

204 After stress, vasodilation of PAs to neuronal stimulation was greatly

205 The effect of ROCK inhibition on the vasodilation of porcine conjunctival vasculature was ass

206 motor function, as measured by flow-mediated vasodilation of the brachial artery, improved by 47% in

207 ng scores) and FMD and nitroglycerin-induced vasodilation of the brachial artery.

208 thelial function was explored by testing the vasodilation of the liver circulation to increasing conc

209 ative modifications in endothelium-dependent vasodilation of the principal nutrient artery (PNA) of t

210 Functionally, the intrinsic vasodilation of the vessel wall decreased at 12 weeks co

211 ne (P < 0.001) but did not induce additional vasodilation (P > 0.05) during obestatin.

212 erine administration resulted in significant vasodilation (p < 0.05).

213 n gene-related peptide artificially activate vasodilation pathways in rat brain and induce contrast c

214 In addition to enhancing vasodilation, PDE5Is seem to protect the myocardium thro

215 s in cardiovascular physiology, specifically vasodilation, platelet aggregation, and leukocyte rollin

216 regulate physiological functions, including vasodilation, platelet aggregation, and neurotransmissio

217 trol diverse physiological processes such as vasodilation, platelet aggregation, and synaptic plastic

218 rdiopulmonary bypass, levosimendan induces a vasodilation, preferentially of preglomerular resistance

219 ction, an effect that likely counteracts the vasodilation produced by activation of neuronal TRPV1.

220 ular processes, including neurotransmission, vasodilation, proliferation, and apoptosis in various ce

221 ease of the drug, produce pulmonary specific vasodilation, reduce the systemic exposure of the drug,

222 ence is activation of EC IK(Ca) channels and vasodilation, reducing the myogenic tone that underpins

223 rlying red blood cell (RBC)-mediated hypoxic vasodilation remain controversial, with separate roles f

224 ivate beta1-containing BK channels and evoke vasodilation remain unknown.

225 In contrast, acetylcholine-mediated vasodilation remained unchanged with TEA in healthy subj

226 and the corresponding need for pharmacologic vasodilation require that all imaging be completed withi

227 Vasodilation required neuronal NMDARs and NOS stimulatio

228 onist) enhanced constriction by removing the vasodilation response to curcumin.

229 Rac1 in vSMCs causes defective nitric oxide vasodilation responses and hypertension.

230 sfusion and effects on endothelial-dependent vasodilation responses to acetylcholine have not been fu

231 In vivo and ex vivo vasodilation responses, the reduction of nitrite to NO.,

232 Hypoxic vasodilation studies in myoglobin and endothelial and in

233 to hypercapnia, which triggers blood vessel vasodilation, suggests its dependence on vascular effect

234 taCys93Ala mutation are deficient in hypoxic vasodilation that governs blood flow autoregulation, the

235 ncreased with impaired endothelium-dependent vasodilation that is restored by thiol-specific reducing

236 may be related to increased CBF rather than vasodilation; these results contradict most previous stu

237 from C57BL/6 mice uncovered insulin-induced vasodilation; this vasodilation was abrogated with PVAT

238 pregnancy, contributing to profound maternal vasodilation through endothelial and nitric oxide (NO)-d

239 fumarate, itself a GPR109A agonist, provokes vasodilation through the same cells.

240 Although astrocytes can induce vasodilations through the release of prostaglandins, the

241 portal perfusion pressure and a decrease in vasodilation to acetylcholine (sinusoidal endothelial dy

242 plex class I inhibited endothelium-dependent vasodilation to acetylcholine.

243 pendent signalling mechanism is required for vasodilation to ADO.

244 l brain blood flow by providing steady-state vasodilation to arterioles via resting astrocyte Ca(2+)

245 ling mechanism linking endothelium-dependent vasodilation to bone remodeling.

246 However, vasodilation to endothelium-independent NO donor sodium

247 omega)-nitro-l-arginine methyl ester reduced vasodilation to flow in adipose as well as atrial vessel

248 EET) substrates, so they accumulate inducing vasodilation to lower blood pressure (BP).

249 reduced vasoconstriction to iberiotoxin and vasodilation to NS1619, BK channel inhibitors and activa

250 However, the endothelium-independent vasodilation to sodium nitroprusside was unaffected.

251 astrocyte endfeet and inversion of NVC from vasodilation to vasoconstriction in brain slices obtaine

252 of the neurovascular coupling response from vasodilation to vasoconstriction.

253 The endothelium-dependent NO-mediated vasodilations to bradykinin and stepwise increases in lu

254 terioles developed stable basal tone and the vasodilations to endothelium-dependent nitric oxide (NO)

255 ic heat-defense responses, such as cutaneous vasodilation, to skin-warming challenges.

256 (2+)-activated K(+)- and KV (KV1.5)-mediated vasodilation toward a large-conductance Ca(2+)-activated

257 es consistent with "upstream" propagation of vasodilation toward the cortical surface along the divin

258 cium-evoked release of PgE2 is decreased and vasodilation triggered by increased astrocyte [Ca(2+)]ii

259 ations of short dosing intervals, peripheral vasodilation, unwanted side effects, and restricted use

260 the therapeutic potential of selective renal vasodilation using serelaxin as a new treatment for rena

261 The TEBV exhibited flow-mediated vasodilation, vasoconstriction after exposure to 1 muM p

262 ice exhibited impaired endothelium-dependent vasodilation versus mock-ND at 9 and 12 weeks and endoth

263 via A2A receptors located on ECs to produce vasodilation via activation of KATP channels located on

264 rite may contribute to physiological hypoxic vasodilation via reactions with vascular myoglobin to fo

265 uncovered insulin-induced vasodilation; this vasodilation was abrogated with PVAT from db/db mice.

266 Acetylcholine-induced vasodilation was also impaired in ECSHIP2(Delta/+) mice,

267 PNA endothelium-dependent vasodilation was assessed in vitro using acetylcholine.

268 Cinaciguat-induced vasodilation was associated with a time- and concentrati

269 Insulin-mediated vasodilation was blunted in ECSHIP2(Delta/+) mice, as wa

270 ogenous estrogen (i.e., intact and old OVX), vasodilation was correlated with BV/TV (R(2) = 0.630; P<

271 In SRF(SMKO), vasodilation was decreased in aorta and carotid arteries

272 In young rats, vasodilation was diminished by OVX and restored with est

273 Coronary endothelium-dependent vasodilation was examined by infusing acetylcholine (10(

274 Tonic vasodilation was insensitive to TTX, as well as a variet

275 ith this hypothesis, intact stimulus-induced vasodilation was observed in inositol 1,4,5-triphosphate

276 This vasodilation was predominantly mediated by enhanced NO a

277 ; P<0.0001), whereas endothelium-independent vasodilation was similar in the 2 groups.

278 Endothelium-dependent and independent vasodilation was tested by selective infusion of acetylc

279 In old animals, vasodilation was unaffected by OVX but enhanced with est

280 Papaverine-induced pial artery vasodilation was unchanged by fluid percussion brain inj

281 ous and exogenous nitric oxide (NO)-mediated vasodilation were both impaired by repeated hypoglycemia

282 The concentrations to induce half-maximal vasodilation were comparable for nitroprusside (+E, 3.3x

283 ulated vascular cGMP signals associated with vasodilation were detected in vivo in an acutely untouch

284 of cell-signaling pathways and induction of vasodilation were examined in vitro and in isolated arte

285 nd on bradykinin- and acetylcholine-mediated vasodilation were studied.

286 increases in chronotropy, contractility, and vasodilation were unique to HFpEF and resulted in impair

287 Activity-dependent vasodilations were followed by a VP-mediated vasoconstri

288 an important mediator of VSMC relaxation and vasodilation, which acts by increasing cyclic GMP (cGMP)

289 VAT from obese mice inhibits insulin-induced vasodilation, which can be restored by inhibition of JNK

290 enoic acid (EET) induces mesenteric arterial vasodilation, which contributes to the onset of portal h

291 modulate nitric oxide synthesis, and promote vasodilation, which thereby improves blood flow.

292 receptors WIN55212, a CB(1)R agonist, caused vasodilation, which was absent in CB(1)R knock-out mice.

293 Hypoxaemia caused a rapid and sustained vasodilation, which was only partially reversed by non-s

294 ere made at baseline and under pharmacologic vasodilation with adenosine.

295 baseline hemodynamics and acute responses to vasodilation with intravenous sodium nitroprusside in pa

296 pression and whole cell currents, leading to vasodilation, with e1c overexpression inducing the large

297 t 500 Hz significantly increased SCS-induced vasodilation without influencing MT.

298 y of vascular smooth muscle (VSM) allows for vasodilation without lowering of cytosolic Ca(2+).

299 Considering that CO2/H(+)-induced vasodilation would accelerate removal of CO2/H(+) and po

300 Handgrip and forearm vasodilation yielded no haplotype differences, and no co