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1 tion of (131)I-mu81C6 mAb through regions of vasogenic edema.
2 presumed tumor-infiltrated edema from purely vasogenic edema.
3 reased blood-brain barrier permeability, and vasogenic edema.
4 ng lesion surrounded by a variable amount of vasogenic edema.
5 or 'paravasculitis' plays a critical role in vasogenic edema.
6  MMP-9 contributes to the early formation of vasogenic edema after transient FCI.
7 ropriate proinflammatory response leading to vasogenic edema and cerebral microhemorrhage.
8 rates a 1.6-cm right parietal mass with mild vasogenic edema and four additional brain metastases mea
9  (BBB) is associated with the development of vasogenic edema and intracranial hypertension in a numbe
10 ed inflammation (CAA-ri) is characterized by vasogenic edema and multiple cortical/subcortical microb
11 d other chronic retinal diseases, results in vasogenic edema and neural tissue damage, causing vision
12 tein and enzyme activity could contribute to vasogenic edema and the pathogenesis of neuronal dysfunc
13  the lesions are likely caused by reversible vasogenic edema and transient breakdown of the blood-bra
14 en in the brain and bone, decreased cerebral vasogenic edema, and improved survival, despite increasi
15 with intracranial evidence of hydrocephalus, vasogenic edema, central venous thrombosis, and/or mass
16 in animals with focal injury associated with vasogenic edema (cryogenic injury).
17  imaging abnormalities, interpreted as focal vasogenic edema, develop in some epileptic patients afte
18 These observations support the proposal that vasogenic edema due to cerebrovascular autoregulatory dy
19                   Proteinase-3 might mediate vasogenic edema during diabetic ketoacidosis, and select
20 nt with this hypothesis, showing no signs of vasogenic edema, even in ApoE4 carriers.
21         This establishes that Pb(2+)-induced vasogenic edema formation develops via a Flk-1-dependent
22                                              Vasogenic edema had a minimal effect on MT ratio, and ra
23 e rapid growth of solid brain metastases and vasogenic edema in patients with advanced cancer, leadin
24 s can all result in the clinical syndrome of vasogenic edema in the central nervous system leading to
25 performed in the emergency department showed vasogenic edema in the right temporal parietal region, w
26 focus 5 mm or less in diameter surrounded by vasogenic edema that extended less than 7 mm in radius b
27 lleled that of known resistance to spread of vasogenic edema, which suggested that anisotropy may be
28 he metabolic enzyme TYMS; and association of vasogenic edema with the oncogene FOXP1 and PIK3IP1, whi

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