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1 cause of its lack of agonist activity at the vasopressin V2 receptor.
2 and beta2-adrenergic receptors, but not the vasopressin (V2) receptor.
4 than the mixed vasopressin type 1a receptor/vasopressin V2 receptor agonist arginine vasopressin bec
5 treatment of selepressin with the selective vasopressin V2 receptor agonist desmopressin were simila
6 This study sought to examine the effects of vasopressin V2 receptor antagonism with tolvaptan on the
7 d effects were blocked by treatment with the vasopressin V2 receptor antagonist SR121463B, but not by
9 aptan (OPC-41061), a novel, oral, nonpeptide vasopressin V2-receptor antagonist in patients with chro
10 he effect of long-term administration of the vasopressin V2-receptor antagonist tolvaptan (30 mg/day)
11 atinine clearance, >/=60 ml per minute), the vasopressin V2-receptor antagonist tolvaptan slowed the
12 Tolvaptan, an oral, nonpeptide, selective vasopressin V2-receptor antagonist, shows promise in thi
13 ypervolemic hyponatremia, tolvaptan, an oral vasopressin V2-receptor antagonist, was effective in inc
14 inhibition of renal intracellular cAMP using vasopressin V2 receptor antagonists, and somatostatin an
22 eamino-(8-D-arginine)-vasopressin (dDAVP), a vasopressin V2 receptor-selective agonist, for 7 d into
23 unctional, shortened, "diabetic" form of the vasopressin V2 receptor that is the product of incomplet
24 asopressin (AVP) affects kidney function via vasopressin V2 receptors that are linked to activation o
25 II type 1A receptor, neurotensin receptor 1, vasopressin V2 receptor, thyrotropin-releasing hormone r
26 After agonist-induced internalization, the vasopressin V2 receptor (V2R) does not recycle to the pl
28 to recycle and resensitize rapidly, and the vasopressin V2 receptor (V2R), known to recycle and rese
31 ous to the human disorders to test whether a vasopressin V2 receptor (VPV2R) antagonist, OPC31260, wo
32 beta2-adrenergic, angiotensin II type 1 and vasopressin V2 receptors was altered by the beta-arresti
33 -7 cells, we found that angiotensin AT1a and vasopressin V2 receptors, which form stable receptor-bet
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