ライフサイエンスコーパス: vasorelaxant

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1 tion in isolated arteries and oppose certain vasorelaxants.

2 interplay of endothelin-1 with the putative vasorelaxant 2-arachidonoyl glycerol, an endogenous cann

3 (ChAT), which catalyzes the synthesis of the vasorelaxant acetylcholine, relay neural signals.

4 53 does not possess the direct, IM-sensitive vasorelaxant action of troglitazone.

5 ivity relationships for the antiischemic and vasorelaxant activities of KATP openers are distinct.

6 o Allium extracts, including hypotensive and vasorelaxant activities.

7 are favored in the oxygenated structure, and vasorelaxant activity is "linked" to deoxygenation, as i

8 culation, and NPR-C activation underlies the vasorelaxant activity of this peptide.

9 d cardiac tissues, devoid of chronotropy and vasorelaxant activity.

10 The vasorelaxant and anti-mitogenic activities of the atrial

11 support that there is no correlation between vasorelaxant and cardioprotective potencies of KATP open

12 t in the kidney, where it exerts significant vasorelaxant and neuromodulatory effects.

13 Hydrogen sulfide is a vasorelaxant and proangiogenic gas with therapeutic pote

14 The vasorelaxant and protective activity of CNP and cANF(4-2

15 the normal response to endothelium-dependent vasorelaxants and myocardial perfusion in response to FG

16 asma levels of adrenomedullin (AM), a potent vasorelaxant, are increased in septic shock and possibly

17 anandamide has recently been identified as a vasorelaxant but the underlying mechanisms are controver

18 mparable (ED(50) 1.7 microM) to 14,15-EET as vasorelaxants but were approximately 10-35 times less po

19 and direct intracavernosal injection of the vasorelaxant drug papaverine cause rapid increases in ph

20 The alpha-tocopherol moiety alone had no vasorelaxant effect at concentrations up to 300 micromol

21 NPR-A mediates the vasorelaxant effect of ANP in pulmonary vasculature.

22 The vasorelaxant effect of LPS was blocked by L-NIL.

23 domethacin (IM; 10 micromol/l; n = 12), this vasorelaxant effect of troglitazone was abolished (P < 0

24 on-dependent (1-30 microM) inhibition of the vasorelaxant effects of abn-cbd and anandamide.

25 r-853 were not observed, indicating that the vasorelaxant effects of cGKI are not through dephosphory

26 The vasorelaxant effects of the endothelium-derived relaxing

27 lates flow by releasing endothelium-mediated vasorelaxant factors such as NO.

28 l dysfunction or drugs that are large-artery vasorelaxants may be effective in reducing large-artery

29 , indicating increased release of the potent vasorelaxant nitric oxide.

30 more potent, respectively, than 14,15-EET as vasorelaxants; on the other hand, their ability to block

31 of 27 results from a combination of reduced vasorelaxant potency and enhanced cardioprotective poten

32 andidate to investigate the role of residual vasorelaxant potency of 2 toward its cardioprotective ac

33 hemic myocardium is achieved by reduction of vasorelaxant potency rather than enhancement in antiisch

34 r designing drugs to counter the noxious and vasorelaxant properties of general anesthetics and may p

35 the hypothesis that the cardioprotective and vasorelaxant properties of K(ATP) openers follow distinc

36 In conclusion, in cirrhotic rat livers, the vasorelaxant response to NTG is impaired owing to both a

37 normal livers, NTG and SNAP induced similar vasorelaxant responses (P =.44).

38 epressed L-arginine (10(-4) mol/L)-dependent vasorelaxant responses in O rings to those of Y.

39 l dysfunction as evidenced by blunted aortic vasorelaxant responses to acetylcholine (ACh) and calciu

40 splay pronounced hypertension with deficient vasorelaxant responses to acetylcholine.

41 The vasorelaxant responses to anandamide have now been exami

42 Cirrhotic livers exhibited lower vasorelaxant responses, compared with normal livers, to

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