1 ooth muscle alpha2-ARs may contribute to the vasospastic activity that is a prominent feature of the

2  with >/=50% angiographic stenosis or use of vasospastic agents were excluded.

3 ormation of an intimal bump in patients with vasospastic angina.

4 ocardial ischemia in patients with stable or vasospastic angina.

5  reductions in the frequency and severity of vasospastic attacks.

6 k, ascending aorta or a bronchial artery, or vasospastic cerebral infarction, or the cause was unesta

7  alpha2c-adrenergic receptors antagonists in vasospastic conditions with encouraging results.

8 es for evaluation of the hand vasculature in vasospastic disorders of the hand.

9 atients with NTG show a higher prevalence of vasospastic disorders.

10 NA abolished both MAPK inactivation and anti-vasospastic effects by r-OPN.

11 (perfusate G2) more effectively reversed the vasospastic effects of ischemia/reperfusion for NHBD tha

12     Long-term ST segment analysis may detect vasospastic episodes but has not been reported.

13  channel antagonist and successfully reduced vasospastic episodes.

14 mational change of arterial structure in the vasospastic lesion with optical coherence tomography.

15 nction of these channels might be related to vasospastic phenomena in human cerebral circulation.

16        This is the first report suggesting a vasospastic role of 5-fluoruracil in 5-FU associated opt