1 hed sympathetically induced and drug-induced venoconstriction.

2  forces (venocompression) rather than active venoconstriction.

3  significant peripheral vasoconstriction and venoconstriction (20% capacitance change).

4              Infusion of L-NMMA alone caused venoconstriction (9.1+/-6.4% increase in venous tone, P=

5 to L-NMMA primarily reflects a difference in venoconstriction between agents, such that phenylephrine

6 [Pro(11)(D)-Ala(12)] Ang I is able to induce venoconstriction in humans in vivo.

7                 SFLLRN caused dose-dependent venoconstriction (P<0.001) that was unaffected by norepi

8                                              Venoconstriction progressed to 47% with increasing conce

9 ge, impaired adrenergic vasoconstriction and venoconstriction result in hypotension.

10                        Norepinephrine exerts venoconstriction that could increase both the mean syste

11 up of low-flow POTS patients had exaggerated venoconstriction to phenylephrine (50% capacitance chang