ライフサイエンスコーパス: venous blood

1 Baseline anemia prevalence was 58% (venous blood).

2 t birth (cord blood) and in early childhood (venous blood).

3 , and their expression, in placenta and cord venous blood.

4 mparing their concentrations in arterial vs. venous blood.

5 LAgP patients were isolated from peripheral venous blood.

6 s were vascularly isolated and perfused with venous blood.

7 and less H(+) release into the great cardiac venous blood.

8 LAgP patients were isolated from peripheral venous blood.

9 consistently higher in the arterial than in venous blood.

10 on of radioactivity into superior mesenteric venous blood.

11 ased and lactate levels increased in patient venous blood.

12 ormal subjects were isolated from peripheral venous blood.

13 venous blood flow and lungs without hepatic venous blood.

14 centrations in arterial, portal, and hepatic venous blood.

15 ntrols were genotyped using genomic DNA from venous blood.

16 Venous blood (5 mL) was obtained from 104 subjects aged

17 entrations of glucose and oxygen relative to venous blood, a comprehensive biochemical characterizati

18 ge volume right lung activity, normalized to venous blood activity, is a good proxy for arterial bloo

19 1 that are specifically increased in hepatic venous blood after CBDL.

20 , analyzed electroencephalography (EEG), and venous blood ammonia responses to an oral glutamine chal

21 A receptor imaging were assessed by PET with venous blood analysis.

22 asma and four DBS cards from anti-coagulated venous blood and a fifth card from finger-prick blood we

23 rstly, platelets were isolated from systemic venous blood and activated with the GPVI specific agonis

24 7%, P < 0.001) compared to DBS prepared from venous blood and handled similarly.

25 nm at basal conditions in both arterial and venous blood and is formed at a level of 0.5-2.5 mum upo

26 ucosal pH, and serum lactate levels of mixed venous blood and mesenteric venous blood were recorded a

27 Venous blood and oral rinse samples were obtained from f

28 n chamber was perfused with unanticoagulated venous blood and PDT evaluated using computerized morpho

29 ions declined rapidly after storage of fresh venous blood and that hypoxic vasodilation by banked RBC

30 , they are recalled, values are confirmed in venous blood and thyroxine replacement therapy (TRT) is

31 Venous blood and urine samples were collected at enrollm

32 concentrations in brain tissue and cerebral venous blood are associated with respiratory depression

33 A) directly diverts the splanchnic and renal venous blood assuring a good portal inflow to the graft.

34 We sampled peripheral venous blood at 0, 3, 6, 12, 24, 48, 72 and 168 hours po

35 s were vascularly isolated and perfused with venous blood at pulsatile pressures designed to simulate

36 partial pressure of O(2) (PO(2)) in cubital venous blood at rest, during handgrip exercise, and duri

37 port rat delivers constant pressure systemic venous blood at stable physiologic parameters to the ex

38 d calcium values of intraosseous and central venous blood at the baseline and during 5 mins of CPR wi

39 en patients also had measurements of jugular venous blood at the level of the jugular bulb.

40 f radiotracer to the brain than arterialized venous blood, at least in some patient populations.

41 We collected venous blood before and after the intervention to measur

42 e and insulin concentrations in arterialized venous blood before and during FSIGT were virtually iden

43 ody fat metabolism, as validated by parallel venous blood beta-hydroxybutyrate (BOHB) measurements.

44 Intraosseous and central venous blood biochemical and hemoglobin values were simi

45 The venous blood BK1-5:bradykinin ratio correlated with plas

46 13-ketooctadecadienoic acid (KODE)] into the venous blood both in vivo and during perfusion.

47 th different combinations of cord and infant venous blood, breast milk, or a section of the placenta.

48 , it appears that increases in the tissue or venous blood CO(2) concentration are neither sensitive n

49 upport the notion that changes in tissue and venous blood CO(2) concentration during dysoxia reflect

50 hypoxia (dysoxia) to increases in tissue and venous blood CO(2) concentration.

51 Samples of venous blood, collected at various time points, were ana

52 utility for applications in the field where venous blood collection and timely shipment of labile bl

53 ith AD showed a significant narrowing of the venous blood column diameter (131.7 +/- 10.8 microm) com

54 correlated with the percentage decreases in venous blood column diameter (P = 0.031, R(2) = 0.51).

55 e primary outcome was within-person GLP-1 in venous blood (concentrations and area under the curve).

56 Use of venous blood data introduced a large bias in VT (r(2) =

57 cted genomic DNA (gDNA) as an alternative to venous blood-derived gDNA from premature neonates for mo

58 were unable to cooperate with fingerstick or venous blood draw.

59 PCR test results for HIV DNA on venous blood drawn from children ages 0-2 days and 3-5 m

60 Plasma was collected from infants if venous blood draws were ordered by pediatricians.

61 n which inspiration-induced downward flow of venous blood due to reduced intrathoracic pressure is co

62 e acid-base status of intraosseous and mixed venous blood during cardiopulmonary resuscitation; and c

63 x gene Prox1 is necessary and sufficient for venous blood endothelial cells (BECs) to acquire a lymph

64 the number of neutrophils was greater in the venous blood entering the lungs than in the arterial blo

65 evelop PAVM compared with lungs with hepatic venous blood flow (12/12 and 3/16 respectively, p < 0.01

66 arterial blood flow (F(a)), absolute portal venous blood flow (F(p)), absolute total liver blood flo

67 Enhancing the portal venous blood flow (PVBF) has been shown to reduce portal

68 a significant improvement in post-LPS portal venous blood flow (PVBF, 79% of baseline vs. 45% of base

69 acquisitions, one optimized for arterial and venous blood flow (velocity encoding range, +/-50 cm/sec

70 ers performed three determinations of portal venous blood flow and hepatic arterial resistive index b

71 t echocardiograms between lungs with hepatic venous blood flow and lungs without hepatic venous blood

72 Lungs with no hepatic venous blood flow are more likely to develop PAVM than l

73 n of a meal was estimated to increase portal venous blood flow by 96.3 mL/min (P < .001)--a change in

74 the portal vein, and disturbances in portal venous blood flow could contribute to the formation of b

75 y sonographers A and B, respectively, portal venous blood flow increased from 144.2 to 201.7 mL/min a

76 Concomitant analysis of brain venous blood flow indicated that CSF and venous flux act

77 The prandial effect on portal venous blood flow is only marginally greater than the in

78 crom, P = 0.01), and a significantly reduced venous blood flow rate (9.7 +/- 3.1 microL/min) compared

79 Change in portal venous blood flow rates did not have an effect on the si

80 Liver portal venous blood flow was assessed during perfusion, and at

81 Lungs with no hepatic venous blood flow were more likely to develop PAVM compa

82 Infrahepatic IVC and portal venous blood flow were shunted to the axillary vein usin

83 venous stasis, reduces intraoperative portal venous blood flow, decreases intraoperative urinary outp

84 tic IRI model, adjunctive BV improved portal venous blood flow, increased bile production, and decrea

85 HO-1 (Ad-HO-1) significantly improved portal venous blood flow, increased bile production, and decrea

86 mitant analyses of CSF dynamics and cerebral venous blood flow, that is, in epidural veins at cervica

87 For venous blood flow, the ICC with Bayesian multipoint MR i

88 ath, there was a large resurgence of femoral venous blood flow.

89 kely to develop PAVM than lungs with hepatic venous blood flow.

90 n lesions is unaffected by changes in portal venous blood flow.

91 t inverse relationship to the rate of portal venous blood flow.

92 ps showed significant restoration of retinal venous blood flow.

93 rtial pressure of a given inert gas in mixed-venous blood flowing back to the lungs is calculated fro

94 multibed scanning over 4 h with sampling of venous bloods for radioactivity and radioactive metaboli

95 Anticoagulated peripheral venous blood from 19 patients with stable CAD and 19 nor

96 Venous blood from 72 Schistosoma haematobium-exposed par

97 Venous blood from diabetic patients was exposed to air t

98 Venous blood from healthy volunteers was tonometered to

99 ve immunoselection from the PBMC fraction of venous blood from healthy volunteers, and monocyte-deriv

100 dGuo in the DNA of lymphocytes isolated from venous blood from healthy young male volunteers in sever

101 Venous blood from human volunteers was stimulated with L

102 It is feasible and safe to collect portal venous blood from patients undergoing EUS.

103 MPs were prepared from ECs and venous blood from patients with ACS (n=30) and from heal

104 Venous blood from seven of the subjects was sampled for

105 ormed and corresponding arterial and central venous blood gas and lactate measurements were made.

106 mal ulnar nerve stimulation and arterialized venous blood gas determinations were obtained before, du

107 Hemodynamic variables, systemic and mixed venous blood gas tensions and oxygenation, arterial lact

108 Arterial and venous blood gas values, glucose, and cardiac output wer

109 th ITPR-CPR for 15 minutes, and arterial and venous blood gases were collected at baseline and minute

110 Cerebral venous blood gases were drawn from a jugular bulb venous

111 Arterial and deep venous blood gases were measured and oxygen consumption

112 Arterial and mixed venous blood gases were measured at baseline, 1 min afte

113 the first minute of CPR, arterial and mixed venous blood gases were superior in the 3 experimental g

114 of 90 mins, vital signs, arterial and mixed venous blood gases, and intramucosal PCO2 values were ob

115 Arterial and venous blood gases, hemodynamics, and pulmonary mechanic

116 ge-pressure monitoring, measurement of mixed venous blood gases, or monitoring of cardiac output by o

117 d inotropic requirements, arterial and mixed venous blood gases, urine output, and biochemical and he

118 nted for measurement of arterial and central venous blood gases.

119 and blood flow were measured with pulmonary venous blood gases.

120 rdiac output by thermodilution, arterial and venous blood gases; electrolytes; lactate; base excess;

121 during periods of rapid rise and decline of venous blood glucose concentration was tested.

122 sions by comparing the CGM glucose values to venous blood glucose measurements taken every 15 min.

123 All 12 lungs with no perfusion of hepatic venous blood had positive contrast echocardiographic stu

124 As expected, ACZ led to lower arterial and venous blood [HCO3-], pH and lactate levels (P < 0.05),

125 lse-positive heel-stick screening but normal venous blood hormone levels; (b) hypoplasia-ectopia in 3

126 lood [PVB]) and outflow compartment (hepatic venous blood [HVB]) of a training (n = 55) and validatio

127 near normal because of sequestered cerebral venous blood in capillaries and venous capacitance vesse

128 n of circulating tumor cells from peripheral venous blood in clinical practice.

129 of circulating tumor cells using peripheral venous blood in clinical practice.

130 Arterial blood (arterialized venous blood in healthy subjects) was collected for the

131 saturations were substituted for true mixed venous blood in oxygen transport or pulmonary venous adm

132 In CAS patients, glutamate concentrations in venous blood increased immediately after the procedure (

133 vels of inflammatory cytokines in the portal venous blood, induced activation of resident liver dendr

134 be a nonspecific feature of impaired portal venous blood inflow, whatever the cause, or a feature of

135 Venous blood ionized calcium was used as an index of int

136 ral blood (PPB) and intraoperative pulmonary venous blood (IPVB) could predict poor long-term surviva

137 Leukocytes were separated from the patient's venous blood, labeled with (18)F-FDG, and reinjected int

138 Forearm venous blood lactate was measured every 2 to 3 min.

139 Here we describe and evaluate the changes in venous blood lead level (VBLL) associated with DMSA trea

140 ation was increased by approximately 7.5% in venous blood leaving the CNS capillary bed compared to a

141 r and sodium values were higher than central venous blood levels.

142 Analysis of cecal venous blood obtained after intracecal instillation of H

143 Human venous blood of different concentrations, various fluids

144 ncentration in arterial blood ([K+]a) and in venous blood of hindlimb muscle ([K+]v) before and at th

145 ry in concentration between the arterial and venous blood of human patients.

146 The reduction in mixed venous blood oxygen saturation and the increase in blood

147 Post-occlusive transient changes in venous blood oxygenation might provide a new measure of

148 differential equation to calculate the mixed-venous blood partial pressure may be in error.

149 ovolemic conditions, especially when central venous blood pressure is critically low.

150 ternative explanations, like measurements of venous blood pressure, did not show correlation with hyd

151 Their arterial and central venous blood pressures were continuously monitored by in

152 he endotracheal tube tip, aortic and central venous blood pressures, arterial blood gases, and respir

153 Arterial blood flow, arterial and venous blood pressures, total vascular resistance, tissu

154 in arterial, hepatic venous (HV) and portal venous blood (PV).

155 were detected in the hepatic inflow (portal venous blood [PVB]) and outflow compartment (hepatic ven

156 ion ratios and taking into account pulmonary venous blood remixing yielded our lung-scale model.

157 Right lung activity, corrected to venous blood, represents the whole arterial blood curve

158 field units) of the systemic blood and renal venous blood, respectively, and CT(PRE) is the precontra

159 inferior vena cava and allows uninterrupted venous blood return during the anhepatic phase.

160 dequate forward blood flow out of the heart, venous blood return must be drawn back into the heart.

161 tion with inflammatory markers measured in a venous blood sample at the time of cognitive testing.

162 urements of known serum radioactivity from a venous blood sample obtained at the time of PET/CT.

163 A venous blood sample was collected and blood pressure was

164 A venous blood sample was taken at baseline and at 6 and 1

165 A sample of unstimulated whole saliva and a venous blood sample were obtained from each subject, and

166 ent of B-type natriuretic peptide level from venous blood sample were performed in 150 consecutive pa

167 P), and serum ferritin concentrations from a venous blood sample.

168 Venous blood samples (0 h predose through 28 h after dos

169 Venous blood samples (12 mL) were obtained from each of

170 CMRGlu quantified with venous blood samples (n = 6) showed excellent agreement

171 ompare pH and Pco2 of intraosseous and mixed venous blood samples after sequential infusions of fluid

172 O2 max , direct Fick by femoral arterial and venous blood samples and Doppler ultrasound blood flow m

173 Femoral arterial and venous blood samples and muscle biopsy samples were coll

174 etics were derived from femoral arterial and venous blood samples and vastus lateralis muscle biopsie

175 Venous blood samples and vastus lateralis muscle biopsy

176 In addition, IDIF results obtained with venous blood samples and with a transformed venous-to-ar

177 Peripheral venous blood samples at 24 and 72 hrs after the onset of

178 Venous blood samples can be used for absolute quantifica

179 le angina pectoris undergoing PCI had serial venous blood samples drawn before PCI, after PCI, and at

180 cs were determined from femoral arterial and venous blood samples during a primed-constant d5-phenyla

181 ation by polymerase chain reaction in serial venous blood samples for 100 days and in tissue specimen

182 mples to quantify S. japonicum infection and venous blood samples for hemograms and measures of iron

183 provided stools to quantify reinfection and venous blood samples for hemograms and measures of iron

184 ood samples could serve as an alternative to venous blood samples for the diagnosis of EVD in resourc

185 vels were measured in adrenal and peripheral venous blood samples from 2 patients.

186 H and PB NK cells in paired liver biopsy and venous blood samples from 70 patients with chronic HCV i

187 Venous blood samples from healthy volunteers were expose

188 We obtained peripheral venous blood samples in 23 subjects with NNV-ARMD or tre

189 Venous blood samples obtained before scanning were expos

190 ably frozen mononuclear cells separated from venous blood samples obtained from 111 infants born to H

191 Venous blood samples of 20 healthy volunteers were expos

192 corticotropin levels were higher in adrenal venous blood samples than in peripheral venous samples,

193 ance between measurements of fingerprick and venous blood samples using the standard hematology analy

194 Concurrent venous blood samples were acquired for blood metabolite

195 Peripheral and portal venous blood samples were assayed for midazolam and [15N

196 Venous blood samples were collected and assayed to exami

197 Venous blood samples were collected at weeks 0, 4, and 8

198 Additionally, venous blood samples were collected before (18)F-FLT inj

199 Venous blood samples were collected before blue dye infu

200 Dynamic scans were acquired and venous blood samples were collected during the (18)F-FLT

201 Fasted venous blood samples were collected for iron isotopic an

202 Venous blood samples were collected from 44 patients wit

203 s on TLR expression and function, peripheral venous blood samples were collected from healthy volunte

204 y on TLR expression and function, peripheral venous blood samples were collected from healthy volunte

205 Venous blood samples were collected from healthy, premen

206 Simultaneous arterial and venous blood samples were drawn at baseline and in respo

207 Growth was assessed at monthly intervals and venous blood samples were drawn at entry into the study

208 Venous blood samples were drawn for thrombelastograpy (T

209 Venous blood samples were drawn from healthy, exclusivel

210 Serial venous blood samples were drawn over an 11-h period (8:3

211 Serial venous blood samples were drawn to assess concentrations

212 nical range from fingerprick (capillary) and venous blood samples were measured and compared using a

213 rization before and after the procedure, and venous blood samples were obtained 24, 48, and 72 hours

214 Serial venous blood samples were obtained every 30 min for the

215 Serial venous blood samples were obtained for radioactivity mea

216 Experiment 2: Arterial and mixed venous blood samples were obtained from 100 percutaneous

217 Venous blood samples were obtained from all participants

218 Venous blood samples were obtained.

219 Venous blood samples were taken at 0, 15, 30, 60, and 12

220 Venous blood samples were taken immediately after each b

221 Anthropometric measures and venous blood samples were taken monthly.

222 Vital signs, arterial and mixed venous blood samples, saline tonometry samples, and reci

223 Venous blood samples, taken from volunteers were culture

224 Using 2 mixed venous blood samples, the method accurately assesses the

225 (EF) of FTHA, measured from LAD arterial and venous blood samples, was compared to beta-oxidation rat

226 Patients and controls provided venous blood samples.

227 ers of endothelial function were measured on venous blood samples.

228 th imaging-derived input function (IDIF) and venous blood samples.

229 ntial muscle biopsies, and femoral arterial, venous blood sampling allowed determination of glucose a

230 en FDG blood clearance, obtained from serial venous blood sampling and from a hybrid method of early

231 f early cardiac blood pool imaging, and late venous blood sampling was analyzed.

232 graphical analysis, dynamic FDG imaging and venous blood sampling were made.

233 PET acquisitions (10-20 min) and venous blood sampling were performed every 30-60 min thr

234 SPECT acquisitions (30 min) and venous blood sampling were performed every 60 min throug

235 The MRFDG, determined by venous blood sampling, had a 6% average overestimate, co

236 Arterial and femoral venous blood sampling, thermodilution blood flow measure

237 es were obtained with concurrent arterial or venous blood sampling.

238 ment with constant infusion of (18)F-FDG and venous blood sampling.

239 Thirteen of these patients also underwent venous blood sampling.

240 90- to 140-min interval after injection with venous blood sampling.

241 On day 20, a venous blood specimen tested negative for Ebola virus by

242 imultaneously collected arterial and central venous blood specimens were obtained on 148 occasions fr

243 examination, structured clinical interview, venous blood specimens, and masked grading of seven stan

244 imultaneously collected arterial and central venous blood specimens; b) to test the hypothesis that e

245 0.0004) 55.2% +/- 22.5% increase in retinal venous blood speed accompanied the decreases in diameter

246 h, nitrite levels are higher in arterial vs. venous blood (suggesting systemic nitrite consumption),

247 CC xenografts that receive both arterial and venous blood supplies.

248 with clearance of CNS-derived Abeta into the venous blood supply with no increase from a peripheral c

249 In each experiment, venous blood taken before and immediately after exercise

250 ed cell NO with appreciably higher values in venous blood than arterial blood.

251 lood that delivers nutrients to tissues, and venous blood that removes the metabolic by-products.

252 Based on venous blood, the analysis used mean corpuscular volume

253 heoretical hepatotrophic molecules in portal venous blood (Theme I) and with the contemporaneous para

254 omic right-to-left shunts, allowing systemic venous blood to bypass gas exchange and pulmonary capill

255 us malformations (PAVMs) that allow systemic venous blood to bypass the pulmonary capillary bed throu

256 continually exposed, via gut-derived portal venous blood, to potential antigens and bacterial produc

257 Clonogenic MSCs were detected in venous blood up to 1 hour after infusion in 13 of 21 pat

258 Increasing perfusion using venous blood (VB) would be an attractive option because

259 of factors control the ratio of arterial and venous blood vessel types during angiogenesis.

260 d for the proper development of arterial and venous blood vessels, and that a major role of Notch sig

261 femoral arterial and venous, and peripheral venous blood vessels.

262 topographic relationship of overt damage to venous blood vessels.

263 he feasibility and safety of sampling portal venous blood via endoscopic ultrasound (EUS) to count po

264 ,O2) due to the influence of the intervening venous blood volume and the contribution of body O2 stor

265 Venous blood was also collected and centrifuged to obtai

266 were measured breath by breath; arterialized venous blood was analyzed for blood gas determinations a

267 In addition, 2 mL venous blood was collected by venipuncture from all part

268 t baseline and at the end of NEFA elevation; venous blood was collected for measurement of lipids and

269 Venous blood was collected for measurements of plasma ca

270 Venous blood was collected from 13 patients with CP desp

271 g (month 0) and end (month 10) of the study, venous blood was collected from family members >18 years

272 Venous blood was collected from normal subjects and peri

273 Internal jugular venous blood was drawn from both left and right sides an

274 Venous blood was drawn to compare protein binding, paren

275 Venous blood was measured for hemoglobin (Hb).

276 Fresh venous blood was obtained and stained with monoclonal an

277 Coronary venous blood was obtained by coronary sinus catheterizat

278 placebo or inhaled enoxaparin (2 mg/kg), and venous blood was obtained for analysis of plasma antifac

279 During coronary angioplasty, venous blood was obtained for flow cytometric detection

280 Venous blood was obtained from the rabbits before and af

281 Venous blood was sampled before and after the exposure a

282 Arterialized venous blood was sampled for 2 h, and measured meal GI a

283 Arterial and hepatic venous blood was sampled in postabsorptive (n = 6; study

284 Arterialized venous blood was sampled throughout the 2-h postchalleng

285 Venous blood was sampled to measure N-terminal (NT)-proA

286 nously; arterial, portal venous, and hepatic venous blood was sampled; and liver and visceral fat wer

287 duration of labeled MSCs in the circulation, venous blood was serially drawn from five additional rat

288 Six ml of venous blood was taken for the measurement of serum uric

289 group, the perfusion rate was unchanged, but venous blood was used as the LAD perfusate.

290 ision and accuracy of the method with use of venous blood were also tested.

291 ples and placental tissue and umbilical cord venous blood were collected and analyzed for choline and

292 Three-milliliter samples of venous blood were collected before, during (at 15, 30, 4

293 The separated leukocytes from 80-ml fresh venous blood were incubated with three different ages (i

294 ments on serum and blood spots prepared from venous blood were performed in 71 healthy subjects, 41 o

295 levels of mixed venous blood and mesenteric venous blood were recorded at baseline, after pericardia

296 Arterial and venous blood were sampled frequently using a peristaltic

297 O2, with flow-independent T2 measurements of venous blood, were obtained at different times.

298 rprick samples accurately reflect those from venous blood, which confirms the potential of capillary

299 relative change in volume for arteriole vs. venous blood within primary vibrissa cortex of awake, he

300 on of spillover between arterial and hepatic venous blood without portal venous data.