ライフサイエンスコーパス: venous occlusion

1 in improved specificity in the detection of venous occlusions (0.99 vs 0.96, P = .03), in reader con

2 lar stroke volume and their changes during a venous occlusion and release maneuver to a calibrated ao

3 alfunction (30%), and other (device upgrade, venous occlusion, and advisory leads; 20%).

4 urements on melanocytic nevus, vitiligo, and venous occlusion conditions were performed in volunteers

5 Presumably, venous occlusion could occur owing to thrombus formation

6 venting excessive tissue swelling leading to venous occlusion during catheter ablation procedures.

7 plored the potential for tissue swelling and venous occlusion during radiofrequency (RF) catheter abl

8 e treated due to symptomatic portomesenteric venous occlusion of thrombotic origin.

9 lium-dependent vasodilation, by both forearm venous occlusion plethysmography (93 +/- 67% vs. 145 +/-

10 asma fibrinolytic factors were assessed with venous occlusion plethysmography and blood sampling duri

11 Forearm venous occlusion plethysmography and intra-arterial infu

12 sured resting forearm blood flow (FBF) using venous occlusion plethysmography before and after intra-

13 and 45% of maximal voluntary contraction by venous occlusion plethysmography before and after region

14 ndependent vasodilatation were assessed with venous occlusion plethysmography before and during intra

15 t period, forearm blood flow was measured by venous occlusion plethysmography during an intrabrachial

16 Forearm blood flow was measured by venous occlusion plethysmography during intrabrachial in

17 ceptor antagonist BQ-123 were assessed using venous occlusion plethysmography in 10 patients with syn

18 Forearm blood flow was measured with venous occlusion plethysmography in 12 cigarette smokers

19 Forearm blood flow was measured by venous occlusion plethysmography in 16 volunteers during

20 Forearm blood flow was measured by venous occlusion plethysmography in response to serial i

21 erial vasodilator responses were assessed by venous occlusion plethysmography in the brachial circula

22 Forearm blood flow was measured with venous occlusion plethysmography in the resting forearm.

23 Supine venous occlusion plethysmography showed no differences b

24 ine gingival capillary density (GCD); and 3) venous occlusion plethysmography to assess endothelium-d

25 l function was assessed by bilateral forearm venous occlusion plethysmography using acetylcholine and

26 Venous occlusion plethysmography was used to assess fore

27 Venous occlusion plethysmography was used to assess resi

28 To address this question, venous occlusion plethysmography was used to measure for

29 conduit vessel response), and in 6 subjects, venous occlusion plethysmography was used to measure for

30 Venous occlusion plethysmography was used to measure pea

31 Subjects (n=157) underwent venous occlusion plethysmography with acetylcholine, bra

32 al function was assessed by standard forearm venous occlusion plethysmography with acetylcholine, nit

33 was measured simultaneously in both arms by venous occlusion plethysmography with mercury-in-Silasti

34 trials by measuring forearm blood flow (FBF; venous occlusion plethysmography) after 5 minutes of art

35 ocol 1, we measured forearm blood flow (FBF; venous occlusion plethysmography) and calculated the vas

36 We measured forearm blood flow (venous occlusion plethysmography) and calculated vascula

37 ilution), forearm vascular conductance (FVC, venous occlusion plethysmography) and cutaneous vascular

38 ulated from forearm blood flow (measured via venous occlusion plethysmography) and intra-arterial blo

39 acebo, allopurinol improved peak blood flow (venous occlusion plethysmography) in arms (+24%, P=0.027

40 healthy young men, forearm blood flow (FBF; venous occlusion plethysmography) responses to brachial

41 e (BP), heart rate (HR), forearm blood flow (venous occlusion plethysmography), FVR, and MSNA (obtain

42 Forearm blood flow was determined by venous occlusion plethysmography, and dose-response curv

43 Blood flow was measured by venous occlusion plethysmography, and the percentage of

44 Forearm blood flow was measured by venous occlusion plethysmography, before, and 8 min afte

45 erial pressure were measured using bilateral venous occlusion plethysmography, bioimpedance cardiogra

46 ar pressure, and cardiac output were made by venous occlusion plethysmography, Doppler flow wire and

47 Endothelial function was assessed by forearm venous occlusion plethysmography, flow-mediated dilation

48 orearm and calf blood flow were evaluated by venous occlusion plethysmography, MSNA by microneurograp

49 ity, carotid-radial pulse wave velocity, and venous occlusion plethysmography.

50 and sodium nitroprusside were assessed using venous occlusion plethysmography.

51 al function was assessed by standard forearm venous occlusion plethysmography.

52 elation was computed for every subject using venous occlusion plethysmography.

53 earm blood flow was measured using bilateral venous occlusion plethysmography.

54 ceptor antagonist (0.3 and 1 nmol/min) using venous occlusion plethysmography.

55 of patients with coronary artery disease by venous occlusion plethysmography.

56 FBF was measured in 10 cirrhotic patients by venous occlusion plethysmography.

57 Forearm blood flow was determined by venous occlusion plethysmography.

58 red in the resting non-dominant forearm with venous occlusion plethysmography.

59 Forearm blood flow was determined by venous occlusion plethysmography.

60 dothelial nitric oxide production by forearm venous occlusion plethysmography.

61 Forearm blood flow was determined by venous occlusion plethysmography.

62 hile forearm blood flow (FBF was measured by venous occlusion plethysmography.

63 In the remaining six patients, portal venous occlusion precluded access to the extrahepatic po

64 the dependent human foot using a single-step venous occlusion protocol.

65 RFA and EVLT offer comparable venous occlusion rates at 3 months after treatment of pr

66 Forearm blood flow was measured using venous-occlusion, strain-gauge plethysmography.

67 cclusive peak reactive hyperemia) and during venous occlusion (venous congestion), as assessed with s