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1  should not be used as surrogate for central venous oxygen saturation.
2 arterial lactate concentrations, and central venous oxygen saturation.
3 ul for predicting low cardiac index or mixed venous oxygen saturation.
4 ral venous oxygen saturation for a low mixed venous oxygen saturation.
5 ght atrial pressure cardiac index, and mixed venous oxygen saturations.
6 ht atrial pressure, cardiac index, and mixed venous oxygen saturations.
7 atients, 31 catheters) and 65 pairs of mixed venous oxygen saturation (20 patients, 28 catheters) wer
8 - 0.9 to 15.7 +/- 2.4; p < .0001); and mixed venous oxygen saturation (26 +/- 5 to 36 +/- 5, p < .05)
9 olume (54 +/- 27 to 65 +/- 38 ml), and mixed-venous oxygen saturation (69 +/- 8 to 73 +/- 10%), all p
10  a significantly higher mean (+/-SD) central venous oxygen saturation (70.4+/-10.7 percent vs. 65.3+/
11               Results for changes of femoral venous oxygen saturation and central venous oxygen satur
12         Correlation and agreement of femoral venous oxygen saturation and central venous oxygen satur
13 ed, including the difference between femoral venous oxygen saturation and central venous oxygen satur
14 rrelation between obtained values of femoral venous oxygen saturation and central venous oxygen satur
15   There is lack of agreement between femoral venous oxygen saturation and central venous oxygen satur
16           We concurrently determined femoral venous oxygen saturation and central venous oxygen satur
17 ermore, we determined simultaneously femoral venous oxygen saturation and central venous oxygen satur
18 ill patients, the difference between femoral venous oxygen saturation and central venous oxygen satur
19 tly increased systemic hypotension and mixed venous oxygen saturation and decreased pulmonary artery
20 ose of our study was to determine if central venous oxygen saturation and femoral venous oxygen satur
21               Six-minute walk results, mixed venous oxygen saturation and initial treatment randomiza
22           We compared the ability of central venous oxygen saturation and markers of anaerobic metabo
23 e determined correlation of baseline central venous oxygen saturation and mixed venous oxygen saturat
24 e central venous oxygen saturation and mixed venous oxygen saturation and predictive value of a low c
25 r resistance indices, and arterial and mixed venous oxygen saturation and reduced pulmonary hypertens
26 vere sepsis, the correlation between central venous oxygen saturation and tissue oxygen saturation at
27 roblems with oxygenation, ventilation, mixed venous oxygen saturation, and cardiac output.
28  P = .02), improved hemodynamics and central venous oxygen saturation, and less organ dysfunction.
29 on, oxygen delivery, mixed venous PO2, mixed venous oxygen saturation, and PmO2 responded with simila
30  change of arterial oxygen saturation, mixed venous oxygen saturation, and PO2.
31  and MSNA, reduced forearm velocity, lowered venous oxygen saturation, and raised venous lactate and
32 cant decrease in Pa(O2 ), arterial and mixed venous oxygen saturation, and ratio of Pa(O(2)) to fract
33 ics, as well as global cardiac output, mixed venous oxygen saturation, and systemic and cerebral oxyg
34                     Perfusion, Intravascular Venous Oxygen saturation, and T2* (PIVOT), a recently de
35 ior probability that cardiac index and mixed venous oxygen saturation are normal and physical examina
36  under the curve 0.66; 0.46-0.86) or central venous oxygen saturation (area under the curve 0.56; 0.3
37  arterial pressure, cardiac index, and mixed venous oxygen saturation, as well as significantly highe
38                              CBF and jugular venous oxygen saturation both increased significantly at
39 to 15% was not predicted by baseline central venous oxygen saturation but by high baseline lactate an
40 central venous oxygen saturation and femoral venous oxygen saturation can be used interchangeably dur
41                          We recorded central venous oxygen saturation continuously for 0 to 6 hrs of
42 e, and low 24-hr fluid output; and low mixed venous oxygen saturation correlated with knee mottling a
43          The bias and precision of the mixed venous oxygen saturation data were -0.57% and 3.76%, res
44 etabolites would be increased during jugular venous oxygen saturation desaturations (<50%) and determ
45 femoral venous oxygen saturation and central venous oxygen saturation.Despite significant correlation
46 ike markers of anaerobic metabolism, central venous oxygen saturation did not allow the prediction of
47 ration and predictive value of a low central venous oxygen saturation for a low mixed venous oxygen s
48 lism should be considered instead of central venous oxygen saturation for starting hemodynamic resusc
49 n saturation was better predictor of central venous oxygen saturation &gt;70% than thenar tissue oxygen
50 fied patients with severe sepsis and central venous oxygen saturation &gt;70%.
51 venous oxygen saturation <60%, but a central venous oxygen saturation &gt;or=70% may be useful to exclud
52 HR, BP, MSNA, forearm flow velocity, forearm venous oxygen saturation, H(+), and lactate.
53 femoral venous oxygen saturation and central venous oxygen saturation in 30 surgical patients and in
54 femoral venous oxygen saturation and central venous oxygen saturation in a group of 100 stable cardia
55 of global cerebral oxygenation using jugular venous oxygen saturation in all 126 patients.
56 femoral venous oxygen saturation and central venous oxygen saturation in both stable and unstable med
57 measures continuous cardiac output and mixed venous oxygen saturation in the clinical setting.
58 femoral venous oxygen saturation and central venous oxygen saturation including its range of variatio
59  to 90 mmHg +/- 32 after thrombectomy, mixed venous oxygen saturation increased from 48% +/- 19% to 6
60        The systolic blood pressure and mixed venous oxygen saturation increased from 75 (IQR:15) mm H
61 res, pulmonary vascular pressures, and mixed venous oxygen saturation is a unique feature; c) additio
62                                      Jugular venous oxygen saturation is monitored after traumatic br
63 " is based on continuing resuscitation until venous oxygen saturation is normalized.
64 ous oxygen saturation <70% predicted a mixed venous oxygen saturation &lt;60% with a sensitivity 84%,spe
65 ation <70% does not accurately predict mixed venous oxygen saturation &lt;60%, but a central venous oxyg
66 3% and 86%) for cardiac index <2.5 and mixed venous oxygen saturation &lt;60%, respectively.
67 ation >or=70% may be useful to exclude mixed venous oxygen saturation &lt;60%.
68                                      Central venous oxygen saturation &lt;70% does not accurately predic
69                                      Central venous oxygen saturation &lt;70% predicted a mixed venous o
70 culated the area under the curve for central venous oxygen saturation &lt;70%.
71 ntricular systolic dysfunction had a central venous oxygen saturation&lt;70%.
72 system was calibrated every 24 hrs for mixed venous oxygen saturation monitoring.
73 tolic blood pressure of 50-70 mm Hg, a mixed venous oxygen saturation of 25% to 40%, and a UO <10% of
74 sue oxygen saturation, brain tissue PO2, and venous oxygen saturation of the superior sagittal sinus
75 ffect of mannitol on brain tissue PO2 and on venous oxygen saturation of the superior sagittal sinus
76 , mean arterial pressure, and either central venous oxygen saturation or lactate clearance.
77 oid tissue oxygen saturation but not central venous oxygen saturation or thenar tissue oxygen saturat
78 ue hypoxia (reflected by lactate and central venous oxygen saturation), organ dysfunction, and mortal
79 s physiologic monitoring (arterial and mixed venous oxygen saturation, oxygen consumption, etc.) may
80  cardiac output (r(s) =.52, p<.05) and mixed venous oxygen saturation (r(s) =.61, p<.05).
81 oxygen transport (r=-.68, P=.005), and mixed venous oxygen saturation (r=-.79, P<.0001).
82 x, and PVRI, whereas cardiac index and mixed venous oxygen saturation remained unchanged.
83                                 When central venous oxygen saturation remains low, despite achieving
84 femoral venous oxygen saturation and central venous oxygen saturation (rs = 0.55; p < .001), the limi
85 (P(SL)CO2), lactate concentration, and mixed venous oxygen saturation (S(MV)O2) in hemodynamically un
86 dissociation curves, we plotted arterial and venous oxygen saturation (SaO2 and SvO2 ) against oxygen
87                                      Central venous oxygen saturation (ScvO2) in the superior vena ca
88 e (or lactate >4 mM), and continuous central venous oxygen saturation (Scvo2) monitoring for quantita
89 itiate central venous pressure (CVP)/central venous oxygen saturation (Scvo2) monitoring within 2 hrs
90                                Thus, femoral venous oxygen saturation should not be used as surrogate
91 sure, urine output, central venous (or mixed venous) oxygen saturation should be targeted.
92 an arbitrary choice of Sat(Thresh), systemic venous oxygen saturation (SsvO(2)) may be a useful param
93  simultaneous reflectance spectrophotometry (venous oxygen saturation StO2 and relative tissue hemogl
94 w), continuous cardiac output (Q), and mixed venous oxygen saturation (Sv(O(2))) were measured by flo
95                                   Perfusion, venous oxygen saturation SvO2, and T2* were each quantif
96 termining weaning outcome, we recorded mixed venous oxygen saturation (SvO2) continuously in eight ve
97                                        Mixed venous oxygen saturation (SVO2) was also compared with P
98 nts were transfused on bypass solely for low venous oxygen saturation (SvO2), ie, <55%, without regar
99 -1.5 mmol/L, respectively, and minimum mixed venous oxygen saturation, systemic oxygen delivery, and
100 ectrocardiogram, electroencephalogram, mixed venous oxygen saturation, temperature (core and blood),
101  patients at H0 and H6, whereas mean central venous oxygen saturation was preserved but significantly
102                                A low jugular venous oxygen saturation was treated in both groups, min
103 nuous monitoring of cardiac output and mixed venous oxygen saturation was used with either an 8.5-Fr
104 tissue hypoxia (as quantified by low central venous oxygen saturation) was not associated with major
105 femoral venous oxygen saturation and central venous oxygen saturation were assessed, including the di
106 femoral venous oxygen saturation and central venous oxygen saturation were similar.
107 onary artery pressure, heart rate, and mixed venous oxygen saturation, were obtained at baseline, aft
108 l venous pressure, fluid output, and central venous oxygen saturation with parameters from a pulmonar

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