ライフサイエンスコーパス: venous stasis

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1 lymph node (RPLN) metastases that may cause venous stasis and increase the risk of VTE development.

2 sequential compression devices to minimizes venous stasis, and optimize intravascular volume to mini

3 erative joint disease, urinary incontinence, venous stasis, and pseudotumor cerebri.

4 mple of Virchow's triad: hypercoagulability, venous stasis, and vascular damage; together these facto

5 Surgically induced weight loss corrected the venous stasis disease in almost all patients as well as

6 The GBP database was queried for venous stasis disease including pretibial venous stasis

7 ificantly more frequent in the patients with venous stasis disease than for those without.

8 Severe venous stasis disease was associated with a significantl

9 is associated with a risk of lower extremity venous stasis disease, pretibial ulceration, cellulitis,

10 r than in patients who underwent GBP without venous stasis disease.

11 h the development of the upright posture and venous stasis in the lower extremities.

12 hypertension and general infradiaphragmatic venous stasis may compound the damage.

13 e increased intraabdominal pressure enhances venous stasis, reduces intraoperative portal venous bloo

14 All eyes had varying degrees of venous stasis retinopathy and intraretinal edema overlyi

15 ation of P multocida via his cat licking the venous stasis ulcers on his legs.

16 Venous stasis ulcers resolved in all but three patients.

17 Thirty-seven patients had pretibial venous stasis ulcers, 4 had bronze edema, 23 had both, a

18 or venous stasis disease including pretibial venous stasis ulcers, bronze edema, and cellulitis.

19 complexes also occurred in wound fluid from venous stasis ulcers, but neither was detected in mastec

20 eep apnea, obesity hypoventilation syndrome, venous stasis ulcers, intestinal leak, small bowel obstr

21 Examining T-cell recruitment in human venous stasis, we show that superficial varicose veins p

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