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1 halin immunoreactivity, both markers for the ventral pallidum.
2 l but not into the core, dorsal striatum, or ventral pallidum.
3 ced reinstatement of cocaine seeking via the ventral pallidum.
4 men, bed nucleus of the stria terminalis, or ventral pallidum.
5 orsal-lateral BF, in regions occupied by the ventral pallidum.
6 t projections from the nucleus accumbens and ventral pallidum.
7 blocking the motor response by DAMGO in the ventral pallidum.
8 us accumbens, globus pallidus and the medial ventral pallidum.
9 als from iMSNs than their projections to the ventral pallidum.
10 the dorsolateral part of the subcommissural ventral pallidum.
11 tate identification of subterritories in the ventral pallidum.
12 the mid-insula and the ventral striatum and ventral pallidum.
13 o mouse nucleus accumbens projections to the ventral pallidum.
14 spot circuits in nucleus accumbens (NAc) and ventral pallidum.
15 on the dorsal striatum, prelimbic cortex, or ventral pallidum.
16 his manner, with both cell types innervating ventral pallidum.
17 ther vector functioned when infused into the ventral pallidum.
18 ctively lost in D2, but not D1 inputs to the ventral pallidum.
19 circuits involving the nucleus accumbens and ventral pallidum.
20 coded in neural signals coursing through the ventral pallidum.
21 0 increased extracellular GABA levels in the ventral pallidum.
22 ), rostromedial lateral globus pallidus, and ventral pallidum.
23 days withdrawal in the nucleus accumbens and ventral pallidum.
24 mbic projections of the NAcc, such as to the ventral pallidum.
25 and in the globus pallidus (+9%; p=0.06) and ventral pallidum (+11%; p=0.1), whereas binding was slig
26 ll (77 and 83% of control, respectively) and ventral pallidum (82% of control) as well as in the caud
28 ver pressing by cocaine was blocked by intra-ventral pallidum administration of the mu receptor antag
29 he stimulation of mu-opioid receptors in the ventral pallidum also elicits motor activation, and this
30 nsmission in the rostral anterior cingulate, ventral pallidum, amygdala, and inferior temporal cortex
31 teral cluster spanning the ventral striatum, ventral pallidum, amygdala, midbrain, and orbitofrontal
32 left dlPFC functional connectivity with the ventral pallidum, an AVP receptor-rich region previously
33 al band nuclei), lateral VTA and medial SNC (ventral pallidum and anterior half of substantia innomin
34 in other brain regions studied (hippocampus, ventral pallidum and cerebellum), or of the effects of c
36 and monosynaptic inhibitory currents in the ventral pallidum and lateral habenula, though the net ef
37 the ventromedial part of the subcommissural ventral pallidum and pallidal parts of the olfactory tub
38 d PHA-L-labeled presynaptic terminals in the ventral pallidum and substantia innominata were found to
41 xtrapyramidal nuclei, such as the globus and ventral pallidum and the substantia nigra, pars reticula
42 ic projections from the nucleus accumbens to ventral pallidum and ventral tegmental area (VTA) are in
43 ampus, nucleus accumbens, prefrontal cortex, ventral pallidum and ventral tegmentum, and more intense
44 cell types send GABAergic projections to the ventral pallidum and were found to differentially promot
45 in a number of limbic (nucleus accumbens and ventral pallidum) and cortical (medial/ventral orbital a
46 nsmission in the rostral anterior cingulate, ventral pallidum, and amygdala was correlated with the i
47 ide substantial GABAergic innervation to the ventral pallidum, and chemogenetic inhibition of ventral
48 en specific neurons in nucleus accumbens and ventral pallidum, and how these changes are associated w
49 ntal cortex, including the ventral striatum, ventral pallidum, and mediodorsal thalamus, the amygdala
50 conditioning (through the ventral striatum, ventral pallidum, and PPTN) and the other to adaptively
51 ioral function for D1-MSN innervation of the ventral pallidum, and suggest that losing LTDGABA in D2-
52 ion in ventral tegmentum, nucleus accumbens, ventral pallidum, and the orbitofrontal prefrontal corte
53 iated with reduced extracellular GABA in the ventral pallidum, and the reduction in GABA was also pre
54 een in both pallidal segments, including the ventral pallidum, and the subthalamic nucleus, consisten
55 minata, bed nucleus of the stria terminalis, ventral pallidum, and ventrolateral globus pallidus.
57 the dorsolateral part of the subcommissural ventral pallidum, anterograde labeling was weak in the m
58 In addition, following injections into the ventral pallidum, anterogradely transported biotinylated
59 bed nucleus of the stria terminalis, medial ventral pallidum, arcuate nucleus, and ventral tegmental
60 brain particularly the nucleus accumbens and ventral pallidum as well as actions within the ascending
61 o sadness-induced mu-opioid activation (left ventral pallidum, bilateral anterior cingulate cortices,
63 VTA (by baclofen) or GABAA receptors in the ventral pallidum (by muscimol) inhibit the motor respons
64 rminals in the pallidum (globus pallidus and ventral pallidum) can be modulated by locally applied op
65 umbens, bed nucleus of the stria terminalis, ventral pallidum, central nucleus of the amygdala, and c
66 D1- versus D2-MSN GABAergic synapses in the ventral pallidum could explain the differential regulati
67 l ventral pallidum (RVP), but not the caudal ventral pallidum (CVP), were robustly Fos activated duri
68 ay to the dorsolateral subcompartment of the ventral pallidum (dlVP) and through the direct pathway t
69 ons in subregions of medial accumbens shell, ventral pallidum, elements of extended amygdala, and lat
70 s from these, including the globus pallidus, ventral pallidum, entopeduncular nucleus and substantia
71 ry bulbs, frontal cortex, or nucleus basalis/ventral pallidum following hormone treatment; however, a
72 nsory cortex, and in contralateral amygdala, ventral pallidum, globus pallidus, and hippocampus, as w
73 al and lateral septum, diagnoal band nuclei, ventral pallidum, globus pallidus, substantia innominata
74 circuitry in both the nucleus accumbens and ventral pallidum has been reported to mediate taste-reac
75 sion of opto-MOR in GABAergic neurons of the ventral pallidum hedonic cold spot led to real-time plac
76 umbens and the GABA(A) agonist muscimol into ventral pallidum (i.e., "disconnection" methods) also im
77 tside the nucleus accumbens (such as rostral ventral pallidum), immediately medial and adjacent to th
78 posing a role for mu-opioid receptors in the ventral pallidum in mediating the reinstatement of cocai
79 0 nl) into the tubercle but not the shell or ventral pallidum induced conditioned place preference.
83 rebrain and brainstem targets, including the ventral pallidum, lateral and magnocellular preoptic nuc
84 connections with the hippocampus, amygdala, ventral pallidum, lateral hypothalamus, and ventral tegm
85 r cingulate, left orbitofrontal cortex, left ventral pallidum, left amygdala, and left inferior tempo
86 acity of morphine (0.01-10 microm) to reduce ventral pallidum levels of extracellular GABA was augmen
87 g LTDGABA in D2-MSN, but not D1-MSN input to ventral pallidum may promote cue-induced reinstatement o
88 uronal activity in the nucleus accumbens and ventral pallidum may underlie the augmented behavioral r
89 es in areas including the nucleus accumbens, ventral pallidum, medial preoptic area, medial amygdala,
92 of sucrose in behavior and firing signals of ventral pallidum neurons, and likewise, they increased i
93 ons (caudate putamen, nucleus accumbens, and ventral pallidum) of mice that were given either water (
95 lockade of kappa-opioid receptors within the ventral pallidum or mu-opioid receptors with the specifi
98 ager outputs to the forebrain, mainly to the ventral pallidum, preoptic-lateral hypothalamic continuu
99 projection from the nucleus accumbens to the ventral pallidum regulates the reinstatement of cocaine
102 the ventromedial part of the subcommissural ventral pallidum resulted in robust anterograde labeling
103 ounted in series of sections cut through the ventral pallidum, rostral globus pallidus, nucleus of th
104 und that projections to VTA from the rostral ventral pallidum (RVP), but not the caudal ventral palli
105 same reward within the nucleus accumbens to ventral pallidum segment of mesocorticolimbic circuitry.
106 d that accumbens neurons that project to the ventral pallidum showed adenosine A(2A) receptors immuno
108 We further show that, in hippocampus and ventral pallidum, spinophilin is occasionally present in
110 tional significant changes were noted in the ventral pallidum, superior colliculus, dentate gyrus (in
112 dial septum, the diagonal band of Broca, the ventral pallidum, the globus pallidus, the bed nucleus o
113 ata in the ventromedial globus pallidus, the ventral pallidum, the internal capsule, and the substant
114 ibution pattern, with high expression in the ventral pallidum, the islands of Calleja and pars compac
115 nveyed through the subiculum, accumbens, and ventral pallidum to the VTA where it contributes (along
116 ent dedicated neuronal subpopulations in the ventral pallidum tracked signal enhancements for hedonic
118 bited a pattern of nucleus accumbens OTR and ventral pallidum V1aR binding different from that associ
119 In parallel with nucleus accumbens (NAS), ventral pallidum (VP) also receives a dopaminergic proje
120 her the nucleus accumbens shell (NAc) or the ventral pallidum (VP) amplifies hedonic "liking" reactio
121 t, Area X and surrounding MSt project to the ventral pallidum (VP) and dorsal thalamus via pallidal-l
124 The role of the nucleus accumbens (NAC) and ventral pallidum (VP) in food reward modulation was inve
125 of ionotropic glutamate receptors within the ventral pallidum (VP) in the expression of conditioned p
126 e amygdala (AMG), nucleus accumbens (NA) and ventral pallidum (VP) influence goal-oriented behaviors.
134 ere, we present evidence suggesting that the ventral pallidum (VP) may participate in this process.
135 of parvalbumin-positive (PV) neurons in the ventral pallidum (VP) projecting to either the lateral h
138 e found that cue-evoked neural firing in the ventral pallidum (VP) reflected the strength of incentiv
141 mino acid (EAA)-containing projection to the ventral pallidum (VP), a major limbic system output regi
142 o block alcohol-maintained responding in the ventral pallidum (VP), a novel alcohol reward substrate,
143 e to show that optogenetic inhibition of the ventral pallidum (VP), a region known for processing rew
147 lized in the nucleus accumbens (NAC) and the ventral pallidum (VP), areas known to be important compo
148 rophic factor (BDNF) immunoreactivity in the ventral pallidum (VP), as determined by optical density
150 ons involves a projection from area X to the ventral pallidum (VP), which in turn projects to dopamin
151 amic nucleus (MD) via its projections to the ventral pallidum (VP), with the core and shell regions o
152 l segment (GPe), internal segment (GPi), and ventral pallidum (VP)-in 8 HD cases compared with 7 matc
158 (PFCd), nucleus accumbens core (NAcore), and ventral pallidum (VP)] blocked the ability of footshock
159 core of the nucleus accumbens (NAcore), and ventral pallidum (VP)] prevented cocaine-induced reinsta
161 y was associated with V1aR expression in the ventral pallidum (VPall) or lateral septum, areas causal
162 umbens shell (AcbSh) terminate in the medial ventral pallidum (VPm) and neurons in the VPm project to
163 he membrane insertion of mu receptors in the ventral pallidum was altered by withdrawal from cocaine,
165 bic structures such as nucleus accumbens and ventral pallidum (where opioid/endocannabinoid/orexin si
166 and vasopressin-immunoreactive fibers in the ventral pallidum, with males showing a greater density o
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