ライフサイエンスコーパス: ventral root

1 oked by antidromic stimulation of the distal ventral root.

2 PTN protein was more abundant in denervated ventral root.

3 einnervated cutaneous nerve but minimally by ventral root.

4 actor was upregulated to a greater degree in ventral root.

5 mature Schwann cells within the degenerating ventral root.

6 lia and several ectopic sites, including the ventral root.

7 ogenesis and motor axonal fasciculation into ventral roots.

8 receding that of their ipsilateral segmental ventral roots.

9 measured with extracellular recordings from ventral roots.

10 axons extending out of the spinal cord into ventral roots.

11 function and atrophy of nerve fibers in the ventral roots.

12 was also found in motoneurons as well as in ventral roots.

13 d rhythms recorded from cervical spinal cord ventral roots.

14 to direct polarization of cranial nerves or ventral roots.

15 tion of the peripheral ends of cut L7 and S1 ventral roots.

16 y of intercostal muscles coupled with the C4 ventral root activity assessed in a medulla-spinal cord

17 the lamprey, the contralaterally alternating ventral root activity that defines this behavior is driv

18 connective tissue bridges and the dorsal and ventral roots adjacent to the transection site.

19 motor-like activity was recorded from lumbar ventral roots after short trains of stimuli (50 Hz for 0

20 was performed on normal cutaneous nerve and ventral root and on graft preparations 5, 15, and 30 d a

21 -specific markers, axon projections into the ventral root and peripheral nerves, ultrastructure, dend

22 ograde transport of fluorescent tracers from ventral roots and axons in the ventrolateral funiculus,

23 present in motoneurons, primarily along the ventral roots and developing intramuscular nerves.

24 Morphometric analysis of L5 ventral roots and horns revealed that 4 weeks prior to a

25 xons exit the spinal cord via their adjacent ventral roots and project rostrally or caudally along th

26 ls was verified by axon counts of dorsal and ventral roots, as well as facial and optic nerves that r

27 bules (MTs), and reduced axonal transport in ventral roots, as well as spinal cord gliosis and motor

28 erwent sham surgery (n = 6), bilateral L5-S2 ventral root avulsion (VRA) injury (n = 5), or bilateral

29 Here, we demonstrate that after ventral root avulsion and immediate re-implantation, mod

30 We developed a rodent lumbosacral ventral root avulsion injury model of cauda equina injur

31 the retrograde effects of a unilateral L5-S2 ventral root avulsion on efferent preganglionic parasymp

32 We conclude that lumbosacral ventral root avulsions progressively deplete autonomic a

33 resymptomatically and before degeneration of ventral root axons and denervation of NMJs.

34 positive DRG neurons and small myelinated L5 ventral root axons decreases to about 35% of control cou

35 be the underlying cause of severe damage to ventral root axons resulting in a Wallerian-like degener

36 In affected cats, significant loss of L5 ventral root axons was observed at 12 weeks.

37 Ventral root axons were shrunken but were normal in numb

38 numbers of spinal motor neurons, diameter of ventral root axons, and extent of neuroinflammation in t

39 In the NF-M-null mutant atrophic ventral root, axons show an age-related depletion of neu

40 d the timing of their spikes relative to the ventral root burst became more variable.

41 y lagged behind the onset of the ipsilateral ventral root burst by a mean phase of 0.21 during electr

42 dorsal roots (DRs) could elicit coordinated ventral root bursting at both cervical and lumbar levels

43 Iberiotoxin extended the duration of ventral root bursts during fictive swimming in larvae at

44 activity was assessed using the synchronized ventral root bursts generated by both bath application o

45 preparation, was to increase the duration of ventral root bursts throughout the spinal cord during sw

46 ther the ipsilateral or the contralateral L2 ventral root bursts.

47 enervated cutaneous nerve than in denervated ventral root, but that PTN protein was more abundant in

48 sory phenotype by prolonged reinnervation of ventral root by cutaneous axons.

49 activity was recorded in vitro from cervical ventral roots (C1 or C4) using the isolated brainstem-sp

50 tivity was recorded from the fourth cervical ventral root (C4 VR), which contains the axons of phreni

51 re was extensive labeling of motoneurons and ventral root efferents-in particular, in an extremely de

52 In mSOD1 mice with clear muscle tremor, the ventral roots exhibited spontaneous synchronized bursts,

53 In ventral root fibers of pigeons, mice, and humans, the al

54 duced by electrical stimulation of L7 and S1 ventral roots in anesthetized cats (n=12).

55 stimulating the cut ends of L4 and L5 spinal ventral roots in Sprague-Dawley rats (300-400 g).

56 F and recording the potentials evoked in the ventral roots, in the VLF, or in individual interneurons

57 acute implantation of the avulsed L6 and S1 ventral roots into the conus medullaris (n = 6).

58 surgical implantation of avulsed lumbosacral ventral roots into the spinal cord can promote functiona

59 how that implantation of avulsed lumbosacral ventral roots into the spinal cord promotes reinnervatio

60 Transection of the fourth and fifth lumbar ventral roots (L4/5 ventral rhizotomy) of the rat is hig

61 ized at nodes of dorsal root sensory but not ventral root motor axons.

62 contraction was evoked by stimulating L7-S1 ventral roots (n = 7).

63 e electrotonically recorded in situ from the ventral root of isolated, hemisected amphibian spinal co

64 33W) mice lose large myelinated axons in the ventral root of the spinal cord and demonstrate myelin t

65 derived motor axons were observed within the ventral roots of each animal, whereas none were observed

66 crease in the level of NF proteins in the L5 ventral roots of G93A mice and a concomitant reduction i

67 endent axonal atrophy develops in the lumbar ventral roots of mice with a null mutation in the mid-si

68 ythmic locomotor-like activity in the lumbar ventral roots of the isolated neonatal rat spinal cord.

69 d cell differentiation in sciatic nerves and ventral roots of the laminin-alpha2-deficient (Lama2(-/-

70 slow axonal transports were impaired in the ventral roots of these mice coincidental with the appear

71 Stimulation of the S2 ventral root or the spinal cord both indicated that 15 H

72 short-latency depolarizing potential in the ventral roots, or in interneurons, that was probably med

73 l regions occurred in mouse motor axons from ventral roots, phrenic nerve and intramuscular branches.

74 0 microM, 60 min) was also able to attenuate ventral root potentials after single shock electrical st

75 Cumulative ventral root potentials evoked by long and short dorsal

76 At the beginning of an episode, ventral root potentials started before the VLF discharge

77 o effect on the amplitude of afferent-evoked ventral root potentials until the second postnatal week,

78 synchronized, cycle by cycle, with rhythmic ventral root potentials.

79 pported cutaneous axon regeneration, whereas ventral root preferentially supported motor axon regener

80 Short-latency reflexes (SRs) in ventral roots, presumably monosynaptic, were evoked by e

81 eurons and population synaptic potentials in ventral roots provided evidence that conventional transm

82 of lumbar (L2) dCINs fired rhythmically with ventral root-recorded motor activity, although their fir

83 f of V2a interneurons fire rhythmically with ventral root-recorded motor activity; the rhythmic V2a i

84 Using extracellular ventral root recordings, we showed that, while the thora

85 bserved in sections of dorsal roots, whereas ventral roots remained intact.

86 dal FHF2 staining within the optic nerve and ventral root, respectively.

87 econd, using L6-S1 ganglionectomies or L6-S1 ventral root rhizotomies we limited viral transport to t

88 Stimulation of sacral ventral roots (S1-S3) revealed that the S2 efferent outf

89 Ventral root stimulation was used to evoke currents from

90 rt latency depolarising potentials following ventral root stimulation, and by the transfer of the low

91 ncreases in MAP and Glu concentration during ventral root stimulation.

92 ann cells, oligodendrocyte progenitors cross ventral root transition zones and myelinate motor axons.

93 urons (MNs) and the locomotor-like bursts in ventral roots, until the motor pattern ceases.

94 rossed projection by assessing LVST-mediated ventral root (VR) response latencies, manipulating synap

95 lated bursts recorded in cervical and lumbar ventral roots was substantially weakened, although not a

96 By stimulating the ventral roots we have demonstrated the presence of short

97 nd motor nerve, grafts of cutaneous nerve or ventral root were denervated, reinnervated with cutaneou

98 ord and the peripheral cut ends of L7 and S1 ventral roots were stimulated electrically to induce mus