ライフサイエンスコーパス: ventricular contraction

1 tentials and pace-maps matching VT/premature ventricular contraction.

2 icient cardiac function requires synchronous ventricular contraction.

3 ardiac morphogenesis following uncoordinated ventricular contraction.

4 ium mutants can maintain circulation through ventricular contraction.

5 servocommand that was still synchronous with ventricular contraction.

6 n=10) and 1 subject diagnosed with premature ventricular contractions.

7 blocks, as well as bradycardia and premature ventricular contractions.

8 s, polyphasic R-waves and frequent premature ventricular contractions.

9 (10 with frequent [>1000/24 hours] premature ventricular contractions, 14 with ventricular tachycardi

10 er of hourly episodes of nocturnal premature ventricular contractions (66+/-117 versus 18+/-20, P=0.0

11 consecutive patients undergoing VT/premature ventricular contraction ablation, 7 patients underwent r

12 d for left ventricle outflow tract premature ventricular contraction ablation, an aortic valve closur

13 ibrillation accounted for >50% and premature ventricular contractions accounted for <10% of CRT loss

14 diac MRI provides a mechanism to assess left ventricular contraction and diagnose inducible myocardia

15 lation plays a critical role in accelerating ventricular contraction and speeding relaxation to match

16 soproterenol infusion also induced premature ventricular contractions and atrioventricular heart bloc

17 bility, including the frequency of premature ventricular contractions and sustained ventricular arrhy

18 Premature ventricular contractions and ventricular tachycardia wer

19 ycardia/atrial fibrillation; 16.6% premature ventricular contractions; and 8.6% captured as episodes

20 Ventricular contraction asynchrony with short-term RV ap

21 stolic peaks and nadirs of CF are because of ventricular contractions at the large majority of pulmon

22 on elicited arrhythmias, including premature ventricular contractions, atrioventricular heart block,

23 Fast cine MRI can be used to assess left ventricular contraction, but its utility for detection o

24 In 3-OST-7 morphants, ventricular contraction can be rescued by overexpression

25 Atrial and ventricular contractions could be discerned from E9.5, a

26 ac function, and direct measurements of left ventricular contraction demonstrated that PLCepsilon(-/-

27 ng in heart failure with dyssynchronous left ventricular contraction (DHF) and its restoration by car

28 xtures decreased LVDP, baseline rate of left ventricular contraction (dP/dtmaximum), and baseline rat

29 ific to dyssynchronous versus resynchronized ventricular contraction during hemodynamic decompensatio

30 owed major kinetic changes in left and right ventricular contraction (ejection) and relaxation (filli

31 age 41.5+/-16 years) referred for premature ventricular contractions evaluation or suspected ARVC.

32 wn of 3-OST-7 in zebrafish uncouples cardiac ventricular contraction from normal calcium cycling and

33 ar apical (RVA) pacing creates abnormal left ventricular contraction, hypertrophy, and reduced pump f

34 kinje system (HPS) is required for efficient ventricular contraction in an apex-to-base direction.

35 Resynchronization of ventricular contraction in patients with heart failure i

36 red with 0 of 30 (0%) (p = 0.048), premature ventricular contractions in 17 of 30 (57%) compared with

37 ed in 3 of 18 (17%), superior axis premature ventricular contractions in 21 of 25 (84%), and new term

38 The return of ventricular contractions in four of these 18 patients a

39 2-stimulation promoted Ca-mediated premature ventricular contractions in heart failure.

40 de, or hypovolemia, and signal the return of ventricular contractions in patients with initially abse

41 In contrast, VT or premature ventricular contractions in the setting of a structurall

42 us pauses, atrioventricular block, premature ventricular contractions, non-sustained ventricular arrh

43 Superior axis premature ventricular contractions occurred only in gene carriers.

44 epilepsy (petit mal) episodes, and premature ventricular contractions of the heart.

45 icular ectopic activity, including premature ventricular contractions of the outflow tract alternatin

46 ma (PI3Kgamma(-/-)) showed runs of premature ventricular contractions on adrenergic stimulation that

47 on 59+/-7.3%) with drug refractory premature ventricular contractions or ventricular tachycardia unde

48 orderline if polymorphic couplets, premature ventricular contractions, or nonsustained monomorphic VT

49 vs. 1 of 10 [10%], p = 0.006), and premature ventricular contractions originating from the outflow tr

50 Premature ventricular contractions originating in the left ventric

51 ventricular arrhythmias (-70+/-22% premature ventricular contractions; P<0.05).

52 icular pacing may promote a more coordinated ventricular contraction pattern in these patients.

53 Three diverse ventricular contraction patterns were defined by cardiov

54 Variability in premature ventricular contraction (PVC) coupling interval (CI) inc

55 High idiopathic premature ventricular contractions (PVC) burden has been associate

56 ght to examine whether suppressing premature ventricular contractions (PVC) using radiofrequency abla

57 achycardia (NSVT) in patients with premature ventricular contractions (PVCs) and heart failure treate

58 for catheter ablation suggest that premature ventricular contractions (PVCs) are a modifiable risk fa

59 achycardia (VT) and high burden of premature ventricular contractions (PVCs) are common in arrhythmog

60 his study was to determine whether premature ventricular contractions (PVCs) arising from the aortic

61 icular (LV) dyssynchrony caused by premature ventricular contractions (PVCs) has been proposed as a m

62 Monomorphic premature ventricular contractions (PVCs) have been shown to initi

63 er, certain patients with frequent premature ventricular contractions (PVCs) or VT and tachycardiomyo

64 ic ventricular tachycardia (VT) or premature ventricular contractions (PVCs) originating from the myo

65 Premature ventricular contractions (PVCs), couplets, and nonsustai

66 tricular tachycardia (NSVT), or 3) premature ventricular contractions (PVCs).

67 ars, 21 female) with outflow tract premature ventricular contractions (PVCs)/VT.

68 ) and frequent ventricular ectopy (premature ventricular contractions [PVCs] >10/h) was assessed from

69 Premature ventricular contraction rate increased with exercise, an

70 ycardia, ventricular couplets, and premature ventricular contractions showed greater amounts of inter

71 shape deepening despite similar magnitude of ventricular contraction, suggestive of ventricular-annul

72 rous cardiac arrhythmias including premature ventricular contractions, tachycardia, and high-degree h

73 tricular tachycardia, couplets, or premature ventricular contractions, the extent of intermediate LGE

74 the development of closely coupled premature ventricular contractions via a phase 2 reentrant mechani

75 Normal left-ventricular contraction was shown in seven patients exam

76 e base of the heart in synchrony with native ventricular contractions was evaluated with the use of a

77 ory of hypertension and occasional premature ventricular contractions was found on routine blood work

78 with left ventricle outflow tract premature ventricular contraction were included.

79 Premature ventricular contractions were common in ARVC patients wi

80 s in the electrocardiogram caused by delayed ventricular contraction (wide QRS complex), is a common

81 used to predict whether regions of abnormal ventricular contraction will improve after revasculariza

82 there were either (1) polymorphic premature ventricular contractions with >/=1 couplet or (2) sustai

83 logy of ventricular tachycardia or premature ventricular contractions with left bundle branch block/i

84 inus beats competing with numerous premature ventricular contractions with right and/or left bundle b